{"id":110852,"date":"2023-07-28T13:28:53","date_gmt":"2023-07-28T13:28:53","guid":{"rendered":"https:\/\/learnexams.com\/blog\/?p=110852"},"modified":"2025-07-19T07:13:03","modified_gmt":"2025-07-19T07:13:03","slug":"lmr-georgettes-pmhnp-certification-exam-latest-2023-2024-questions-and-correct-answersverified-answers","status":"publish","type":"post","link":"https:\/\/www.learnexams.com\/blog\/2023\/07\/28\/lmr-georgettes-pmhnp-certification-exam-latest-2023-2024-questions-and-correct-answersverified-answers\/","title":{"rendered":"LMR Georgette\u2019s PMHNP Certification Exam Latest 2023-2024 Questions And Correct Answers(Verified Answers)"},"content":{"rendered":"\n<p>Which patient is at highest risk for SI<\/p>\n\n\n\n<p>A. 30y\/o married AA female with previous SI attempt *1 risk factor<\/p>\n\n\n\n<p>B. 35 y\/o single Asian male with previous SI attempt *3 risk factors<\/p>\n\n\n\n<p>C. 38 y\/o single AA male who is a manager of a bank *2 risk factors<\/p>\n\n\n\n<p>D. 68 y\/o single white male with depression *5 risk factors (age, male, white, depression)<\/p>\n\n\n\n<p>D. 68 y\/o single white male with depression *5 risk factors (age, male, white, depression)<\/p>\n\n\n\n<p>Count the risk factors<\/p>\n\n\n\n<p>When interview teenagers (16 y\/o) that arrive with their parents what should you do?<br>interview them separately from parents.<br>-This helps Build therapeutic rapport with teens by telling them the info is confidential. Parents may be upset but remember you are advocating for the child.<\/p>\n\n\n\n<p>Which Ethnic group has the highest rate of suicide?<br>Native Americans<\/p>\n\n\n\n<p>Example A patient is being treated for schizophrenia with olanzapine. Which of the following is the most common side effect of olanzapine?<br>A. Increased waist circumference<br>B. EPS (not as common in atypical antipsychotics d\/t 5HT2A)-receptor antagonism<br>C. Increased Lipids<br>D. Metabolic Syndrome<br>D. Metabolic Syndrome (UMBRELLA ANSWER)<\/p>\n\n\n\n<p>Which antipsychotics have the least weight gain?<br>Latuda, Abilify, (also least sedating), Geodon-if patient has metabolic syndrome consider switching to one of the medications above. Or if the patient is overly sedated try switching to ABILIFY<\/p>\n\n\n\n<p>Which mood stabilizer have the least weight gain?<br>Lamictal<br>-But remember all mood stabilizers cause some weight gain<\/p>\n\n\n\n<p>When presented with a question about typical vs atypical antipsychotic the answer is usually to start of a<br>atypical<\/p>\n\n\n\n<p>A client presents with complains of changes in appetite, feeling fatigued, problems with sleep-rest cycle, and changes in libido. What is the neuroanatomical area of the brain that is responsible for the normal regulation of these functions?<\/p>\n\n\n\n<p>A. Thalamus<\/p>\n\n\n\n<p>B. Hypothalamus<\/p>\n\n\n\n<p>C. Limbic System<\/p>\n\n\n\n<p>D. Hippocampus<\/p>\n\n\n\n<p>Hypothalamus<br>A, B, &amp; D are all part of the limbic system so you can rule that out<\/p>\n\n\n\n<p>When a patient is hesitant to participate in treatment you should encourage?<br>Bring a support person like a husband<\/p>\n\n\n\n<p>Thyroid-Stimulating hormone normal level<br>0.5-5.0 Mu\/L<\/p>\n\n\n\n<p>When T4 and T3 are high and TSH is low what is the diagnosis<br>HYPERTHYROIDISM, TSH secretion decreases: TSH LOW \u00e0 key symptoms HEAT INTOLERANCE<\/p>\n\n\n\n<p>Key symptoms of Heat Intolerance<br>Hyperthyroidism<\/p>\n\n\n\n<p>When T4 and T3 are Low and TSH is high what is the diagnosis<br>(HYPOTHYROIDISM) TSH secretion increased: TSH HIGH \u00e0 COLD INTERANCE<\/p>\n\n\n\n<p>Key symptoms of Cold Intolerance<br>Hypothyroidism<\/p>\n\n\n\n<p>Hyperthyroid can mimic<br>Mania<\/p>\n\n\n\n<p>Hypothyroid can mimic<br>Depression<\/p>\n\n\n\n<p>A patient on depakote complains of RUQ pain and has reddish\/brown urine<br>Hepatoxicity<br>-Check LFTs<\/p>\n\n\n\n<p>Signs of Depakote toxicity<br>Disorientation, confusion, lethargy<\/p>\n\n\n\n<p>You suspect depakote toxicity what do you do?<br>Check<br>-LFT<br>-Ammonia<br>-Depakote Level<\/p>\n\n\n\n<p>What herbal supplement can cause hepatoxicity?<br>Kava Kava<\/p>\n\n\n\n<p>When taking Kava Kava in combinations with other medications you should caution about<br>Risk of Hepatoxicity and Sedation<\/p>\n\n\n\n<p>TCAs carry a risk of<br>Hepatotoxicity<\/p>\n\n\n\n<p>Signs of Stevens-Johnson Syndrome<br>-fever, mouth pain, swelling, burning eyes, blisters, skin pain<\/p>\n\n\n\n<p>two psychotropics known to cause steven johnson syndrome<br>lamictal and tegretol<\/p>\n\n\n\n<p>What nationality is most suseptible of getting steven johnson?<br>Asians<\/p>\n\n\n\n<p>When treating asians with tegretal screen for?<br>HLAB-1502 Allele<\/p>\n\n\n\n<p>What two medications cause agranulocytosis?<br>Clozaril &amp; Tegretal<\/p>\n\n\n\n<p>Agranulocytosis when to discontinue medication<br>Less than 1000<\/p>\n\n\n\n<p>When monitoring for agranulocytosis in patients look for s\/s of what?<br>Infection<br>-Fever, sore throat, fatigue, chills<\/p>\n\n\n\n<p>Before starting any mood stabilizer in a female of childbearing age be sure to check?<br>HCG<\/p>\n\n\n\n<p>Which two medications may decrease the risk of suicide?<br>clozaril and lithium<\/p>\n\n\n\n<p>Medications that increase lithium level<br>NSAID-ibuprofen, INDOCIN<br>THIAZIDES-hydrochlorithiazide ACE INHIBITORS-lisinopril<\/p>\n\n\n\n<p>Ace inhibitors are treatment of choice for?<br>Heart Failure<\/p>\n\n\n\n<p>Certain medications are known to increase lithium level, but HOW?<br>by reducing renal clearance<\/p>\n\n\n\n<p>When educating a patient about lithium teach them about<br>Hyponatremia<br>Dehydration-hot days, exercise<\/p>\n\n\n\n<p>Normal Lithium Level<br>0.6-1.2<\/p>\n\n\n\n<p>Lithium Toxicity<br>1.5 or above<br>Discontinue and re-order lithium level<\/p>\n\n\n\n<p>Lithium level of 1.4<br>Monitor for toxicity<\/p>\n\n\n\n<p>Labs before starting lithium<br>TSH, BUN, CREATININE, HCG, U\/A to check for presence of protein in the urine (4+ protein is concerning for renal impairment)\u00e04+ protein in urine=MONITOR FOR TOXICITY<\/p>\n\n\n\n<p>4+ protein in the urine of a patient on lithium<br>4+ protein is concerning for renal impairment<\/p>\n\n\n\n<p>4+ protein in urine=MONITOR FOR TOXICITY<\/p>\n\n\n\n<p>Lithium side effects<br>hypothyroid, leukocytosis, maculopapular rash, t-wave inversion, Coarse Hand Tremor, GI upset (nausea, vomiting, anorexia)<\/p>\n\n\n\n<p>-Some of these are also signs of toxicity<\/p>\n\n\n\n<p>Signs of lithium toxicity<br>confusion, ataxia, GI upset, palpitation, tremor<\/p>\n\n\n\n<p>NMS<br>muscle rigidity, mutism (because of muscle rigidity), increased CPK (caused by muscle contraction and muscle destruction), increase WBC, increased WBC, myoglobinuria (also from muscle destruction)<\/p>\n\n\n\n<p>Cherry colored urine in a patient that exercises a lot<br>test for myoglobinuria may be a sign of rhabdo<\/p>\n\n\n\n<p>Serotonin Syndrome<br>With any drug that increases 5-HT (e.g., MAO inhibitors, SNRIs, TCAs) hyperthermia, confusion, myoclonus, cardiovascular instability, flushing, diarrhea, seizures.<br>-Treatment: cyproheptadine (5-HT2 receptor antagonist).<\/p>\n\n\n\n<p>Treatment for NMS<br>Stop Offending Medication<\/p>\n\n\n\n<p>-Dantrolene (muscle relaxer)<\/p>\n\n\n\n<p>-Bromocriptine (Dopamine D2 agonist).<\/p>\n\n\n\n<p>*In question focus on what they are asking for\u2026.dopamine agonist vs muscle relaxer<\/p>\n\n\n\n<p>Treatment for Serotonin Syndrome<br>Stop Med (1 or more SSRI, SSNRI, TCA, MOAI)<\/p>\n\n\n\n<p>-Cyproheptadine<\/p>\n\n\n\n<p>Triptans<br>Used for MIGRAINES<\/p>\n\n\n\n<p>-These meds increase serotonin<\/p>\n\n\n\n<p>example SUMATRIPTAN<\/p>\n\n\n\n<p>patient taking Prozac and started on sumatriptan<\/p>\n\n\n\n<p>-call PCP to ask them to switch the migraine med if patient already on SUMATRIPTAN do not start antidepressant without talking to PCP<\/p>\n\n\n\n<p>How long do you wait when switching between an SSRI to an MAOI?<br>2 weeks<\/p>\n\n\n\n<p>How long do you wait when switching between Prozac and MAOI?<br>5-6 weeks wash out period<\/p>\n\n\n\n<p>What is the first line treatment for depression and why?<br>SSRI-First line treatment for depression due to less risk of injury from OVERDOSE<\/p>\n\n\n\n<p>If a cancer patient has depression what should you consider?<br>Treating with a medication with minimal drug\/drug side effects like Lexapro<\/p>\n\n\n\n<p>Patient with depression worries about sexual dysfunction what would be the medication of choice?<br>Wellbutrin<\/p>\n\n\n\n<p>Primary symptoms of depression include fatigue and low energy what med would you chose?<br>Wellbutrin<\/p>\n\n\n\n<p>Wellbutrin is contraindicated in patients with<br>Seizures and anorexia<\/p>\n\n\n\n<p>Which medications are best for neuropathic pain?<br>SNRI<br>Gabapentin<br>TCA<\/p>\n\n\n\n<p>Secondary to the black box warning providers caring for patients on antidepressants should assess for?<br>Suicidality, frequency, and severity at EVERY appointment<\/p>\n\n\n\n<p>Which meds have the worse serotonin discontinuation syndrome<br>Those with short half lives<br>such as zoloft<\/p>\n\n\n\n<p>Symptoms of serotonin withdrawal syndrome<br>Fever, achiness, soreness, lethargy, fatigue, impaired memory, decreased concentration, GI UPSET<\/p>\n\n\n\n<p>Shits and Shivers<\/p>\n\n\n\n<p>Ages of onset for schizophrenia in males vs females<br>-MALES 18-25 years<\/p>\n\n\n\n<p>-FEMALE 25-35 years<\/p>\n\n\n\n<p>Schizophrenia increases the risk for<br>SUICIDE<\/p>\n\n\n\n<p><em>HIGH RISK OF SI in SCHIZOPHRENIA<\/em><\/p>\n\n\n\n<p>Just having schizophrenia increases your risk of suicide.<\/p>\n\n\n\n<p>MUST ASK ABOUT SI, EVERYTIME (frequency, severity of thoughts)<\/p>\n\n\n\n<p>What increases the causes or increases the risk or schizophrenia<br>excessive pruning of synapses<\/p>\n\n\n\n<p>-inadequate synapse formation,<\/p>\n\n\n\n<p>-intrauterine insults such as maternal exposure to toxins, viral agents, maternal substance use, maternal illness, maternal malnutrition, fetal oxygen deprivation,<\/p>\n\n\n\n<p>-first order relative (mom\/dad)<\/p>\n\n\n\n<p>MRI or PET scan what is seen in schizophrenia<br>EVERYTHING DECREASES EXCEPT VENTRICLES<\/p>\n\n\n\n<p>-You will see VENTRICULAR ENLARGEMENT<\/p>\n\n\n\n<p>Stimulants can potentiate the release of what neurotransmitter?<br>Dopamine which can worsen symptoms of schizophrenia<\/p>\n\n\n\n<p>Assertive Community Treatment (ACT)<br>a form of rehabilitation post hospitalization, in home treatment<\/p>\n\n\n\n<p>What level of prevention is ACT?<br>Tertiary<\/p>\n\n\n\n<p>What adjunctive treatment is important in schizophrenia<br>-social skills training<br>-Exercise<\/p>\n\n\n\n<p>Exercise for mental health patients can promote<br>Cognition<br>Quality of Life<br>Long-term health<\/p>\n\n\n\n<p>ACT is ideal for patients with a history of<br>Treatment non-compliance<\/p>\n\n\n\n<p>-Think about making the treatment convenient for them&#8211;&gt;bringing it to their home<\/p>\n\n\n\n<p>What diagnosis has the highest risk of Homicidality<br>Antisocial<\/p>\n\n\n\n<p>In the MMSE how do you test for abstraction?<br>proverb interpretation (everyone that lives in glass houses shouldn\u2019t throw stones) Are they able to think abstractly<\/p>\n\n\n\n<p>Thought Process-Tangential<br>means that their response has nothing to do with the question<\/p>\n\n\n\n<p>Circumstantial<br>means that their response goes in circles instead of getting to the point of the question<\/p>\n\n\n\n<p>Mental Status-Thought Content includes<br>SI\/HI\/AH\/VH<\/p>\n\n\n\n<p>Another name for MMSE<br>Folstein Scale<\/p>\n\n\n\n<p>How to assess concentration on MMSE<br>Serial 7s or perform an activity backwards i.e list the days of the week backwards<\/p>\n\n\n\n<p>Assess ability to learn new material<br>repeat 3 words after me<\/p>\n\n\n\n<p>Assess ability to recall<br>repeat 3 words after 5 minutes<\/p>\n\n\n\n<p>Assess fund of knowledge<br>Who is the president<\/p>\n\n\n\n<p>What is a quick and easy way to assess for neurological issues<br>Clock drawing test<\/p>\n\n\n\n<p>If patient is unable to draw a clock this indicates<br>Problem with the right hemisphere, cerebrum, or parietal lobe<\/p>\n\n\n\n<p>mesolimbic pathway<br>Hyperactivity of dopamine in the this pathway mediates positive psychotic symptoms<\/p>\n\n\n\n<p>-Antagonism of D2 receptors in this pathway treats positive psychotic symptoms<\/p>\n\n\n\n<p>mesocortical pathway<br>-Decreased dopamine in the this projection to the dorsolateral prefrontal cortex is postulated to be responsible for negative and depressive symptoms of schizophrenia<\/p>\n\n\n\n<p>Nigrostriatal Pathway<br>-This pathway mediates motor movements<\/p>\n\n\n\n<p>-Dopamine blockade in this pathway can lead to increase acetylcholine levels<\/p>\n\n\n\n<p>-Blockade of dopamine (D2) receptors in this pathway can lead to EPS, i.e dystonia, parkinsonian symptoms and akathisia<\/p>\n\n\n\n<p>Low Dopamine in the nigrostriatal pathway increases which neurotransmitter<br>-Dopamine has a reciprocal relationship with acetylcholine (Ach) (LOW DOPAMINE INCREASE Ach)<\/p>\n\n\n\n<p>Long-standing D2 blockade in the nigrostriatal pathway can lead to<\/p>\n\n\n\n<p>tardrive dyskinesia<\/p>\n\n\n\n<p>Tuberoinfundibular pathway<br>-Blockade of D2 receptors in this pathway can lead to increase prolactin levels leading to hyperprolactinemia which clinically manifests as amenorrhea, galactorrhea, and sexual dysfunction, gynecomastia<\/p>\n\n\n\n<p>-DECREASE DOPAMINE INCREASED PROLACTIN<\/p>\n\n\n\n<p>Long-term hyperprolactinemia can be associated with what condition<\/p>\n\n\n\n<p>osteoporosis<\/p>\n\n\n\n<p>Normal Prolactin Level in Men<br>level less than 20ng\/ml<\/p>\n\n\n\n<p>Normal Prolactin Level in Women<br>less than 25ng\/ml<\/p>\n\n\n\n<p>Which medication is the highest offender for increasing prolactin<br>Risperdal<\/p>\n\n\n\n<p>Acute Dystonia + Treatment<br>neck stiffness, muscle spasm of upper body especially neck\/face\/tongue<\/p>\n\n\n\n<p>-Treatment is IM COGENTIN + continue PO COGENTIN for several days<\/p>\n\n\n\n<p>Akathisia + Treatment<br>may mimic anxiety, restlessness, can\u2019t sit still, rocking, pacing<\/p>\n\n\n\n<p>-First line Treatment is BETA-BLOCKERS like PROPANOLOL (Inderal)<\/p>\n\n\n\n<p>-Second line treatment is COGENTIN<\/p>\n\n\n\n<p>-Third line treatment is benzos<\/p>\n\n\n\n<p>Beta-Blockers such as Inderal are contraindicated with what type of asthma medication<br>-DO NOT GIVE WITH BROCHODIALATOR such as ALBUTERAL this combination can cause bronchospasm<\/p>\n\n\n\n<p>akinesia\/bradykinesia + treatment<br>A. difficulty initiating movement; slowness of movement<br>-Treatment Cogentin<\/p>\n\n\n\n<p>PSEUDOPARKINSON or PARKINSONIAN + Treatment<br>caused by dopamine blockade, results in muscle rigidity, mask like facial expression, may look blunted, pill rolling tremors in fingers, shuffling gait, motor slowing<\/p>\n\n\n\n<p>-Treatment COGENTIN<\/p>\n\n\n\n<p>tardive dyskinesia + Treatment<br>abnormal facial movements, grinding teeth, lip smacking, protruding tongue<\/p>\n\n\n\n<p>-Treatment DECREASE DOSE OF MED, DISCONTINUE MED, Switch to CLOZARIL, Switch to different med, VINPAT<\/p>\n\n\n\n<p>Does Cogentin Treat TD<br>COGENTIN MAKES TD WORSE<\/p>\n\n\n\n<p>Typical onset of TD<br>OCCURS 1-2 years TYPICALLY, but can be ACUTE ONSET ALSO<\/p>\n\n\n\n<p>What non-psych med can cause TD?<br>REGLAN (Metoclopramide) can CAUSE Tardive Dyskinesia must educate patient that this med or the combination of this PLUS antipsychotic can increase risk of TD*** encourage them to discontinue reglan if TD develops<\/p>\n\n\n\n<p>InDucers CYP450<\/p>\n\n\n\n<p>DECREASE<\/p>\n\n\n\n<p>Carbamazepine<\/p>\n\n\n\n<p>Rifampin<\/p>\n\n\n\n<p>Alcoholics (chronic)<\/p>\n\n\n\n<p>Phenytoin<\/p>\n\n\n\n<p>Grisiofulvin<\/p>\n\n\n\n<p>Phenobarb<\/p>\n\n\n\n<p>Sulphonylureas<\/p>\n\n\n\n<p>Crap GPS Induces me to Madness!<\/p>\n\n\n\n<p>InhIbitors of CYP450<\/p>\n\n\n\n<p>INCREASE<\/p>\n\n\n\n<p>Ciprofloxacin<br>Ritonavir<br>Amiodarone<br>Cimetidine<br>Ketoconazole<\/p>\n\n\n\n<p>Acute Etoh<br>Macrolides<br>INH<br>Grapefruit Juice<br>Omeprazole<\/p>\n\n\n\n<p>Crack Amigos<\/p>\n\n\n\n<p>Erythromycin and Clarithromycin can cause<br>Increased tegretol levels<\/p>\n\n\n\n<p>Patient started on Clozaril or Zyprexa and two months later starts smoking<\/p>\n\n\n\n<p>as a provider you know that the smoking can decrease the medication effectiveness<\/p>\n\n\n\n<p>-Increase medication dose<\/p>\n\n\n\n<p>Patient has been a chronic smoker and has been stable on Zyrexa but tells you that he recently quit smoking cold turkey<br>as a provider you know that you must now decrease the dose of the antipyshcotic<\/p>\n\n\n\n<p>Medications that cause mania<br>Steroids, Disulfiram (Antabuse), Isoniazid (INH), Antidepressants in persons with bipolar<\/p>\n\n\n\n<p>-If a patient must take steroids, the provider should increase the mood stabilizer<\/p>\n\n\n\n<p>Medications that cause depression<br>steroids, beta blockers, interferon, Accutane (isotrentinoin), some retroviral drugs, antineoplastic drugs, benzodiazepines, progesterone<\/p>\n\n\n\n<p>-may need to increase antidepressant<\/p>\n\n\n\n<p>Accutane (isotretinoin)<br>Can cause depression and birth defects<\/p>\n\n\n\n<p>Flonase<br>As a provider you know that flonase is a STEROID so it may exacerbate mood symptoms<\/p>\n\n\n\n<p>Increase mood stabilizer to maintain stability, steroids can also trigger depression<\/p>\n\n\n\n<p>Flonase can trigger mood instability but it can also cause an increase in<br>Psychosis<\/p>\n\n\n\n<p>patient is taking flonase while on antipsychotic but you find that the antipsychotic is ineffective it is likely because the flonase is exacerbating psychosis<\/p>\n\n\n\n<p>-increase the dose of antipsychotic<\/p>\n\n\n\n<p>Neurotransmitters involved in Addiction<br>Dopamine and GABA<\/p>\n\n\n\n<p>Symptoms of Stimulant Abuse<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>agitation\/aggression<\/li>\n\n\n\n<li>impaired judgment<\/li>\n\n\n\n<li>euphoria<\/li>\n\n\n\n<li>elevated BP<\/li>\n\n\n\n<li>tachycardia<\/li>\n\n\n\n<li>dilated pupils<\/li>\n\n\n\n<li>hallucinations<\/li>\n\n\n\n<li>TREMORS<\/li>\n\n\n\n<li>IMSOMNIA<\/li>\n<\/ol>\n\n\n\n<p>If an anorexic patient complains of pain or bloating after eating this may indicate<br>delayed gastric emptying<\/p>\n\n\n\n<p>Medications that delay gastric emptying<br>Omeprazole, ranitidine, famotidine<\/p>\n\n\n\n<p>Proton Pump Inhibitors (omeprazole &amp; Protonix)<br>Decrease absorption of antipsychotics &amp; SSRI<\/p>\n\n\n\n<p>-MUST WAIT TWO HOURS BEFORE TAKING ANTIPSYCHOTIC OR SSRI<\/p>\n\n\n\n<p>When initiating an SSRI on an elderly patient you should advise about<br>increased anxiety<\/p>\n\n\n\n<p>Paradoxical effect<br>when meds cause the opposite effect than expected<\/p>\n\n\n\n<p>Apoptosis<br>programmed cell death\/neuronal loss<\/p>\n\n\n\n<p>At age 45 and above the patient displays mania for first time what should be ruled out<\/p>\n\n\n\n<p>MEDICAL CONDITION<\/p>\n\n\n\n<p>Patient with bipolar disorder presents with depressed mood &amp; emotional lability<\/p>\n\n\n\n<p>Give Depakote<\/p>\n\n\n\n<p>Hallmark sx of Borderline Personality<br>Recurrent self harm<\/p>\n\n\n\n<p>Treatment for Borderline Personality<br>DBT<\/p>\n\n\n\n<p>Creator of DBT<br>Marsha Linehan<\/p>\n\n\n\n<p>What activity is helpful in making a diagnosis of borderline personality<br>Journaling or diary keeping<\/p>\n\n\n\n<p>Conversion Disorder<br>STRESS leads to neurological symptoms such as seizures, paresthesia, blindness, mutism<\/p>\n\n\n\n<p>Adjustment Disorder<br>adjusting to a situation resulting in depression or anxiety or both or mixed disturbance of emotions and conduct (this type is more common in children: insomnia, peer conflict, verbal altercations, truancy, crying)<\/p>\n\n\n\n<p>-Symptoms occur within 3 months of the stressor<\/p>\n\n\n\n<p>If question states recently moved, recent death\u2026.THINK ADJUSTMENT<\/p>\n\n\n\n<p>factitious disorder<br>when patients introduce foreign substances into their body or contaminate their food<\/p>\n\n\n\n<p>-Faking illness but NO MOTIVE BEHIND IT<\/p>\n\n\n\n<p>Malingering<br>Faking illness for financial gain<\/p>\n\n\n\n<p>Reactive Attachment<br>common in children in foster care, abuse from parents<\/p>\n\n\n\n<p>-Withdrawn and shows no emotion towards caregiver<\/p>\n\n\n\n<p>ODD<br>They deliberately annoy others, no aggression, defiance of authority<\/p>\n\n\n\n<p>-Family Therapy is mainstay<\/p>\n\n\n\n<p>-Child management \/Parent management skills is the focus in therapy<\/p>\n\n\n\n<p>-Positive reinforcement<\/p>\n\n\n\n<p>-Boundary Setting<\/p>\n\n\n\n<p>Conduct Disorder<br>violence, criminal, fire setting, killing animals, gang activity, +AGGRESSION, NO REMORSE<\/p>\n\n\n\n<p>-May need meds and therapy<\/p>\n\n\n\n<p>-Goal of therapy is to target MOOD &amp; AGGRESSSION (mood stabilizers, antipsychotics, alpha agonists\/alpha 2 adrenergic receptor blockers such as guanfacine and clonidine)<\/p>\n\n\n\n<p>-Monitor BP with guanfacine and clonidine<\/p>\n\n\n\n<p>Acute Stress Disorder<br>similar to PTSD but the timeline differs<\/p>\n\n\n\n<p>-heightened arousal, nightmares, flashbacks<\/p>\n\n\n\n<p>-LESS THAN ONE MONTH<\/p>\n\n\n\n<p>PTSD<br>-OVER ONE MONTH<\/p>\n\n\n\n<p>-3 HALLMARK SXS: intrusive re-experiencing of trauma, increased arousal, avoidance<\/p>\n\n\n\n<p>-May also have NIGHTMARES\u00e0 GIVE PRAZOSIN<\/p>\n\n\n\n<p>-Non-pharm tx of PTSD- EMDR, CBT<\/p>\n\n\n\n<p>Panic attack vs Panic disorder (treatment)<br>Panic attack = BZ<br>Panic disorder = SSRI<\/p>\n\n\n\n<p>Panic Attack is ACUTE<br>Panic Disorder is CHRONIC<\/p>\n\n\n\n<p>Feels like impending doom<\/p>\n\n\n\n<p>Tourette&#8217;s Syndrome<br>Criteria for diagnosis<\/p>\n\n\n\n<p>-TWO moto tics and ONE vocal tics<\/p>\n\n\n\n<p>-LASTS more than ONE YEAR<\/p>\n\n\n\n<p>-By age 18<\/p>\n\n\n\n<p><strong>CHILDREN MAY NORMALLY HAVE TICS so if they have one tic only THIS IS NORMAL<\/strong><\/p>\n\n\n\n<p>Child presents with one tic and the parent is worried<br><strong>CHILDREN MAY NORMALLY HAVE TICS so if they have one tic only THIS IS NORMAL<\/strong><\/p>\n\n\n\n<p>Neurotransmitters involved in Tourettes<br>DNS: Dopamine, Norepinephrine, Serotonin<\/p>\n\n\n\n<p>Treatment for tourettes<br>Treatment: Haldol, Pimozide, Abilify, Guanfacine, clonidine<\/p>\n\n\n\n<p>What type of medication can cause tics or exacerbate them<br>Stimulants<\/p>\n\n\n\n<p>Neurotransmitters involved in mood disorders<br>DNS: Dopamine, Norepinephrine, Serotonin + GABA<\/p>\n\n\n\n<p>Neurotransmitters involved in ADHD<br>DNS: Dopamine, Norepinephrine, Serotonin<\/p>\n\n\n\n<p>part of brain implicated in ADHD<br>prefrontal cortex<br>basal ganglia<br>reticular activating system<\/p>\n\n\n\n<p>ADHD inattentive type is caused in what part of the brain<br>Prefrontal Cortex which is known to regulate ATTENTION and EXECUTIVE FUNCTION<\/p>\n\n\n\n<p>dorsolateral prefrontal cortex<br>Attention<br>Executive Function<br>Cognition<br>Processing<br>Working Memory<br>Problem Solving<\/p>\n\n\n\n<p>Deficit in the <strong>_<\/strong> can lead to ADHD inattentive type<br>Prefrontal Cortex<\/p>\n\n\n\n<p>Teacher reports that the stimulant only works for first few hours of class<\/p>\n\n\n\n<p>medication has worn off too fast. Order multiple dosing throughout the day<\/p>\n\n\n\n<p>When does the aftercare plan start<br>on admission<\/p>\n\n\n\n<p>If parents become anxious while you are educating about a new diagnosis what should you do<br>-Provide patient and parents information immediately don\u2019t wait till discharge<\/p>\n\n\n\n<p>-Parents may become anxious after a diagnosis of mental illness such as ADHD, stop teaching offer support because they will not absorb the education. Provide supportive therapy<\/p>\n\n\n\n<p>Neurotransmitters involved in OCD<br>serotonin, dopamine, glutamate &amp; GABA<\/p>\n\n\n\n<p>A tic may also be a <em>_<\/em><br>Compulsion<\/p>\n\n\n\n<p>Facts about OCD<br>Obsession\/Compulsion<\/p>\n\n\n\n<p>-A tic may be a compulsion<\/p>\n\n\n\n<p>-If first order relative has OCD the child\u2019s risk of developing OCD is increased<\/p>\n\n\n\n<p>-Streptococcal infections increase risk of OCD<\/p>\n\n\n\n<p>-Treatment SSRI-prozac, Zoloft, if adult you may also use TCA such as clomipramine<\/p>\n\n\n\n<p>If question asks if the patient has Tourette\u2019s vs OCD listen for mention of streptococcal treatment this will trigger you to think OCD<\/p>\n\n\n\n<p>DMDD<br>6-17 years ONLY<\/p>\n\n\n\n<p>-Irritability for no reason, sad, depressed mood, tantrums, crying, moody, always mad<\/p>\n\n\n\n<p>If patient presents with irritability or labile mood and you need help further delineating symptoms<br>Administer MOOD QUESTIONAIRE<br>7\/13 Bipolar Diagnosis Likely<\/p>\n\n\n\n<p>Sleep Disorders are often <strong>_<\/strong><br>So what should you assess if a parent reports that a child is having nightmares<br>GENETIC<\/p>\n\n\n\n<p>ask if someone in the family has a similar issue with sleep\u2026look for family patterns of sleep problems<\/p>\n\n\n\n<p>GAD<br>Worry, apprehension, fear must LAST ATLEAST 6 MONTHS<\/p>\n\n\n\n<p>Delirium<br>-ACUTE (within hours to days) onset of disturbance of LOC, COGNITION,<\/p>\n\n\n\n<p>inattention<\/p>\n\n\n\n<p>-Urinary Tract Infections are common cause for DELIRIUM always check UA<\/p>\n\n\n\n<p>-Treatment is antipsychotics like HALDOL<\/p>\n\n\n\n<p>Dementia<br>-Chronic and slow onset (months to years to develop)<\/p>\n\n\n\n<p>-Mental decline in cognition, irritability, personality changes<\/p>\n\n\n\n<p>-When asked questions they may try to answer or MAKE UP ANSWERS (confabulate)<\/p>\n\n\n\n<p>Low levels of what labs may mimic dementia<br>Vit B12 and Folic Acid<\/p>\n\n\n\n<p>Cortical Dementia<br>Language and memory (aphasia and amnesia)<\/p>\n\n\n\n<p>Subcortical Dementia<br>Motor abnormalities\/Mood issues like apathy, depression, irritability<\/p>\n\n\n\n<p>HIV Dementia is a type of subcortical dementia<\/p>\n\n\n\n<p>Early signs of HIV dementia<br>subcortical form of dementia<\/p>\n\n\n\n<p>COGNITIVE, MOTOR, BEHEAVIOR for example a patient with lack of coordination, unsteady gait<\/p>\n\n\n\n<p>Treatment for HIV dementia<br>Antivirals<\/p>\n\n\n\n<p>Pseudo Dementia<br>Depression causes the memory issues, common in older adults<\/p>\n\n\n\n<p>-Also assess onset of symptoms, pseudo dementia is more acute onset<\/p>\n\n\n\n<p>-When asked questions they often say \u201cI DON\u2019T KNOW\u201d<\/p>\n\n\n\n<p>Instruments to use to differentiate between dementia and pseudo dementia<br>-Use instrument to further screen out cognitive issues such as SLUMS, MOCHA, MMSE<\/p>\n\n\n\n<p>-Older individuals with depression may present with irritability and agitation<\/p>\n\n\n\n<p>If question is asking you to differentiate between depression and dementia look at the amount of time that the symptoms have been present<\/p>\n\n\n\n<p>hallmark of lewy body dementia<br>visual hallucinations<\/p>\n\n\n\n<p>Frontotemporal lobe Dementia<br>PICKs Disease<\/p>\n\n\n\n<p>-Hallmark is personality changes, language difficulties, poor impulse control, and behavioral changes<\/p>\n\n\n\n<p>-May see slurred speech or difficulty getting words out<\/p>\n\n\n\n<p>What lobe is associated with ability to understand what others are saying (comprehending speech)<br>Temporal Lobe<\/p>\n\n\n\n<p>Neurotransmitters involved in Autism<br>GABA, Glutamate, Serotonin<\/p>\n\n\n\n<p>Autism<br>a disorder that appears in childhood and is marked by deficient communication, social interaction, Poor eye contact, May not respond when you call their name, Stereotypical movement<\/p>\n\n\n\n<p>When play they often like to line up their toys, stack them in tidy rows<\/p>\n\n\n\n<p>Broken Mirror Theory of Autism<br>Explains that the child\u2019s presentation is caused by the mirror neuron i.e dysfunction in the mirror neuron<\/p>\n\n\n\n<p>Risk Factors for Autism<br>Male gender, genetic loading, intellectual disability, parents ages, preterm<\/p>\n\n\n\n<p>Screening tools for Autism<br>ADOS-G (autism diagnostic observation schedule-genetic)<br>ASQ (ages and stages questionnaire)<br>M-CHAT (modified-checklist for autism-toddler)<\/p>\n\n\n\n<p>Where is Norepinephrine produced?<br>locus coeruleus and medullary reticular formation<\/p>\n\n\n\n<p>Where is serotonin produced?<br>raphe nuclei<\/p>\n\n\n\n<p>Where is dopamine produced?<br>substantia nigra, ventral tegmental area, nucleaus accumbens<\/p>\n\n\n\n<p>Where is acetylcholine synthesized?<br>Basal nucleus of Meynert<\/p>\n\n\n\n<p>Hippocampus<br>a neural center located in the limbic system; helps process memory and manage stress<\/p>\n\n\n\n<p>Limbic System<br>The limbic system is the part of the brain involved in our behavioral and emotional responses, especially when it comes to behaviors we need for survival: feeding, reproduction and caring for our young, and fight or flight responses.<br>-Hippocampus<br>-Amygdala<br>-Hypothalamus<br>-Thalamus<\/p>\n\n\n\n<p>Amygdala function<br>Responsible for the response and memory of emotions, especially fear<\/p>\n\n\n\n<p>Thalamus function<br>relay station for sensory impulses, pain<\/p>\n\n\n\n<p>hypothalamus function<br>homeostasis, temperature, thirst, appetite, sex drive, sleep cycle, emotions<\/p>\n\n\n\n<ul class=\"wp-block-list\">\n<li>believed to serve a regulatory role in aggression<\/li>\n<\/ul>\n\n\n\n<p>anterior cingulate cortex<br>brain region that regulates cognitive function, decision making, empathy, impulse control, and emotions<\/p>\n\n\n\n<p>Cerebellum<br>Balance and coordination<\/p>\n\n\n\n<p>signs of lead toxicity<br>developmental delay, learning diff., irritability, loss of appetite, weight loss, sluggishness, fatigue, abdominal pain, vomiting, constipation, hearing loss, seizures, eating non-food items PICA<\/p>\n\n\n\n<p>Hint home built before 1970\u2019s<\/p>\n\n\n\n<p>TEST FOR LEAD<\/p>\n\n\n\n<p>When caring for an infant that is about to die?<br>GIVE THE BABY TO THE PARENTS and allow them to grieve<\/p>\n\n\n\n<p>Risk factors for osteoporosis<br>Age<br>smoking<br>caffeine<br>lack of exercise<br>diet lacking calcium and vit D<\/p>\n\n\n\n<p>Provide Education<\/p>\n\n\n\n<p>If discharging a patient that is not following up with outpatient care, organize ways to help the patient get to the appointment REMEMBER STAY INVOLVED IN THE CARE<\/p>\n\n\n\n<p>Assume you are doing group therapy and there is a patient that is not comfortable sharing but you are trying to promote interpersonal learning. What should you do?<\/p>\n\n\n\n<p>Provide adjunctive individual session that will help facilitate group participation<\/p>\n\n\n\n<p>Cognitive Therapy<br>-Aaron Beck<\/p>\n\n\n\n<p>Replacing irrational or distortive thoughts with positive thoughts<\/p>\n\n\n\n<p>Behavioral Therapy<br>-Arnold Lazarus<br>focuses on changing behavior by identifying problem behaviors, replacing them with appropriate behaviors<br>-Exposure<br>-Relaxation<br>-Skills training<br>-Role Playing<\/p>\n\n\n\n<p>Humanistic Therapy<br>-Carl Rogers<br>person-centered therapy<br>-Self-actualization<br>-Self-Directive Growth<br>-Everyone has the potential to actualize and find meaning in life<\/p>\n\n\n\n<p>Existential Therapy<br>Victor Frankl -an insight therapy that focuses on the elemental problems of existence, such as death, meaning, choice, and responsibility, emphasized making courageous life choices.<br>-Emphasizes accepting freedom and making responsible choices<br>-Focus on the present<\/p>\n\n\n\n<p>Why am I here, What is my purpose<\/p>\n\n\n\n<p>Interpersonal Therapy<br>Gerald Kierman &amp; Myrna Weissman<\/p>\n\n\n\n<p>&#8211;Used for people who have trouble interacting with others, relationship distress<\/p>\n\n\n\n<p>-Marital conflict<\/p>\n\n\n\n<p>-12-16 weeks (3-4 months)<\/p>\n\n\n\n<p>EMDR Phases<br>Desensitization Phase: visualize the trauma, verbalize negative thoughts but remain attentive to physical sensations<\/p>\n\n\n\n<p>Installation Phase: Installs and increases strength of the positive thoughts that the patient has declared as a replacement<\/p>\n\n\n\n<p>Body Scan: Visualize the trauma along with the positive thought and then scan ones body mentally to identify any tension within<\/p>\n\n\n\n<p>Group therapy: Installation of hope<br>participants develop hope for creating a different life; they gain hope from others<\/p>\n\n\n\n<p>Group therapy: Universality<br>people have similar problems, thoughts, and feelings and they are NOT ALONE<\/p>\n\n\n\n<p>Group Therapy: Altruism<br>sharing of oneself with another and helping another<\/p>\n\n\n\n<p>Group Therapy: Imitative Behavior<br>Patients can increase their skills by imitating the bx of others<\/p>\n\n\n\n<p>Group Therapy: Interpersonal learning<br>interacting with others increases adaptive interpersonal relationships<\/p>\n\n\n\n<p>Group Therapy: Group Cohesiveness<br>Patients develop an attraction to the group and other members as well as a sense of belonging<\/p>\n\n\n\n<p>Group Therapy: Catharsis<br>Patients openly express their feelings which were previously suppressed<\/p>\n\n\n\n<p>Group Therapy: Existential Factors<br>Groups enable participants to deal with the mean of their own existance<\/p>\n\n\n\n<p>Group Therapy: Corrective Refocusing<br>Participants reexperience family conflicts in the group, which allows them to recognize and change behaviors that may be problematic<\/p>\n\n\n\n<p>Group Phases<br>forming, storming, norming, performing, adjourning<\/p>\n\n\n\n<p>Family Systems Therapy<br>Murray Bowen<\/p>\n\n\n\n<p>-a person&#8217;s problematic bx may serve a function for the family or be a symptoms of dysfunctional patterns<\/p>\n\n\n\n<p>KEY WORDS*<\/p>\n\n\n\n<p>Self-Differentiation, Triangulation,<\/p>\n\n\n\n<p>Triangles<\/p>\n\n\n\n<p>Structural Family Therapy<br>Salvador Minuchin<\/p>\n\n\n\n<p>-How, when, and who whom family members relate<\/p>\n\n\n\n<p>KEY WORDS*<\/p>\n\n\n\n<p>Mapping<\/p>\n\n\n\n<p>Hierarchies<\/p>\n\n\n\n<p>Boundaries<\/p>\n\n\n\n<p>Strategic Therapy<br>Jay Haley<\/p>\n\n\n\n<p>-Symptoms are a way to communicate metaphorically in a family<\/p>\n\n\n\n<p>-Symptom focused<\/p>\n\n\n\n<p>KEY WORDS*<\/p>\n\n\n\n<p>Straightforward directive<\/p>\n\n\n\n<p>Paradoxical directive (reverse psychology)<\/p>\n\n\n\n<p>Reframing (you are not jealous of your sister you just care for her so much)<\/p>\n\n\n\n<p>Solution Focused Therapy<br>-MIRACLE QUESTIONS<\/p>\n\n\n\n<p>-EXCEPTION-BASED FINDING -SCALING QUESTIONS<\/p>\n\n\n\n<p>Meditation<br>if teaching about meditation must tell them about MUSCLE RELAXATION<\/p>\n\n\n\n<p>If patient tells you something BEFORE you ASSESS- EXPRESS EMPATHY \u201cI\u2019m sorry this happened to you.\u201d<\/p>\n\n\n\n<p>USE OPEN ENDED QUESTIONS unless talking to a child or someone that is unable to construct a narrative then use CLOSED ENDED or YES\/NO<\/p>\n\n\n\n<p>PICOT<br>P: Population<br>I: Intervention<br>C: Comparison<br>O: Outcome<br>T: Time<\/p>\n\n\n\n<p>If a patient has rheumatoid arthritis check<br>ESR<\/p>\n\n\n\n<p>Therapy session with husband and wife &amp; only one shows up<\/p>\n\n\n\n<p>Reschedule<\/p>\n\n\n\n<p>A patient\u2019s mother calls and tells you that her son has been sodomized by their 15 year old brother<\/p>\n\n\n\n<p>tell mother DO NOT LEAVE THE CHILD ALONE WITH THE BROTHER<\/p>\n\n\n\n<p>-Provider calls CPS<\/p>\n\n\n\n<p>-Arrange crisis therapy for family<\/p>\n\n\n\n<p>Patient is moving out of state<\/p>\n\n\n\n<p>if there is no imminent danger then provide enough medication for them to establish a new provider<\/p>\n\n\n\n<p>Level 1 evidence<br>systematic reviews of random control trials (RCTs) or Meta-analysis or RCT-highest internal validity due to randomizations<br>At least (2)<\/p>\n\n\n\n<p>Level 2 Evidence<br>systematic reviews of cohort studies<br>-little bias because the subjects are identified prior to outcome &#8211; randomization is lost<\/p>\n\n\n\n<p>Shrill Cry<br>Intracranial pressure<\/p>\n\n\n\n<p>Child between the ages of 3-6 masterbating<br>Normal to play with genitals (PHALLIC STAGE) NORMAL do not assume they have been abused*<\/p>\n\n\n\n<p>Mom is concerned that her son age 10 has swelling in his chest and she is concerned he is developing breasts<br>Young boys ages 9-16 years old often have NORMAL BREAST ENLARGEMENT which disappears within 6 months<\/p>\n\n\n\n<p>*reassure them that this is normal<\/p>\n\n\n\n<p>Elderly female presents with decreased sex drive<br>Check Testosterone level<\/p>\n\n\n\n<p>Sex Hormone-Testosterone is involved in sex drive<\/p>\n\n\n\n<p>-MUST KNOW THAT WOMAN have TESTOSTERONE TOO<\/p>\n\n\n\n<p>alcohol dehydrogenase<br>an enzyme active in the stomach and the liver that metabolizes alcohol<\/p>\n\n\n\n<p>-WOMAN HAVE LOWER ALCOHOL DEHYDROGENASE (metabolizes alcohol)<\/p>\n\n\n\n<p>-This is why woman get drunk faster<\/p>\n\n\n\n<p>-Lower levels of this enzyme may also cause a higher propensity to develop LIVER DISEASE<\/p>\n\n\n\n<p>When OB wants to hire psych providers<\/p>\n\n\n\n<p>they want to increase mental health access to those that need it the most<\/p>\n\n\n\n<p>Normalizing grief and loss in children<\/p>\n\n\n\n<p>Don\u2019t tell them what to do because grief responses vary<\/p>\n\n\n\n<p>-i.e Don\u2019t tell them to stop working that is prescriptive advise<\/p>\n\n\n\n<p>-With children the most important thing is to reinforce FAMILY support an supportive therapy such as group therapy so they can learn from other children who have experienced similar events<\/p>\n\n\n\n<p>palmar grasp reflex<br>normal up to 5-6 months<br>If older baby still has this reflex-&gt;refer to specialist<\/p>\n\n\n\n<p>Moro (startle) reflex<br>Normal till 5-6 months<br>If present past normal range-&gt;refer to specialist<br>If not present within the normal age-&gt;Xray may be a sign of a broken bone, nerve injury, or spinal injury<\/p>\n\n\n\n<p>Babinski reflex<br>Normal up to 2 years<br>If present past normal range-&gt;refer to specialist<\/p>\n\n\n\n<p>PDE-5 inhibitors<br>Sildenafil (Viagra)<\/p>\n\n\n\n<p>Vardenafil (Levitra)<\/p>\n\n\n\n<p>Tadalafil (Cialis)<\/p>\n\n\n\n<p>RAPIDLY ABSORBED<\/p>\n\n\n\n<p>Used for erectile disfunction<\/p>\n\n\n\n<p>Difference between BMI in anorexia vs Bulemia<br>Anorexia-Low BMI<br>Bulemia-Normal BMI<\/p>\n\n\n\n<p>Parent brings in 16-year-old with BMI 12, Pulse LOW, BP LOW and you determine the patient needs to be sent for medical evaluation but the parents refuse<\/p>\n\n\n\n<p>Contact CPS<\/p>\n\n\n\n<p>You read and article that says that most children with ADHD abuse substances\u2026<\/p>\n\n\n\n<p>-To translate this information into practice what should you do?<\/p>\n\n\n\n<p>-Screen ALL children for ADHD for SUBSTANCE USE<\/p>\n\n\n\n<p>-OR Screen ALL children with SUBSTANCE USE for ADHD<\/p>\n\n\n\n<p>accupuncture<br>used for pain and depression<\/p>\n\n\n\n<p>Habeas Corpus<\/p>\n\n\n\n<p>legal concept that protects patients from unlawful hospitalization<\/p>\n\n\n\n<p>-May be a reason to leave AMA<\/p>\n\n\n\n<p>Disseminated Encephalomyelitis<\/p>\n\n\n\n<p>inflammation of nervous system<\/p>\n\n\n\n<p>-MUST DO NEURO EXAM<\/p>\n\n\n\n<p>-EXAM: May present with ASSYMETRICAL BODY MOVEMENTS<\/p>\n\n\n\n<p>Assume you are interviewing a patient and you want them to provide information in a specific timeline, but they are unable<\/p>\n\n\n\n<p>TO help ask them specific questions which helps to ANCHOR their memory<\/p>\n\n\n\n<p>Before you administer a medication, you must educate them about the medication but first you should assess<\/p>\n\n\n\n<p>BUT FIRST ASSESS WHAT THEY KNOW ALREADY or WHAT THERE BELIEVES ARE ABOUT THE MEDICATION<\/p>\n\n\n\n<p>Patient presents with iatrogenic effect<br>assess ALL the medications that the patient is taking<\/p>\n\n\n\n<p>-Don\u2019t assume that it is from the medication you prescribed<\/p>\n\n\n\n<p>When trying to pass a policy and your co-workers are against it what should you do<br>educate them on how the policy will benefit patient care<\/p>\n\n\n\n<p>To promote a policy how do you get the word out there<br>Think most FEASIBLE option with WIDE net or audiance<\/p>\n\n\n\n<p>Working in outpatient setting and you want to ensure continuous improvement in quality of care.<\/p>\n\n\n\n<p>-Create an instrument to monitor clinical outcomes (this helps to identify what you are doing right or wrong)<\/p>\n\n\n\n<p>Autoimmune disease can lead to increased<br>Cytokine level<\/p>\n\n\n\n<p>If a child is urinating the bed<br>-Teach parents to use alarm clock to wake up to urinate (NON PHARM FIRST)<\/p>\n\n\n\n<p>-If that doesn\u2019t work try DESMOSPRESSIN (decreased enuresis)<\/p>\n\n\n\n<p>Are you allowed to look up a patient on social media?<br>No it violates their trust<\/p>\n\n\n\n<p>Assume you started a patient on a medication and they go home and find out that there is a black box warning on the medication that you were unaware of. They call with concerns\u2026<\/p>\n\n\n\n<p>-First, go online and do your own research<\/p>\n\n\n\n<p>-Research the RISK vs BENEFIT before you tell the patient to stop the medication<\/p>\n\n\n\n<p>Risk factors for sleep apnea<br>excessive weight, obesity, diabetes, smoking<\/p>\n\n\n\n<p>Tolerance<br>you need higher doses of the medication in order for the medication to be effective<\/p>\n\n\n\n<p>PHQ-9<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>5 mild depression<br>10 moderate<br>15 moderately severe<br>20 severe<\/p>\n<\/blockquote>\n\n\n\n<p>Max score 27<\/p>\n\n\n\n<p>HAM-D<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>10 Mild<br>14 Moderate<br>17 Severe<\/p>\n<\/blockquote>\n\n\n\n<p>HAM-A<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>8 Mild<br>15 Moderate<br>24 Severe<\/p>\n<\/blockquote>\n\n\n\n<p>Beck Depression Inventory (BDI)<br>0-13 Subclinical<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>14 Mild<br>20 Moderate<br>29 Severe<br>40 EXTREME<\/p>\n<\/blockquote>\n\n\n\n<p>Max score 63<\/p>\n\n\n\n<p>GAD-7 Scoring<br>0-4: Minimal Anxiety<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>5 Mild Anxiety<br>10 Moderate Anxiety<br>15 Severe Anxiety<\/p>\n<\/blockquote>\n\n\n\n<p>Max score 21<\/p>\n\n\n\n<p>COWS<br>Medicate with PRNS at score of 7 or above<\/p>\n\n\n\n<p>Consider Subutex or Suboxone at 13 or above<\/p>\n\n\n\n<p>Remember Methadone is the LEAST safe option due to cardiac issues<\/p>\n\n\n\n<p>CIWA<br>Begin PRN medication at 8 or above<\/p>\n\n\n\n<p>Scores of 15 or above consider scheduled medications<\/p>\n\n\n\n<p>Patient in alcohol withdrawal and you are choosing medication for CIWA, check what?<br>LFT<\/p>\n\n\n\n<p>if liver disease use ATIVAN because of short half life over VALIUM<\/p>\n\n\n\n<p><em>REMEMBER DETOX SHOULD NOT OCCUR OUTPATIENT&#8211;&gt;residential or inpatient is needed<\/em> especially if pregnant. REMEMBER SAFETY FIRST<\/p>\n\n\n\n<p>Idealization<br>Seeing someone else as perfect, ideal, or more worthy than everyone else<\/p>\n\n\n\n<p>This is often seen in grieving before acceptance of the loss<\/p>\n\n\n\n<p>Appreciative Inquiry<br>is an approach to organizational change which focuses on strengths rather than on weakness<\/p>\n\n\n\n<p>Example do not focus on what the employee does wrong focus on what they do well<\/p>\n\n\n\n<p>Reflective Practice<br>Links theory to practice with a goal of correcting practices that are incorrect. Example Debriefing after a restraint to find out what went wrong or what went right DEBFRIEFING MAY BE A KEY WORD<\/p>\n\n\n\n<p>-After an incident&#8211;&gt; Debrief<\/p>\n\n\n\n<p>Assume you started the patient on an antidepressant and now they complain of insomnia<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>Before changing medication, CHANGE THE TIME OF DAY THEY ARE TAKING THE MED<\/li>\n<\/ol>\n\n\n\n<p>Conflict of interest between pharmaceutical companies and Nurse Practitioners i.e promises to sponsor NP loan forgiveness. You want to create a policy to address this. First you must examine\u2026<\/p>\n\n\n\n<p>study the relationship between the industry provided samples and industry sponsored education<\/p>\n\n\n\n<p>If a patient is involuntarily admitted can they still refuse medications?<br>they can still refuse medications, UNLESS it is an emergency or if the court determines they must take medications<\/p>\n\n\n\n<p>Scope of practice is determined by<br>State board of Nursing<\/p>\n\n\n\n<p>Scope of practice defines<br>NP roles and actions<br>-Varies broadly state to state<\/p>\n\n\n\n<p>If you would like to perform ECT as an NP what should you review<br>The state scope of practice standards to see if it is allowed and what certification is needed<\/p>\n\n\n\n<p>Standard of practice is determined by<br>ANA<\/p>\n\n\n\n<p>-Provides a way to judge nature of the care provided<\/p>\n\n\n\n<p>The PMHNP is required by law to carry out care in accordance with what other reasonably prudent nurses would do in the same or similar circumstances. Thus, provision of high-quality care consistent with established standards is critical<\/p>\n\n\n\n<p>Exceptions to Confidentiality<br>-Answering court orders, subpoenas, or summonses *high yield (if you don\u2019t release or lie about knowing this is PERJURY)<\/p>\n\n\n\n<p>-Insurance companies<\/p>\n\n\n\n<p>-Giving information to attorneys involved in litigation<\/p>\n\n\n\n<p>-Intent to harm self or others<\/p>\n\n\n\n<p>-Meeting state of federal requirements for reporting disease states<\/p>\n\n\n\n<p>-When the need for information outweighs the principle of confidentiality i.e unconscious patient and their life is at stake<\/p>\n\n\n\n<p>Tarasoff principle<br>1976 &#8211; duty to warn victims of potential harm from client<\/p>\n\n\n\n<p>may vary by state so you must check with your state board of nursing first\u2026it may not be your responsibility to notify<\/p>\n\n\n\n<p>If you are taking care of a patient and during the process of interview she tells you that her husband just texted her that her husband is going to kill self<\/p>\n\n\n\n<p>-Call the police, provide the address<\/p>\n\n\n\n<p>informed consent<br>-Communication process between the provider and client that results in client\u2019s acceptance or rejection of proposed treatment<\/p>\n\n\n\n<p>-Ensure they understand the risks vs benefits<\/p>\n\n\n\n<p>If patient is able to reiterate the risks vs benefits of procedure or treatment, they are able to give consent*<\/p>\n\n\n\n<p>Example patient comes to the hospital, and they are unable to give consent i.e to sick to agree to treatment you must<\/p>\n\n\n\n<p>assess need for involuntary treatment<\/p>\n\n\n\n<p>Justice<br>doing what is fair, fairness in all aspects of care<\/p>\n\n\n\n<p>Nonmaleficence<br>do no harm<\/p>\n\n\n\n<p>Beneficence<br>doing good\/promoting well-being<\/p>\n\n\n\n<p>Fidelity<br>being true and loyal<\/p>\n\n\n\n<p>Veracity<br>telling the truth, patients have the right to know the truth about their treatment<\/p>\n\n\n\n<p>Autonomy<br>doing for self (right to self-determination)<\/p>\n\n\n\n<p>New male patient has a 10 year history of substance abuse, depression, and anxiety. He is requesting Xanax. Which principle should the PMHNP employ moving forward?<\/p>\n\n\n\n<p>A. Beneficence<\/p>\n\n\n\n<p>B. Fidelity<\/p>\n\n\n\n<p>C. Non-Maleficence<\/p>\n\n\n\n<p>D. Veracity<\/p>\n\n\n\n<p>C. Non-Maleficence<\/p>\n\n\n\n<p><em>Do no harm, giving Xanax would endanger him secondary to the high abuse potential and imminent danger if he were to overdose on Xanax<\/em><\/p>\n\n\n\n<p>If no history of substance use, then beneficence would be appropriate because the Xanax would help the anxiety<\/p>\n\n\n\n<p>Acute agitation and anxiety vs acute agitation and psychosis<\/p>\n\n\n\n<p>IM ordered<br>IM ativan for agit\/anx<\/p>\n\n\n\n<p>IM antipsychotic for agit\/psychosis<\/p>\n\n\n\n<p>Patients have the right to be treated in the Least<br>Restrictive Setting<\/p>\n\n\n\n<p>The PMHNP is asked to consult with a local inpatient psychiatric facility to provide nursing staff development. After meeting with the administrator to identify the nature of the problem requiring the consultation, the PMHNP&#8217;s next step is to:<br>A. Create interdisciplinary teaching team<br>B. Develop Outcome measures<br>C. Market the educational plan<br>D. Utilize a survey to assess the educational needs of the staff<br>D. Utilize a survey to assess the educational needs of the staff<\/p>\n\n\n\n<p><em>3 Interventions vs 1 Assessment<\/em> FIRST YOU NEED TO ASSESS FIRST<\/p>\n\n\n\n<p>What is the best way to reduce stigma<br>THROUGH EDUCATION<\/p>\n\n\n\n<p>*THINK WIDEST AUDIENCE<\/p>\n\n\n\n<p>Just Culture<br>individuals are continually learning, designing safety systems, and managing behavioral choices<\/p>\n\n\n\n<p>The PMHNP is concerned about access-to-care issues in the local community and wants to help develop health care policy to help patients access care more effectively.<br>A. Asking the clinical manager to explore options for access<br>B. Organizing a political protest<br>C. Working with the local chapter of the nurses professional association<br>D. Writing letters to the editor of the local newspaper<br>C. Working with the local chapter of the nurses professional association<\/p>\n\n\n\n<p><em>Remember strength in numbers but STAY INVOLVED i.e asking the manager just passes off<\/em><\/p>\n\n\n\n<p>If a patient from a specific culture is refusing to accept any diagnosis of mental health disorders because of shame what could be done to address this barrier?<\/p>\n\n\n\n<p>A. Educate the family<\/p>\n\n\n\n<p>B. Political advocacy<\/p>\n\n\n\n<p>C. Public Health Concern<\/p>\n\n\n\n<p>D. Community education programs<\/p>\n\n\n\n<p>D. Community education programs<\/p>\n\n\n\n<p>*Narrow down to education A\/D\u2026then think WIDE NET =Community EDUCATION<\/p>\n\n\n\n<p>A client with Biolar I disorder presents to your PMHNP office for a follow-up visit. During the visit the client informs you he no longer wants to be treated with medication. , and he does not have bipolar disorder, that was a misdiagnosis, He further informs you he stopped all his medications 2 months ago and is here to thank you for your care and tell you he no longer needs appointments. Understanding ethical conflict, you use which of the following ethical principles?<br>A. Autonomy<br>B. Nonmaleficence<br>C. Justice<br>D. Beneficence<br>A. Autonomy<\/p>\n\n\n\n<p><em>Patient\u2019s have the right to self-determination<\/em><\/p>\n\n\n\n<p>Recovery Model * RELAPSE IS A LEARNING OPPORTUNITY<\/p>\n\n\n\n<p>-Treatment approach that does not focus on full symptom resolution but emphasizes on resilience and control over problems in life<\/p>\n\n\n\n<p>-Self-Direction (do not tell them what to do)<\/p>\n\n\n\n<p>-Individualized and Person-Centered<\/p>\n\n\n\n<p>-Non-Linear, Recovery is not a step-by-step process, but one based on continual growth, occasional setbacks, and learning from experience<\/p>\n\n\n\n<p>In counseling a 23 y\/o married Hispanic mother who brought her 4 year old son to the clinic for &#8220;mal de ojo&#8221; with symptoms of fitful sleep, diarrhea, vomiting, and fever the PMHNO;<br>A. Identifies what steps the mother has already tried in caring for the child<br>B. Explain that the symptoms are viral infection<br>C. Educates about importance of fluid electrolyte imbalance<br>D. Respects the mother&#8217;s understanding of the child&#8217;s illness<br>A. Respects the mother\u2019s understanding of the child\u2019s illness<\/p>\n\n\n\n<p>*In cultural questions remember RESPECT FIRST! Even before assessment<\/p>\n\n\n\n<p>Quality Improvement<\/p>\n\n\n\n<p>Projects designed to improve systems, decrease cost, and improve productivity<\/p>\n\n\n\n<p>What is an example of a quality improvement process?<br>Plan, Do, Study, Act<\/p>\n\n\n\n<p>Retrospective Chart Review is an example of a Quality Improvement Process<\/p>\n\n\n\n<p>If they ask HOW the NP would do a quality Improvement Process the answer may be Plan, Do, Study, Act<\/p>\n\n\n\n<p>The NP is responsible for initiating quality improvement at a community clinic. The effective strategy for evaluating the clients services is to<\/p>\n\n\n\n<p>A. Chart review analysis<\/p>\n\n\n\n<p>B. A root cause analysis<\/p>\n\n\n\n<p>C. Plan DO Study Act<\/p>\n\n\n\n<p>D. Failure effect mode analysis<\/p>\n\n\n\n<p>C. Plan Do Study Act<\/p>\n\n\n\n<p>SBIRT<br>Screening, Brief Intervention, and Referral to Treatment<\/p>\n\n\n\n<ul class=\"wp-block-list\">\n<li>Use to screen substance use disorders<\/li>\n<\/ul>\n\n\n\n<p>Erikson&#8217;s stages of psychosocial development<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>trust vs. mistrust<\/li>\n\n\n\n<li>autonomy vs. shame and doubt<\/li>\n\n\n\n<li>initiative vs. guilt<\/li>\n\n\n\n<li>industry vs. inferiority<\/li>\n\n\n\n<li>identity vs. role confusion<\/li>\n\n\n\n<li>intimacy vs. isolation<\/li>\n\n\n\n<li>generativity vs. stagnation<\/li>\n\n\n\n<li>integrity vs. despair<\/li>\n<\/ol>\n\n\n\n<p>Piaget&#8217;s stages of cognitive development<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>sensorimotor<\/li>\n\n\n\n<li>preoperational<\/li>\n\n\n\n<li>concrete operational<\/li>\n\n\n\n<li>formal operational<\/li>\n<\/ol>\n\n\n\n<p>Preoperational Stage includes<br>2-7 y.o- MAGICAL THINKING IS NORMAL, if they believe that monsters can fly this is NORMAL<\/p>\n\n\n\n<p>Egocentric<\/p>\n\n\n\n<p>Understand language<\/p>\n\n\n\n<p>formal operational stage<br>12+ y\/o during which people begin to think logically about abstract concepts<\/p>\n\n\n\n<p>KEY WORD is LOGIC think like a scientist or do a science project you must be able to use logic ABSTRACT THINKING such as doing algebra<\/p>\n\n\n\n<p>sensorimotor stage<br>in Piaget&#8217;s theory, the stage (from birth to about 2 years of age) during which infants know the world mostly in terms of their sensory impressions and motor activities<br>Object permanance<\/p>\n\n\n\n<p>concrete operational stage<br>in Piaget&#8217;s theory, the stage of cognitive development (from about 6 or 7 to 11 years of age) during which children gain the mental operations that enable them to think logically about concrete events<br>i.e finding similarities in objects, grouping things<\/p>\n\n\n\n<p>Risk factors for suicide<br>Sex (Male),<br>Age (Teenager or Elderly),<br>Depression,<br>Previous Attempt, Ethanol or Drug Use, Loss of rational thinking, Sickness (medical illness),<br>3 or more prescription medications,<br>Organized plan,<br>No spouse (divorced, widowed, or single especially if childless). Social support lacking.<br>WHITE<\/p>\n\n\n\n<p>Women try more often. Men succeed more often.<\/p>\n\n\n\n<p>A 72 year old female brought in by her husband with increasing forgetfulness, decreased activity, and decreased appetite for 2 months. She has a history of HTN and is being treated with Lisinopril. The exam is normal and the MMSE provides a score of 24 but she declines to answer some questions and needs to be urged to participate in the assessment. What is the likely diagnosis?<\/p>\n\n\n\n<p>A. Alzheimer<\/p>\n\n\n\n<p>B. Vascular Dementia<\/p>\n\n\n\n<p>C. Depression<\/p>\n\n\n\n<p>D. Medication Toxicity<\/p>\n\n\n\n<p>C. Depression<\/p>\n\n\n\n<p>*2 months=too soon for Alzheimer\u2019s, 24 is mild MMSE, HTN is not enough info to diagnose vascular dementia HALLMARKS FOR VASCULAR DEMENTIA are carotid bruits fundoscopic abnormalities and enlarged cardiac chambers, remember PSEUDO dementia is DEPRESSION<\/p>\n\n\n\n<p>HALLMARKS FOR VASCULAR DEMENTIA<\/p>\n\n\n\n<p>carotid bruits fundoscopic abnormalities and enlarged cardiac chambers,<\/p>\n\n\n\n<p>Patient comes to the office, and you score them on HAM-D a 23 and you start an antidepressant on dose Xmg, 2 weeks later they score a 16 on the HAM-D. What would you do?<\/p>\n\n\n\n<p>Leave the dose where it is<\/p>\n\n\n\n<p>Patient is taking Zoloft 200mg and on the GAD 7 they score a 2, what do you do<\/p>\n\n\n\n<p>Leave the dose where it is<\/p>\n\n\n\n<p>Zung Depression Scale Scoring<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>50 Mild<br>60 Moderate<br>70 Severe<\/p>\n<\/blockquote>\n\n\n\n<p>25-49 is NORMAL RANGE<br>100 is max score<\/p>\n\n\n\n<p>MMSE scoring<br>0-10 severe<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>10 moderate<\/p>\n\n\n\n<p>20 mild<\/p>\n\n\n\n<p>25 Normal<\/p>\n<\/blockquote>\n\n\n\n<p>Kids under 10 years old are severely challenging to teach<\/p>\n\n\n\n<p>HIGHER THE BETTER!<\/p>\n\n\n\n<p>Teratogenic Effects<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>Lithium<\/li>\n\n\n\n<li>Carbamazepine<\/li>\n\n\n\n<li>Depakote<\/li>\n\n\n\n<li>Benzo<\/li>\n\n\n\n<li>Epstein Anomaly<\/li>\n\n\n\n<li>Neural Tube<\/li>\n\n\n\n<li>Neural Tube (specifically spina bifida, atrial septal defect, cleft palate)<\/li>\n\n\n\n<li>Floppy Baby<\/li>\n<\/ol>\n\n\n\n<p>Primary prevention<br>Efforts to prevent an injury or illness from ever occurring.<br>-Education<br>-Safety Initiatives<br>-Modifying environment<\/p>\n\n\n\n<p>Secondary Prevention<br>Efforts to limit the effects of an injury or illness that you cannot completely prevent.<br>-Early findings<br>-Screening<br>-Prompt and effective treatment<br>Example: Crisis hotline, disaster response<\/p>\n\n\n\n<p>Tertiary Prevention<br>-aims to prevent the long-term consequences of a chronic illness or disability and to support optimal functioning<br>-Rehab Services<br>-Day treatment<br>-Case management<br>-Social Skills training<\/p>\n\n\n\n<p>Pharmacokinetics<br>what the body does to the drug<\/p>\n\n\n\n<p>Pharmacodynamics<br>what the drug does to the body<\/p>\n\n\n\n<p>messenger RNA codes for<br>amino acids<\/p>\n\n\n\n<p>Poor relationships, lack of future hope, suspicious of others indicates developmental failure of what stage<br>infancy, trust vs mistrust<\/p>\n\n\n\n<p>Poor self-esteem, low self control, self-doubt, lack of independence indicates failure of what stage<br>early childhood 1-3, autonomy vs shame and doubt<\/p>\n\n\n\n<p>Lack of self-initiative, lack of goal orientation indicates failure of what stage<br>Late childhood 3-6 y\/o initiative vs guilt<\/p>\n\n\n\n<p>sense of inferiority, difficulty with working\/learning indicates a failure of what stage<br>school age 6-12 y\/o industry vs inferiority<\/p>\n\n\n\n<p>identity confusion, poor self-identification in groups indicates failure of what stage<br>adolescence 12-20 y\/o identity vs inferiority<\/p>\n\n\n\n<p>emotional isolation, egocentrism indicates a failure of what stage<br>early adulthood 20-35 y\/o intimacy vs isolation<\/p>\n\n\n\n<p>self-absorption, inability to grow and change as a person, inability to care for others indicates a failure at what stage<br>middle adulthood 35-65 y\/o generativity vs stagnation<\/p>\n\n\n\n<p>bitterness, sense of dissatisfaction with life, despair over impending death indicates failure of what stage<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>65 y\/o integrity vs despair<\/p>\n<\/blockquote>\n\n\n\n<p>agonist effect<br>Drug binds to receptors and activates a biological response<\/p>\n\n\n\n<p>Inverse agonist effect<br>Drug causes the opposite effect of agonist<\/p>\n\n\n\n<p>partial agonist effect<br>Drug does not fully activate the receptors<\/p>\n\n\n\n<p>Antagonist effect<br>Drug binds to the receptor but does not activate a biological response<\/p>\n\n\n\n<p>Herbals that interact with warfarin<br>Vitamin E<br>Omega-3<\/p>\n\n\n\n<p>Black Cohosh<br>herbal used for menopause<\/p>\n\n\n\n<p>Bellandonna<br>herbal used for anxiety<\/p>\n\n\n\n<p>chamomile<br>herbal used for sedation and anxiety<\/p>\n\n\n\n<p>Ginko<br>Herbal used to treat memory, dementia, &amp; sexual dysfunction from SSRIs<\/p>\n\n\n\n<p>Ginseng<br>Herbal product used for stress reduction, fatigue, and depression<\/p>\n\n\n\n<p>Valerian<br>herbal used for sedation<\/p>\n\n\n\n<p>Hypertensive crisis can occur when MAOI are taken with<br>Meperidine<br>Decongestants<br>TCAs<br>Atypical Antipsychotics<br>St.Johns wart<br>L-Tryptophan<br>Stimulants<br>Asthma meds<\/p>\n\n\n\n<p>Microcytic anemia<br>iron deficiency<\/p>\n\n\n\n<p>macrocytic anemia<br>due to folate or vitamin B12 deficiency<\/p>\n\n\n\n<p>Labs: Folic Acid, B12, ESR\/CRP, HGB, MCV<\/p>\n\n\n\n<p>14 y\/o with no axillary hair and no period<br>Normal tanner stage, start by 16<\/p>\n\n\n\n<p>Two classes of cardiac meds that should not be used together<br>ACES and ARBS (angiotensin receptor blocker)<br>Together can cause renal dysfunction<\/p>\n\n\n\n<p>Abnormal Trendelenburg Test<br>Hip disease, refer child out, assessed during head to toe<\/p>\n\n\n\n<p>too little acetylcholine<br>too much acetylcholine<br>Alzheimer&#8217;s<br>Parkinson&#8217;s and EPS<\/p>\n\n\n\n<p>4 D&#8217;s Discover, Dream, Design, Destiny<br>Appreciative Inquiry<\/p>\n\n\n\n<p>Cranial Nerve V<br>Trigeminal<br>Clenched Teeth<\/p>\n\n\n\n<p>WBC 1500-2000<br>Biweekly labs<br>Less than 1000 Stop<\/p>\n\n\n\n<p>How can you assess cranial nerve XII?<br>Ask patient to stick out their tongue<\/p>\n\n\n\n<p>First sign of metabolic syndrome<br>large waist circumference<\/p>\n\n\n\n<p>Grade 2\/5 hoarse systolic heart murmur<br>aortic stenosis<\/p>\n\n\n\n<p>Ibuprofen + lithium<br>increases the serum level of lithium up to double<\/p>\n\n\n\n<p>Kleinfelter&#8217;s Syndrome<br>male with more than one X chromosome (XXY)<br>-Decreased sperm, fertility issues<\/p>\n\n\n\n<p>Mental Health Parity Act<br>forbids health plans from placing lifetime or annual limits on mental health coverage that are less generous than those placed on medical or surgical benefits<\/p>\n\n\n\n<p>what does nuchal rigidity indicate?<br>meningitis<\/p>\n\n\n\n<p>Patient is on interferon and lexapro, as a provider you understand that<br>interferon can increase depression therefore you may have to increase the lexapro<\/p>\n\n\n\n<p>Patient complains of neuropathic pain and neurontin is not working<br>Try Lyrica (pregabalin) its absorbed quickly and the maximum rate of absorption is 3x of Neurontin<\/p>\n\n\n\n<p>Patient is in hospital with no family and is failing cognitive test what should you do?<br>MRI<br>Tox Screen<\/p>\n\n\n\n<p>Phases of policy making<br>formulation, implementation, evaluation<\/p>\n\n\n\n<p>Rhett Syndrome<br>a rare disorder found virtually exclusively in girls, is a neurodevelopmental disorder in which the child usually develops normally until about 6 to 18 months of age at which characteristics of the syndrome emerge; characteristics include: hypotonia (loss of muscle tone), reduced eye contact, decelerated head growth, and disinterest in play activities<\/p>\n\n\n\n<p>Signs of fetal alcohol syndrome<br>small head, smooth palpebral fissure, inner epicanthal folds, thin upper lip<\/p>\n\n\n\n<p>Tegretol side effects<br>Aplastic anemia, agranulocytosis, steven johnsons, hyponatremia. Watch with cipro and erythro<\/p>\n\n\n\n<p>Telemedicine legal question?<br>Licensing Jurisdiction for the NP must be considered<\/p>\n\n\n\n<p>A person is seen wandering the streets for 2 days<br>Delirium<\/p>\n\n\n\n<p>Medication used for serotonin syndrome<br>Cyproheptadine<\/p>\n\n\n\n<p>Why? it is an H1 blocker but is also has serotonin receptor blocking activity. Specifically, it acts to block 5-HT1A and 5-HT2A receptors which are the ones responsible for serotonin syndrome<\/p>\n\n\n\n<p>When to assess a patient in restraints?<br>initially within 1 hour; then 8 hours<\/p>\n\n\n\n<p>Why would you be concerned with immature reticulocytes?<br>Reticulocytes are involved in conditions affecting RBCs such as anemia.<br>-Low reticulocytes may be seen is iron def. anemia, pernicious anemia, folic acid deficiency, and aplastic anemia<\/p>\n\n\n\n<p>pharm treatment for agoraphobia<br>short term benzo, SSRI, SNRI, TCA, or beta-blocker off label<\/p>\n\n\n\n<p>Anorexia admission Criteria for hospitalization includes:<br>weight loss over 30% over 6 months<br>severe hypothermia temp less than 96.8<br>HR less than 40<br>BP less than 70<br>Hypokalemia less than 3mEq\/L<br>BMI&lt;16<\/p>\n\n\n\n<p>ANOVA<br>ANalysis Of VAriance &#8211; btwn means of 3 or more groups<br>An inferential statistical test for comparing the means of three or more groups<\/p>\n\n\n\n<p>precontemplation stage<br>stage of change in which people are unwilling to change their behavior<\/p>\n\n\n\n<p>Contemplation stage<br>person is considering making a change, aware that there is a problem but is not quite committed to changing<\/p>\n\n\n\n<p>Preparation Stage<br>Person has made the decision to change, is ready for action<\/p>\n\n\n\n<p>Action Stage<br>Person is engaging in specific, overt actions to change<\/p>\n\n\n\n<p>Maintanence stage<br>The person is engaging in behaviors to prevent relapse<\/p>\n\n\n\n<p>These 3 meds cause BIG FREAKING PROBLEMS<br>Strong inhibitors of 2D6<br>Bupropion, Fluoxetine, Paxil<\/p>\n\n\n\n<p>Boy tells you he wishes to be a girl and asks you not to tell the parents<br>Don&#8217;t tell<\/p>\n\n\n\n<p>BRUISE on the padded part of his arms<br>Say I see you have bruises on your arm<br>may I Ask what happened<\/p>\n\n\n\n<p>Can an advanced directive be revoked? How?<br>Yes<br>at any time<\/p>\n\n\n\n<p>Can you take Buspar during pregnancy?<br>Category B &#8211; ok if really needed.<\/p>\n\n\n\n<p>Carb and barb + Coumadin<br>Strong Inducers of 3A4 can decrease INR<\/p>\n\n\n\n<p>Who is in charge of the DEA?<br>State and Federal<\/p>\n\n\n\n<p>Common comorbidities of bipolar<br>anxiety, alcohol, substance use<\/p>\n\n\n\n<p>Conjunctival injection, munchies, psychomotor slowness?<br>Marijuana intoxication<\/p>\n\n\n\n<p>Diary Log<br>CBT<\/p>\n\n\n\n<p>Depakote and Disulfiram<br>increases INR<\/p>\n\n\n\n<p>What 3 atypicals can be used with teens?<br>Zyprexa, Abilify, Seroquel &#8211; low doses<\/p>\n\n\n\n<ul class=\"wp-block-list\">\n<li>also Risperdal<\/li>\n<\/ul>\n\n\n\n<p>Np wants to implement a certain policy in nursing.<br>start with nurse manager<\/p>\n\n\n\n<p>Phenycyclidine (PCP) can cause?<br>Nystagmus<\/p>\n\n\n\n<p>Hildegard Peplau<br>Theory of Interpersonal Relations<br>Nurse as therapeutic tool<br>-Care for the person as well as the illness<br>-Patients are PEOPLE not DIAGNOSES<\/p>\n\n\n\n<p>Patient acting out due to missing session what do you do?<br>You relate to childhood abandonment and talk about it with the patient<\/p>\n\n\n\n<p>Patient on antidepressants for 3 weeks and attempted Suicide<br>stop the medication immediately<\/p>\n\n\n\n<p>Patient shows symptoms of dizziness, tremors, sweating, What Medical Diagnosis<br>hypoglycemia<\/p>\n\n\n\n<p>Pearson&#8217;s r<br>a statistic that measures the direction and strength of the linear relation between two variables that have been measured on an interval or ratio scale<\/p>\n\n\n\n<p>pincer grasp<br>9 months<\/p>\n\n\n\n<p>Problems in the parietal lobe can lead<br>Sensory-perceptual disturbances and agnosia(inability to perceive objects)<br>R-L confusion<br>Difficulty writing (agraphia)<br>Aphasia(difficulty of language)<\/p>\n\n\n\n<p>To promote resilience in a patient with schizophrenia that lives alone consider referral to<br>-ACT<br>-Peer support<\/p>\n\n\n\n<p>Patient on Lithium and Depakote and has temp, right flank pain, brown urine. What do you do FIRST?<br>Check LFT<br>If fine then check creatinine<\/p>\n\n\n\n<p>Pt states &#8220;god did this to me.&#8221;<br>Assess spiritual needs first<\/p>\n\n\n\n<p>Patient taking breathing treatment Albuterol\/Proventil<br>Do not take MAOI or TCA<\/p>\n\n\n\n<p>p-value<br>The probability of results of the experiment being attributed to chance.<\/p>\n\n\n\n<p>Reliability<br>consistency of measurement<\/p>\n\n\n\n<p>Stereogenesis<br>identify an object without sight<br>i.e dice in hand<\/p>\n\n\n\n<p>Tagamet (cimetidine)<br>H2 receptor antagonist (antacid)<br>Increases benzo<br>Increases coumadin<\/p>\n\n\n\n<p>Trazadone concerns<br>EKG-QT prolongation<br>Priapism<br>Glaucoma<\/p>\n\n\n\n<p>Turner Syndrome<br>A chromosomal disorder in females in which either an X chromosome is missing, making the person XO instead of XX, or part of one X chromosome is deleted.<br>-Delayed puberty<br>-Amenorrhea<br>-Web neck, osteoporosis, lymphedema<br>-poor social skills<\/p>\n\n\n\n<p>To start your own firm as an NP and need to examine economic viability<br>Show Revenue and expenses<\/p>\n\n\n\n<p>3 CK muscle enzyme tests?<br>CKMM, CKBB, CKMB (normal 0.3 mmcg\/L)<\/p>\n\n\n\n<p>What are the legal ramifications of treating someone without informed consent?<br>The same as they are with informed consent<br>-Respect<br>-Beneficence<br>-Justice<\/p>\n\n\n\n<p>Two important things to measure when prescribing Zyprexa<br>Waist circumference<br>Lipids<\/p>\n\n\n\n<p>What schedule of controlled substances are NPs allowed to prescribe?<br>II-V<\/p>\n\n\n\n<p>What crania nerve is affected when you ask the patient to shrug their shoulders?<br>XI (11) Spinal Accessory<\/p>\n\n\n\n<p>These medications are renally metabolized<br>Gabapentin<br>Campral<br>Lithium<\/p>\n\n\n\n<p>grapefruit juice<br>inhibitor that can reduce the absorption of the drug by 47% therefore the drug blood levels will be increased<br>-Decrease dose of drug<\/p>\n\n\n\n<p>How do Asians see HC providers?<br>As in a position of authority.<br>Expect to give instructions and help make decisions<\/p>\n\n\n\n<p>How do you protect from the evil eye?<br>Red ribbon on an infant<br>Amulet for adults<\/p>\n\n\n\n<p>T-test<br>assesses whether the means of two groups are statistically different from each other<\/p>\n\n\n\n<p>Treatment for children with panic disorder<br>clonidine<br>guanfacine<\/p>\n\n\n\n<p>What do BCP&#8217;s do to Lamictal?<br>Inducer &#8211; will lower dose of Lamictal<\/p>\n\n\n\n<p>What does an increased retic count indicate<br>Bone marrow disorder or Vitamin Deficiency<br>Normal Range 0.5-1.5<\/p>\n\n\n\n<p>What do you see in Labs with HIV dementia<br>CD4 &lt;200<br>Viral Load is high<br>&lt;20% get it with antiretroviral treatment<\/p>\n\n\n\n<p>What is occuring in the adolescent brain?<br>Dendritic pruning<br>Emotions are controlled by amygdala<br>Prefrontal Cortex is still not fully developed (may be why young boys are risk takers)<\/p>\n\n\n\n<p>Indomethacin is a<br>NSAID- WATCH WITH LITHIUM<\/p>\n\n\n\n<p>Tramadol<br>Highly serotonergic<\/p>\n\n\n\n<p>Dissemination<br>the act of spreading widely<br>-publication-highest level<br>-Presenting at national conference<br>-Journal club<\/p>\n\n\n\n<p>Sensitivity vs. Specificity<br>sensitivity &#8211; how well a test identifies truly ill people (True positive)<br>specificity &#8211; how well a test identifies truly well people (True negative)<\/p>\n\n\n\n<p>*In medical diagnosis, testing sensitivity is the ability of a test to correctly identify those with the disease (true positive) whereas test specificity is the ability of the test to correctly identify those without the disease (true negative)<\/p>\n\n\n\n<p>What is the purpose of HIPAA?<br>National standards for electronic HC transactions<br>-National ID for providers, health plans and employers.<br>-Not SIMPLY Confidentiality.<\/p>\n\n\n\n<p>DETROL interactions<br>Topamax<br>KCL<br>Zonegran<\/p>\n\n\n\n<p>Yale-Brown Obsessive Compulsive Scale (Y-BOCS)<br>OCD<br>0-7 subclinical<br>8-15 Mild<br>16-23 Moderate<br>24-31 Severe<br>32-40 Extreme<\/p>\n\n\n\n<p>If you want to decrease the use of seclusion who would be considered the primary change agent<br>Unit staff<\/p>\n\n\n\n<p>Rennie vs Klein<\/p>\n\n\n\n<ul class=\"wp-block-list\">\n<li>right to refuse any treatment<br>Until court orders it<br>&#8220;due process&#8221;<\/li>\n<\/ul>\n\n\n\n<p>Roger vs. Oken<br>determined that patients have an absolute right to refuse treatment, but a guardian may authorize their treatment.<\/p>\n\n\n\n<p>ROGERS GUARDIAN<\/p>\n\n\n\n<p>Donalson vs O&#8217;Connor<br>Confinement<br>-It is unconstitutional to commit a person involuntarily who is not imminently dangerous to self or others<\/p>\n\n\n\n<p>Donalson was a patient hospitalized for 15 years<\/p>\n\n\n\n<p>Dusty vs United States<br>incompetent to stand trial<\/p>\n\n\n\n<p>Durham vs King<br>Insanity defense<\/p>\n\n\n\n<p>Riese vs St. Mary&#8217;s Hospital<br>7\/8 8B ruling says that there should be court determination of incompetence for involuntary committed mental persons to receive antipsychotic medications<\/p>\n\n\n\n<p>Stark Law<br>Prohibits physicians or their family members who own health care facilities from referring patients to those entities if the federal government, under Medicare or Medicaid, will pay for treatment.<\/p>\n\n\n\n<p>There is a medication class that is contraindicated when a patient has tics. What is that class of medication?<br>Stimulants &#8211;often used for ADHD<\/p>\n\n\n\n<p>What action do you take if a patient reports being a victim of abuse.<br>Safety first!<br>Provide patient with an environment of safety and reassure them of their safety in the clinic\/hospital.<\/p>\n\n\n\n<p>If you work inpatient, what do you do before discharging a patient who says they have been abused?<br>Make sure the patient will be safe.<\/p>\n\n\n\n<p>If you work inpatient, is a safety contract sufficient to determine that you are releasing a patient into a safe home?<br>No. A safety contract is not enough. The NP has to confirm that the patient is going to be safe for themselves or their environment is safe.<\/p>\n\n\n\n<p>Inpatient&#8211;for safety, where should the NP interview the patient and why?<br>Safety is more important than privacy. In an office with a door open or partially open. The day room would be a breach of confidentiality and a closed door or in their room would give the patient access to harming the NP.<\/p>\n\n\n\n<p>If a child comes into the office with their parents and say they have been abused, what action does the NP take?<br>interview the child separately from the parents THEN report to CPS<\/p>\n\n\n\n<p>If a child comes into an appointment without their parents and plays with a toy in a sexual way, what action does the NP take?<br>This makes the NP suspect abuse so initial action is to immediately report to CPS.<\/p>\n\n\n\n<p>What is the cornerstone of building a therapeutic alliance with adolescents?<br>Confidentiality with the explanation that confidentiality must be broken if they are a danger to self or others<\/p>\n\n\n\n<p>Do we interview adolescents with their parents in the room?<br>No because the adolescent may have info that they want kept confidential from their parents.<\/p>\n\n\n\n<p>If an adolescent comes in with their parents, do you keep the parents and adolescent together or separate theme?<br>Separate them and speak to adolescent alone because the adolescent has a right to confidentiality.<\/p>\n\n\n\n<p>Which ethnic group has the highest incidence of suicide and suicide attempts ?<br>Native American<\/p>\n\n\n\n<p>Native Americans believe in their healing stick. If a staff member tries to take it away, what should the NP do?<br>Educate the staff member in cultural competency and sensitivity.<\/p>\n\n\n\n<p>If a patient wants a traditional healer to accompany them to an appointment, what does the NP have to do ?<br>Get consent from the patient and allow the traditional healer to attend as well. With permission from the patient, you can include the healer in the care plan as follow up.<\/p>\n\n\n\n<p>Which labs should be taken before treating for depression or mania?<br>TSH<\/p>\n\n\n\n<p>What is the normal range of TSH?<br>0.5-5.0 mu\/L<\/p>\n\n\n\n<p>Symptoms of hypothyroidism mimic which mental illness?<br>Depression<\/p>\n\n\n\n<p>Symptoms of hyperthyroidism mimic which mental illness?<br>Mania<\/p>\n\n\n\n<p>If TSH is low i.e. 0.4 or lower, what is happening to T4 an T3 and what condition does patient have?<br>T4 and T3 will be high and patient will have hyperthyroidism<\/p>\n\n\n\n<p>If TSH is high e.g. 7.0, what is happening to T4 an T3 and what condition does patient have?<br>T4 and T3 will be low and patient will have hypothyroidism.<\/p>\n\n\n\n<p>What are the symptoms of hyperthyroidism that can mimic mania? And what are other symptoms?<br>agitation, anxiety, irritability, mood swings, weight loss.<\/p>\n\n\n\n<p>Other symptoms are heat intolerance and tachycardia<\/p>\n\n\n\n<p>What are symptoms of hypothyroidism that mimic depression? And what are other symptoms?<br>lethargy, weight gain, decreased libido.<\/p>\n\n\n\n<p>And, cold intolerance<\/p>\n\n\n\n<p>What is the Black Box warning for Depakote?<br>Pancreatitis<\/p>\n\n\n\n<p>What are physical side effects\/ dangers of Depakote (Divalproex)&#8211;for patient or fetus<br>Spina bifida in fetuses<br>Hepatotoxicity<\/p>\n\n\n\n<p>If a patient taking depakote has signs of hepatotoxicity what would those signs and symptoms be ?<br>Abdominal pain in upper right quadrant of abdomen<br>Reddish brown urine<br>Yellowing of the skin and whites of eyes<br>Fatigue<\/p>\n\n\n\n<p>If patient taking depakote has signs of hepatotoxicity, what should the NP do?<br>A liver function test &#8211;check AST and ALT levels<\/p>\n\n\n\n<p>Signs of valproic acid toxicity?<br>Disorientation<br>lethargy<br>Respiratory depression<br>Nausea\/vomiting<\/p>\n\n\n\n<p>What action do we take at signs of valproic acid (Divalproex\/Depakote) toxicity?<br>Dc medication and check depakote levels, do a LFT and check ammonia levels<\/p>\n\n\n\n<p>What are the signs and symptoms of pancreatitis<br>upper adbominal pain<br>abdominal pain that radiates to patient&#8217;s back<\/p>\n\n\n\n<p>tenderness when touching the abdomen<br>fever<br>rapid pulse<br>nausea<br>vomiting<br>oily stools<\/p>\n\n\n\n<p>What in mental illness is Kava (or Kava Kava) used for ?<br>Kava Calms<\/p>\n\n\n\n<p>anxiety<br>stress<br>insomnia<\/p>\n\n\n\n<p>What is the major side effect of Kava? What do we monitor?<br>Liver damage<br>We monitor patient for RUQ pain and do LFTs<\/p>\n\n\n\n<p>Kava is contraindicated with which meds\/med classes?<br>Xanax (Alprazolam)<br>CNS depressants\/ sedative medications<\/p>\n\n\n\n<p>Benzos (CLonazepam\/Klonopin), (Lorazepam\/ Ativan),<\/p>\n\n\n\n<p>phenobarbital (Donnatal)<\/p>\n\n\n\n<p>Zolpidem (Ambien)<\/p>\n\n\n\n<p>Lamictal and weight<br>Lamictal is the mood stabilizer that causes the least weight gain<\/p>\n\n\n\n<p>Lamictal and rash<br>Can cause Stevens Johnson<\/p>\n\n\n\n<p>What are the symptoms of Stevens Johnson syndrome?<br>Body aches, red rash, peeling skin, facial and tongue swelling<\/p>\n\n\n\n<p>Which antipsychotics cause the least weight gain?<br>ZAL<br>Ziprasodone (Geodon)<br>Aripriprazole (Abilify)<br>Lurasidone (Latuda)<\/p>\n\n\n\n<p>For patients taking antipsychotics that have caused weight gain, what routine labs do we check?<br>bmi<br>hip-to-waist ratio<br>glucose<br>lipid panel<\/p>\n\n\n\n<p>Non-pharm treatment of antipsychotic induced weight gain (AIWG)&#8211;1st line<br>Exercise and nutritional counseling<\/p>\n\n\n\n<p>This is first line treatment<\/p>\n\n\n\n<p>Pharm intervention for Antipsychotic Induced Weight Gain<br>Switch to antipsychotic with lower potential for weight gain: ZAL<\/p>\n\n\n\n<p>Black Box Warning for Carbamazepine<br>agranulocytosis, aplastic anemia, Stevens &#8211;Johnson syndrome<\/p>\n\n\n\n<p>symptoms of Aplastic anemia<br>pallor<br>fatigue<br>HA<br>fever<br>nosebleeds<br>bleeding gums<br>skin rash<br>SOB<\/p>\n\n\n\n<p>If prescribing Carbamazepine for an Asian patient, what gene do you screen for ?<br>HLA-B* 1502 allele is highly associated with Carbamazepine-induced Stevens-Johnson syndrome<\/p>\n\n\n\n<p>ANC level that indicates NP should DC clozapine or Carbamazepine?<br>An ANC less than 1000 mm3 whether patient is showing signs of infection or not.<\/p>\n\n\n\n<p>What are signs of infection for which we should monitor patients on Clozapine or Carbamazepine to DC it?<br>Sudden fever<br>Chills<br>sore throat<br>weakness<\/p>\n\n\n\n<p>Lithium and neuroprotection<br>Lithium is neuroprotective treatment of choice for bipolar disorder&#8211; can protect nerve cells from damage.<\/p>\n\n\n\n<p>What is the therapeutic range of Lithium<br>0.6-1.2 mEq\/L<\/p>\n\n\n\n<p>At what Lithium level does lithium toxicity occur?<br>1.5 mEq\/L or higher<\/p>\n\n\n\n<p>Lithium is the gold standard for treating what?<br>Mania<\/p>\n\n\n\n<p>Lithium and suicide.<br>Lithium is the only mood stabilizer with evidence of anti-suicidal effects in bipolar<\/p>\n\n\n\n<p>What is the therapeutic range of depakote?<br>50-125 micrograms (ug\/ml)<\/p>\n\n\n\n<p>What is the toxic range of depakote(valproic acid)?<br>150 microgrms (ug\/ml)<\/p>\n\n\n\n<p>Necessary labs for Lithium<br>TSH<br>Serum creatinine<br>BUN<br>Urinalysis<br>HCG for females age 12-51<\/p>\n\n\n\n<p>Signs of lithium toxicity<br>In mild lithium toxicity, symptoms include WATCAD&#8211;Weakness, Ataxia, Tremor, Concentration poorness and Diarrhea.<\/p>\n\n\n\n<p>weakness, worsening tremor, mild ataxia, poor concentration and diarrhea.<\/p>\n\n\n\n<p>With worsening toxicity, vomiting, the development of a gross tremor, slurred speech, confusion and lethargy emerge<\/p>\n\n\n\n<p>When to DC Lithium<br>serum level of 1.3 or 1.4 and signs of Lithium toxicity.<\/p>\n\n\n\n<p>with or without symptoms if 1.5 serum level &#8211;toxicity level<\/p>\n\n\n\n<p>If creatinine or BUN are high because Lithium is processed through kidney<\/p>\n\n\n\n<p>Preventative tests when prescribing antipsychotics to women<br>Take HCG test\/ aka pregnancy test before placing any woman on antipsychotic -for females of age 12-51.<\/p>\n\n\n\n<p>If patient has 4+ protein in urine while on lithium what do we do ?<br>Monitor closely for lithium toxicity<\/p>\n\n\n\n<p>What are side effects of Lithium?<br>hypothyroidism, ebstein anomaly, and nephrogenic diabetes insipidus, fine hand tremors, Maculopapular rash, GI upset(Diarrhea, vomiting, cramps, anorexia), polyuria, polydispsia, T-wave inversions, Leukocytosis (increased WBCs)<\/p>\n\n\n\n<p>What do we do in cases of Lithium toxicity?<br>DC Li and check serum Li levels<\/p>\n\n\n\n<p>What are some factors that can increase Lithium levels?<br>Kidney disease or drugs that reduce renal clearance<br>NSAIDS(ibuprofen, Indocin)<br>Thiazides (hydrochlorothiazide)<br>ACE inhibitors<br>Medications used for cardiac failure (lisinopril)<br>Dehydration<br>Hyponatremia (low sodium levels)<\/p>\n\n\n\n<p>What causes Neuroleptic Malignant Syndrome (NMS)?<br>Antipsychotics<\/p>\n\n\n\n<p>What are the symptoms of Neuroleptic Malignant syndrome (NMS)?<br>Extreme musclular rigidity<br>Mutism<\/p>\n\n\n\n<p>Elevated CPK (happens due to muscle contraction and destruction)<\/p>\n\n\n\n<p>Myoglobinuria<br>Elevated WBCs(leukocytosis)<br>Elevated LFTs<\/p>\n\n\n\n<p>What is the treatment for NMS?<br>DC the antipsychotic and treat with:<\/p>\n\n\n\n<p>There are two with different MOAs:<\/p>\n\n\n\n<p>Bromocriptine (Parlodel) which is a Dopamine (2) agonist<\/p>\n\n\n\n<p>Dantrolene which is a Muscle Relaxant<\/p>\n\n\n\n<p>What are the signs of both NMS and serotonin syndrome?<br>Hyperthermia<br>Tachycardia<br>Diaphoresis<br>Altered level of consciousness<\/p>\n\n\n\n<p>What are the signs of Serotonin Syndrome?<br>Hyperreflexia<br>Myoclonic jerks<\/p>\n\n\n\n<p>What causes serotonin syndrome?<br>SSRIs\/SNRIs\/TCAs\/MAOIs<\/p>\n\n\n\n<p>How do we treat Serotonin Syndrome?<br>DC the offending agent and treat with<\/p>\n\n\n\n<p>Cyproheptadine<\/p>\n\n\n\n<p>When switching from an SSRI to an MAOI, how long wait before starting the MAOI?<br>wait 14 days<\/p>\n\n\n\n<p>When switching from fluoxetine (Prozac) to MAOIs how long to wait before starting the MAOI?<br>5-6 weeks<\/p>\n\n\n\n<p>When switching from an MAOI to Prozac how long wait until start Prozac?<br>wait 2 weeks<\/p>\n\n\n\n<p>Why the wait times for switching from between SSRIs and MAOIs?<br>need a washout period of 5 half-lives between cessation of previous drug and introduction of new drug. This is the time it takes for the medication to degenerate out of the system<\/p>\n\n\n\n<p>Due to risk of Serotonin Syndrome, which combination of meds to we avoid?<br>Combinations of SSRIs and SNRIs or TCAs or MAOIs or St. John&#8217;s Wort<\/p>\n\n\n\n<p>If we see &#8220;regenerate&#8221; in answers related to serotonin, what do you do ?<\/p>\n\n\n\n<p>eliminate. &#8220;Regenrate&#8221; is wrong answer<\/p>\n\n\n\n<p>Why are SSRIs the 1st line antidepressants used in depression<br>because they are safer in cases of overdose<\/p>\n\n\n\n<p>Serotonin Syndrome and triptans?<br>Triptans can cause serotonin syndrome so no triptans (for migraines) or sumatriptan (imitrex)<\/p>\n\n\n\n<p>Which antidepressant to do we give a patient who is depressed and has cancer?<br>Citalopram or escitalopram because lower incidence of drug-drug interaction.<\/p>\n\n\n\n<p>Which antidepressant to do we give a patient who is depressed and has neuropathic pain?<br>SNRI or TCA<br>An SNRI is safer<\/p>\n\n\n\n<p>What do we prescribe for patients in chronic neuropathic pain?<br>Alpha 2 Delta Ligands= Gabapentin and Pregabalin (Lyrica)<\/p>\n\n\n\n<p>antidepressants and sexual dysfunction<br>SSRIs\/SNRIs can cause sexual dysfunction<\/p>\n\n\n\n<p>Important info about Wellbutrin<br>NDRIs do not cause sexual dysfunction = Wellbutrin<\/p>\n\n\n\n<p>Patient depressed +Low energy +fatigue give Wellbutrin<\/p>\n\n\n\n<p>Wellbutrin contraindicated in hx of seizures or eating d\/o = Wellbutrin increase seizure risk<\/p>\n\n\n\n<p>Black Box Warning on all depressants and responsibiltity of NP<br>increase thoughts of self-harm in adolescents.<\/p>\n\n\n\n<p>assess for frequency and severity of these thoughts at every visit.<\/p>\n\n\n\n<p>Alcohol and depression<br>If patient is depressed, assess alcohol intake because some patients use alcohol to self-medicate and this can become a barrier to treatment<\/p>\n\n\n\n<p>Prozac and Insomnia<br>Prozac can cause insomnia; encourage pt to take Prozac in the morning<\/p>\n\n\n\n<p>Mental illnesses that cause thoughts of self-harm<br>Depression<br>bipolar<br>alcohol abuse<br>eating disorder<br>schizophrenia<\/p>\n\n\n\n<p>mental illness most often associated with Homicidal ideation<br>Antisocial personality disorder<\/p>\n\n\n\n<p>Placebo rate, children, antidepressants<br>few experimental studies exist that investigate the placebo effect of antidepressants in children and adolescent (When compared to adults, children with depression have a reduced placebo representation in studies but a higher placebo rate).<\/p>\n\n\n\n<p>positive symptoms of schizophrenia<br>\u2022Hallucination, Delusion, Loose association<br>\u2022Ideas of reference (paranoia\/ paranoid delusions)<br>\u2022Agitated and bizarre behavior<\/p>\n\n\n\n<p>Negative symptoms of schizophrenia<br>\u2022Avolition &#8211; Asociality (Hygiene, Work)<br>\u2022Anhedonia &#8211; Asociality (Interest, Relationship)<br>\u2022Blunted affect<br>\u2022Paucity of thought<\/p>\n\n\n\n<p>age of onset of schizophrenia<br>18-25 males<br>25-35 in females<\/p>\n\n\n\n<p>What is going on in brain of a schizophrenic that we see in an MRI\/PET scan?<br>Ventricular enlargement<\/p>\n\n\n\n<p>Everything else is decreasing in size<br>&#8211;Different lobes<br>&#8211;Different parts of limbic system<br>&#8211;Cerebral blood flow<\/p>\n\n\n\n<p>What are the parts of the brain in which abnormalities or changes\/deficits cause agression, impulsitivity, and abstract thinking problems in schizophrenia?<br>prefrontal cortex<br>amygdala<br>basal ganglia<br>hippocampus<br>limbic regions<\/p>\n\n\n\n<p>Medications for which schizophrenics have a low tolerability so they are not as neuroprotective for schizophrenics as they are for others<br>Alpha 2 adrenergic receptor agonist (guanfacine and clonidine)<\/p>\n\n\n\n<p>Why should we not give stimulants to schizophrenics?<br>Because stimulants can potentiate dopamine release<\/p>\n\n\n\n<p>Non-pharm management of schizophrenia<br>manualized group therapy and assertive community treatment (ACT)<\/p>\n\n\n\n<p>What is ACT?<br>ACT is a form of rehabilitation post-hospitalization<\/p>\n\n\n\n<p>If a schizophrenic has a long-term history of non-adherence what may they need for in home care?<br>Referral to case management team so a nurse can go to their home and administer their medication. And, referral to aerobic exercise program<\/p>\n\n\n\n<p>What level of care is social skills training for a schizophrenic?<br>Tertiary<\/p>\n\n\n\n<p>How does exercise help schizophrenics?<br>Improve cognition<br>Improve quality of life<br>Improve long term health<\/p>\n\n\n\n<p>A schizophrenic is taking oral Haldol and is at high risk of relapse (multiple hospitalizations), what should we do about administering medication<br>switch to intramuscular Haldol<\/p>\n\n\n\n<p>Dosing for switching from oral Haldol to Haldol Decanoate<br>20 X the total daily oral doses=Decanoate dose&#8211;example:<\/p>\n\n\n\n<p>5 mg PO BID<br>LAI&#8211;20 X 10 mg = 200 mg<\/p>\n\n\n\n<p>What is the dose limit of LAI Haldol that can be given in one week?<br>100 mg<br>If they need 200 mg then give 100 mg on day one and come back in 5-7 days for another 100 mg;<\/p>\n\n\n\n<p>same for 300 mg= 100 mg day one<br>5-7 days for second 100 mg<br>and 5-7 days for third 100 mg<\/p>\n\n\n\n<p>What is a delusion?<br>Firm belief maintained despite evidence to the contrary.<\/p>\n\n\n\n<p>If doing a MSE of preschooler(3-5y\/o), what is the most important approach to<br>listen and observe cues\u2014depends on clinical observation<\/p>\n\n\n\n<p>Components of a Mental Status Exam&#8211;what do you assess with thought process assessment?<br>Assess the organization of the patient&#8217;s thoughts and ideas.<\/p>\n\n\n\n<p>Components of a Mental Status Exam&#8211;what do you assess with thought content assessment?<br>Refers to the themes that occupy the patient&#8217;s thoughts and perceptual disturbances. Ex:<br>Suicidal ideations<br>homicidal ideations<br>SI or HI plan<br>visual hallucinations<br>auditory hallucinations<\/p>\n\n\n\n<p>Mental status exam&#8211;when evaluating thought process or thought content, what are we evaluating? Are we evaluating organization of speech?<br>We are evaluting thoughts and ideas<\/p>\n\n\n\n<p>NO, we are not evaluating organization of speech<\/p>\n\n\n\n<p>The Folstein Mini Mental Status Examination is used with which population and for what ?<br>Adults&#8211;to assess cognitive decline<\/p>\n\n\n\n<p>What are some important components of the mini-mental status examination<br>Concentration\/attention\/ calculation&#8211;examples: count backward from 100 by 7s<\/p>\n\n\n\n<p>Orientation: Year, season, date, day, month, country, town, hospital, floor<\/p>\n\n\n\n<p>Registration\/ability to learn new material: say names of three unrelated objects clearly and slolwy, ask patient to repeat immediately<\/p>\n\n\n\n<p>Recall (memory): Ask patient if they can recall the three object words previously asked to remember 5 minutes after introducing the object words<\/p>\n\n\n\n<p>Fund of knowledge: Who is president\/govenor<\/p>\n\n\n\n<p>What is the purpose of the Clock drawing test ?<br>Very quick way to screen for possible dementia&#8211; takes 1-2 minutes to complete<\/p>\n\n\n\n<p>If there are impairments on the CDT (Clock Drawing Test), which part of the brain may be damaged?<br>right parietal lobe i.e. the right hemisphere of the brains<\/p>\n\n\n\n<p>What makes an antipsychotic atypical?<br>Atypical antipsychotics have both dopamine and 5HT2A receptor antagonism<\/p>\n\n\n\n<p>Lower chance of EPS syndromes<\/p>\n\n\n\n<p>Which type of antipsychotic for first psychotic episode?<br>Atypical antipsychotic that can be administered IM like Invegga, Geodon or Abilify.<\/p>\n\n\n\n<p>Dopamine Pathway: Mesocortical pathway relationship between dopamine and schizophrenic symptoms<br>Meso&#8211;not major depression but meso depression + other negative symptoms<br>decreased dopamine in the mesocortical pathway is thought to be responsible for negative and depressive symptoms of schizophrenia<\/p>\n\n\n\n<p>Nigrostriatal pathway of brain controls what ?<br>Nigrostriatal pathway mediates motor movements<\/p>\n\n\n\n<p>Dopamine blockade in this pathway can lead to increase acetylcholine levels&#8211;increase salivation, teary eyes, diarrhea<\/p>\n\n\n\n<p>Dopamine Pathway: Nigrostriatal pathway relationship between dopamine and EPS<br>Blockade of dopamine receptors in the nigrostriatal pathways can lead to EPs e.g. acute dystonia, parkinsonism, and akathisia<\/p>\n\n\n\n<p>Long standing D2 blockade in the nigrostriatal pathway can lead to Tardive Dyskinesia.<\/p>\n\n\n\n<p>Neurotransmitters and EPS<br>Increased acetylcholine and decreased dopamine can cause EPS&#8217;<\/p>\n\n\n\n<p>EPS and metoclopramide<br>Metoclopramide (Reglan) can cause EPS like Tardive dyskinesia and parkinsonism<\/p>\n\n\n\n<p>How do you treat EPS except Tardive Dyskinesia?<br>Benztropine<\/p>\n\n\n\n<p>How do we treat Tardive Dyskinesia?<br>stop\/decrease meds, switch to new anti psych that doesn&#8217;t cause tardive dyskinesia (possibly clozapine)<\/p>\n\n\n\n<p>Dopamine Pathway: Tuberoinfundibular pathway relationship between dopamine and physical symptoms<br>Blockade of D2 receptors in this pathway can lead to increased prolactin levels leading to hyperprolactinemia which clinically manifests as amenorrhea, galactorrhea (Risperidone), sexual dysfunction, and gynecomastia.<\/p>\n\n\n\n<p>Long term hyperprolactinemia can be associated with osteoporosis.<\/p>\n\n\n\n<p>Normal prolactin levels<br>Men&#8211;less than 20 ng\/ml<br>Women &#8212; less than 25 ng\/ml<\/p>\n\n\n\n<p>impact of Cytocrome P450 enzyme CYP1A2 inducers on drugs metabolized on the pathway<br>decrease serum levels of drugs that are substrates of CYP1A2 enzymes which causes subtherapeutic drug levels<\/p>\n\n\n\n<p>impact of Cytocrome P450 enzyme CYP1A2 inhibitors on drugs metabolized on the pathway<br>Increase serum levels possibly causing toxic levels of drug<\/p>\n\n\n\n<p>Olanzapine (Zyprexa) and Clozapine are both metabolized on which enzyme?<br>Cytochrome P450 Enzyme CYP1A2<\/p>\n\n\n\n<p>If a patient on olanzapine or clozapine stops smoking what do we do with the dose<br>Decrease the dose because smoking is an inducer and it is no longer decreasing the serum levels of the drug.<\/p>\n\n\n\n<p>Which are more worrisome? Inhibitors or Inducers?<br>Inhibitors because they can cause toxic levels of a drug in the body.<\/p>\n\n\n\n<p>Is Tegretol(carbamazapine) an Inducer or Inhibitor ?<br>Inducer<\/p>\n\n\n\n<p>Are clarithromycin and erithromycin inducers or inhibitors?<br>Inhibitors<\/p>\n\n\n\n<p>If a patient is taking tegretol (carbamazapine) and clarithromycin, tegretol an inducer and clarithromycin an inhibitor, what should an NP do?<br>Decrease the tegretol (carbamazapine) to avoid the inhibitors causing a toxic level of tegretol in the blood<\/p>\n\n\n\n<p>Medications that cause mania<br>Steroids<br>Disulfram (Antabuse)<br>Isoniazid(INH)<br>Antidepressants in persons with bipolar<\/p>\n\n\n\n<p>Medications that cause depression<br>Steroids<br>Beta blockeres<br>Interferon<br>Isotretinoin (Accutane)<br>Some retroviral drugs<br>Antineoplastic drugs<br>benzodiazepines<br>progesterone<\/p>\n\n\n\n<p>Steroids and psychosis<br>Steroids can cause psychosis<\/p>\n\n\n\n<p>Are flonase and prednisone steroids?<br>Yes<\/p>\n\n\n\n<p>When taking medications that cause mania and depression, how do you dose the antidepresant or antipsychotic?<br>Increase the dose.<\/p>\n\n\n\n<p>Neurotransmitters involved in addiction<br>Dopamine and GABA<\/p>\n\n\n\n<p>What causes pain in anorexics after eating?<br>Delayed gastric emptying<\/p>\n\n\n\n<p>medications that can delay gastric emptying<br>Ranitidine&#8211;Antihistamine and Antacid<br>Famotidine&#8211;Antihistamine and Antacid<br>Omeprazole&#8211;proton pump inhibitor<\/p>\n\n\n\n<p>If a med decreases the absorption of psychotropic medications, what is the best way to take it?<br>Take 2 hours apart<\/p>\n\n\n\n<p>Some medications that decrease absorption of psychotropics?<br>Antacids<br>PPIs like Protonix, Omeprazole<\/p>\n\n\n\n<p>If a patient aged 65+ is given SSRIs, for what should we monitor them?<br>monitor for increased anxiety<\/p>\n\n\n\n<p>What is a paradoxical effect of a medication?<br>when medication causes opposite effect for which is was prescribed. Avoid giving in the future<\/p>\n\n\n\n<p>Heart issues and Geodon and Citalopram<br>Geodon and Citalopram can cause QT prolongation<\/p>\n\n\n\n<p>What is the maximum recommended dosage per day for Citalopram?<br>40 mg<br>for those 65+&#8211; 20 mg<\/p>\n\n\n\n<p>What is the greatest risk factor for bipolar<br>multigenerational bipolar<\/p>\n\n\n\n<p>If a person&#8217;s first bipolar episode is after the age of 45, what is the likely cause?<br>A medical condition like a stroke<\/p>\n\n\n\n<p>What are the symptoms of Mania?<br>Distractability<br>Insomnia<br>Grandiosity<br>Flight of ideas<br>Agitation\/Activity increase<br>Sexual indiscretions (or other pleasurable activity)<br>Talkativeness<br>DIG FAST<\/p>\n\n\n\n<p>What is apoptosis<br>programmed cell death<\/p>\n\n\n\n<p>Primary symptoms of Borderline Personality Disorder<br>self-harming behavior<br>recurrent suicidal behavior<\/p>\n\n\n\n<p>What is the only treatment for Borderline Personality Disorder?<br>DBT&#8211;decreases suicidality in Borderlines<\/p>\n\n\n\n<p>Who created DBT?<br>Marsha Linehan<\/p>\n\n\n\n<p>What is the diagnostic process for borderline<br>use their journals\/diaries to help diagnose borderlines<\/p>\n\n\n\n<p>Medication for a pateint with borderline presenting with irritability, anger and self-harming behavior<br>Lithium<\/p>\n\n\n\n<p>Specific Medication for a patient with borderline presenting with depressed mood, emotional lability, interpersonal problems, rejection sensitivity, aggresion, hostility<br>Depakote<\/p>\n\n\n\n<p>What is conversion disorder?<br>a mental condition&#8211;usually begining suddenly after a stressful experience&#8211; in which a person has blindness, paralysis, or other nervous system (neurologic) symptoms that cannot be explained by medical evaluation<\/p>\n\n\n\n<p>Treatment is therapy<\/p>\n\n\n\n<p>What is an adjustment disorder?<br>An unhealthy or excessive response to an event or change within 3 months of it happening<\/p>\n\n\n\n<p>What are symptoms of adjustment disorder<br>with depressed mood &#8212; feelings of sadness, decreased interest, sleep disturbance, appetite changes<\/p>\n\n\n\n<p>with mixed disturbance of emotion and conduct &#8212; A child has a mix of symptoms from anxiety, depression and conduct dysfunction&#8211;possible peer conflict, verbal altercations, insomnia, frequent crying<\/p>\n\n\n\n<p>Differentiation between Adjustment disorder and MDD<br>Adjustment disorder has a specific cause.<\/p>\n\n\n\n<p>How do we treat Oppositional Defiant Disorder?<br>Family therapy with emphasis on child management skills; teaching parents about positive reinforcement and boundary settings.<\/p>\n\n\n\n<p>Child and parent problem-solving skills training<\/p>\n\n\n\n<p>In ODD therapy, if no parenting skills are developed, what can ODD turn into<br>Conduct Disorder<\/p>\n\n\n\n<p>What are key symptoms of conduct disorder<br>Aggression toward people or animals and property.<\/p>\n\n\n\n<p>Lack of remorse for ill deeds done<\/p>\n\n\n\n<p>Pharmacological treatment for conduct disorder<br>Targets mood and aggression&#8211;treat with antipsychotics, mood stabilizers, SSRIs and alpha agonists (Clonidine and guanfacine)<\/p>\n\n\n\n<p>Diagnosing Tourette Syndrome<br>At least 2 motor tics and at least 1 vocal tic have been present, not necessarily at the same time&#8211;for more than a year<\/p>\n\n\n\n<p>Tics are not caused by using a subtance or other medical condition<\/p>\n\n\n\n<p>When children have motor tics are they rare and permanent ?<br>No. Children&#8217;s tics are common and often temporary<\/p>\n\n\n\n<p>Primary neurotransmitter involved in Tourette Syndrome<br>Dopamine, Norepinephrine, serotonin (DNS)<\/p>\n\n\n\n<p>Hyperactivity of Dopaminergic systems in brain can lead to Tourette&#8217;s<\/p>\n\n\n\n<p>Pharmacological treatment of Tourette Syndrome<br>Clonidine (Catapres or Kapvay) or Guanfacine (Intuniv)<\/p>\n\n\n\n<p>What is acute stress disorder?<br>Psychiatric diagnosis that may occur in patients within 4 weeks after a traumatic event Features include anxiety, insomnia, re-experiencing, avoidance behaviors&#8211;basically PTSD symptoms but duration of symptoms is less than a month. (PTSD has to last at least one month)<\/p>\n\n\n\n<p>What are hallmark symptoms of PTSD?<br>Intrusive re-experiencing<br>Increased arousal (hyperarousal)<br>Avoidance of stimuli associated with trauma<\/p>\n\n\n\n<p>Pharmacological management of PTSD<br>SSRIs, TCAs, Prazosin for nightmares<\/p>\n\n\n\n<p>Non-Pharmacological management of PTSD<br>EMDR (preferred over CBT)<br>CBT<\/p>\n\n\n\n<p>What are the phases of EMDR?<br>Desensitization phase<br>Installation phase<br>body scan phase<\/p>\n\n\n\n<p>Which parts of the brain are affected in ADHD?<br>Frontal cortex<br>Basal ganglia<br>Abnormalities in prefrontal cortex&#8211;inattentive<br>Abnormalities of reticular activating system<\/p>\n\n\n\n<p>ADHD &#8212; age amphetamines are approved for<br>children age 3 to adult<\/p>\n\n\n\n<p>Heart and ADHD meds<br>Assess cardiac hx before beginning stimulants.<\/p>\n\n\n\n<p>There can be elevated heart rate and BP; increase risk of heart attack and stroke<\/p>\n\n\n\n<p>ADHD &#8212; age methylphenidate are approved for<br>children age 6 to adult<\/p>\n\n\n\n<p>ADHD&#8211; what does the dorsolateral prefrontal cortex control?<br>Executive function<br>Cognitive process such as planning, working memory<br>Problem solving<br>How to direct and maintain attention to a task<\/p>\n\n\n\n<p>Signs of stimulant abuse<br>Insomnia<br>Tremors<br>Increased BP<br>Heart palpitations<\/p>\n\n\n\n<p>ADHD&#8211;If patient starts having symptoms again during the day, what does that indicate; what should be done?<br>Medication has been cleared by the body; consider an extended release dose.<\/p>\n\n\n\n<p>If parent is anxious or scared of child starting stimulants what should be done?<br>address their anxiety\u2014give them some support before continuing with psychoedcation<\/p>\n\n\n\n<p>What is OCD?<br>Presence of anxiety-provoking obsessions (recurrent and persistent thoughts, impulses, or images) or compulsions (for example motor tics) that function to reduce the person&#8217;s subjective anxiety level<\/p>\n\n\n\n<p>Which autoimmune illness should be considered with sudden onset OCD symptoms in children?<br>PANDAS&#8211;Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcal Infections.<\/p>\n\n\n\n<p>Differentiate OCD from Tourettes<br>Tourettes= tics only<\/p>\n\n\n\n<p>OCD = intrusive\/ persistent thoughts and tics<\/p>\n\n\n\n<p>Factitious disorder<br>Condition in which a person presents with physical or mental illness symptoms that are induced.<\/p>\n\n\n\n<p>Malingering<br>symptoms are faked for secodary gain such as avoiding work or prison<\/p>\n\n\n\n<p>factitious disorder imposed on another<br>a condition in which one person induces illness symptoms in someone else<\/p>\n\n\n\n<p>Reactive Attachment Disorder<br>product of severely dysfunctional early relationships between principle caregiver and child &#8212;- results when caregiver disregards child&#8217;s physical\/emotional needs&#8211;&gt; behavioral\/interpersonal problems later in life (fearful\/inhibited\/withdrawn\/apathetic\/shows no emotion towards caregivers\/ disruptive\/disorganized)<\/p>\n\n\n\n<p>GAD (general anxiety disorder)<br>excessive worry for at least 6 months<\/p>\n\n\n\n<p>Panic Attack<br>surge of intense fear or discomfort that reaches a peak within minutes as well as a sense of impending doom<\/p>\n\n\n\n<p>Panic Disorder<br>diagnosis given when patient experiences recurrent panic attacks without apparent triggers<\/p>\n\n\n\n<p>SSRIs for treatment of panic disorder<br>fluoxetine<br>Paroxetine<br>Sertraline<br>Venlafaxine<\/p>\n\n\n\n<p>Definition and Symptoms of DMDD (disruptive mood dysregulation disorder)<br>Childhood (&lt;18) depressive disorder that includes<br>Chronic dysregulated mood (&#8220;moody&#8221;)<br>Frequent intense temper outburts\/temper tantrums<br>Severe irritability<br>Anger<\/p>\n\n\n\n<p>Treatment for panic attacks<br>Betablocker like propranolol.<\/p>\n\n\n\n<p>Contraindications for propranolol<br>Can cause bronchospasms so contraindicated in patients using bronchodilators like albuterol<\/p>\n\n\n\n<p>Anorexia Nervosa symptoms<br>Low BMI (&lt;15)<br>Amenorrhea<br>Emaciation<br>Bradycardia<br>Hypotension<\/p>\n\n\n\n<p>Action if Anorexic with BMI less than 15?<br>Refer for hospitalization. If parent refuses, report to CPS<\/p>\n\n\n\n<p>BMI of bulimia nervosa<br>Usually in normal range<\/p>\n\n\n\n<p>What is the non-pharm treatment for Oppositional Defiant Disorder?<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>Family therapy, with emphasis on child management skills;<\/li>\n\n\n\n<li>teaching parents about positive reinforcement and boundary settings<\/li>\n\n\n\n<li>Child and parent problem-solving training<\/li>\n<\/ol>\n\n\n\n<p>If no parenting skills are developed what can ODD develop into?<br>CD-conduct disorder<\/p>\n\n\n\n<p>What are the primary symptoms of Conduct disorder<br>Aggression towards human and animals and lack of remorse<\/p>\n\n\n\n<p>What is the pharm treatment for conduct disorder?<br>Meds that target mood and aggression:<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>antipsychotics<\/li>\n\n\n\n<li>mood stabilizers<\/li>\n\n\n\n<li>SSRIs<\/li>\n\n\n\n<li>alpha agonists (Clonidine and guanfacine)<\/li>\n<\/ol>\n\n\n\n<p>What are the defining symptoms of Tourette Syndrome\/Disorder<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>At least 2 motor tics and at least 1 vocal tic<\/li>\n\n\n\n<li>Tics are not caused by a substance or other medical conditions<\/li>\n<\/ol>\n\n\n\n<p>What is the expected permanence of children&#8217;s motor tics?<br>They are fairly common and can be temporary.<\/p>\n\n\n\n<p>What are the primary neurotransmitters involved in tourette&#8217;s syndrome?<br>DNS= Dopamine, Norepinephrine, Serotonin<\/p>\n\n\n\n<p>Hyperactivity of Dopaminergic systems in the brain can lead to Tourette&#8217;s<\/p>\n\n\n\n<p>What is the primary pharm treatment for Tourette&#8217;s<br>Clonidine (Catapres, Kapvay)<br>Guanfacine (Intuniv)<\/p>\n\n\n\n<p>What are secondary pharm treatments for Tourette&#8217;s ?<br>Atypical Antipsychotic<br>Haldol, Pimozide, Aripiprazole<\/p>\n\n\n\n<p>DSM-5 of Acute Stress Disorder<br>Acute stress disorder occurs within 4 weeks of traumatic event.<\/p>\n\n\n\n<p>Features include anxiety, insomnia, poor concentration, intense fear or helplessness, re-experiencing the event and avoidance behaviors&#8211;<\/p>\n\n\n\n<p>It presents as PTSD but the onset of symptoms is less than PTSD<\/p>\n\n\n\n<p>What is the minimum length of time that PTSD has to last?<br>One month<\/p>\n\n\n\n<p>Hallmark symptoms of PTSD<br>Intrusive re-experiencing<br>Increased arousal<br>Avoidance of stimuli associated with the trumatic event<\/p>\n\n\n\n<p>What is the pharm management of PTSD?<br>Prazosin for nightmares<br>SSRIs<br>TCAs<\/p>\n\n\n\n<p>What is primary important non-pharm treatment for PTSD?<br>EMDR<\/p>\n\n\n\n<p>What are the different Non-pharm treatments for PTSD?<br>EMDR<\/p>\n\n\n\n<p>CBT<\/p>\n\n\n\n<p>What are the parts of the brain involved in ADHD?<br>BAFaP<\/p>\n\n\n\n<p>Basal ganglia<\/p>\n\n\n\n<p>Abnormalities in reticular activating system<\/p>\n\n\n\n<p>Frontal cortex<\/p>\n\n\n\n<p>abnormalities in the Prefrontal cortex&#8211;inattentive type<\/p>\n\n\n\n<p>Cardiology and ADHD stimulant<br>Assess cardiac history before placing patient on stimulants as they can cause elevated heart rate and bP and increase risk of Heart attack and stroke<\/p>\n\n\n\n<p>If history or family hx of cardiac issues, get an ECG before starting.<\/p>\n\n\n\n<p>What are the ages for amphetamines?<br>3 and up<\/p>\n\n\n\n<p>what are the ages for methylphenidate?<br>Ages 6 and older<\/p>\n\n\n\n<p>What are the ages for Alpha agonist or alpha 2 adrenergic receptors agonist?<br>Age 6 and up<\/p>\n\n\n\n<p>What are the ages for Strattera?<br>Ages 6 and up<\/p>\n\n\n\n<p>Which aspect of ADHD does the Dorsolateral prefrontal cortex control?<br>Executive function<\/p>\n\n\n\n<p>Signs of Stimulant Abuse<br>Insomnia<br>tremors<br>increased blood pressure and HR<br>Heart palpitations<\/p>\n\n\n\n<p>If a patient is having ADHD symptoms during the day after having taken their stiumlant, what does that indicate and what should we do?<br>Indicates the medication has been cleared by the body<\/p>\n\n\n\n<p>We should consider an extended release<\/p>\n\n\n\n<p>What to do if parent is really anxious or scared about Medication for their kid?<br>Address their anxiety&#8211;support them then continue with psychoeducation<\/p>\n\n\n\n<p>Differentiate OCD from Tourettes<br>OCD&#8211;intrusive\/persistent thoughts and tics<\/p>\n\n\n\n<p>Tourettes: Tics only<\/p>\n\n\n\n<p>Symptoms of OCD<br>presence of anxiety provoking obsessions&#8211;recurrent and persistent thoughts, impulses, or images or compulsions for example motor tics that funtion to reduce the person&#8217;s subjective anxiety level<\/p>\n\n\n\n<p>What does PANDAS stand for and which mental illness&#8217; acute onset may be an indication of having PANDAS?<br>Should be considered in all children with sudden onset OCD symptoms<\/p>\n\n\n\n<p>Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcal Infections<\/p>\n\n\n\n<p>Malingering<br>Deliberate faking of a physical or psychological disorder motivated by secondary gain.<\/p>\n\n\n\n<p>factitious disorder<br>Condition in which a person does things to make themselves sick when they are not organically sick<\/p>\n\n\n\n<p>Reactive Attachment Disorder<br>in children, a pattern of inhibited, withdrawn, apathetic behavior toward adult caregivers and show no emotion towards caregivers.<br>The product of a severely dysfuntional early relationship between the principal caregiver and the child<\/p>\n\n\n\n<p>GAD<br>General Anxiety Disorder:<br>excessive worry for at least 6 months<\/p>\n\n\n\n<p>Panic Attack<br>Abrupt surge of intense fear of discomfort that reaches a peak within minutes; a variety of psychological and physical symptoms including a sense of impending doom<\/p>\n\n\n\n<p>Panic Disorder<br>Patient experiences recurrent unexpected panic attacks<\/p>\n\n\n\n<p>Treatment for Panic disorder<br>Fluoxetine<br>Paroxetine<br>sertraline<\/p>\n\n\n\n<p>SNRI&#8211;venlafaxine<\/p>\n\n\n\n<p>Beta Blocker&#8211;this manages the elevated BP that occurs with a panic attack<\/p>\n\n\n\n<p>Can also give benzos which aren&#8217;t as safe<\/p>\n\n\n\n<p>Beta blockers can cause bronchospasms so they are contraindicated with<br>bronchodilators like albuterol<\/p>\n\n\n\n<p>DMDD&#8211;Disruptive mood dysregulation disorder<br>Childhood depressive and mood disorder&#8211;diagnosis age 6-17<\/p>\n\n\n\n<p>Chronic dysregulated mood<br>Frequent intense temper outbursts\/ temper tantrums<\/p>\n\n\n\n<p>Hallmark symptoms of Anorexia Nervosa<br>Low BMI (15 or less than 15)<\/p>\n\n\n\n<p>Amenorrhea<br>Emaciation<br>Bradycardia<br>Hypotension<\/p>\n\n\n\n<p>BMI for Bulimia Nervosa<br>BMI usually in normal range<\/p>\n\n\n\n<p>if a patient has an irritable, depressed, labile mood, what is the first thing we should do ?<br>Administer the mood questionnaire<\/p>\n\n\n\n<p>When should we assess for nightmare disorder?<br>if pt reports a nightmare and parents or other family members have nightmares<\/p>\n\n\n\n<p>Which neurotransmitters are affected in autism?<br>Glutamate, GABA, Serotonin<\/p>\n\n\n\n<p>What are the symptoms of autism?<br>Persistent deficits in social communication and social interaction<br>No response when called by name<br>Nonverbal communication<br>Little or no eye contact<br>often like to line up, stack, or organize objects and toys in long, tidy rows<\/p>\n\n\n\n<p>Broken mirror theory of autism<br>Claims that dysfunction of the mirror neuron system may be the cause of poor social interaction and cognition<\/p>\n\n\n\n<p>If delirium is caused by ETOH or substance abuse what treatment can we give patient?<br>Benzos<\/p>\n\n\n\n<p>What are often the causes of delirium in older people and what should we test them for?<br>Infections<br>We should do a urinalysis with culture and sensitivity<\/p>\n\n\n\n<p>Dementia and which vitamins levels should be checked<br>Progressive mental decline; personality changes occur, irritability<br>Check Vit B12 and Folic Acid levels<\/p>\n\n\n\n<p>Differentiating between depression and dementia<br>with depression it is acute onset of memory problems like happening over 5 months.<\/p>\n\n\n\n<p>Also, in combination with memory issues, they have depression issues.<br>&#8220;I do not know&#8221; responses are commonly depression. In dementia, patient often confabulates answers&#8211;will not say &#8220;I do not know&#8221;<\/p>\n\n\n\n<p>Dementia memory decline happens over time i.e. over 12 months or longer<\/p>\n\n\n\n<p>Pseudodementia<br>Primary diagnosis is depression<br>acute onset of memory problems like happening over 5 months<\/p>\n\n\n\n<p>Which parts of the brain are involved in Dementia<br>subcortical&#8211;motor symptoms: lack of coordination, tremors, ataxia, dystonia<\/p>\n\n\n\n<p>Cortical: Language (aphasia) and memory impairments<\/p>\n\n\n\n<p>Early signs of HIV dementia<br>Cognitive decline<br>motor abnormalities<br>behavioral abnormalities<\/p>\n\n\n\n<p>Lewy Body Disease symptom of importance for the test<br>Presents with recurrent visual hallucinations<\/p>\n\n\n\n<p>Pick&#8217;s disease<br>AKA frontotemporal dementia\/frontal lobe dementia<\/p>\n\n\n\n<p>Personality, behavioral, and language changes (slurred) in early stage<\/p>\n\n\n\n<p>What meds treat HIV dementia?<br>Antiretrovirals<\/p>\n\n\n\n<p>if patient has history of high risk behavior and HIV dementia symptoms, what should we do?<br>give them an HIV test<\/p>\n\n\n\n<p>Pharm treatment of psychosis and agitation in dementia?<br>atypical antipsychotics<\/p>\n\n\n\n<p>What does the amygdala control?<br>aggression<br>fear<br>anxiety<br>emotions<\/p>\n\n\n\n<p>What does the Hippocampus control?<br>emotions<br>stress<br>learning<br>memory<\/p>\n\n\n\n<p>What does dopamine produced in the substantia nigra regulate?<br>motor movements<\/p>\n\n\n\n<p>If you are taking care of a terminal infant what do you do with the parents?<br>give infant to parents and allow them to grieve for their loss<\/p>\n\n\n\n<p>How should we speak of and refer about grief to children<br>Normalize grief and loss in children\u2014psychoeducation on grief responses; group therapy<br>Grief responses vary so do not tell a patient or family how they should grieve<\/p>\n\n\n\n<p>What is the most important factor in children&#8217;s healing from grief?<br>an intact family so they can adapt easily<\/p>\n\n\n\n<p>mood disorder neurotransmitters<br>DNS (dopamine, nor, serotonin+ GABA+ Glutamate )<\/p>\n\n\n\n<p>What are most important risk factors for osteoporosis?<br>Smoking<br>Caffeine<br>Lack of weight bearing exercises<br>Lack of dietary calcium and vitamin D<\/p>\n\n\n\n<p>What drugs should be avoided with Kava Kava?<br>Alprazolam<br>CNS depressants (e.g., phenobarbital, zolpidem)<\/p>\n\n\n\n<p>What is the normal range for TSH?<br>0.5 &#8211; 5.0 mu\/L<\/p>\n\n\n\n<p>What are lab values in Hyperthyroidism<br>Decreased TSH<br>Increased T4 and T3<\/p>\n\n\n\n<p>What are labs in Hypothyroidism?<br>Increased TSH<br>Decreased T4 and T3<\/p>\n\n\n\n<p>What are 5 primary symptoms of Hyperthyroidism?<br>Heat intolerance<br>Agitation, Anxiety, Irritability<br>Tachycardia<br>Mood swings<br>Weight loss<\/p>\n\n\n\n<p>What are 4 primary symptoms of Hypothyroidism?<br>Cold intolerance<br>Lethargy<br>Weight gain<br>Decreased libido<\/p>\n\n\n\n<p>Hypothyroidism mimics symptoms of what psychiatric disorder?<br>Depression<\/p>\n\n\n\n<p>Hyperthyroidism mimics symptoms of what psychiatric disorder?<br>Mania<\/p>\n\n\n\n<p>Valproic acid produces what teratogenic effect?<br>Spina bifida (neural tube defect)<\/p>\n\n\n\n<p>What is the primary organ where valproic acid is toxic?<br>Liver (hepatoxicity)<\/p>\n\n\n\n<p>What are 4 signs of hepatoxicity?<br>Abdominal pain in the URQ<\/p>\n\n\n\n<p>Reddish-brown urine<\/p>\n\n\n\n<p>Yellowing of the skin and sclera<\/p>\n\n\n\n<p>Fatigue<\/p>\n\n\n\n<p>What is the therapeutic range for valproic acid?<br>50-125 mcg\/ml<\/p>\n\n\n\n<p>What is a toxic level of valproic acid?<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>150 mcg\/ml<\/p>\n<\/blockquote>\n\n\n\n<p>What are 4 signs of valproic acid toxicity?<br>Disorientation<br>Lethargy<br>Respiratory depression<br>Nausea\/vomiting<\/p>\n\n\n\n<p>A pt on Depakote C\/O Abdominal pain in the URQ and Reddish-brown urine. What do you suspect? What do you do?<br>Hepatotoxicity; hold med, draw LFT&#8217;s (priority), DVP level<\/p>\n\n\n\n<p>With suspected toxicity with valproic acid, what steps\/labs should be drawn?<br>D\/C drug<br>Check drug level<br>Obtain LFTs<br>Obtain ammonia level<\/p>\n\n\n\n<p>A pregnant pt is taking accutane, zyprexa and abilify; what med needs to be d\/c&#8217;d?<br>Accutane (used for acne; Causes birth Defects!!!)<\/p>\n\n\n\n<p>What herbal supplement is used for anxiety, stress, and insomnia? What is the major side effect with Kava Kava?<br>Kava Kava<br>liver damage<\/p>\n\n\n\n<p>What are 5 symptoms of Stephen&#8217;s Johnson Syndrome?<br>Fever -Key sx to suspect SJS; high yield (HY)<\/p>\n\n\n\n<p>burning of mouth\/eyes -HY<\/p>\n\n\n\n<p>Body aches<\/p>\n\n\n\n<p>Severe red rash<\/p>\n\n\n\n<p>Peeling skin<\/p>\n\n\n\n<p>Facial and tongue swelling<\/p>\n\n\n\n<p>Which mood stabilizer causes the least weight gain?<\/p>\n\n\n\n<p>Lamotrigine (Lamictal)<\/p>\n\n\n\n<p>Which 3 SGAs cause the least weight gain?<\/p>\n\n\n\n<p>Lurasidone (Latuda)<br>Aripiprazole (Abilify)<br>Ziprasidone (Geodon)<\/p>\n\n\n\n<p>Which SGA is the least sedating?<br>Aripiprazole (Abilify)<\/p>\n\n\n\n<p>Your pt is taking Kava and you need to add an AS to manage their SCZ, which do you add?<br>Aripiprazole (least sedating)<\/p>\n\n\n\n<p>What are 2 hallmark symptoms of delirium?<br>Acute onset<\/p>\n\n\n\n<p>disturbance of LOC<\/p>\n\n\n\n<p>impaired cognition<\/p>\n\n\n\n<p>inattention<\/p>\n\n\n\n<p>How are agitation and psychotic symptoms treated in delirium?<br>Low dose Haldol (QTc check first)<\/p>\n\n\n\n<p>16 y\/o C\/O feeling funny after inhaling Acetone (nail polish remover). What is your action?<br>Do UDS; chances are they may be abusing other substances<\/p>\n\n\n\n<p>What 4 drugs can cause mania?<br>&#8220;SAID&#8221;<\/p>\n\n\n\n<p>Steroids<\/p>\n\n\n\n<p>Antidepressants (in BPD)<\/p>\n\n\n\n<p>Isoniazid (INH)<\/p>\n\n\n\n<p>Disulfiram (Antabuse)<\/p>\n\n\n\n<p>What 6 drugs can induce depression?<br>&#8220;SIP BARB&#8221;<\/p>\n\n\n\n<p>Steroids<\/p>\n\n\n\n<p>Interferon,<\/p>\n\n\n\n<p>Progesterone<\/p>\n\n\n\n<p>Beta blockers<\/p>\n\n\n\n<p>Accutane, (Isotretinoin)<\/p>\n\n\n\n<p>Retroviral drugs, Antineoplastic drugs<\/p>\n\n\n\n<p>Benzodiazepines<\/p>\n\n\n\n<p>Steroids can induce what 3 psychiatric states?<br>Mania<br>Depression<br>Psychosis<\/p>\n\n\n\n<p>REMEMBER SAFETY FIRST; pt s\/p assault, 1st thing you do is_____.<br>What is the best environment to interview this pt in?<br>reassure safety, provide environment of safety<br>private area with door open\/partially open<\/p>\n\n\n\n<p>What is the therapeutic range for lithium?<br>0.6-1.2 mEq\/L<\/p>\n\n\n\n<p>Many postpartum women have SI after delivery, yet they don&#8217;t have psych evals before leaving the hospital, what could a possible solution be?<br>collaborating care between psych and OBGYN to provide the best care for that pt<\/p>\n\n\n\n<p>If collaborating with a provider who is not in the same clinic or hospital, the informed consent [does\/does not] apply?<br>Does not, ie pt will need to sign consent for labs\/ chart to be sent<\/p>\n\n\n\n<p>When discharging from inpatient you want [to fax\/print off-give to pt.] labs for the outpatient provider before pt discharge?<br>fax; so the new provider has them for follow-up appointment<\/p>\n\n\n\n<p>Before referring a pt out or discharging them, it is good to ask yourself what question?<br>is there something that I could do for this patient first?<\/p>\n\n\n\n<p>What 3 things are MUST KNOWS about Lithium?<br>has neuroprotective effect for bipolar disorder<\/p>\n\n\n\n<p>the gold standard for treating manic episodes<\/p>\n\n\n\n<p>the mood stabilizer w\/ anti-suicidal effects<\/p>\n\n\n\n<p>What mood stabilizer has anti-suicidal effects?<br>Lithium<\/p>\n\n\n\n<p>What 5 labs should be drawn with lithium?<br>Thyroid panel (TSH)<br>Serum creatinine<br>BUN<br>Urinalysis<br>HCG<\/p>\n\n\n\n<p>What is a normal SerumCr range?<br>0.6-1.2 mg\/dL<\/p>\n\n\n\n<p>What is the normal range for BUN?<br>10-20 mg\/dL<\/p>\n\n\n\n<p>When should you suspect kidney disease with a urinalysis?<br>4+ protein; if pt on Lithium&#8211;&gt;monitor closely toxicity<\/p>\n\n\n\n<p>What are 7 side effects of lithium?<\/p>\n\n\n\n<p>Hypothyroidism<br>Fine hand tremors<br>Maculopapular rash<br>GI upset (N\/V\/D, cramps, anorexia)<br>Polyuria, polydipsia, diabetes insipidus<br>T-wave inversions<br>Leucocytosis<\/p>\n\n\n\n<p>What are 7 signs of lithium toxicity?<\/p>\n\n\n\n<p>Severe nausea, vomiting, diarrhea<\/p>\n\n\n\n<p>Confusion<\/p>\n\n\n\n<p>Drowsiness<\/p>\n\n\n\n<p>Muscle weakness<\/p>\n\n\n\n<p>Heart palpitations<\/p>\n\n\n\n<p>Coarse hand tremor<\/p>\n\n\n\n<p>Unsteadiness while standing or walking<\/p>\n\n\n\n<p>Bold =must knows<\/p>\n\n\n\n<p>Which 3 drugs can increase lithium levels by decreasing renal clearance?<br>NSAIDs (ibuprofen, indomethacin)<br>Thiazides<br>ACE-I<\/p>\n\n\n\n<p>What is a toxic level of lithium?<br>1.5 mEq\/L or higher<\/p>\n\n\n\n<p>At what level do you monitor lithium closely<br>(but not discontinue)?<br>1.3 -1.4 mEq\/L or higher<\/p>\n\n\n\n<p>With suspected lithium toxicity, what do you do first?<\/p>\n\n\n\n<p>D\/C Li and draw Lithium level (not VS)<\/p>\n\n\n\n<p>What is the appropriate action if lithium level is 1.5 mEq\/L?<br>D\/C lithium<br>Check lithium level<\/p>\n\n\n\n<p>What 2 conditions can increase lithium levels?<br>If this pt is going on a hike, what would you recommend ?<br>Dehydration<br>Hyponatremia<\/p>\n\n\n\n<p>Take extra water to avoid dehydration<\/p>\n\n\n\n<p>What birth defect can lithium cause?<br>Ebstein anomaly (congenital heart defect)<\/p>\n\n\n\n<p>When working with adolescents, parents often feel they have a right to know what is going on with their child, but the teen has a right to confidentiality. This can create an<br>Ethical dilemma<\/p>\n\n\n\n<p>When building a therapeutic alliance w\/ adolescents, it is important to stress<br>confidentiality;<br>what they say remains confidential unless they are wanting to harm self\/ someone else,<br><\/p>\n\n\n<p>[or they are in a relationship with someone much older (like 14 y\/o seeing a 30 y\/o)]<\/p>\n\n\n\n<p>if an adolescent C\/O abuse, even if they make the claim in the presence of the parents, what should you do?<br>interview the teen w\/o the parents; call CPS<\/p>\n\n\n\n<p>What 3 neurotransmitters are associated with ADHD?<br>DA, NE, 5HT aka serotonin &#8211;&gt;(DNS)<\/p>\n\n\n\n<p>The Frontal cortex, Basal ganglia, Reticular Activating System are associated with which psychiatric disorder?<br>ADHD<\/p>\n\n\n\n<p>Inattentive Type ADHD demonstrates abnormalities in which part of the brain?<br>Prefrontal cortex<\/p>\n\n\n\n<p>What should be done before placing a patient on stimulants for ADHD (priority)?<br>Cardiac history<\/p>\n\n\n\n<p>Excessive worry, apprehension, or anxiety about events or activities that occurs more days than not for a period of at least 6 months is<br>GAD<\/p>\n\n\n\n<p>if ADHD and tic both present, what is contraindicated?<br>Stimulants<\/p>\n\n\n\n<p>if a stimulant was tried and provided some but not enough benefit (helped, but not long enough) what can this mean?<br>the medication has been cleared from the body<\/p>\n\n\n\n<p>What are the lower age limits for stimulants for ADHD?<br>AMPH = 3yrs MPH = 6yrs<\/p>\n\n\n\n<p>When considering Clonidine or Guanfacine for a pt, the PMHNP knows?<br>Clonidine is more sedating than Guanfacine;<br>Guanfacine lasts longer than Clonidine<\/p>\n\n\n\n<p>What does thought process assess?<br>thoughts and ideas<\/p>\n\n\n\n<p>A normal thought process is described as<br>Logical<br>Linear<br>Coherent<br>Goal-oriented<\/p>\n\n\n\n<p>Moving from thought to thought and never getting to the point is called<br>Tangentiality (&#8220;goes on tangent&#8221;)<\/p>\n\n\n\n<p>Providing unnecessary detail but eventually getting to the point is called<br>Circumstantiality (&#8220;goes in circles&#8221;)<\/p>\n\n\n\n<p>Themes that occupy a patient&#8217;s thoughts and perceptual disturbances is called<br>Thought content<\/p>\n\n\n\n<p>Thought content includes 3 items<br>Suicidal ideation<br>Homicidal ideation<br>Hallucinations\/Delusions<\/p>\n\n\n\n<p>Asking a patient to count backward from 100 by seven (serial 7s), or anything going backwards measures what?<br>Concentration<br>Attention<\/p>\n\n\n\n<p>Asking the year, season, date, month, and location measures<br>Orientation<\/p>\n\n\n\n<p>Asking a patient to repeat &#8220;bed, red, ball&#8221; measures<br>Registration (ability to learn new material)<\/p>\n\n\n\n<p>Asking a patient to repeat objects 5 minutes later measures<br>Recall (memory)<\/p>\n\n\n\n<p>Asking the patient who is the president of the US or governor of state is measuring<br>Fund of Knowledge<\/p>\n\n\n\n<p>What is a simple test that can be administered in a minute or two to measure possible dementia?<br>Clock Drawing Test<\/p>\n\n\n\n<p>Impairments in the clock drawing test (CDT) can be associated with damage to<br>Right parietal lobe (right hemisphere)<\/p>\n\n\n\n<p>Hyperactivity of dopamine in the mesolimbic pathway to the dorsolateral prefrontal cortex (DLPFC) modulates<br>positive psychotic symptoms<\/p>\n\n\n\n<p>The safest drug switch strategy is to have a &#8220;washout period&#8221; of <strong><em>_<\/em><\/strong> half-lives between cessation of old med and the introduction of the new med.<\/p>\n\n\n\n<p>5 1\/2<\/p>\n\n\n\n<p>Decreased dopamine in the mesocortical pathway (dorsolateral prefrontal cortex) leads to what 2 symptoms?<br>Negative symptoms: affect, anhedonia, asociality, alogia, apathy, avolition,<br>Depressive symptoms<\/p>\n\n\n\n<p>The nigrostriatal pathway modulates<br>motor movements<\/p>\n\n\n\n<p>Dopamine has what type of relationship with acetycholine<br>Inverse<\/p>\n\n\n\n<p>Dopamine blockage in the nigrostriatal pathway leads to what 4 side effects?<br>Acute dystonia: stiff neck, muscle spasms neck\/back, painful<br>Parkinsonism syndromes<br>Akathisia<br>Tardive dyskinesia<\/p>\n\n\n\n<p>Blocking DA in the tuberoinfundibular pathway leads to<br>Increased prolactin<\/p>\n\n\n\n<p>What are 5 symptoms of hyperprolactinemia?<br>Amenorrhea<br>Galactorrhea<br>Sexual dysfunction<br>Gynecomastia<br>Osteoporosis (Long-term)<\/p>\n\n\n\n<p>What antipsychotic has the greatest effect on prolactin?<br>Risperidone<\/p>\n\n\n\n<p>What is the black box warning for carbamazepine?<br>Agranulocytosis and<\/p>\n\n\n\n<p>SJS in Asians (+) for HLA-B*1502 allele<\/p>\n\n\n\n<p>What are 8 symptoms of aplastic anemia?<br>Pallor<br>Fatigue<br>Headache<br>Fever<br>Nosebleeds<br>Bleeding gums<br>Skin rash<br>SOB<\/p>\n\n\n\n<p>In which ethnic group must you screen for the HLA-B<em>1502 allele before initiating CBZ (Tegretol) therapy? Why is it necessary? Asians risk for SJS in Asians (+) for HLA-B<\/em>1502 allele<\/p>\n\n\n\n<p>Which 2 drugs have the highest likelihood of causing agranulocytosis?<br>Clozaril<br>Carbamazepine<\/p>\n\n\n\n<p>A sudden fever, chills, a sore throat, and weakness are symptoms of<br>Agranulocytosis<\/p>\n\n\n\n<p>At what ANC should clozapine be stopped (even if asymptomatic)?<br>&lt;1000<\/p>\n\n\n\n<p>What is neutropenia?<br>&lt;2000 PMNs or WBC &lt;2000<\/p>\n\n\n\n<p>What is agranulocytosis?<br>&lt;500 ANC<\/p>\n\n\n\n<p>What does DIGFAST stand for?<br>Distractibility<\/p>\n\n\n\n<p>Indiscretion<\/p>\n\n\n\n<p>Grandiosity<\/p>\n\n\n\n<p>FOI<\/p>\n\n\n\n<p>Activity increased<\/p>\n\n\n\n<p>Sleep decreased<\/p>\n\n\n\n<p>Talkativeness<\/p>\n\n\n\n<p>What neurological symptoms might one see in Conversion Disorder?<br>Blindness<br>Mutism<br>Paralysis<br>Paresthesia (glove stocking syndrome)<br>Seizures<\/p>\n\n\n\n<p>When do symptoms typically begin with Conversion Disorder?<br>After a stressful experience (suddenly)<\/p>\n\n\n\n<p>What are 5 stressful events in a child&#8217;s life that can cause adjustment disorder?<br>Family move<br>Parent divorce\/separation<br>Loss of pet<br>Birth of sibling<br>Sudden or chronic illness<\/p>\n\n\n\n<p>A child with an Adjustment Disorder with disturbances of conduct may have what symptoms?<br>Not going to school, destroying property, driving recklessly, or fighting<\/p>\n\n\n\n<p>What are some key sx of Oppositional Defiant Disorder (ODD)?<br>NOT aggressive (key difference vs CD)<br>loses temper easily<br>annoys others<br>angry\/resentful<br>argues with authority<br>easily annoyed<br>blames others<br>spiteful<br>refuses to comply with rules\/requests from authority figures<\/p>\n\n\n\n<p>What is the mainstay treatment of Oppositional Defiant Disorder (ODD)?<br>Therapy, individual and family<\/p>\n\n\n\n<p>What is the primary therapy used to treat Oppositional Defiant Disorder (ODD)?<br>Child and parent problem-solving skills training, boundary setting<\/p>\n\n\n\n<p>A repetitive and persistent pattern of behavior in which the rights of others or societal norms or rules are violated is what disorder if under age 18?<br>Conduct Disorder<\/p>\n\n\n\n<p>What are 3 &#8220;hallmark&#8221; characteristics of Conduct Disorder?<br>aggression<\/p>\n\n\n\n<p>Destruction of property<\/p>\n\n\n\n<p>Lack of remorse<\/p>\n\n\n\n<p>What 2 factors differentiate Conduct Disorder from ODD?<br>Severity<\/p>\n\n\n\n<p>Aggression<\/p>\n\n\n\n<p>What are 4 pharmacologic treatments for Conduct Disorder?<br>Things to target mood and aggression<\/p>\n\n\n\n<p>&#8211;Antipsychotics<\/p>\n\n\n\n<p>&#8211;Mood stabilizers<\/p>\n\n\n\n<p>&#8211;SSRIs<\/p>\n\n\n\n<p>&#8211;Alpha agonists (Clon. and Guan)<\/p>\n\n\n\n<p>What type of therapy is used in the treatment of Conduct Disorder?<br>Behavioral therapy\/ problem solving skills<\/p>\n\n\n\n<p>What is the goal of multisystemic family therapy (MFT)?<br>Reduce barriers to resources for youth with problematic behavior<\/p>\n\n\n\n<p>What is Multisystemic family therapy?<br>Home-based model for youth (12-17) with serious antisocial problematic bx and criminal offenses by empowering parents with resources and skills and reducing barriers to resources that prevent families from accessing services needed for effective management of youth<\/p>\n\n\n\n<p>helps to develop natural support systems<\/p>\n\n\n\n<p>What is the range for MMSE?<br>score 0-30. Higher the better<\/p>\n\n\n\n<p>25+ normal<\/p>\n\n\n\n<p>21-24 mild<\/p>\n\n\n\n<p>10-20 moderate<\/p>\n\n\n\n<p>0-9 severe<\/p>\n\n\n\n<p>What is the range for SLUM?<br>score 0-30 Higher the better<\/p>\n\n\n\n<p>27+ normal<\/p>\n\n\n\n<p>21-26 mild<\/p>\n\n\n\n<p>&lt;21 dementia<\/p>\n\n\n\n<p>What is the range for HAM-D?<br>range 0-76<\/p>\n\n\n\n<p>0-7 normal<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>8 mild<\/p>\n\n\n\n<p>14 moderate<\/p>\n\n\n\n<p>19 mod-severe<\/p>\n<\/blockquote>\n\n\n\n<p>23+ severe<\/p>\n\n\n\n<p>Just moderate 14-18<\/p>\n\n\n\n<p>What is the moderate range for PHQ-9<br>range 0-27<\/p>\n\n\n\n<p>0-4 normal<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>5 mild<\/p>\n\n\n\n<p>10 moderate<\/p>\n\n\n\n<p>15 mod-severe<\/p>\n\n\n\n<p>20 severe<\/p>\n<\/blockquote>\n\n\n\n<p>Just moderate 10-14<\/p>\n\n\n\n<p>What is the moderate range for BDI?<br>range 0-63<\/p>\n\n\n\n<p>0-9 normal<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>10 mild<\/p>\n\n\n\n<p>19 moderate<\/p>\n\n\n\n<p>30 severe<\/p>\n<\/blockquote>\n\n\n\n<p>Just moderate 19-29<\/p>\n\n\n\n<p>What is the moderate range for HAM-A?<br>0-56<\/p>\n\n\n\n<p>0-17 mild<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>18 moderate<\/p>\n\n\n\n<p>25 severe<\/p>\n<\/blockquote>\n\n\n\n<p>Just moderate 18-24<\/p>\n\n\n\n<p>What is the moderate range for GAD-7<br>range 0-23<\/p>\n\n\n\n<p>0-4 normal<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>5 mild<\/p>\n\n\n\n<p>10 noderate<\/p>\n\n\n\n<p>15 severe<\/p>\n<\/blockquote>\n\n\n\n<p>Just moderate 10-14<\/p>\n\n\n\n<p>What is the moderate range for COWS?<br>13-24<\/p>\n\n\n\n<p>If a patient&#8217;s COWS score is between 5 &#8211; 12, what do you do?<br>Administer clonidine (mild score)<\/p>\n\n\n\n<p>If a patient&#8217;s COWS score is between 13 &#8211; 24, what do you do?<br>Administer buprenorphine (Suboxone) (moderate score)<\/p>\n\n\n\n<p>When do you administer clonidine on the COWS?<br>Definitely at score of &gt;7; prior to that (scores 5-6) it can be given<\/p>\n\n\n\n<p>When do you administer buprenorphine (Suboxone) on the COWS?<br>moderate symptoms (13-24)<\/p>\n\n\n\n<p>What is a moderate range on the CIWA?<br>16-20<\/p>\n\n\n\n<p>When do you administer prn meds such as benzodiazepine or for N\/V\/D on the CIWA?<br>Mild symptoms (8 or greater)<\/p>\n\n\n\n<p>When do you administer scheduled benzodiazepine +PRN&#8217;s on the CIWA?<br>moderate symptoms (15 or higher)<\/p>\n\n\n\n<p>If a patient has severe depression (over 18 on HAM-D) (over 14 on PHQ-9) (over 29 on BDI), how do you treat it?<\/p>\n\n\n\n<p>Medication and\/or therapy<\/p>\n\n\n\n<p>Assess for suicidal ideation<\/p>\n\n\n\n<p>If a patient has moderate depression (14 &#8211; 18 on HAM-D) (10 &#8211; 14 on PHQ-9) (19 &#8211; 29 on BDI), how do you treat it?<\/p>\n\n\n\n<p>Medication and\/or therapy<\/p>\n\n\n\n<p>If a patient has mild depression (&lt;14 on HAM-D) (&lt;10 on PHQ-9) (&lt;19 on BDI), how do you treat it?<\/p>\n\n\n\n<p>Therapy or nothing<\/p>\n\n\n\n<p>If a patient has mild anxiety (&lt;18 on HAM-A) (&lt;10 on GAD-7), how do you treat?<\/p>\n\n\n\n<p>Therapy or nothing<\/p>\n\n\n\n<p>If a patient has severe anxiety (&gt;24 on HAM-A) (&gt;14 on GAD-7) how do you treat?<\/p>\n\n\n\n<p>Medication and\/or therapy<\/p>\n\n\n\n<p>If a patient has moderate anxiety (18-24 on HAM-A) (10 &#8211; 14 on GAD-7) how do you treat it?<\/p>\n\n\n\n<p>Medication and\/or therapy<\/p>\n\n\n\n<p>What are 4 areas in the brain that can cause aggression, impulsivity, and difficulty with abstract thinking?<br>Prefrontal cortex<br>Amygdala<br>Basal ganglia<br>Hippocampus<\/p>\n\n\n\n<p>Abnormalities in the Prefrontal cortex, Amygdala, Basal ganglia, and<br>Hippocampus can cause what 3 symptoms?<br>Aggression<br>Impulsivity<br>Difficulty with abstract thinking<\/p>\n\n\n\n<p>What are 6 symptoms of NMS?<br>*Extreme muscular rigidity<\/p>\n\n\n\n<p>*Mutism<\/p>\n\n\n\n<p>*Elevated labs CPK (creatine phosphokinase), LFT&#8217;s and WBC&#8217;s<\/p>\n\n\n\n<p>Myoglobinuria<\/p>\n\n\n\n<p>autonomic instability vBP, ^HR\/RR<\/p>\n\n\n\n<p>Fever<\/p>\n\n\n\n<p>*= top 3 ways to differ from SS<\/p>\n\n\n\n<p>Elevated CPK (creatine phosphokinase) in NMS is caused from<br>muscle contraction and destruction<\/p>\n\n\n\n<p>What are 2 differentiating symptoms (Key indicators) of Serotonin Syndrome?<br>Hyperreflexia<br>Myoclonic jerks<\/p>\n\n\n\n<p>S\/S of serotonin syndrome include:<\/p>\n\n\n\n<p>what are the 4 most often seen (in bold)?<\/p>\n\n\n\n<p>\u201cShits and Shivers\u201d<\/p>\n\n\n\n<p>\u00a7 Diarrhea (shits)<\/p>\n\n\n\n<p>\u00a7 Shivering,<\/p>\n\n\n\n<p>\u00a7 Hyperreflexia\/myoclonic jerks<\/p>\n\n\n\n<p>\u00a7 Increased temperature<\/p>\n\n\n\n<p>\u00a7 Vital sign instability<\/p>\n\n\n\n<p>\u00a7 Encephalopathy<\/p>\n\n\n\n<p>\u00a7 Restlessness\/anxiety<\/p>\n\n\n\n<p>\u00a7 Sweating<\/p>\n\n\n\n<p>How do you treat NMS?<br>DC offending agent<br>Bromocriptine or<br>Dantrolene<\/p>\n\n\n\n<p>What does Dantrolene treat in NMS?<br>Muscle rigidity<\/p>\n\n\n\n<p>How do you treat Serotonin Syndrome?<br>DC offending agent<\/p>\n\n\n\n<p>Cyproheptadine: H1 antihistamine that acts to block<\/p>\n\n\n\n<p>5-HT1A and 5-HT2A receptors<\/p>\n\n\n\n<p>What combination of medications increases the risk of Serotonin Syndrome? What herbal supplement also increases the risk?<br>What class of migraine medications raises the risk?<br>multiple antidepressants (SSRI\/SNRI\/TCA\/MOAI)<br>St John&#8217;s wort &#8211;&gt; with any of the above AD classes<br>Triptans &#8211;&gt; with any of the above AD classes<\/p>\n\n\n\n<p>When switching from an MAOI to SSRI\/SNRI, how long should you wait? Why?<br>2 weeks;<\/p>\n\n\n\n<p>for the MAO to regenerate (remember MAO-I deplete MAO)<\/p>\n\n\n\n<p>When switching from Prozac to any antidepressant (TCA, SNRA, or MAOI), how long should you wait?<br>5-6 weeks<\/p>\n\n\n\n<p>When switching from an SSRI\/SNRI to a MAOI, how long should you wait? Why?<br>2 weeks; for the SSRI to degenerate<\/p>\n\n\n\n<p>What is the function of compulsions in OCD?<br>Reduce subjective anxiety level<\/p>\n\n\n\n<p>What is PANDAS? When would you suspect PANDAS?<br>Pediatric Autoimmune Neuropsychiatric Disorder associated with streptococcal infections in children<\/p>\n\n\n\n<p>new onset OCD sx- recent strep throat<\/p>\n\n\n\n<p>If a child has both intrusive thoughts and tics, his most likely diagnosis is<br>OCD<\/p>\n\n\n\n<p>If a child has multiple tics (at least 2 motor and 1 vocal) for at least 1 yr, and no ruminating thoughts, his diagnosis is most likely?<br>Tourette&#8217;s, NOTE: (the tics do not necessarily have to occur at same time)<\/p>\n\n\n\n<p>What 2 neurotransmitters are associated with OCD?<br>Serotonin<br>Norepinephrine<\/p>\n\n\n\n<p>vocal tics<br>can include coughing, grunting, throat clearing, sniffling, or making sudden, vocal outbursts<\/p>\n\n\n\n<p>A child between 7-17 years with a chronic dysregulated mood (moody for no reason), frequent intense temper outbursts, severe irritability, and anger is most likely to have what diagnosis?<br>DMDD<\/p>\n\n\n\n<p>What are some symptoms associated with lead poisoning?<br>Developmental delay (learning difficulties)<\/p>\n\n\n\n<p>Pica (paint chips)<\/p>\n\n\n\n<p>irritability<\/p>\n\n\n\n<p>gastrointestinal sx<\/p>\n\n\n\n<p>low weight<\/p>\n\n\n\n<p>Which 2 demographic characteristics are associated with lead poisoning?<br>rural areas<br>houses built in 1970&#8217;s (lead was in the paint)<\/p>\n\n\n\n<p>A child with developmental delay who eats things (such as paint chips) may have<br>Lead poisoning<\/p>\n\n\n\n<p>What 4 assessments should be done in a patient receiving an antipsychotic that causes weight gain?<br>BMI<br>Hip-to-waist ratio (waist circumference)<br>Glucose<br>Lipid panel<\/p>\n\n\n\n<p>Where is norepinephrine produced?<br>Locus coeruleus<br>Medullary reticular formation<\/p>\n\n\n\n<p>Serotonin is produced in<br>raphe nuclei in the brainstem<\/p>\n\n\n\n<p>What 3 areas of the brain is dopamine produced?<br>Substantia nigra<br>Ventral tegmental area (VTA)<br>Nucleus Accumbens<\/p>\n\n\n\n<p>Where is acetylcholine synthesized?<br>Basal nucleus of Meynert<\/p>\n\n\n\n<p>What is the function of the amygdala?<br>Emotional memories (aggression, fear, anxiety, rage [think amygdala], stress)<\/p>\n\n\n\n<p>What are 5 functions of the hippocampus?<br>Emotions<\/p>\n\n\n\n<p>Stress<\/p>\n\n\n\n<p>Learning<\/p>\n\n\n\n<p>Motivation<\/p>\n\n\n\n<p>Memory conversion ST to LT<\/p>\n\n\n\n<p>BOLD =HY<\/p>\n\n\n\n<p>What disorder is associated with persistent deficits in social communication and social interactions across multiple settings?<br>Autism Spectrum Disorder<\/p>\n\n\n\n<p>A child who likes to line up, stack, or organize objects and toys in tidy rows, little or no eye contact and does not respond when called by name may have<br>Autism<\/p>\n\n\n\n<p>What theory claims that dysfunction of a particular neuron system may be a cause of the poor social interaction and cognition in autism?<br>Broken Mirror Theory<\/p>\n\n\n\n<p>What is the age criteria for a DSM-5 diagnosis of Tourette&#8217;s Disorder?<br>tics appeared before age 18 yrs<\/p>\n\n\n\n<p>What is the pharmacological treatment of Tourette&#8217;s Disorder?<br>Antipsychotic<br>(haloperidol, pimozide, aripiprazole*)<\/p>\n\n\n\n<p>What are the 3 primary neurotransmitters involved in Tourette&#8217;s Disorder? What is special about DA?<br>DA, NE, 5HT or (DNS)<br>hyperactivity of DA can lead to tourettes<\/p>\n\n\n\n<p>What are 11 risk factors for suicide?<br>Previous suicide attempt<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>45 and older ( &gt; 55 for women)<\/p>\n<\/blockquote>\n\n\n\n<p>Male gender<\/p>\n\n\n\n<p>Divorced, single, or separated<\/p>\n\n\n\n<p>White (Caucasian)<\/p>\n\n\n\n<p>Living alone<\/p>\n\n\n\n<p>Psychiatric disorder<\/p>\n\n\n\n<p>Physical illness<\/p>\n\n\n\n<p>Substance abuse<\/p>\n\n\n\n<p>Family history of suicide<\/p>\n\n\n\n<p>Recent loss<\/p>\n\n\n\n<p>bold (must knows)<\/p>\n\n\n\n<p>What are 5 physical characteristics of Anorexia Nervosa?<br>Low BMI (below 18.5 is underweight)<br>Amenorrhea<br>Emaciation (abnormally thin)<br>Bradycardia<br>Hypotension<\/p>\n\n\n\n<p>What is the BMI in a patient with Bulimia Nervosa<br>Normal range (18.5-24.9)<\/p>\n\n\n\n<p>Intense anxiety and fear, helplessness, reexperiencing the event and avoidance behaviors within 4 weeks of a traumatic event is DSM-5 criteria for what disorder?<br>Acute Stress Disorder<\/p>\n\n\n\n<p>What are the 3 hallmark symptoms of PTSD?<br>Intrusive re-experiencing of the trauma<br>Increased arousal (hyperarousal)<br>Avoidance of stimuli associated with trauma<\/p>\n\n\n\n<p>What are 3 pharmacological treatments for PTSD<br>SSRIs<br>TCAs<br>Prazosin for nightmares<\/p>\n\n\n\n<p>What 2 nonpharmacologic treatments for PTSD?<br>EMDR<\/p>\n\n\n\n<p>CBT<\/p>\n\n\n\n<p>What are the 12 components\/symptoms of the COWS?<br>Pulse<br>Sweating<br>Restlessness<br>Pupil size<br>Bone aches<br>Running nose or tearing<br>GI upset<br>Yawning<br>Tremors<br>Anxiety\/irritability<br>Gooseflesh skin<\/p>\n\n\n\n<p>What are the 9 components\/symptoms of the CIWA Scale?<br>Nausea\/vomiting<br>Tremor<br>Sweats<br>Anxiety<br>Agitation<br>Tactile disturbances<br>Auditory disturbances<br>Headaches<br>Orientation<\/p>\n\n\n\n<p>What are 3 treatments for Alcohol Use Disorder?<br>Acamprosate (Campral)<br>Disulfiram (Antabuse)<br>Naltrexone (Vivitrol, ReVia)<\/p>\n\n\n\n<p>Which agent for the treatment of Alcohol Use Disorder is not metabolized by the liver?<br>Acamprosate (Campral)<\/p>\n\n\n\n<p>What to avoid while taking Acamprosate and for at least 2 weeks post use?<br>anything with ETOH, mouthwash, aftershave, vinegars, perfume, cough\/cold meds<\/p>\n\n\n\n<p>What are 4 therapeutic factors in Yalom&#8217;s Group Therapy?<br>Instillation of hope<br>Universality<br>Group cohesiveness<br>Altruism<\/p>\n\n\n\n<p>What is an approach to organizational change which focuses on strengths rather than weaknesses?<br>Appreciative Inquiry<\/p>\n\n\n\n<p>What are the 3 goals of Quality Improvement?<br>Projects designed to<br>&#8211;improve systems,<br>&#8211;decrease cost,<br>&#8211;improve productivity<\/p>\n\n\n\n<p>The process\/strategy of Quality Improvement is called<br>PDSA Cycle<\/p>\n\n\n\n<p>Plan<\/p>\n\n\n\n<p>Do<\/p>\n\n\n\n<p>Study<\/p>\n\n\n\n<p>Act<\/p>\n\n\n\n<p>Process, Policy reform, Policy environment, and Policy makers are the 4 components of<br>Health Policy<\/p>\n\n\n\n<p>Changes in programs and practices in Health Policy is called<br>Policy Reform<\/p>\n\n\n\n<p>What is the &#8220;policy environment&#8221; component of Health Policy?<br>The arena the process takes place in (government, media, public)<\/p>\n\n\n\n<p>What is the &#8220;policy makers&#8221; component of Health Policy?<br>Key players and stake holders<\/p>\n\n\n\n<p>What is the first action when developing Health Policy?<\/p>\n\n\n\n<p>Assess\/address organizational barriers and facilitators<\/p>\n\n\n\n<p>A treatment approach that does not focus on full symptom resolution but emphasizes resilience and control over problems and life is called<\/p>\n\n\n\n<p>Recovery Model<\/p>\n\n\n\n<p>What are 3 characteristics of the Recovery Model?<br>Nonlinear recovery, continual growth and occasional setbacks; Learning from experience (e.g. relapse)<\/p>\n\n\n\n<p>Self-directed -pt is not told what to do<\/p>\n\n\n\n<p>Individualized and Person-Centered-pt is the center of the therapy<\/p>\n\n\n\n<p>What is the SBIRT, and what is it used for?<br>Screening<\/p>\n\n\n\n<p>Brief<\/p>\n\n\n\n<p>Intervention<\/p>\n\n\n\n<p>Referral<\/p>\n\n\n\n<p>Treatment<\/p>\n\n\n\n<p>Screens for Substance Use Disorders<\/p>\n\n\n\n<p>What is the Tarasoff Principle<br>Duty to warn victim of imminent danger of homicidal patients\u2026varies by state<\/p>\n\n\n\n<p>What is the Rennie vs Klein Court case?<\/p>\n\n\n\n<p>&#8220;Rennie&#8217;s Right to Refuse and appeal&#8221;<\/p>\n\n\n\n<p>An involuntarily committed patient who has not been found incompetent, absent an emergency, has a qualified right to refuse psychotropic medications<\/p>\n\n\n\n<p>What is the Donaldson vs. O&#8217;Connor court case?<\/p>\n\n\n\n<p>&#8220;You can&#8217;t confine Donald&#8221;<\/p>\n\n\n\n<p>You cannot confine (commit a person involuntarily) who is not imminently dangerous to self or others<\/p>\n\n\n\n<p>What are 4 key components of Strategic Therapy?<br>Problem and Symptom focused<\/p>\n\n\n\n<p>Paradoxical directive\/intervention (reverse psychology) when pt non-compliant<\/p>\n\n\n\n<p>Straight forward directive- when pt compliant<\/p>\n\n\n\n<p>Reframe belief system<\/p>\n\n\n\n<p>Miracle Questions, Exception-finding questions, and Scaling Questions are used in which therapy?<br>Solution-Focused therapy<\/p>\n\n\n\n<p>Which therapy uses triangles\/triangulation, and self-differentiation?<br>Family Systems Therapy<\/p>\n\n\n\n<p>The paradoxical directive, a technique to be used with caution, is used in which therapy?<br>Strategic Therapy<\/p>\n\n\n\n<p>Hierarchies, Boundaries, and Genograms are characteristics of what therapy?<br>Structural Family Therapy<\/p>\n\n\n\n<p>Genograms are used in which 2 family therapies?<br>Family system therapy<br>Structural therapy<\/p>\n\n\n\n<p>What type of therapy should be performed when a patient presents with a cultural syndrome?<br>Brief supportive therapy<\/p>\n\n\n\n<p>What type of therapy should be used in a patient who has just lost their job?<br>Brief supportive therapy<\/p>\n\n\n\n<p>What is the most important concept in working with patients from different cultues?<br>Respect<\/p>\n\n\n\n<p>What should you teach a patient interested in meditation?<br>muscle relaxation<\/p>\n\n\n\n<p>Which ethnic group views mental illness as an imbalance between an individuals&#8217; relationship with world?<br>Native American<\/p>\n\n\n\n<p>Which ethnic group has the highest incidence of suicidal attempt and completion?<\/p>\n\n\n\n<p>Native American<\/p>\n\n\n\n<p>A culturally expected response to a stressor is called<br>Cultural Syndrome<\/p>\n\n\n\n<p>How does the PMHNP perform health promotion education in a community setting with different ethnicities<br>provide multi-cultural teaching\/education by using ethno-specific assessment parameters<\/p>\n\n\n\n<p>If a patient is regularly taking Kava, what lab should you get?<br>LFTs<\/p>\n\n\n\n<p>Releasing information to a traditional healer<br>is okay after you get clearance for informed consent<\/p>\n\n\n\n<p>Which antipsychotic has the least weight gain?<br>Ziprasidone (Geodon)<\/p>\n\n\n\n<p>If your patient doesn&#8217;t understand English, the PMHNP should<br>get a translator\/interpreter for them so they can understand you and know you are culturally competent<\/p>\n\n\n\n<p>NativeAmericans have healing sticks that can heal them of illnesses, if the patient is hospitalized inpatient, what should you do?<br>inform\/educate the staff members of cultural competency\/sensitivity re: the healing stick, the healing stick doesn\u2019t need to be taken away (make accommodations for the patient )<\/p>\n\n\n\n<p>Which SGA is the least sedating?<br>Aripiprazole (Abilify)<\/p>\n\n\n\n<p>Which 3 antipsychotic causes weight gain?<br>Quetiapine (Seroquel)<\/p>\n\n\n\n<p>Olanzapine (Zyprexa)<\/p>\n\n\n\n<p>Clozapine (Clozaril)<\/p>\n\n\n\n<p>What is the first action to take with a patient who is gaining weight from an SGA?<br>nonpharmacologic<\/p>\n\n\n\n<p>nutritional counseling (diet)<\/p>\n\n\n\n<p>exercise<\/p>\n\n\n\n<p>CBT<\/p>\n\n\n\n<p>When should clozapine be discontinued?<br>ANC &lt; 1,000 (even if asymptomatic)<\/p>\n\n\n\n<p>One of your patients receiving psychiatric medication develops an infection. What do you consider?<br>Agranulocytosis<\/p>\n\n\n\n<p>What antipsychotic is anti-suicidal in schizophrenia?<\/p>\n\n\n\n<p>Clozapine<\/p>\n\n\n\n<p>When a young woman is suspected of a toxic dose of lithium, what lab should you obtain initially?<br>HCG<\/p>\n\n\n\n<p>If a patient has signs\/symptoms of lithium toxicity but you have no labs available, what do you do?<br>DC lithium<\/p>\n\n\n\n<p>A patient with what medical condition is at risk for lithium toxicity?<br>Cardiac failure (hyponatremia)<\/p>\n\n\n\n<p>What are the key indicators for NMS?<br>Extreme muscle rigidity<\/p>\n\n\n\n<p>Increased CPK<\/p>\n\n\n\n<p>If a depressed patient is taking a triptan for migraines, what should you prescribe for depression?<br>Bupropion<\/p>\n\n\n\n<p>What is first line treatment for MDD? Why?<br>SSRIs<br>Safer in OD<\/p>\n\n\n\n<p>What are the 2 antidepressants of choice in a depressed patient with cancer? Why?<br>Citalopram (Celexa)<br>Escitalopram (Lexapro)<br>less risk for Drug-Drug Interaction<\/p>\n\n\n\n<p>What is the antidepressant of choice in a depressed patient with sexual dysfunction?<br>Bupropion<\/p>\n\n\n\n<p>What is the antidepressant of choice in a depressed patient with decreased energy?<br>Bupropion<\/p>\n\n\n\n<p>What are 3 treatments for depressed patients with neuropathic pain?<br>SNRI<\/p>\n\n\n\n<p>TCA (think safety tho)<\/p>\n\n\n\n<p>Gabapentin\/Pregabalin (alpha2 delta ligands)<\/p>\n\n\n\n<p>BOLD = know<\/p>\n\n\n\n<p>Which SSRI is most likely to cause insomnia?<br>Fluoxetine (Prozac)<\/p>\n\n\n\n<p>What question is important to ask in the social history with a depressed patient?<br>Alcohol intake (self-medicating)<\/p>\n\n\n\n<p>Suicidal ideation for children, adolescents, and young adults &lt;24 years is a BBW with what medications?<br>all Antidepressants<\/p>\n\n\n\n<p>What question should ask all adolescent patients at each visit?<br>Self harm (frequency and severity)<\/p>\n\n\n\n<p>Which 2 groups is it especially important to ask about SI at each visit?<br>Adolescence<br>Schizophrenics<\/p>\n\n\n\n<p>Alpha 2 adrenergic receptor blockers (Clonidine and Guanfacine) have lower tolerability in patients with schizophrenia. What effects from these meds are limited in this population?<br>the neuroprotective effects<\/p>\n\n\n\n<p>Why do children have a decreased placebo response with antidepressants?<br>Few evidence-based studies<\/p>\n\n\n\n<p>What is the onset of schizophrenia in males?<br>18-25<\/p>\n\n\n\n<p>What is the onset of schizophrenia in females?<br>25-35<\/p>\n\n\n\n<p>What question should ask schizophrenic patients at each visit?<br>SI<\/p>\n\n\n\n<p>What is the most likely diagnosis in a patient with homicidal ideation?<br>Antisocial Personality Disorder<\/p>\n\n\n\n<p>What would yo expect to see on an MRI\/Pet scan in a patient with schizophrenia?<br>All structures decreased EXCEPT Ventricles, they are enlarged<\/p>\n\n\n\n<p>Which class of agents should be avoided in patients with schizophrenia?<br>Stimulants (will increase DA)<\/p>\n\n\n\n<p>Abnormalities, changes\/deficits in which structures of the brain are associated with aggressive and impulsive behavior ?<br>Prefrontal cortex<br>Amygdala<br>Hippocampus<br>Basal ganglia<br>limbic Regions<\/p>\n\n\n\n<p>What is Assertive Community Treatment (ACT)?<br>Form of rehabilitation post hospitalization for Severe Mental Illness (SMI)<br>Case management approach<br>24\/7 services<\/p>\n\n\n\n<p>Can patients receive Assertive Community Treatment (ACT) while in the hospital?<\/p>\n\n\n\n<p>No; think &#8220;community&#8221; in ACT<\/p>\n\n\n\n<p>What is the best treatment program for SMI patients with a long Hx of poor medication adherence?<br>Assertive Community Treatment (ACT)<\/p>\n\n\n\n<p>Social skills training in a schizophrenic patient is what type of prevention?<br>Tertiary<\/p>\n\n\n\n<p>What are 3 benefits of aerobic exercise in schizophrenic patients?<br>Increased cognition<\/p>\n\n\n\n<p>mproves Quality of Life<\/p>\n\n\n\n<p>improves Long-term health<\/p>\n\n\n\n<p>What is a delusion?<br>A firm belief despite contrary evidence<br>Ex: Church members are part of a cult<\/p>\n\n\n\n<p>How often is Haldol Decanoate typically adminitered?<br>monthly<\/p>\n\n\n\n<p>If a patient is receiving Haldol 5 mg PO bid, how much Haldol Decanoate should they receive for the 1st month?<br>20 x [total daily dose] = first month dose<br>What is their maintenance dose?<br>20 x 10 mg = 200 mg (first month)<br>Maintenance: 10-15 x previous daily oral dose<\/p>\n\n\n\n<p>When converting oral Haldol to depot form, what the maximum dose should administer at a time?<br>100 mg R\/T adverse SE;<br>then 5-7 days later then give an additional 100 mg<\/p>\n\n\n\n<p>How do you conduct a MSE in a preschooler (3-5 yo)<br>Clinical observation (listen\/observe)<\/p>\n\n\n\n<p>What is included in thought content?<br>SI\/HI, plan<\/p>\n\n\n\n<p>What are the 5 components of the MMSE (Folstein&#8217;s)<br>Concentration\/attention\/calculation<br>Orientation<br>Registration\/ability to learn new material<br>Recall\/memory<br>Fund of Knowledge<\/p>\n\n\n\n<p>What pharmacologic characteristic makes SGA&#8217;s unique? What does this help prevent?<br>5-HT2A receptor antagonism<br>EPS<\/p>\n\n\n\n<p>What antipsychotic should you give to a patient with their first psychotic episode?<br>If they are a harm to themselves or others how should it be administered?<br>What is the risk of this type of administration?<br>SGA<\/p>\n\n\n\n<p>IM Invega, Geodon or Abilify<\/p>\n\n\n\n<p>increase risk for EPS (acute dystonia)<\/p>\n\n\n\n<p>Which 4 SGAs are available in IM form?<br>Ziprasidone (Geodon)<\/p>\n\n\n\n<p>Olanzapine (Zyprexa)<\/p>\n\n\n\n<p>Aripiprazole (Abilify)<\/p>\n\n\n\n<p>Paliperidone (Invega)<\/p>\n\n\n\n<p>What agent other than antipsychotics can increase prolactin and can cause TD?<\/p>\n\n\n\n<p>Metoclopramide (Reglan)<\/p>\n\n\n\n<p>What do you do if a patient is on olanzapine (Zyprexa) and stops smoking?<br>Decrease olanzapine dose (he stopped the inducer)<\/p>\n\n\n\n<p>What effect does tobacco have on CYP450?<br>1A2 inducer<\/p>\n\n\n\n<p>What class of drugs are primarily inhibitors?<br>Antibiotics<\/p>\n\n\n\n<p>How does erythromycin and clarithromycin affect CYP450?<br>Inhibitors<\/p>\n\n\n\n<p>What do you do if a patient on carbamazepine is given an inhibitor like erythromycin?<br>Decrease CBZ dose<br>(avoid aplastic anemia)<\/p>\n\n\n\n<p>If a depressed patient is receiving a drug that is associated with depression (like interferon), what do you do?<br>Increase dose of antidepressant<\/p>\n\n\n\n<p>What 7 signs\/symptoms are associated with stimulant abuse?<br>Insomnia<br>Tremors<br>Irritability,<br>Mood swings.<br>Agitation,<br>Anxiety<br>CV (increased HR\/BP, palpitations)<\/p>\n\n\n\n<p>A patient who c\/o pain after eating, bloating and fullness, what do you suspect?<br>Delayed gastric emptying from anorexia<\/p>\n\n\n\n<p>What 2 classes of medications delay gastric emptying?<br>H2 antagonists: Famotidine (Pepcid)<\/p>\n\n\n\n<p>PPIs: Omeprazole (Prilosec)<\/p>\n\n\n\n<p>What 2 classes of medications interfere with the absorption of psychiatric medications and should be spaced apart by 2 hours?<br>Antacids<br>PPIs: Omeprazole (Prilosec), Pantoprazole (Protonix)<\/p>\n\n\n\n<p>What is a common side effect of SSRIs in the older population<br>Anxiety<\/p>\n\n\n\n<p>What is a paradoxical effect of benzodiazepines?<br>Increased anxiety<br>(avoid in future)<\/p>\n\n\n\n<p>What is apotosis?<br>Cell death\/neuronal loss<\/p>\n\n\n\n<p>What is the greatest risk factor for bipolar disorder?<br>What is the first question you ask with a patient with suspected bipolar disorder?<br>genetics (very heritable)<\/p>\n\n\n\n<p>fm Hx<\/p>\n\n\n\n<p>What should you consider in a 45 y\/o patient that presents with manic symptoms?<br>Medical condition<\/p>\n\n\n\n<p>if a pt w\/ SCZ is controlled on Risperdal; but has been prescribed a steroid by the PCP, what might you need to do?<br>increase the anti-psychotic<\/p>\n\n\n\n<p>What are 2 differences between mania and hypomania?<br>More severe<br>Increased duration (7 days vs. 4 days)<\/p>\n\n\n\n<p>A patient who is manic, irritable mood, and uncooperative is most likely to have what disorder?<br>bipolar disorder<\/p>\n\n\n\n<p>What are key symptoms to consider in a patient you suspect of mania or hypomania?<br>Grandiosity or exaggerated self esteem<br>lack of sleep (and not tired)<br>talkativeness or pressured speech<\/p>\n\n\n\n<p>What is a hallmark feature of borderline personality disorder?<br>Self-harming behavior (recurrent)<\/p>\n\n\n\n<p>What therapy in borderline personality disorder been shown to decrease suicidal ideation?<br>DBT<\/p>\n\n\n\n<p>Who developed DBT?<br>Marsha Linehan<\/p>\n\n\n\n<p>In DBT, diagnosis of borderline disorder requires activity?<br>journaling\/diary<\/p>\n\n\n\n<p>A patient with borderline personality disorder with symptoms of depressed mood, rejection sensitivity, and emotional lability should be prescribed?<\/p>\n\n\n\n<p>A patient with borderline personality disorder with symptoms of irritability, anger, self-harm could be given?<br>Valproic acid\/Depakote\/DVP<\/p>\n\n\n\n<p>Lithium<\/p>\n\n\n\n<p>A hospitalized patient with a reaction to a stressful event is most likely to have what diagnosis?<br>Adjustment Disorder<\/p>\n\n\n\n<p>A child with a history of juvenile detention (e.g., fire setting, forced sex) is most likely to have what diagnosis?<br>Conduct Disorder<\/p>\n\n\n\n<p>Is a child with ODD or Conduct Disorder more likely to need medication?<br>Conduct Disorder<\/p>\n\n\n\n<p>If a child develops tics after beginning a stimulant, should he be diagnosed with Tourette&#8217;s?<br>No<br>(tics caused by a substance is an exclusionary diagnosis)<\/p>\n\n\n\n<p>Are motor tics permanent?<br>Not always<\/p>\n\n\n\n<p>What is factitious disorder imposed on another?<br>Munchausen syndrome by proxy, caretakers make up or produce physical illnesses in the individual receiving care. Usually parent &#8211;&gt; child<\/p>\n\n\n\n<p>What is required if you suspect a factitious disorder imposed on another (Munchausen syndrome)?<br>Report to CPS<br>Child abuse (duty to report)<\/p>\n\n\n\n<p>What is Factitious Disorder<br>Faking illness to get medical attention or sympathy<\/p>\n\n\n\n<p>A patient with factitious disorder is most likely to have what type of demographic?<br>Unmarried healthcare worker<\/p>\n\n\n\n<p>What is malingering?<br>Feigning illness for secondary gain ($)<\/p>\n\n\n\n<p>(not a psychiatric diagnosis)<\/p>\n\n\n\n<p>A patient who claims disability or worker&#8217;s comp when there is no injury is most likely<br>Malingering (not a psychiatric diagnosis)<\/p>\n\n\n\n<p>A child who has been neglected, abuse, or comes from a foster home, is most likely to have what diagnosis?<br>Reactive Attachment Disorder<\/p>\n\n\n\n<p>A child who shows no emotion or is withdrawn may have what disorder?<br>Reactive Attachment Disorder<\/p>\n\n\n\n<p>A disorder that presents like PTSD, but symptoms last less than a month is most likely to have<br>Acute Stress Disorder<\/p>\n\n\n\n<p>How does a panic attack differ from Panic Disorder?<\/p>\n\n\n\n<p>acute (peaks w\/in minutes), impending doom<\/p>\n\n\n\n<p>How does Panic Disorder differ from a panic attack?<\/p>\n\n\n\n<p>Chronic, recurrent, unexpected<\/p>\n\n\n\n<p>How do you treat a patient with panic disorder who is taking albuterol for asthma?<\/p>\n\n\n\n<p>SSRI or benzodiazepine<\/p>\n\n\n\n<p>(don&#8217;t give beta blocker)<\/p>\n\n\n\n<p>What is the treatment of choice for acute symptoms of panic disorder?<\/p>\n\n\n\n<p>Beta blocker (not addictive)<\/p>\n\n\n\n<p>How do you treat test anxiety for maintenance therapy?<br>SSRI<\/p>\n\n\n\n<p>How do you treat text anxiety acutely?<br>Beta blocker<\/p>\n\n\n\n<p>Benzodiazepine<\/p>\n\n\n\n<p>What structure of the brain is involved with attention, executive function, planning and cognitive processes (working memory, problem solving) that affects ADHD?<br>Dorsolateral prefrontal cortex<\/p>\n\n\n\n<p>Children with inattentive ADHD are most likely to have an abnormality in which part of the cortex?<br>prefrontal<\/p>\n\n\n\n<p>What do you recommend to a mother of a child with ADHD as an aftercare plan on admission?<\/p>\n\n\n\n<p>Brief supportive therapy (not after dc)<\/p>\n\n\n\n<p>If a patient present with irritability, depression, and lability, which questionnaire do you administer?<br>Mood Disorder Questionnaire (MDQ)<\/p>\n\n\n\n<p>What disorder resembles bipolar disorder in children (7 and 17 years)?<br>Disruptive Mood Dysregulation Disorder (DMDD)<\/p>\n\n\n\n<p>What should you assess initially in a child who presents with nightmares?<\/p>\n\n\n\n<p>If other family members have the same problem<\/p>\n\n\n\n<p>aftercare starts<br>upon admission<\/p>\n\n\n\n<p>What are 2 key factors in the diagnosis of Autism Spectrum Disorder?<br>Persistent deficits in social communication and social interaction across multiple settings<\/p>\n\n\n\n<p>A child that does not establish eye contact is likely to have<br>autism spectrum disorder<\/p>\n\n\n\n<p>When is the only time you should give a benzodiazepine to a patient with delirium?<br>Alcohol or substance withdrawal<\/p>\n\n\n\n<p>An older female has delirium and you suspect a UTI, what 2 labs should you order?<br>UA<br>C\/S<\/p>\n\n\n\n<p>An older patient who has had mental decline (&gt;1 year) with chronic irritability and personality changes should be assessed for<br>Dementia<\/p>\n\n\n\n<p>What are 2 labs that should be obtained in an older patient suspected of having a metabolic cause of dementia?<br>B12<br>Folic acid<\/p>\n\n\n\n<p>What is the initial treatment for a patient with dementia and psychosis?<br>Nonpharmacologic treatment<\/p>\n\n\n\n<p>If a patient with dementia does not respond to nonpharmacologic treatment, what do you do?<br>Administer SGA<\/p>\n\n\n\n<p>What type of dementia is HIV-related? cortical or subcortical<br>Subcortical<\/p>\n\n\n\n<p>A patient who presents with a cognitive deficit, decreased coordination, and behavioral symptoms should be assessed for what type of dementia?<br>HIV-related<\/p>\n\n\n\n<p>In a patient with suspected HIV-related dementia, what is your initial action?<br>Obtain HIV test<\/p>\n\n\n\n<p>What is first-line pharmacologic treatment for a patient with HIV-related dementia?<br>Antiretroviral treatment<\/p>\n\n\n\n<p>An older adult who has had cognitive impairment for over a year and who tries to answer (may not be able to) or confabulates is most likely to have a diagnosis of<br>Dementia<\/p>\n\n\n\n<p>An older adult who has had cognitive impairment for less than 5 months and who answers your questions with &#8220;I don&#8217;t know,&#8221; most likely has a diagnosis of<br>Depression (pseudodementia)<\/p>\n\n\n\n<p>An older patient who has symptoms of irritability, agitation, hallucinations, and delusions is most likely to have<br>Depression (vs. dementia)<\/p>\n\n\n\n<p>What are 2 ways to determine whether an older person with decreased memory has depression or dementia?<br>screening tools (SLUMS, Mini-Cog, MMSE)<\/p>\n\n\n\n<p>onset of symptoms<\/p>\n\n\n\n<p>A patient who presents with difficulties with executive function (poor judgment), slurred speech, personality and behavior changes, difficulties with language comprehension, lack of empathy, and poor social skills is most likely to have what type of dementia?<br>Frontotemporal (Pick&#8217;s)<\/p>\n\n\n\n<p>A patient with visual hallucinations is most likely to have what type of demenetia?<br>Lewy Body<\/p>\n\n\n\n<p>A patient who presents with cognitive dysfunction (decision making), lack of empathy, and difficulty with impulse control and emotions is most likely to have damage in what brain structure?<br>Anterior cingulate<\/p>\n\n\n\n<p>A patient with damage to the cerebellum will demonstrate what?<br>Lack of balance<\/p>\n\n\n\n<p>What condition may cause pica (eating things that are not food &#8211; paint chips)?<br>Lead poisoning<\/p>\n\n\n\n<p>What 5 neurotransmitters are associated with mood disorders?<br>Dopamine<br>Norepinephrine<br>Serotonin<br>GABA<br>Glutamate<\/p>\n\n\n\n<p>What do you do when an infant is about to die?<br>Give him to his parents to grieve<\/p>\n\n\n\n<p>What 2 activities should be encouraged with grieving patients?<br>Family support<br>Support groups<br>(don&#8217;t give prescriptive advice)<\/p>\n\n\n\n<p>Smoking, Caffeine, Low calcium\/vitamin D in diet, Decreased weight,<br>and Lack of weight bearing exercises are risk factors for<br>Osteoporosis<\/p>\n\n\n\n<p>long term prolactinemia is associated with?<br>Osteoporosis<\/p>\n\n\n\n<p>Is Interpersonal Therapy offered individually as well as in group therapy?<br>Yes<\/p>\n\n\n\n<p>If a patient is reluctant to participate in group therapy, what do you do?<br>Continue group therapy<br>Start individual therapy<\/p>\n\n\n\n<p>Which is more effective &#8211; cognitive therapy or CBT?<br>CBT<\/p>\n\n\n\n<p>Problem-solving, Skills training, Exposure therapy, Roleplaying\/modeling, and relaxation are techniques used in<br>Behavioral Therapy<\/p>\n\n\n\n<p>What are 2 common techniques used in CBT?<br>Cognitive restructuring<br>Journaling<\/p>\n\n\n\n<p>Self-directed growth and self-actualization to find meaning in life are key concepts of what type of therapy?<br>Humanistic (person-centered)<\/p>\n\n\n\n<p>What is the best therapy for dealing with conflicts with others (e.g., spouse, co-workers)?<br>Interpersonal therapy<\/p>\n\n\n\n<p>How long does interpersonal therapy typically last?<br>12-16 weeks<\/p>\n\n\n\n<p>If a patient is noncompliant, what is a technique that is used in Strategic Therapy?<br>Paradoxical directive\/intervention<\/p>\n\n\n\n<p>What is the benefit of paradoxical directive<br>Promote self-awareness (ie med adherence or fear of failure)<\/p>\n\n\n\n<p>If a patient is compliant, what is a technique that is used in Strategic Therapy?<br>Straight Forward Directive<\/p>\n\n\n\n<p>What type of therapy is a therapist using when he asks &#8220;what worked before when you had this problem&#8221;<br>Solution-focused therapy<\/p>\n\n\n\n<p>How can a child&#8217;s body image, resilience, relationships, and social anxiety be improved?<br>Physical activity<\/p>\n\n\n\n<p>When may a closed-ended question (yes\/no) be necessary rather than an opened-ended question?<br>Children<br>(when a narrative cannot be constructed)<\/p>\n\n\n\n<p>If a child does not respond or open or closed-ended questions, what to you do?<br>talk to parents<\/p>\n\n\n\n<p>What therapeutic factor does open-ended questions instill?<br>empathy<\/p>\n\n\n\n<p>Example of open ended questions<br>How did you fall?<br>Why do you think you are feeling sad?<br>Tell me more<\/p>\n\n\n\n<p>If only the wife shows up for a couple appointment, what do you do?<br>Reschedule the appointment<\/p>\n\n\n\n<p>A mother reports that her child was sodomized. What 2 things do you do?<br>Contact CPS<br>Crisis therapy<\/p>\n\n\n\n<p>A mother reports her 5-y\/o was raped by his 16 y\/o brother. What 2 things do you do?<br>crisis therapy<br>separate brothers<\/p>\n\n\n\n<p>What lab do you draw in a patient with rheumatoid arthritis<br>ESR<\/p>\n\n\n\n<p>What does PICOT stand for?<br>P- Patient population of interest<br>I &#8211; Intervention of interest<br>C &#8211; Comparison of interest<br>O &#8211; Outcome<br>T &#8211; Time<\/p>\n\n\n\n<p>How do you provide evidenced-based care?<br>Have access to current journals<\/p>\n\n\n\n<p>A baby with a shrill cry is likely to have<br>Increased intracranial pressure<\/p>\n\n\n\n<p>What do you tell a mother when she reports her preschool child is playing with their genitals?<br>That is normal between 3-6 yrs old (Phallic stage)<\/p>\n\n\n\n<p>What do you tell an male adolescent who is concerned about nipple swelling and tenderness and breast enlargement?<br>This is normal between 9-16 yo.<br>It will disappear within 6 mo.<\/p>\n\n\n\n<p>How do you treat decreased sexual drive in a older female patient?<br>Testosterone<br>(improves blood flow to pelvic region)<\/p>\n\n\n\n<p>Why are women more likely to get intoxicated and have alcohol-induced hepatotoxicity?<br>Decreased alcohol dehydrogenase<\/p>\n\n\n\n<p>What primitive reflex causes a newborn to grasp vigorously any object touching the palm or fingers or placed in the hand? when does it go away?<br>Grasp\/palmar reflex; disappears by 5-6 mons<\/p>\n\n\n\n<p>Infant reflex where a baby will startle in response to a loud sound or sudden movement is called______and it disappears by <strong><em>_<\/em><\/strong>?<br>Moro\/startle reflex; disappears by 5-6 mons<\/p>\n\n\n\n<p>What is the Babinski (Plantar) reflex? When does the Babinski (Plantar) reflex disappear?<br>the big toe is extended and the other toes fan in response to the brushing of the sole of the foot<br>disappears at 2 yrs<\/p>\n\n\n\n<p>What is the absorption rate of PDE-5 inhibitors (Viagra)?<br>Rapid absorption<\/p>\n\n\n\n<p>What 2 deficiencies are associated with<br>macrocytic anemia?<br>Folic acid<br>B12<\/p>\n\n\n\n<p>With normocytic, macrocytic anemia, what 3 labs do you obtain?<br>B12, folic acid, Ferritin(iron)<\/p>\n\n\n\n<p>What deficiency is associated with microcytic anemia?<br>Iron<\/p>\n\n\n\n<p>Acute or chronic blood loss (GI bleeding, heaving menses) can cause what type of anemia?<br>Normocytic<\/p>\n\n\n\n<p>When vital signs are not normal in a thin women, you suspect<br>Anorexia nervosa<\/p>\n\n\n\n<p>You see a patient with suspected anorexia nervosa. What do you do?<br>Refer out for medical evaluation<\/p>\n\n\n\n<p>If a patient with anorexia nervosa refuses to see a PCP, what do you do?<br>Contact CPS<\/p>\n\n\n\n<p>If AN patient is medically unstable and parents refuse hospital treatment, what do you do?<br>contact CPS<\/p>\n\n\n\n<p>What alternative medicine treatment is used with pain and depression?<br>Acupuncture<\/p>\n\n\n\n<p>What act prevents a patient from unlawful hospitalization (can leave AMA)?<br>Habeus corpus<br>(usually max of 28 days)<\/p>\n\n\n\n<p>A pedi patient presents with paresthesia, fatigue, unsteady gait, confusion, arm\/leg weakness and asymmetric body movement of extremities. What do you suspect and what do you do?<br>Disseminated Encephalomyelitis<br>Neuro exam<\/p>\n\n\n\n<p>If a patient is unable to remember events, how do you establish a diagnosis?<br>Provide anchor (memorable events) to establish diagnosis<\/p>\n\n\n\n<p>How do you implement policy affecting NPs nationwide?<br>Host online forum\/give survey to obtain information (vs. letters)<\/p>\n\n\n\n<p>How do you implement policy that coworkers are against?<br>Tell them how the policy will increase quality of patient care<\/p>\n\n\n\n<p>What is the initial step in providing continuous improvement of Quality of Care in an outpatient setting?<br>Create an instrument to monitor outcomes<\/p>\n\n\n\n<p>What will you see in a laboratory test in a patient with an autoimmune disease?<br>Increased cytokine levels<\/p>\n\n\n\n<p>What are 2 ways to treat enuresis (night-time bedwetting)?<br>Alarm clock<br>Desmopressin (DDAVP)<\/p>\n\n\n\n<p>What is the mechanism of action of Desmopressin (DDAVP)?<br>Decreases urine production<br>(via antidiuretic hormone &#8211; vasopressin)<\/p>\n\n\n\n<p>When considering the relationship between receiving samples from a drug rep and sponsored education, what is there a concern for, and what should you do?<br>Conflict of Interest<br>Create policy to address COI<\/p>\n\n\n\n<p>A conflict of interest may occur in what 2 scenarios?<br>Receiving samples of drug rep<br>Attending an industry sponsored educational program<\/p>\n\n\n\n<p>A patient calls because he read on the internet about a BBW on his medication. What do you do?<br>You don&#8217;t need to discontinue drug.<br>Research benefits and risks<\/p>\n\n\n\n<p>What are 2 things you should do when you prescribe off-label?<br>Document<br>Provide full disclosure (risks\/benefits)<\/p>\n\n\n\n<p>When you prescribe trazodone to a male patient, what should you do?<br>Warn about priapism and document<\/p>\n\n\n\n<p>Smoking, increased weight\/obesity, and DM are risk factors for what disorder?<br>Sleep Apnea<\/p>\n\n\n\n<p>What is tolerance?<br>Decreased effect with continuous use<\/p>\n\n\n\n<p>What is the goal or Reflective Practice?<br>Improve practice<\/p>\n\n\n\n<p>Providing debriefing techniques, feedback to staff and strategies to learn from experiences is called<\/p>\n\n\n\n<p>Reflective Practice<\/p>\n\n\n\n<p>If a patient has mild symptoms on the COWs, what should you do?<br>Give clonidine or a muscle relaxant<\/p>\n\n\n\n<p>If a patient scores 8 or more on the COWS during opiate withdrawal, what should you do?<br>Administer clonidine or a muscle relaxant<\/p>\n\n\n\n<p>If a patient has moderate symptoms on the COWS what should you do?<br>Administer buprenorphine\/naloxone (Suboxone)<\/p>\n\n\n\n<p>When should you administer buprenorphine\/naloxone (Suboxone) in a patient withdrawing from opiates?<br>Moderate symptoms (13 or higher)<\/p>\n\n\n\n<p>If a patient scores 13 or higher on the COWS scale during opiate withdrawal, what do you do?<br>Administer buprenorphine\/naloxone (Suboxone)<\/p>\n\n\n\n<p>Why should methadone not be used to treat opiate withdrawal?<br>Arrhythmias<\/p>\n\n\n\n<p>Which benzodiazepine is used most often to treat alcohol withdrawal?<br>Diazepam (longer half-life)<\/p>\n\n\n\n<p>If a patient with liver dysfunction or hepatitis is undergoing alcohol withdrawal, what benzodiazepine should he receive?<br>Lorazepam<\/p>\n\n\n\n<p>If a patient presents with opiate withdrawal, what should you do?<br>Refer to residential center to administer protocol (don&#8217;t initiate in outpatient setting)<\/p>\n\n\n\n<p>If a pregnant patient presents with opiate withdrawal, what do you do?<br>Refer to residential center (not CPS)<\/p>\n\n\n\n<p>What do you suspect when a patient presents with CNS activation (hand tremor, insomnia, anxiety, psychomotor agitation, seizures) and<\/p>\n\n\n\n<p>Autonomic hyperactivity (sweating, increased HR, increased BP) and<\/p>\n\n\n\n<p>N\/V and hallucinations\/illusions?<\/p>\n\n\n\n<p>alcohol withdrawal<\/p>\n\n\n\n<p>What do you suspect when a patient who presents with muscle cramps, bony aches, diarrhea\/abdominal cramps, rhinorrhea, lacrimation, goosebumps, pupil dilation increased BP\/HR and yawning?<br>Opiate withdrawal<\/p>\n\n\n\n<p>adolescents have a right to confidentiality, but not<br>privacy<\/p>\n\n\n\n<p>Instillation of Hope, Universality, Altruism, Increased development of socialization skills, Imitative behaviors, Interpersonal learning, Group cohesiveness, Catharsis, Existential factors, and Corrective refocusing are<\/p>\n\n\n\n<p>BOLD= focus on more<\/p>\n\n\n\n<p>Therapeutic Factors which differentiate group therapy from individual therapy<\/p>\n\n\n\n<p>Experiencing optimism through observing the improvement of others in the group is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Instillation of Hope<\/p>\n\n\n\n<p>Learning to give to others is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Altruism<\/p>\n\n\n\n<p>Sharing experiences\/feelings in group clients realize they are not alone is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Universality<\/p>\n\n\n\n<p>The &#8220;natural laboratory&#8221; is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Increased development of social skills<\/p>\n\n\n\n<p>Group members modeling their behavior after other members of group\/therapist is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Imitative behavior<\/p>\n\n\n\n<p>Developing a sense of belonging in the group is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Group cohesiveness<\/p>\n\n\n\n<p>Interacting with others to increase adaptive interpersonal relationships is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Interpersonal learning<\/p>\n\n\n\n<p>Group members dealing with the meaning of their existence is which of Yalom&#8217;s therapeutic factors?<\/p>\n\n\n\n<p>Existential factors<\/p>\n\n\n\n<p>Participants reexperience family conflicts, allows them to recognize and change\/correct behaviors that may be problematic is which of Yalom&#8217;s Therapeutic Factors?<\/p>\n\n\n\n<p>Corrective refocusing<\/p>\n\n\n\n<p>Openly expressing their feelings which were previously suppressed is which of Yalom&#8217;s Therapeutic Factors?<\/p>\n\n\n\n<p>Catharsis<\/p>\n\n\n\n<p>Focusing on the strength of the group is key to what organizational change strategy?<\/p>\n\n\n\n<p>Appreciative Inquiry<\/p>\n\n\n\n<p>Who determines the Scope of Practice?<br>State legislature (BON)<br>(varies state to state)<\/p>\n\n\n\n<p>Who determines the Standard of Practice?<\/p>\n\n\n\n<p>ANA<br>(doesn&#8217;t change)<\/p>\n\n\n\n<p>What are 8 exceptions to confidentiality? (know all)<br>Unconscious (to save a life)<\/p>\n\n\n\n<p>Intends to harm self\/others<\/p>\n\n\n\n<p>Court order (subpoena, summons)<\/p>\n\n\n\n<p>Attorneys in litigation<\/p>\n\n\n\n<p>Insurance companies<\/p>\n\n\n\n<p>Mandatory reporting (diseases)<\/p>\n\n\n\n<p>Tarasoff (duty to warn)<\/p>\n\n\n\n<p>Child or elder abuse<\/p>\n\n\n\n<p>What are the 5 elements of informed consent?<br>Nature and purpose of treatment\/procedure<br>Risks and benefits of treatment<br>Risks and benefits of not undergoing treatment<br>Alternative procedures or treatments<br>Diagnosis and prognosis<\/p>\n\n\n\n<p>What are the 7 ethical principles?<br>Justice<br>Beneficence<br>Nonmaleficence<br>Fidelity<br>Autonomy<br>Veracity<br>Respect<\/p>\n\n\n\n<p>Fairness (according to gender, orientation, or ethnicity) is what ethical principle?<br>Justice<\/p>\n\n\n\n<p>Discontinuing a medication that causes side effects is an example of what ethical principle?<br>Nonmaleficence<\/p>\n\n\n\n<p>Denying a benzodiazepine to a patient with a SUD is an example of what ethical principle?<br>Nonmaleficence<\/p>\n\n\n\n<p>&#8220;Doing good&#8221; and promoting well-being is what ethical principle?<br>Beneficence<\/p>\n\n\n\n<p>Giving a patient a medication to treat symptoms is an example of<br>Beneficence<\/p>\n\n\n\n<p>Being true and loyal is what ethical principle?<br>Fidelity<\/p>\n\n\n\n<p>&#8220;Telling the truth&#8221; is what ethical principle?<br>Veracity<\/p>\n\n\n\n<p>A patient&#8217;s right to refuse medication is an example of what ethical principle?<br>Autonomy<\/p>\n\n\n\n<p>Treating everyone with equal respect is what ethical principle?<br>Respect<\/p>\n\n\n\n<p>What are 2 important roles to perform as a client advocate?<br>support the client&#8217;s best interest while respecting family&#8217;s role<br>reduce stigma of Mental illness<\/p>\n\n\n\n<p>What is the best way to reduce stigma?<br>Education (esp. large audience)<\/p>\n\n\n\n<p>A work environment that improves patient safety through continuous learning designing safe systems is<\/p>\n\n\n\n<p>Just Culture<\/p>\n\n\n\n<p>What treatment approach does not focus on mental illness, is nonlinear (occasional setbacks), and stresses learning from experience?<br>Recovery Model<\/p>\n\n\n\n<p>In the recovery model, the clinician focuses less on the Dx and tries to focus\/foster the the pts <em>__<\/em>?<br>interests, abilities and dreams<\/p>\n\n\n\n<p>What is the PDSA cycle<br>A quality improvement process or strategy<\/p>\n\n\n\n<p>What is an example of a Quality Improvement Project?<br>RETROSPECTIVE Chart review<\/p>\n\n\n\n<p>When implementing health policy, what is the first thing you do?<br>Assess barriers and facilitators<br>(prior to meeting with stakeholders)<\/p>\n\n\n\n<p>What should you do before warning someone about harm (Tarisoff act)?<br>Contact Board of Nursing<\/p>\n\n\n\n<p>A key component of Piaget&#8217;s Sensorimotor stage is<br>Object permanence<\/p>\n\n\n\n<p>Object permanence develops in which of Piaget&#8217;s developmental stages?<br>Sensorimotor<\/p>\n\n\n\n<p>What are 2 key components of Piaget&#8217;s Preoperational Stage?<br>Magical thinking<br>Symbols and language<br>thoughts of monsters being real, if they think of someone getting hurt and it happens, they think they caused it.<\/p>\n\n\n\n<p>According to Piaget, at what stage do children display magical thinking and use language and symbols more?<\/p>\n\n\n\n<p>Preoperational<\/p>\n\n\n\n<p>What is a key component of Piaget&#8217;s Concrete Operations Stage?<br>See things from other&#8217;s perspectives (less egocentric)<br>develops concepts of<br>&#8211;conservation: clay is always clay<br>&#8211;reversibility: water to ice turns back to water<\/p>\n\n\n\n<p>According to Piaget, during what stage do children see things from other&#8217;s perspectives &#8211; less egocentric?<\/p>\n\n\n\n<p>Concrete operations<\/p>\n\n\n\n<p>Conservation and reversibility occur during what stage per Piaget?<br>Concrete<\/p>\n\n\n\n<p>What is the key component of Piaget&#8217;s Formal Operations stage?<br>Abstract logic (science projects, hypotheses)<\/p>\n\n\n\n<p>According to Erikson, what period (age) is the development of meaningful relationships important?<\/p>\n\n\n\n<p>Infancy (0 &#8211; 1 yr.)<\/p>\n\n\n\n<p>According to Erikson, what period (age) is self-control a key component?<\/p>\n\n\n\n<p>Early childhood (1 &#8211; 3 yrs.)<\/p>\n\n\n\n<p>According to Erikson, what period (age) is being a &#8220;self-starter&#8221; (self-directed) a key factor?<\/p>\n\n\n\n<p>Late childhood (3 &#8211; 6 yrs.)<\/p>\n\n\n\n<p>According to Erikson, what period (age) is personal sense of identity a key factor?<\/p>\n\n\n\n<p>Adolescence (12 &#8211; 20 yrs.)<\/p>\n\n\n\n<p>According to Erikson, what period (age) is competence important, especially with classmates?<\/p>\n\n\n\n<p>School age (6 &#8211; 12 yrs.) Industry vs inferiority<\/p>\n\n\n\n<p>According to Erikson, what period (age) are committed relationships a key factor?<\/p>\n\n\n\n<p>Early adulthood (20 &#8211; 35 yrs.) intimacy vs isolation<\/p>\n\n\n\n<p>According to Erikson, what period (age) is the ability to care for others important (e.g., parenting)?<\/p>\n\n\n\n<p>Middle adulthood (35 &#8211; 65 yrs.) generativity vs stagnation<\/p>\n\n\n\n<p>According to Erikson, what period (age) does fulfillment and comfort with life occur?<\/p>\n\n\n\n<p>Late adulthood (&gt;65) Integrity vs despair<\/p>\n\n\n\n<p>What 3 neurotransmitters are associated with MDD?<br>DNS (all decreased)<\/p>\n\n\n\n<p>What 5 neurotransmitters are associated with bipolar disorder?<br>DNS<br>Glutamate (increased)<br>GABA<\/p>\n\n\n\n<p>What 2 neurotransmitters are involved with Addictive Disorders?<br>DA (decreased)<br>GABA (decreased)<\/p>\n\n\n\n<p>What 2 neurotransmitters are involved with OCD?<br>5HT (decreased)<br>NE (decreased)<\/p>\n\n\n\n<p>What 4 neurotransmitters are associated with schizophrenia?<br>DA (increased)<br>Glutamate (increased)<br>GABA (decreased)<br>5HT (decreased)<\/p>\n\n\n\n<p>What 3 neurotransmitters are associated with Autism Spectrum Disorder?<br>Glutamate (increased)<br>GABA (decreased)<br>5HT (decreased)<\/p>\n\n\n\n<p>What neurotransmitters are associated with anxiety?<br>NE (increased)<br>5HT (decreased)<br>GABA (decreased)<\/p>\n\n\n\n<p>What neurotransmitters are associated with Alzheimer&#8217;s?<br>ACh (decreased)<br>Glutamate (decreased)<\/p>\n\n\n\n<p>What neurotransmitters are associated with Parkinson&#8217;s Disease?<br>DA (decreased)<br>ACh (Increased)<\/p>\n\n\n\n<p>What 4 psychiatric disorders have dysregulation of dopamine, norepinephrine, and serotonin (DNS)?<br>ADHD<br>MDD<br>BPD<br>Tourette&#8217;s<\/p>\n\n\n\n<p>Which 5 psychiatric disorders have dysregulation of the GABA receptor?<br>BPD<br>Schizophrenia<br>Autism<br>SUD<br>Anxiety<\/p>\n\n\n\n<p>What 4 psychiatric disorders have dysregulation of glutamate receptors?<br>BPD<br>Schizophrenia<br>ASD<br>Alzheimer&#8217;s<\/p>\n\n\n\n<p>Which 2 psychiatric disorder have dysregulation of acetylcholine receptor?<br>Alzheimer&#8217;s<br>Parkinson&#8217;s<\/p>\n\n\n\n<p>What 3 disorders have dysregulation of glutamate and gabapentin?<br>autism spectrum disorder<br>bipolar disorder<br>schizophrenia<\/p>\n\n\n\n<p>You are concerned a 5-yo child may be sexually abused. How do you conduct the interview?<br>Interview child and parents separately if child can communicate<\/p>\n\n\n\n<p>You see a child playing with a toy in a sexual manner. What do you do?<br>contact CPS<\/p>\n\n\n\n<p>What are the 2 most important aspects of interviewing an adolescent?<br>Rapport<br>Confidentiality (explain exceptions)<\/p>\n\n\n\n<p>How should an adolescent be interviewed<br>separately form parents<\/p>\n\n\n\n<p>If an adolescent is dating someone the same age, do you need to tell the parents?<br>No<\/p>\n\n\n\n<p>If an adolescent is dating someone of the same gender, do you need to tell the parents?<br>No<\/p>\n\n\n\n<p>What should you ensure before discharging a patient from the hospital?<br>can they stay safe and cope effectively<\/p>\n\n\n\n<p>Should a patient be interviewed in a day room?<br>No (not confidential)<\/p>\n\n\n\n<p>When do you draw a T3 or T4<br>If TSH is abnormal<\/p>\n\n\n\n<p>What symptom occurs with high levels of ammonia?<br>confusion<\/p>\n\n\n\n<p>What 4 symptoms are similar with serotonin syndrome and NMS?<br>Hyperthermia<br>Diaphoresis<br>Tachycardia<br>Altered LOC<\/p>\n\n\n\n<p>Thoughts of self-harm are greatest in what 5 disorders (according to order) [I left this one\u2026but I&#8217;ve not heard of it ranked]<br>MDD<br>BPD<br>Alcohol use disorder<br>Eating disorder<br>schizophrenia<\/p>\n\n\n\n<p>Are oral contraceptives inducers or inhibitors?<br>inducers<\/p>\n\n\n\n<p>A person with pressured speech, no sleep and inflated self-esteem, may suspect<br>bipolar disorder<\/p>\n\n\n\n<p>If you can determine an identifying factor (an event) to why a person is depressed, what is their most likely diagnosis?<br>Adjustment disorder with depressed [or anxious, both etc.] mood<\/p>\n\n\n\n<p>Can tics be a normal behavior?<br>yes<\/p>\n\n\n\n<p>Language and memory are the 2 primary deficits in which type of dementia?<br>What are 2 examples?<br>Cortical<br>ALZ and Mad cow Ds<\/p>\n\n\n\n<p>Motor and behavioral (depression, irritability, apathy) are the 2 primary deficits in which type of dementia?<br>What are 3 examples?<br>Subcortical<br>HIV dementia, Huntington&#8217;s and Parkinson&#8217;s<\/p>\n\n\n\n<p>What is the value of integrative medicine (E.g., ObGyn + PMHNP)?<br>increased mental health access to those who need it most<\/p>\n\n\n\n<p>What is the most important factor in a child who is grieving?<br>Intact family system<\/p>\n\n\n\n<p>How do you treat a child who is grieving?<br>Normalize grief and loss<br>(don&#8217;t tell them what to do\/how to grieve)<br>(responses vary)<br>supportive group therapy<\/p>\n\n\n\n<p>How do you apply your knowledge of a relationship between ADHD and substance abuse to practice?<br>Screen ADHD patients for substance use<br>Screen SUD patients for ADHD<\/p>\n\n\n\n<p>When you look up data about a patient in social media, what are you violating?<br>patient&#8217;s trust<\/p>\n\n\n\n<p>Prior to sending prescriptions to a pharmacy, what do you need to do?<br>obtain patient consent<\/p>\n\n\n\n<p>If patient is unable to give consent, what do you do?<br>assess for involuntary treatment (don&#8217;t forcefully give medication)<\/p>\n\n\n\n<p>The ethical principle that is associated with preventing imminent danger to patient is?<br>nonmaleficence<\/p>\n\n\n\n<p>Sending a patient to a crisis stabilization unit rather than a hospital is honoring what patient right?<br>least restrictive environment<\/p>\n\n\n\n<p>You plan to teach the patient about their illness. What do do you do first?<br>assess knowledge<\/p>\n\n\n\n<p>In what age groups should you obtain HCG?<br>12-51<\/p>\n\n\n\n<p>What are normal prolactin levels for males and females?<br>Male: &lt;20<br>Female: &lt;25<\/p>\n\n\n\n<p>serotonin is implicated in what 2 areas?<br>mood and sleep<\/p>\n\n\n\n<p>What 2 symptoms might you see in a patient with a frontal lobe tumor?<br>social skill deficit<br>inappropriate affect<\/p>\n\n\n\n<p>What is the term for symptoms caused by a medical treatment or drug therapy? How do you assess it?<br>Iatrogenic S\/S, assess medical history to ID if S\/S are caused by the medication or not<\/p>\n\n\n\n<p>If you refer a patient to a residential center for alcohol withdrawal, what can you provide them for symptom control?<br>Hydroxyzine or buspirone (not benzodiazepine)<\/p>\n\n\n\n<p>What does perjury mean?<br>Lying or withholding information under oath<\/p>\n\n\n\n<p>Which 2 parties can you not disclose information without the patient&#8217;s consent?<br>family member<br>another provider<\/p>\n\n\n\n<p>As part of reflective practice, what strategy is used post event?<br>debriefing strategies<\/p>\n\n\n\n<p>When a medication error occurs, what is the first thing to assess per just culture?<br>Pt safety; assess the patient<\/p>\n\n\n\n<p>What cardiac SE is geodon (ziprasidone) known for?<br>QTc prolongation<\/p>\n\n\n\n<p>the max dose of Citalopram is 20mg\/day in the elderly client, why?<br>risk for QTc prolongation (Note:max 40mg in adults &lt;65)<\/p>\n\n\n\n<p>Parkinson&#8217;s disease is caused by<br>damage or loss of the dopamine-producing cells of the midbrain, leading to dopamine depletion in the basal ganglia<\/p>\n\n\n\n<p>Pseudoparkinsonism<br>caused by DA blockade in the nigrostriatal pathway causing sx resembling Parkinson&#8217;s Ds; tremor, shuffling gait, drooling, rigidity<\/p>\n\n\n\n<p>In a trauma focused cognitive interview, what questions help build rapport?<br>non-judgmental questions<br>&#8220;how has that trauma affected you?&#8221;<br>&#8220;When did this happen&#8221;<\/p>\n\n\n\n<p>There are 3 phases to EMDR, they are\u2026<br>&#8220;DIB&#8221;<br>Desensitization<br>installation<br>Body Scan<\/p>\n\n\n\n<p>the goal of EMDR is<br>to achieve adaptive resolution<\/p>\n\n\n\n<p>Having the pt visualize the trauma, verbalize the negative thoughts or maladaptive beliefs; remain attentive to physical sensations while the also maintaining rhythmic eye movements is part of which EMDR Phase?<br>Desensitization phase<\/p>\n\n\n\n<p>Instructing the pt to block out negative thoughts, to breathe deeply, and verbalize what they are thinking, feeling, or imagining occurs during which EMDR Phase?<br>Desensitization phase<\/p>\n\n\n\n<p>Which EMDR phase closely resembles Cognitive Therapy and why?<br>Installation phase; because replacing negative thought with positive thought<\/p>\n\n\n\n<p>Having the pt visualize the trauma alongside the positive thought, then scan their body for tension is which EMDR phase? Why is this important to do?<br>Body Scan; successful resolution will result in the pt having no body tension when seeing the trauma and the positive memory side by side; if (+) tension, pt is still working through to achieve resolution<\/p>\n\n\n\n<p>telemedicine\/telehealth legal issues include<br>confidentiality<\/p>\n\n\n\n<p>med errors<\/p>\n\n\n\n<p>Jurisdictions<\/p>\n\n\n\n<p>Authority over licensure<\/p>\n\n\n\n<p>if the PMHNP want to change an old Tx for a newer way to Tx a condition, what should it be based on?<br>the change should be evidence based<\/p>\n\n\n\n<p>How are rating scales best used?<br>At baseline -pre-treatment, then at regular intervals to monitor response to Tx<\/p>\n\n\n\n<p>When deciding if a client is ready to move to a less intensive level of treatment (Inpt to PHP or PHP to IOP etc); what does the PMHNP look for?<br>signs not ready: no coping skills, still blaming others<\/p>\n\n\n\n<p>Signs they are ready: (+) coping skills, taking accountability<\/p>\n\n\n\n<p>prevent\/promotion, classes, safety initiatives, education, classes, modifying environment is what level of prevention?<br>Primary<\/p>\n\n\n\n<p>screen-early detection, crisis hotlines, disaster is what level of prevention?<br>Secondary<\/p>\n\n\n\n<p>treat- to prevent further deterioration, rehab, restoration, day treatment, social skills is what level of prevention?<br>Tertiary<\/p>\n\n\n\n<p>When questions ask for a priority action\u2026think about\u2026<br>ABC, airway breathing, circulation<br>Maslows hierarchy<\/p>\n\n\n\n<p>If undecided on an answer due to high similarities, choose:<br>the umbrella answer<\/p>\n\n\n\n<p>What is the most common side effect of olanzapine\/zyprexa<br>metabolic syndrome<\/p>\n\n\n\n<p>what is the difference between typical and atypical antipsychotics<br>Atypical 5HT2A specific<\/p>\n\n\n\n<p>1st psychotic break\u2026 two actions to take<br>UDS and r\/o sub<br>Consider IM Geodon or Invega<\/p>\n\n\n\n<p>Three AP with least weight gain<br>Latuda, Abilify, Geodon<\/p>\n\n\n\n<p>Always encourage interprofessional collaboration<br>between therapists\/pcps\/SW\/RN, the ENTIRE team<\/p>\n\n\n\n<p>TSH High, then\u2026.t3\/t4<br>T3, T4 low<\/p>\n\n\n\n<p>TSH low, then\u2026t3\/t4<br>T3, T4 high<\/p>\n\n\n\n<p>cold\/hot sensitivity with t3\/t4 relationship<br>T3\/T4 low, hypothyroid, cold, slow<br>T3\/T4 high, hyperthyroid, hot, flushed, tachy<\/p>\n\n\n\n<p>What birth defect can be caused by depakote?<br>Spina bifida<\/p>\n\n\n\n<p>What organ does depakote cause toxicity? and what sx would you expect to see? labs to run?<br>Hepatotoxicity: RUQ pain, reddish brown urine-<br>Do LFTs<\/p>\n\n\n\n<p>kava kava is used to treat<br>anxiety and insomnia<\/p>\n\n\n\n<p>Rash and fever associated with tegretol, suspect<\/p>\n\n\n\n<p>What allele is HLAB 1502 associated?<br>Asians. They CANNOT have tegretol. Test all asians for this allele.<\/p>\n\n\n\n<p>what rare and dangerous side effects are associated with tegretol<br>Aplastic anemia<br>Agranulocytosis-DC at ANC less than 1000<\/p>\n\n\n\n<p>Sx&#8217;s of agranulocytosis<br>unusual bleeding or bruising, mouth sores, infections, fever, sore throat, fatigue<\/p>\n\n\n\n<p>if starting a woman on lithium what test should be done? why?<br>HCG&#8211;risk of ebstein anomaly<\/p>\n\n\n\n<p>adverse s\/e of lamictal\/lamotrigine<br>SJS<\/p>\n\n\n\n<p>labs to checke BEFORE starting on lithium<br>BUN<br>CRE<br>urine protein<\/p>\n\n\n\n<p>What does protein in urine indicate<br>kidney impairment; 4+ protein in urine=you cannot start on lithium<\/p>\n\n\n\n<p>best choice med for decreasing si in bipolar disorder.<br>lithium<\/p>\n\n\n\n<p>best choice med for si in schizophrenia<br>clozaril<\/p>\n\n\n\n<p>best choice med for SI in borderline<br>lithium<\/p>\n\n\n\n<p>What medications will INCREASE Li levels<br>NSAIDS<br>ACE&#8217;s<br>Thiazides\/HCTZ<\/p>\n\n\n\n<p>Besides medications, what else can cause increased Li levels<br>dehydration<br>hyponatremia<br>lithium s\/e inc N\/V, which will effect electrolytes, and dehydration status<\/p>\n\n\n\n<p>what type of tremors will you see with lithium toxicity?<br>course tremors<\/p>\n\n\n\n<p>lithium can cause what other comorbidities?<br>hypothyroidism<br>maculopapular rash<br>leukocytosis<br>twave inversion<\/p>\n\n\n\n<p>what is a defining characteristic of NMS vs SS<br>muscle rigidity<\/p>\n\n\n\n<p>Sx&#8217;s\/labs associated with NMS<br>Inc CPK, WBC, LFT<br>Rhabdomyolosis<br>myoglobinuria<br>Can lead to mutism<\/p>\n\n\n\n<p>myoglobinuria\/rhabdo can cause cherry colored urine<\/p>\n\n\n\n<p>Treatment for NMS and what each does<br>DC the offending agent<br>bromocriptin-D2 agonis<br>dantrolene: muscle relaxant<br>Make sure if ? is asking for agonist or relaxant<\/p>\n\n\n\n<p>Sx of SS<br>HYPERREFLEXIA<br>myoclonic jerks<\/p>\n\n\n\n<p>treatment for SS<br>ciproheptadine<\/p>\n\n\n\n<p>how to best PREVENT SS<br>follow proper transition protocols<br>SSRI to MAOI=14 days<br>Prozac to MAOI=5-6 weeks<br>Triptans (Imitrex sumatriptan) can also cause SS due to serotonin increase with the use<br>give any NDRI welbutrin<\/p>\n\n\n\n<p>Why are SSRIs considered the safest for use in depression<br>safest for OD first line treatment for depression<\/p>\n\n\n\n<p>antidepressant for cancer<br>citalopram and escitalopramless drug to drug interactions<\/p>\n\n\n\n<p>depressed patient presents with fatigue and low energy, consider:<br>NDRI wellbutrin<\/p>\n\n\n\n<p>sexual s\/e with ssri? try\u2026<br>wellbutrin due to lower risk of sexual s\/e<\/p>\n\n\n\n<p>What medication must be avoided if client has seizure history or eating disorder? why?<br>wellbutrin due to decreasing the seizure threshold<\/p>\n\n\n\n<p>if client has depression and neuropathic pain<br>SNRI or TCA for treatment of BOTH<\/p>\n\n\n\n<p>What med class treats neuropathic pain well<br>alpha 2 delta ligands<br>Gabapentin<br>Lyrica<\/p>\n\n\n\n<p>What medication class is good for depression with comorbid CA<br>SSRI<br>least chance of drug drug interactions<br>Celexa and lexapro are good choices<\/p>\n\n\n\n<p>Black box warning on SSRI<br>increase SH kids, and young adult<\/p>\n\n\n\n<p>Required education for rx ssri<br>long time for effect<br>side effects esp n\/v\/d<br>NO ABRUPT stopping d\/t Serotonin discontinuation syndrome<\/p>\n\n\n\n<p>what to ask when client is depressed (additional question)<br>alcohol use<\/p>\n\n\n\n<p>Sx&#8217;s of serotonin discontinuation syndrome<br>fever, shivering, muscle aches and nausea diarrhea, agitation, cog impairment\u2026 (think flu like sx&#8217;s)<br>disequilibrium<\/p>\n\n\n\n<p>What are some scenarios that place patients at risk of a hypertensive crisis?<br>MAOI and tyramine<br>MAOI and TCA<br>MAOI and Atypical AP<br>MAOI and decongestant<br>MAOI and stimulants<br>MAOI and asthma meds<\/p>\n\n\n\n<p>Sx of Hypertensive crisis<br>HA<br>Diaphoresis<br>fever<br>facial flushing<br>pupillary dilation<br>palpitation<\/p>\n\n\n\n<p>Treatment for HTN Crisis<br>DC Agent<br>Give Phenolamine<\/p>\n\n\n\n<p>age of onset for male \/ female schizophrenia<br>male 18-25<br>female 25-35<\/p>\n\n\n\n<p>schizophrenia has high rate of suicide<br>always be assessing for SI<\/p>\n\n\n\n<p>what is the cause of schizophrenia<br>inadequate synapse formation<br>excessive pruning of synapses<br>intrauterine insult (drugs\/toxin\/viral agent\/malnutrition\/substance use, mental illness, o2 deprivation)<\/p>\n\n\n\n<p>on MRI \/PET schizonphrenia<br>ventrical enlargement<\/p>\n\n\n\n<p>positive sx of schizophrenia caused by\u2026<br>excess DA in mesolimbic pathway<\/p>\n\n\n\n<p>positive sx of schizophrenia on a stimulant?<br>will potentiate DA release (worsening schizophrenias positive symtpoms) bc stimulants tap into the reward and addiction pathway inc dopamine<\/p>\n\n\n\n<p>What is ACT<br>Assertive community treatment<br>Post hospital DC<br>not in the hospital<\/p>\n\n\n\n<p>delusions, you respond how?<br>do not try to disprove<br>If HARMFUL delusions, notify authority<br>Also notify potential victim<\/p>\n\n\n\n<p>how to assess mental abstraction<br>interpret a proverb<\/p>\n\n\n\n<p>assess thought process why? and potential findings<br>to assess organization of patients thoughts<br>tangential: no rip to a ?<br>circumstantial: gets around to anss after going in circles with unnecessary details<\/p>\n\n\n\n<p>MMSE thought content include<br>SI HI<br>plan<br>Hallucinations<br>delusions<\/p>\n\n\n\n<p>MMSE\/ Folstein Test assesses?<br>tool used to assess cognitive status in adults<\/p>\n\n\n\n<p>concentration\/attention\/calculation assessed how?<br>spell a word backward or serial 7&#8217;s<\/p>\n\n\n\n<p>Registration\/ability to learn new material assessed how?<br>remember 3 words<\/p>\n\n\n\n<p>orientation is assessed by<br>Person place time<\/p>\n\n\n\n<p>fund of knowledge assessed how?<br>Who is the president? Governor?<\/p>\n\n\n\n<p>clock drawing assesses?<br>takes 1-2 minutes<br>easy to administer<br>tests right hemisphere health,cannot do it they have a prob in R side.<\/p>\n\n\n\n<p>First generation AP<br>haldol<br>fluphenazine<br>Chlorpromazine<br>thioridazine<\/p>\n\n\n\n<p>2nd gen AP<br>rispeidone<br>olanzapine<br>seroquel<br>abilify<br>ziprasidone<br>lurasidone<br>clozipine<\/p>\n\n\n\n<p>excess DA in mesolimbic pathway<br>positive sx of schizophrenia<\/p>\n\n\n\n<p>decreased DA in mesocoritcal pathway<br>negative sx of schizophrenia<br>anhedonia<br>mask face<br>slow speech<br>isolative<\/p>\n\n\n\n<p>nigrostriatal pathway<br>excess DA in this pathway but it doesn&#8217;t cause any sx,<br>but when DA is decreased in this pathway due to AP (dopamine blockade) increases acetylcholine=EPS sx<\/p>\n\n\n\n<p>Tuberoinfundibular pathway<br>excess DA is normal here<br>but when it is decreased with AP leads to increased prolactin and hyperprolactinemia<\/p>\n\n\n\n<p>what does high prolactin levels lead to<br>breast DC<br>amenorrhea<br>osteoporosis<br>breast enlargement<\/p>\n\n\n\n<p>which AP is most closely associated with hyperprolactinemia<br>risperidone<\/p>\n\n\n\n<p>Male prolactin<br>female prolactin&#8212;normals<br>male less than 20ng\/ml<br>female less than 25ng\/ml<\/p>\n\n\n\n<p>EPS caused by<br>DA blockade in the NS pathway<\/p>\n\n\n\n<p>acute dystonia<br>acute sustained contraction of muscles, usually of the head and neck<br>spasms<br>painful<\/p>\n\n\n\n<p>treatment of acute dystonia<br>cogentin\/benztropine IM, but may need Oral follow up for several days<\/p>\n\n\n\n<p>akathisia<br>ANXIETY<br>restless<br>cannot sit still<br>pacing rocking<\/p>\n\n\n\n<p>1st line treatment for akathisia? And which population to AVOID use in? 2nd line? 3rd line?<br>propranolol\/or beta blocker<br>client with asthma on bronchodilators due to increased risk of bronchospasm when given beta blockers<\/p>\n\n\n\n<p>2nd line: cogentin\/benztropine<\/p>\n\n\n\n<p>3rd line: benzos<\/p>\n\n\n\n<p>akanesia<br>difficulty iniating motion<\/p>\n\n\n\n<p>treatment for akanesia<br>cogentin\/benztropin<\/p>\n\n\n\n<p>Sx of akinesia<br>DIFFICULTY INITIATING MOVEMENT;<br>PSEUDO PARKINSONIAN SX&#8217;S:<br>muscle rigidity<br>shuffling gait<br>mask like facial expression<br>affect may be flat or blunted<br>pill rolling tremors (motor slowing)<\/p>\n\n\n\n<p>Tardive dyskinesia (TD)\/timeframes<br>chewing\/lip smacking<br>facial dyskinesia<br>Treatment: dec dose of offending agent or switch to a different AP or can switch to clozaril.<br>can have onset between a few weeks and 2 years post starting of medication<\/p>\n\n\n\n<p>Reglan (metoclopramide) and compazine (prochlorperazine) can both cause?<br>TD<\/p>\n\n\n\n<p>what medication can worsen TD?<br>benztropine\/cogentin<\/p>\n\n\n\n<p>Inducers<br>cause low serum levels<\/p>\n\n\n\n<p>inhibitors<br>cause high serum levles<\/p>\n\n\n\n<p>reglan and compazine can both cause<br>TD<\/p>\n\n\n\n<p>Smoking and dosing of meds<br>smoking is a strong inducer.<br>therefore smoking increases drug metablolism and you may need to dose higher. If they stop smoking serum levels will increase.<br>Always assess for smoking\/cessation of.<\/p>\n\n\n\n<p>Antibiotics\/macroglides are INHIBITORS and greatly effect what medication<br>tegratol<\/p>\n\n\n\n<p>what meds can cause mania<br>disulfram<br>steroids<br>isoniazide<br>antidepressants<\/p>\n\n\n\n<p>what meds can cause depression<br>steroids<br>beta blockers<br>interferon<br>accutane-can cause birth defects too<\/p>\n\n\n\n<p>if patient taking meds for a mood disorder and is on flonase or prednisone. What do you do with your dose of oxcarbazepine<br>increase the dose to adjust for the medication (steroids) influencing mania<\/p>\n\n\n\n<p>treating a patient with zoloft and starts taking interferon. what do you do with zoloft dosing?<br>increase dose of zoloft to adjust for the increase in depression for the interferon<\/p>\n\n\n\n<p>addiction neurotransmitters<br>DA<br>GABA<\/p>\n\n\n\n<p>Gamma-aminobutyric acid main function<br>major inhibitory neurotransmitter<\/p>\n\n\n\n<p>Sx&#8217;s of stimulant abuse<br>irritability<br>insomnia<br>tremors<br>delayed gastric emptying-feeling fullness\/bloated<\/p>\n\n\n\n<p>Pt with anorexia complains of pain after eating\/bloating\/fullness<br>Signs of delayed gastric emptying<\/p>\n\n\n\n<p>what meds can delay gastric empying?<br>PROTON PUMP INHIBITORS<br>famotidine<br>omeprazole<br>ranitidine<\/p>\n\n\n\n<p>ANTACIDS \/PPI do what to psychotropic medications?<br>Decrease absorption of psychotropic medication<\/p>\n\n\n\n<p>advise client to take other meds 2 hours AFTER antacids\/PPI<\/p>\n\n\n\n<p>Older adults and SSRIs for anxiety?<br>May increase anxiety<br>paradoxical effect<\/p>\n\n\n\n<p>Older adults and benzos?<br>May increase agitation<br>paradoxical effect<\/p>\n\n\n\n<p>apoptosis<br>neuronal loss or cell death<\/p>\n\n\n\n<p>BP1 pneumonic<br>DIGFAST<\/p>\n\n\n\n<p>DIGFAST<br>Distractible<br>Impulsive choices<br>Grandiosity<br>Flight of ideas<br>Active<br>Sleep not needed<br>Talkative<\/p>\n\n\n\n<p>Borderline sx in regard to impulsivity<br>impulsivity is often with associated with recurrent SI and self harming behavior<\/p>\n\n\n\n<p>Therapy for BPD\/goal\/founder<br>DBT<br>to decrease recurrent SI<br>Marsha Linehan<\/p>\n\n\n\n<p>Conversion Disorder (Functional Neurological Symptom Disorder)<br>can be result of a stressful experience<\/p>\n\n\n\n<p>present with neuro sx:<br>parasthesia<br>paralysis<br>blindness<br>Mutism<\/p>\n\n\n\n<p>adjustment disorder (anxiety\/depressive\/mixed)<br>an emotional disturbance caused by ongoing stressors within the range of common experiences (dx of a new disease) (recent move) (loss of loved one)<\/p>\n\n\n\n<p>factitious disorder<br>Condition in which a person acts as if he or she has a physical or mental illness when he or she is not really sick.<br>Ie drinking contaminated urine<\/p>\n\n\n\n<p>Reactive Attachment Disorder<br>common in kids from foster care<br>kid goes back to real parent and may appear withdrawn, no emotions toward caregiver,<br>FOSTER is key word<br>Doesn&#8217;t seek comfort when distressed<\/p>\n\n\n\n<p>ODD<br>NO AGGRESSION in ODD<br>defiant to authority<br>deliberately annoy others<\/p>\n\n\n\n<p>treatment for ODD<\/p>\n\n\n\n<ul class=\"wp-block-list\">\n<li>Family Therapy<br>with emphasis on child management skills<\/li>\n<\/ul>\n\n\n\n<p>CD conduct disorder<br>no remorse<br>violent<br>raping\/beating<br>theft<br>arson<br>INTENSE<\/p>\n\n\n\n<p>AGE 6-17<br>When 18; think of Antisocial personality disorder<\/p>\n\n\n\n<p>Treatment for CD<br>Medication and therapy<br>to target symptoms of mood and aggression<\/p>\n\n\n\n<p>Alpha agonist aka alpha 2 adrenergic receptor blockers (clonidin, guanfacine)<\/p>\n\n\n\n<p>Family therapy<\/p>\n\n\n\n<p>acute stress disorder<br>An anxiety disorder in which fear and related symptoms are experienced soon after a traumatic event and last LESS THAN 1 MONTH<\/p>\n\n\n\n<p>Over 1 month is PTSD<\/p>\n\n\n\n<p>PTSD cluster of sx<br>Increased arousal<br>Reexperiencing the event<br>avoidance<br>nightmares<\/p>\n\n\n\n<p>nightmare trmt in ptsd<br>prazosin\/minipress<\/p>\n\n\n\n<p>PTSD treatment<br>EMDR<br>SSRI<br>CBT<\/p>\n\n\n\n<p>phases of EMDR<br>see purple book<\/p>\n\n\n\n<p>Panic attack vs disorder<br>panic attack: acute\/seldom<\/p>\n\n\n\n<p>disorder: chronic leads to feelings of impending doom<br>propanolol<\/p>\n\n\n\n<p>Tourette dx criteria<br>2 motor tic<br>1 vocal tic<br>for ONE year (even if ? says 6 months) its still most likely the correct answer<\/p>\n\n\n\n<p>NT in tourette<br>DA-da dysfunction is major NT in tourette<br>NE<br>S<\/p>\n\n\n\n<p>tics can be caused by what type of medication<br>stimulant<\/p>\n\n\n\n<p>how do you treat a kid with tics for adhd<br>non stimulant<\/p>\n\n\n\n<p>guanfacine and clonidine are good choice for adhd in kid with tics<br>TRUE<\/p>\n\n\n\n<p>Meds for tourettes<br>FDA approved<br>haldol<br>abilify<br>pimozide<br>guanfacine<br>clonidine<br>esp if kid is having impulse control problems<\/p>\n\n\n\n<p>are tics in kids normal<br>yes<br>normal.<br>normally by teen years they disappear. no treatment unless meeting dsm criteria for tourettes. tourettes needs trmt. 2 motor 1 vocal<\/p>\n\n\n\n<p>brain region affected in adhd<br>frontal cortex<\/p>\n\n\n\n<p>basal ganglia-<\/p>\n\n\n\n<p>dorsolateral PFC&#8211;executive function, attention, cognitive processes (problem solving, working memory)<\/p>\n\n\n\n<p>reticular activating<\/p>\n\n\n\n<p>OCD NT&#8217;s<br>S<br>NE<\/p>\n\n\n\n<p>Pandas<br>causes OCD<br>from strep infection<br>treat with SSRI prozac in kids, in adults use sertraline.<br>adults can also get TCA like clomipramine<\/p>\n\n\n\n<p>DMDD (disruptive mood dysregulation disorder)<br>in kids, mood d\/o 6-17yo<br>kid has bipolar disorder but is not 18 yet<\/p>\n\n\n\n<p>moody for no reason<br>irritible for no reason<br>tantrums for no reason<br>Mad\/Sad for no reason<\/p>\n\n\n\n<p>IED (intermittent explosive disorder)<br>reacts grossly out of proportion to the situation<\/p>\n\n\n\n<p>violent<br>aggressive<br>HAVE REMORSE, feel bad about it all later<\/p>\n\n\n\n<p>irritibility, depression, labile mood\u2026utilize a________<br>mood questionnaire<\/p>\n\n\n\n<p>nightmares in children can be genetic or psychological<br>assess the family tree\/hx for this<\/p>\n\n\n\n<p>GAD (Generalized Anxiety Disorder)<br>AT LEAST SIX MONTHS<br>If less it is ASD<\/p>\n\n\n\n<p>Autism Spectrum Disorder NTs<br>Gaba<br>glutamate<br>SE<\/p>\n\n\n\n<p>impaired communication<br>poor cognition<br>broken mirror theory of autism-<\/p>\n\n\n\n<p>what two things do you have to have to diagnose adhd<br>a teacher and a parent evaluation<\/p>\n\n\n\n<p>Do stimulants increase DA?<br>Yes, so don&#8217;t give a vmat 2 and a stimulant together. wipes one another out<\/p>\n\n\n\n<p>P450 Inducer meds<br>carbamazepine<br>rifampin<br>alcohol<br>phenytoin<br>griseofulvin<br>phenobarbital<br>sulfonylureas<\/p>\n\n\n\n<p>P450 inhibitor<br>Valproate<br>ketoconazole<br>isoniazid<br>sulfonomides<br>choramphenical<br>amiodarone<br>erythromycin<br>quinidine<br>grapefruit juice<\/p>\n\n\n\n<p>pneumonic for inducers<br>CRAP GPS induces my rage<br>Carbamazepine<br>Rifampin<br>Alcohol<br>Phenytoin<br>Griseofulvin<br>Phenobarbital<br>Sulfonylureas<\/p>\n\n\n\n<p>450 subtypes<br>1A2<br>2E1<br>2C9<br>2D6<br>3A4<br>1A2. AcetAminophen- 1 big word with 2A<br>2E1 &#8211; 21 years to drink ETOH<br>2C9- Warfarin factors 2, C, 9<br>2D6- 2D echo-cardiac drug metabolism<br>3A4- most common, metabolizes up to 60% of ALL medications, so if its not one of the above it is likely 3A4.<\/p>\n\n\n\n<p>What NT are involved with OCD<br>NE<br>SE<\/p>\n\n\n\n<p>Trmt med for kid with ocd<br>SSRI: prozac<\/p>\n\n\n\n<p>trmt med for adult with ocd<br>ssri: sertralin<br>or TCA (clomipramine)<\/p>\n\n\n\n<p>GAD pneumonic<br>WATCHERS x 6 months<br>Worry<br>Anxiety<br>Tension<br>Concentration<br>Hyperarousal<br>Energy Loss<br>Restlessness<br>Sleep issues<\/p>\n\n\n\n<p>Stages of change<br>precontemplation: Not acknowledged an issue. &#8221; I don&#8217;t have a drug problem&#8221;<\/p>\n\n\n\n<p>contemplation: acknowledges, but NOT YET ready to change. &#8220;i know heroin is killing me, but i have no time for rehab&#8221;\u2026 some self doubt<\/p>\n\n\n\n<p>preparation: acknowledges issue, intends to eventually act. &#8220;Ive been thinking about rehab or buprenorphine, what do you think?&#8221; No action or behavioral modification yet\u2026 researching<\/p>\n\n\n\n<p>action: behavioral change occurs. &#8220;I&#8217;m in inpatient rehab and I&#8217;m feeling much better!&#8221;<\/p>\n\n\n\n<p>maintenance: effort made to sustain the change. &#8220;Ive been on buprenorphine for a year and I&#8217;m still doing well&#8221;<\/p>\n\n\n\n<p>relapse: reversion to a previous stage\u2026 goes back to a prior stage to start over.<\/p>\n\n\n\n<p>Autism hallmarks<br>impaired communication<br>impaired social interaction<br>poor eye contact<br>sensory issues<br>poor cognition<\/p>\n\n\n\n<p>Broken mirror theory of autism-responsible for childs presentation of autism symptoms<br>mirror neuron is dysfunctional<\/p>\n\n\n\n<p>Risk factors for autism<br>male<br>intellectual disability<br>genetic loading-inc risk with family member with autism<\/p>\n\n\n\n<p>screeners for autism?<br>M-Chat: modified checklist for autism<\/p>\n\n\n\n<p>ADOS-G: autism diagnostic observation schedule-generic<\/p>\n\n\n\n<p>ASQ: ages and stages questionnaire<\/p>\n\n\n\n<p>nightmares in children<br>can be genetic<br>so ASSESS family for same problem<\/p>\n\n\n\n<p>NT in Autism Spectrum disorder<br>Gaba and Glutamate<\/p>\n\n\n\n<p>If patient presents with irritibility, personality changes, check their:<br>Vit B12 and folic acid<\/p>\n\n\n\n<p>if ? presents lack of coordination, slowing, motor symptoms, and apathy, depression, irritability think about this dx<br>subcortical dementia<\/p>\n\n\n\n<p>cortical dementia effects mostly\u2026<br>memory and language<\/p>\n\n\n\n<p>cognitive deficits<br>motor, behavioral<\/p>\n\n\n\n<p>pseudo dementia<br>cognitive screening<br>present with:<br>agitation<br>irritability<\/p>\n\n\n\n<p>what disorder does a provider hear a lot of &#8220;I don&#8217;t know answers&#8221;<br>pseudo dementia<\/p>\n\n\n\n<p>aphasia is associated with which region of the brain<br>prefrontal cortex<\/p>\n\n\n\n<p>visual hallucinations are associated with which type of dementia<br>lewy body<\/p>\n\n\n\n<p>frontotemoporal dementia<br>frontal lobe<br>picks disease<br>personality changes<br>behavioral<br>language (slurred)<\/p>\n\n\n\n<p>Signs of lead abuse<br>developmental delay<br>learning difficulties<br>irritability<br>loss of appetite<br>weight loss<br>sluggishness<br>fatigue<br>abd pain<br>vomiting<br>constipation<br>hearing loss<br>seizures<br>pica<\/p>\n\n\n\n<p>TEST LEAD LEVELS<\/p>\n\n\n\n<p>Components of the COW Scale<br>Pulse<br>sweating<br>restlessness<br>pupil size<br>bone aches<br>running nose<br>tearing<br>GI upset<br>yawning<br>tremors<br>anxiety\/irritability<br>goosebumps<\/p>\n\n\n\n<p>anterior cingulate is responsible for what<br>cognitive functions<br>decision making<br>emotions<br>impulse control<\/p>\n\n\n\n<p>NE is produced in the locus ceruleus and the \u2026<br>medullary reticular formation<\/p>\n\n\n\n<p>SE is produced in the_________? And is involved in <strong>__<\/strong>?<br>raphe nuclei of the brainstem\/sleep<\/p>\n\n\n\n<p>DA is produce in the substantia nigra and <strong>__<\/strong>?<br>the ventral tegmental area VTA<br>and nucleus accumbens<\/p>\n\n\n\n<p>Acetylcholine is synthesized by the basal nucleus of <strong>_<\/strong>?<br>Meynert<\/p>\n\n\n\n<p>NT in mood disorders<br>SE and NE<\/p>\n\n\n\n<p>risk factors for osteoporosis<br>smoking<br>caffeine<br>lack of exercise<br>diet low in cal and vit D<\/p>\n\n\n\n<p>What is the hippocampus responsible for?<br>ST to LT memory<br>emotions<br>stress<br>learning<\/p>\n\n\n\n<p>Amygdala<br>fear<br>anger<br>anxiety<br>aggression<br>stress<\/p>\n\n\n\n<p>cerebellum<br>balance<\/p>\n\n\n\n<p>anterior cingulate<br>cognitive functions<br>decision making<br>empathy<br>impulse control<br>emotions<br>decision making<\/p>\n\n\n\n<p>frontal lobe issues<br>social skills<br>tumor here can cause behavioral problems<\/p>\n\n\n\n<p>If you are caring for an infant that is dying and the parents are present, what action should you take<br>give the infant to the parents to hold and<br>grieve their loss<\/p>\n\n\n\n<p>osteoporosis can be prevented by a diet rich in what? and other ways to prevent<br>calcium and vitamin d<br>weight bearing exercises<br>not using tobacco<\/p>\n\n\n\n<p>Cognitive theory basics<br>trying to replace automatic negative beliefs\/irrational thoughts with positive \/ functional thoughts<\/p>\n\n\n\n<p>Humanistic therapy basics<br>Person centered<br>self actualization<br>self directed growth<\/p>\n\n\n\n<p>Behavioral therapy<br>problem solving techniques<br>role playing<br>skills training<br>relaxation<\/p>\n\n\n\n<p>SKILLS SKILLS<\/p>\n\n\n\n<p>interpersonal therapy (IPT)<br>interpersonal distress with lots of people and in several settings.<br>also used in marital conflict for a 12-16 weeks.<br>Think relationship distress with this<\/p>\n\n\n\n<p>Family systems therapy<br>triangles<br>triangulation<br>self differentiation<br>genograms<\/p>\n\n\n\n<p>structural family therapy<br>hierarchies keyword<br>structural mapping<br>genograms<\/p>\n\n\n\n<p>strategic family therapy<br>problem focused\/symptom focused<br>paradoxical strategies<br>or a straight forward directive<br>Reframe patients belief system<\/p>\n\n\n\n<p>solution focused family therapy<br>miracle question<br>miracle solution<br>exception based finding questions<br>scaling questions<\/p>\n\n\n\n<p>Appreciative inquiry<br>focused on the strengths of client<\/p>\n\n\n\n<p>if question answers give closed ended questions and one open ended question and all are appropriate, choose open ended because open ended questions promote the relationship<br>promote the relationship between client\/provider. if they won&#8217;t talk, you can f\/u with closed ended questions if the child remains avoidant\u2026 stays avoidant, then go to parents as a last resort<\/p>\n\n\n\n<p>therapy session with a couple and only one party shows up, what do you do?<br>reschedule both of them. they must both be present.<\/p>\n\n\n\n<p>if someone has lost someone, ask them\u2026.?<br>how is that loss affecting you\u2026how do you feel about it?<br>Not affected? maybe you don&#8217;t need to focus on it too much<\/p>\n\n\n\n<p>If client is moving to another state, how can you assist in not abruptly stopping medications?<br>give plenty of meds until they can see a new provider\u2026maybe three month supply<\/p>\n\n\n\n<p>Sibling abuse 5yo sodomized by his 15yo borther reported to you. Inform parent to <strong><em><strong><em>, and call<\/em><\/strong><\/em><\/strong>?<br>tell parents to separate the siblings<br>cps<\/p>\n\n\n\n<p>suspect rheumatoid arthritis, check\u2026..?<br>ESR level, which indicates inflammation<\/p>\n\n\n\n<p>PICOT<br>Population<br>Intervention<br>Comparison<br>Outcome<br>Time<\/p>\n\n\n\n<p>Level 1 evidence<br>systematic reviews of random control trials (RCTs) -highest internal validity due to randomizations<\/p>\n\n\n\n<p>Level 2 evidence<br>at least 1 RCT<\/p>\n\n\n\n<p>how do you continue to give evidence based care?<br>by reading current up to date journals<\/p>\n\n\n\n<p>Shrill cry in infant<br>intracranial pressure; inconsolable<\/p>\n\n\n\n<p>when do you start aftercare plan?<br>when patient is admitted<\/p>\n\n\n\n<p>masturbating is normal in what age range? Freud stage?<br>3 to six year old<br>phallic stage<\/p>\n\n\n\n<p>normal for young boys to develop breast enlargement\/swelling\/tenderness. Usually goes away in\u2026.?<br>six months<\/p>\n\n\n\n<p>sex drive is driven by what hormone?<br>testosterone. so check this if c\/o dec libido.<\/p>\n\n\n\n<p>Testosterone decreases as we\u2026?<br>age<\/p>\n\n\n\n<p>if male or female c\/o dec sex drive, chec\u2026?<br>testosterone<\/p>\n\n\n\n<p>Alcohol dehydrogenase<br>etoh enzyme<br>women have less than men so get intoxicated faster compared to men<\/p>\n\n\n\n<p>best way to normalize the grieving process in children?<br>supportive group therapy<\/p>\n\n\n\n<p>do psychoeducation<\/p>\n\n\n\n<p>do not give prescriptive advise<\/p>\n\n\n\n<p>these reflexes go away at what age:<br>grasp\/palmar<br>moro\/startle<br>babinski<br>grasp\/palmar 5-6 months<br>moro\/startle 5-6 months<br>babinski 9-12 months<\/p>\n\n\n\n<p>what class of medications are PDE5?<br>sexual dysfunction<br>viagra<br>short absorption, rapidly absorbed<\/p>\n\n\n\n<p>know diff between AN and BN<br>AN BMI is very low, high chance of hospital admission<br>VS changes like bradycardia. if parents won&#8217;t admit call cps. bmi to admit are 12, 13, 14.<\/p>\n\n\n\n<p>Screen all adhd presentations for substance use<\/p>\n\n\n\n<p>high risk for sub use in depression population<br>use early screening and early intervention<\/p>\n\n\n\n<p>recommend accupuncture for<br>depression and pain<\/p>\n\n\n\n<p>inform adolescents all info is confidential except<br>harm to self or others<\/p>\n\n\n\n<p>what is habeus corpus<br>legal, not medical concept, that protects patients from unlawful hospitalization<\/p>\n\n\n\n<p>substance induced psychosis has a very high rate of ?<br>HI. even more than ASPD.<\/p>\n\n\n\n<p>disseminated encephalomyelitis<br>nervous system is affected<\/p>\n\n\n\n<p>asymmetrical body movements<\/p>\n\n\n\n<p>do a neuro exam<\/p>\n\n\n\n<p>patient has a mood disorder. started depakote and been stable. BMI 25 now 30. eating alot of sweets and juices. recommendations?<br>A. switch med<br>b. dec depakote<br>c. ask them to exercise<\/p>\n\n\n\n<p>how can you help your client remember something by anchoring their memory. how do you do thta?<br>ask a specific question that will provide an anchor to a timeline, like a wedding, bday, new job, etc.<\/p>\n\n\n\n<p>ask what they know about medications before rxing<br>assesses their knowledge of meds<\/p>\n\n\n\n<p>polypharmacy can lead to adverse side effects<br>assess medication hx<\/p>\n\n\n\n<p>iatrogenic<br>an iatrogenic illness is an illness that is caused by a medication or physician.<\/p>\n\n\n\n<p>tardive dys: caused by a med rx&#8217;d by a doc<\/p>\n\n\n\n<p>post online forms to give responses to influence<br>policy making<br>advocacy<\/p>\n\n\n\n<p>autoimmune diseases can lead to increased\u2026.?<br>cytokine levels<\/p>\n\n\n\n<p>Desmopressin (DDAVP)<br>for nighttime bed wetting<br>reduces urine production esp at noc<\/p>\n\n\n\n<p>guanfacine and clonidine is disliked by some patients due to?<br>have low tolerability due to lowers BP<\/p>\n\n\n\n<p>cannot look up patietns information on Social media<br>violates trust<\/p>\n\n\n\n<p>treatment for nighttime wetting under 12yo?<br>nonpharm<br>setting alarms<br>bladder training<\/p>\n\n\n\n<p>if addicts are still blamining others they are not ready for\u2026.?<br>discharge. If still blaming others they are not ready. Need to take responsibility for their actions.<\/p>\n\n\n\n<p>non compliant with medication?<br>intervention based on why they are not compliant<\/p>\n\n\n\n<p>assess data before changing anything<\/p>\n\n\n\n<p>if patient comes to your with concerns make sure you<br>validate their concerns to promote communication<\/p>\n\n\n\n<p>come to office or discuss by phone<\/p>\n\n\n\n<p>documentation for off label use? do this\u2026.<br>document why.<br>and document support for off label use.<br>MSL or trials<\/p>\n\n\n\n<p>risk factors for sleep apnea<br>excessive weight<br>obesity<br>diabetes<br>smoking<br>HTN<br>narrowed airways<\/p>\n\n\n\n<p>what is macrocytic anemia<br>vit B12 deficiency<\/p>\n\n\n\n<p>MMSE high score means?<br>Good! low score is severe cognitive deficit<\/p>\n\n\n\n<p>if depression is &#8220;severe&#8221; assess for<br>SI<\/p>\n\n\n\n<p>MMSE scale<br>25-30 is normal<br>21-24 mild<br>10-20 moderate<br>0-9 severe<\/p>\n\n\n\n<p>SLUM scale<br>27-30 Normal<br>21-26 Mild<br>0-20 dementia<\/p>\n\n\n\n<p>HAM D<br>0-7 Normal<br>8-13 mild<br>14-18 moderate<br>19-22 severe<br>23 plus very severe<\/p>\n\n\n\n<p>PHQ 9<br>0-4 normal<br>5-9 mild<br>10-14 moderate<br>15-19 moderate to severe<br>20-27 severe<\/p>\n\n\n\n<p>Beck<br>0-9 normal<br>10-18 mild<br>19-29 moderate<br>30-63 severe<\/p>\n\n\n\n<p>HAM A<br>under 17 mild<br>18-24 moderate<br>25 plus severe<\/p>\n\n\n\n<p>GAD<br>0-4 normal<br>5-9 mild<br>10-14 moderate<br>15-21 severe<\/p>\n\n\n\n<p>COWs<br>0-4 none<br>5-12 mild<br>13-24 moderate<br>25-35 mod to severe<br>35 plus severe<\/p>\n\n\n\n<p>CIWA<br>0-9 none<br>10-15 mild<br>16-20 moderate<br>21 plus severe<\/p>\n\n\n\n<p>cows criteria<br>mod: treatment at 13 and above for scheduled. consider buprenorphine or Suboxone. methadone consider safety concerns and under supervision<br>pulse<br>sweating<br>restlessness<br>pupil size-dilated<br>bone aches<br>runny nose<br>tearing<br>gi upset<br>yawning<br>tremors<br>anxi\/irritable<br>goosebumps<\/p>\n\n\n\n<p>ciwa<br>scheduled medication starts at score of 15<br>Nausea<br>vomiting<br>tremor<br>sweats<br>anxiety<br>agitation<br>tactile disturbances<br>auditory disturbances<br>HA<br>orientation<\/p>\n\n\n\n<p>etoh abuse treatment<br>disulfram no etoh for two weeks after stopping\/none 12 hours before starting<\/p>\n\n\n\n<p>aversion therapy<\/p>\n\n\n\n<p>naltrexone<\/p>\n\n\n\n<p>idealization<br>exaggeration of good qualities of the person or object lost, followed by acceptance for the loss. widow exaggeraes how amzing her not so amazing husban is<\/p>\n\n\n\n<p>false positive screen for pcp<br>tramadol<br>dextromethorphan<br>alprazolam<br>clonazepam<br>carvedilol<br>diphenhydramine<\/p>\n\n\n\n<p>rationalization<br>always to work late and tells boss &#8220;I&#8217;m not the only one&#8221;<\/p>\n\n\n\n<p>intellectualization<br>new diagnosis so you go home and do tons of research<\/p>\n\n\n\n<p>scope of practice<br>varies state to state<br>defines role and action<br>varies broadly<\/p>\n\n\n\n<p>standard of practice<br>determined by american nurses association<br>judges care given<br>reasonably prudent is standard<\/p>\n\n\n\n<p>confidentiality<br>the assurance that messages and information are available only to those who are authorized to view them<\/p>\n\n\n\n<p>exceptions to confidentiality<br>when info outweighs risk (think murder trial)<\/p>\n\n\n\n<p>intent to harm self or others<\/p>\n\n\n\n<p>info given to atty in litigation<\/p>\n\n\n\n<p>releasing records to insurance companies<\/p>\n\n\n\n<p>answering court orders, subpoenas, summons<\/p>\n\n\n\n<p>Meeting state requirements for mandatory reporting of dx of condition<\/p>\n\n\n\n<p>informed consent in apt with dementia<br>if client can repeat the benefits and risks o the med\/<\/p>\n\n\n\n<p>autonomy is:<br>right to self determination<br>EXCEPT<br>rennie vs klein: right to refuse medication if NOT-found incompetent<\/p>\n\n\n\n<p>Rights of patient re environment<br>least restrictive first<\/p>\n\n\n\n<p>just culture<br>safety<br>continual learning<br>designing safe systems<br>managing behavioral choices<\/p>\n\n\n\n<p>Recovery model<br>treatment approach doesn&#8217;t really focus on the dx\/illness<br>looking beyond the dx and search out abilities dreams goals<br>self direction<br>finding meaning in life despite the dx<br>person centered<br>individualized<br>NON LINEAR not step by step but based on growth setbacks and learning experiences<\/p>\n\n\n\n<p>quality improvement projects focus on\u2026<br>improving systems<br>decrease cost<br>improve productivity<\/p>\n\n\n\n<p>a retrospective chart review would be a type of\u2026.<br>quality improvement initiative<\/p>\n\n\n\n<p>PDSA is a process<br>not a project<br>an effective strategy<\/p>\n\n\n\n<p>reflective practice<br>linking theory to practice<br>providing feedback<\/p>\n\n\n\n<p>industry vs inferiority<br>school age 6-12<br>becoming industrious and getting confidence about what they can do\u2026 fx is feeling bad and inferior<\/p>\n\n\n\n<p>intimacy vs isolation<br>age 20-35<br>in a loving relationship<br>starting a family<br>fx is feeling alone and isolated<\/p>\n\n\n\n<p>piaget age 2-7<br>magical thinkeers<br>preoperational<br>&#8220;if i think about a new dress I will get one&#8221;<\/p>\n\n\n\n<p>piaget 11 and up<br>formal operations<br>logical and abstract<br>algebra etc<\/p>\n\n\n\n<p>primary prevention example<br>screening and community education<\/p>\n\n\n\n<p>secondary prevention<br>crisis intervention<br>hotlines<br>disaster response<\/p>\n\n\n\n<p>tertiary prevention<br>rehab<br>active treatment<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Which patient is at highest risk for SI A. 30y\/o married AA female with previous SI attempt *1 risk factor B. 35 y\/o single Asian male with previous SI attempt *3 risk factors C. 38 y\/o single AA male who is a manager of a bank *2 risk factors D. 68 y\/o single white male [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"default","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","ast-disable-related-posts":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"","background-image":"","background-repeat":"repeat","background-position":"center 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