{"id":111191,"date":"2023-07-29T12:09:20","date_gmt":"2023-07-29T12:09:20","guid":{"rendered":"https:\/\/learnexams.com\/blog\/?p=111191"},"modified":"2023-07-29T12:09:24","modified_gmt":"2023-07-29T12:09:24","slug":"barkley-pre-test-2023-2024-actual-exam-100-questions-and-correct-detailed-answers-100-verified-answers-already-graded-a","status":"publish","type":"post","link":"https:\/\/www.learnexams.com\/blog\/2023\/07\/29\/barkley-pre-test-2023-2024-actual-exam-100-questions-and-correct-detailed-answers-100-verified-answers-already-graded-a\/","title":{"rendered":"BARKLEY PRE TEST 2023-2024 ACTUAL EXAM 100 QUESTIONS AND CORRECT DETAILED ANSWERS (100% VERIFIED ANSWERS) |ALREADY GRADED A+"},"content":{"rendered":"\n<p>You have been treating Jason, 35 y\/o M, for HIV infection, and the course of his treatment requires that he be hospitalized for a few days. His medical bill is paid by his Aunt, Sylvia, who wants to know why her nephew was in the hospital, but Jason is not willing to share that information. What is the appropriate course of action?<br>the NP should not release the details of Jason&#8217;s health status to his aunt<\/p>\n\n\n\n<p>Which of the following best describes a II\/VI heart murmur?<br>Audible but faint<\/p>\n\n\n\n<p>21 y\/o M comes complaining of a painful bump with small, red-looking halo on his penis. The affected area is about 5 mm in diameter, soft and TTT. he states that the halo has been there for about a week and is painful. he doubts that he has a STI, since the only unprotected sex he has ever had has been with his current, longtime GF. he adds that his GF &#8220;can be trusted&#8221; and that she has never had an STI. What is the most likely diagnosis?<br>Chancroid<\/p>\n\n\n\n<p>While treating 29 y\/o F for vulvovaginitis, you inquire about her sexual hx. She explains that she has been sexually active for about 10 years. There are many cases of vulvovaginitis, but only one is an STI. Which one?<br>Trichomoniasis<\/p>\n\n\n\n<p>35 y\/o sarah says she her partner are in committed relationship and are looking for long lasting, effortless BC method. She wants to explore her BC options but mentions that she has an allergy to estrogen=based BC. What choice should be recommended?<br>Implanon<\/p>\n\n\n\n<p>Differences b\/t glaucoma and cataracts. Which test would be test?<br>T test<\/p>\n\n\n\n<p>Which of the following CN does NOT control eye movement<br>CN II (Olfatory)<\/p>\n\n\n\n<p>A man who recently attempted suicide is brought to your office by his wife. Having conducted a significant amount of research on her own about suicide prevention, she has many questions. Which of the following choice is the LEAST effective treatment for her husband?<br>? Intervention is the pt&#8217;s risk is escalating<\/p>\n\n\n\n<p>During a physical exam, Brett mentions that he wants to quit his job but is worried about losing health insurance. you inform him that HIPAA will provide protection for workers when they change or lose their jobs. This includes certain private information that pertains to Brett&#8217;s experience with the healthcare system. What is NOT true about HIPAA?<br>? All sharing of pt&#8217;s information requires written authorization<\/p>\n\n\n\n<p>Using the Dubowitz\/Ballard exam to estimate the gestational age of a Caucasian newborn, you determine him to be post-term. Which of the following is a finding NOT related to a criterion included in the Dubowitz\/Ballard exam?<br>Elasticity of cartilage in nose<\/p>\n\n\n\n<p>plantar creases over sole of feet<br>thickness and size of breast tissue<br>Hypertonic flexion of knees are included<\/p>\n\n\n\n<p>There is concern at your clinic that some children with developmental delays are not being identified soon enough. You are examining a 6 y\/o M. Which is the best tool to identify risk for developmental delay in this child?<br>Denver II<\/p>\n\n\n\n<p>Doris, 77 y\/o F, reports abd pain in the LLQ. For the last several weeks, Doris experienced frequent constipation and has taken laxatives for relief. However, her abd pain has become increasing severe and has been accompanied by n\/v. Her lab work shows an ESR of 45 mm\/hour and leukocytosis. Additionally, a sigmoidoscopy reveals inflamed mucosa. What is the most likely diagnosis?<br>Diverticulitis<\/p>\n\n\n\n<p>Donna, 25, took pregnancy test a week ago and learned that she is pregnant. Her mother is currently on insulin therapy to control diabetes, and Donna has BMI of 31. Since she is in her first trimester, which of the following diagnostic tests should you first order to assess the risk specific to this pt?<br>Urinalysis<\/p>\n\n\n\n<p>Kaitlin is 18 y\/o and has recently become sexually active. She requests a contraceptive prescription at your clinic. A physical exam indicates that she is 5&#8217;6 and 205lbs. Based on her ht and wt, which would be the LEAST effective form of contraception?<br>? Nuva ring<\/p>\n\n\n\n<p>Tanya is 10 y\/o, 4&#8217;8, and 110 lbs. Tanya&#8217;s mother is concerned about her wt and eating habits, as she has found junk food wrappers hidden around the house. She adds that Tanya is very defiant when it comes to food restrictions. Which of the following is the most appropriate way of approaching the topic of diet with Tanya?<br>Review the new MyPlate standards with Tanya and help her understand why a balanced diet and maintaining a healthy wt are important<\/p>\n\n\n\n<p>Dori, 64, has experienced recurring bouts of shingles. although you note during todays appointment that her skin eruptions have disappeared, she complains of pain and burning sensations that remain in the areas of outbreak. These sensations, she adds, have been severe enough to interfere with her sleep. What would be prescribed?<br>Gabapentin<\/p>\n\n\n\n<p>24 y\/o comes in for sore throat, nasal congestion, and cough. Pt repeatedly clears her watery congestion into tissue. Pt does not have fever, and PE is unremarkable. What is the most likely cause?<br>Common cold<\/p>\n\n\n\n<p>59 y\/o M is having FU eval 2 years after conclusion of chemo for leukemia. He tells you that he has been feeling &#8220;run down&#8221; and report unexplained wt loss, pain below the ribs on his left side, and night sweats. You determine he also has a fever. You consider chronic myelogenous leukemia Which finding is hallmark?<br>? Lymphocytosis<\/p>\n\n\n\n<p>15 y\/o penelope, who complains of regularly being tired. You note that she has dry skin, thinning hair, and intolerance to cold. Which of the following should be tested for in order to verify the most likely diagnosis?<br>? Elevated TSH and decreased T4<\/p>\n\n\n\n<p>65 y\/o in recent months noticed persistent, sharp, and painful spasms in her left cheek. They occur primarily when she is applying makeup and do not occur on the other side of her face. The pain feels like &#8220;an electric shock&#8221;. Which fo the following would be LEAST appropriate treatment for this condition?<br>Calcium channel blocker<\/p>\n\n\n\n<p>*Anti-seizure drugs<br>Muscle relaxants<br>Tricyclic antidepressants<\/p>\n\n\n\n<p>What is the purpose of case mgmt?<br>To mobilize, monitor, and control resources used by pt during illness<\/p>\n\n\n\n<p>16 y\/o M, with no abnormalities in his medical records, presents with unusual body proportions and underdeveloped sexual characteristics. Blood test indicates Klinefelter syndrome. Which is NOT typical feature of klinefelter syndrome?<br>Short stature<\/p>\n\n\n\n<p>*Hypogonadism<br>learning disability<br>Gynecomastia<\/p>\n\n\n\n<p>Elderly have a blunted fever response to infection due to an impairment in which body system?<br>Nervous system<\/p>\n\n\n\n<p>Prophylactic treatment of Pneumocystis jirovecii<br>Co-trimoxazole (trimethoprim-sulfamoxazole)<\/p>\n\n\n\n<p>ECG changes in hypokalemia<br>broad T wave and prominent U waves<\/p>\n\n\n\n<p>Atypical UTI symptoms usually found in elderly pts<br>lethargy<br>decreased appetite<br>incontinence<\/p>\n\n\n\n<p>acidosis and hyperthermia have what affect on the oxyhemoglobin dissociation curve<br>oxygen has a decreased affinity for hemoglobin shifting the curve to the right + to the tissues<\/p>\n\n\n\n<p>alkalosis and hypothermia have what effect on the oxyhemoglobin dissociation curve<br>oxygen has a greater affinity for hemoglobin shifting the curve to the left = to the blood<\/p>\n\n\n\n<p>What does it mean when there is a right shift in the oxyhemoglobin dissociation curve?<br>means that a higher PO2 is required to achieve hemoglobin saturation compared to baseline<br>*oxygen is more willing to unload into the tissues<\/p>\n\n\n\n<p>What does it mean when there is a left shift in the oxyhemoglobin dissociation curve?<br>it means that less PO2 is required to achieve hemoglobin saturation compared to baseline<br>*oxygen is less willing to unload into the tissues<\/p>\n\n\n\n<p>JNC 8 threshold for treatment of HTN in the elderly<br>150\/90<\/p>\n\n\n\n<p>Immediate treatment after diagnosis of pheochromocytoma is what?<br>Phentolamine to stabilize before surgery<\/p>\n\n\n\n<p>Criteria for diminished renal reserve<br>nephron loss of 50% and double baseline Cr<\/p>\n\n\n\n<p>Criteria of renal insuficiency<br>nephron loss of 75% and mild azotemia<\/p>\n\n\n\n<p>Treatment of bacterial vaginosis<br>metronidazole or clindamycin<\/p>\n\n\n\n<p>End-stage renal disease criteria<br>90% nephron loss, azotemia, and metabolic alterations<\/p>\n\n\n\n<p>American Burn Association: Burn Center Referral Criteria<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>Partial thickness &gt;10% TBSA<br>Burns that involve the face, hands, feet, genitalia, perineum, or major joints<\/li>\n\n\n\n<li>3rd degree<\/li>\n\n\n\n<li>electrical<\/li>\n\n\n\n<li>chemical<\/li>\n\n\n\n<li>inhalation injury<\/li>\n\n\n\n<li>burns with preexisting medical disorders that could complicate<\/li>\n\n\n\n<li>burn + trauma<\/li>\n\n\n\n<li>burned children<\/li>\n\n\n\n<li>burns that will require special social, emotional, or rehabilitation intervention<\/li>\n<\/ol>\n\n\n\n<p>dislocation<br>displacement of a bone from its joint<\/p>\n\n\n\n<p>subluxation<br>partial dislocation<\/p>\n\n\n\n<p>Labs in temporal arteritis<br>high ESR<br>normal WBC<\/p>\n\n\n\n<p>Temporal arteritis treatment<br>High-dose steroids<\/p>\n\n\n\n<p>Normal CVP<br>2-6<\/p>\n\n\n\n<p>CVP is elevated in conditions that cause a(n) <strong><em><strong>_<\/strong><\/em><\/strong> in the amount of fluid in the right atrium<br>increase<\/p>\n\n\n\n<p>Shock states that cause an elevated CVP<br>cardiogenic shock<br>obstructive shock<br>*also FVO<\/p>\n\n\n\n<p>CVP is decreased in conditions that cause a(n) <strong><em><strong>_<\/strong><\/em><\/strong> in the amount of fluid in the right atria<br>decrease<\/p>\n\n\n\n<p>Shock states that cause a decreased CVP<br>distributive shock<br>hypovolemic shock<\/p>\n\n\n\n<p>MAP formula<br>(SBP + 2DBP)\/3<\/p>\n\n\n\n<p>Normal pulmonary wedge pressure<br>6-12<\/p>\n\n\n\n<p>Shock states with elevated PWP<br>cardiogenic<br>low then high &#8211; septic<\/p>\n\n\n\n<p>Shock states that cause low PWP<br>hypovolemic<br>obstructive<br>anaphylactic<br>neurogenic<\/p>\n\n\n\n<p>PWP is a measure of what<br>left ventricular function<br>end-diastolic left ventricular pressure<\/p>\n\n\n\n<p>PWP is increased in conditions that <strong><em><strong>__<\/strong><\/em><\/strong> the pressure in the left ventricle at the end of diastole<br>increase<\/p>\n\n\n\n<p>Conditions that increase PWP<br>FVO<br>decreased elasticity of the left ventricle<\/p>\n\n\n\n<p>PWP is decreased in conditions that <strong><em><strong><em>___<\/em><\/strong><\/em><\/strong> the pressure in the left ventricle at the end of diastole<br>decrease<\/p>\n\n\n\n<p>Normal CVP<br>2-6 mmHg<\/p>\n\n\n\n<p>Normal PWCP<br>6-12 mmHg<\/p>\n\n\n\n<p>Cardiac Output<br>HR x SV<\/p>\n\n\n\n<p>Normal CO<br>4-8 L\/min<\/p>\n\n\n\n<p>Normal CI<br>2.5-4<\/p>\n\n\n\n<p>SVR<br>the resistance of systemic circulation<\/p>\n\n\n\n<p>Normal SVR<br>800-1200<\/p>\n\n\n\n<p>Where is SvO2 measured?<br>Pulmonary artery<\/p>\n\n\n\n<p>What is SvO2<br>a measurement of effectiveness of O2 delivery<\/p>\n\n\n\n<p>Normal SvO2<br>60-80%<\/p>\n\n\n\n<p>What does low SvO2 indicate<br>increased tissue extraction of O2<br>decreased O2 supply (decreased CO, decreased FiO2, anemia)<br>increased O2 demand (fever, shivering, increased WOB)<\/p>\n\n\n\n<p>What does high SvO2 indicate?<br>decreased tissue extraction of O2<br>increased O2 supply (FiO2 &gt; need)<br>decreased O2 demand (hypothermia)<br>decreased effectiveness of O2 delivery and uptake by cells (sepsis, shift of oxyhemoglobin dissociation curve to the left)<\/p>\n\n\n\n<p>Hypovolemic shock hemodynamics<br>SVR HIGH, everything else low<br>CO\/CI &#8211; low<br>CVP &#8211; Low<br>PWP &#8211; low<br>SVR &#8211; HIGH<br>SvO2 &#8211; low<\/p>\n\n\n\n<p>Cardiogenic shock hemodynamics<br>LOW CO\/CI &amp; SvO2 everything else high<br>CO\/CI &#8211; low<br>CVP &#8211; HIGH<br>PWP &#8211; HIGH<br>SVR &#8211; HIGH<br>SvO2 &#8211; low<\/p>\n\n\n\n<p>septic shock hemodynamics<br>CO\/CI: High then low (only shock state with high CO)<br>everything else: low then high<\/p>\n\n\n\n<p>Anaphylactic shock hemodynamics<br>everything low<\/p>\n\n\n\n<p>obstructive shock hemodynamics<br>LOW CO\/CI and normal to low PWCP; everything else HIGH<br>CO\/CI &#8211; low<br>CVP &#8211; HIGH<br>PWP normal\/low<br>SVR &#8211; HIGH<br>SvO2 &#8211; HIGH<\/p>\n\n\n\n<p>Neurogenic shock hemodynamics<br>everything low<\/p>\n\n\n\n<p>Herbal agents that increase risk of bleeding<br>if it starts with G it increases bleeding<br>Ginger<br>Ginko Biloba<br>Garlic<br>Ginseng<\/p>\n\n\n\n<p>Herbal agents that increase clotting<br>St. Jon&#8217;s Wart<\/p>\n\n\n\n<p>Use of St. Johns wart<br>depression<br>BPH<br>Sleep<\/p>\n\n\n\n<p>Normal Plts<br>150,000-400,000\/mm3<\/p>\n\n\n\n<p>Lower UTI drugs<br>trimethoprim-sulfamethoxazole (Bactrim) &lt;20% resistance nitrofurantoin (Macrobid) &gt;20% resistance<br>Fosfomycin (expensive)<\/p>\n\n\n\n<p>Upper UTI drugs<br>Ciprofloxacin<br>Levofloxacin<br>Ceftriaxone (IV)<\/p>\n\n\n\n<p>Prerenal Diagnostics<br>Serum BUN:Cr &#8211; &gt;10:1<br>urine Na &#8211; &lt;20 mmol SG &#8211; &gt;1.015<br>Fractional Excretion of Na &lt;1<br>Urinary Sediment &#8211; normal\/few hyaline casts<\/p>\n\n\n\n<p>Intrarenal diagnostics<br>Serum BUN:Cr &#8211; 10:1<br>Urine Na &#8211; &gt;40 mmol<br>SG &lt;1.015 Fractional Excretion of Na &gt;3<br>Urine Sediment Granular with white casts<\/p>\n\n\n\n<p>Postrenal diagnostics<br>Serum BUN:Cr &#8211; 10:1<br>Urine Na &#8211; &gt;40 mmol<br>SG &lt;1.015 Fractional Excretion of Na &#8211; usually &gt;3<\/p>\n\n\n\n<p>Management of Prerenal AKI<br>expand volume<\/p>\n\n\n\n<p>Management of intrarenal AKI<br>maintain perfusion, stop nephrotoxic drugs, RRT<\/p>\n\n\n\n<p>Management of post renal AKI<br>remove obstruction<\/p>\n\n\n\n<p>Criteria of dialysis<br>A &#8211; acidosis (metabolic)<br>E &#8211; electrolyte abnormalities<br>I &#8211; intoxication<br>O &#8211; oliguria &lt;400 cc\/day<br>U &#8211; uremia<\/p>\n\n\n\n<p>Most common nephrotoxic drugs<\/p>\n\n\n\n<ul class=\"wp-block-list\">\n<li>antifungals (&#8220;fungins&#8221;, &#8220;azoles&#8221; and Amphotericin B {amphoterrible})<\/li>\n\n\n\n<li>aminoglycoside abx (vancomycin, streptomycin, tobramycin, gentamycin)<\/li>\n\n\n\n<li>NSAIDs<\/li>\n\n\n\n<li>ACE inhibitors<\/li>\n<\/ul>\n\n\n\n<p>Differential for testicular pain<br>renal stone (no relief with testicular lift)<br>epididymitis (relief with testicular lift)<br>torsion (no relief with testicular lift)<\/p>\n\n\n\n<p>Management of nephrolithiasis<br>CT scan<br>PRN morphine or dilaudid and x1 toradol, and x1 reglan<br>possible lithotripsy (large stone)<\/p>\n\n\n\n<p>Black box warning for Reglam<br>EPS &#8211; tardive dyskinesia in long term use<\/p>\n\n\n\n<p>BPH onset<br>50 yo<\/p>\n\n\n\n<p>Use of Saw Palmetto<br>BPH<br>no evidence for Prostate Cancer prevention!!<\/p>\n\n\n\n<p>Normal GFR<br>125 mL\/min<\/p>\n\n\n\n<p>GFR diminishes <strong>__<\/strong>% per decade after 30yo<br>10<\/p>\n\n\n\n<p>Normal Renal Gerontology changes<br>decreased kidney size and number of nephrons<br>reduced response to vasopressin and impaired Na conservation<br>decreased bladder tone, elasticity, and capacity<br>prostate enlargement<\/p>\n\n\n\n<p>GFR formula<br>(140 &#8211; age) x (WT x .85 {for women})\/73 x Serum Cr<\/p>\n\n\n\n<p>Which patients do we treat for asymptomatic bacteriuria<br>pregnancy<br>urologic interventions<br>renal transplant<\/p>\n\n\n\n<p>Valve activity in S1<br>mitral\/tricuspid close<br>aortic\/pulmonic open<\/p>\n\n\n\n<p>Valve activity in S2<br>aortic\/pulmonic close<br>mitral\/tricuspid open<\/p>\n\n\n\n<p>S3 heart sound<br>Increased ventricular filling pressure (e.g., mitral regurgitation, HF), common in dilated ventricles, pregnancy<\/p>\n\n\n\n<p>S4 heart sound<br>Atrial Gallup;<br>related to stiffness of the ventricular myocardium to rapid filling<\/p>\n\n\n\n<p>Murmur grades<br>I\/VI &#8211; barely audible<br>II\/VI: audible but faint<br>III\/VI: moderately loud<br>IV\/VI: loud with a thrill<br>V\/VI: very loud<br>VI\/VI: loudest<\/p>\n\n\n\n<p>Ms. Ard and Mr. Ass<br>Mitral Stenosis &amp; Aortic Regurgitation = Diastolic<br>Mitral regurgitation &amp; Atrial Stenosis = Systolic<\/p>\n\n\n\n<p>Murmur locations<br>Aortic &#8211; Right sternal boarder 2nds ICS<br>Pulmonic &#8211; left sternal boarder 2nd ICS<br>Tricuspid &#8211; left sternal boarder 4th ICS<br>Mitral &#8211; 5th ICS at midclavicular line<\/p>\n\n\n\n<p>NYHA Classification of HF<br>class 1 -no symptoms<br>class 2 &#8211; slight symptoms on exertion<br>class 3 &#8211; marked symptoms on exertion<br>class 4 &#8211; symptoms at rest<\/p>\n\n\n\n<p>Treatment of acute pulmonary edema<br>low flow O2<br>4 morphine<br>40 mg lasix x2 q10<\/p>\n\n\n\n<p>decreased cortisol levels<br>-Adrenal hyperplasia<br>-Addison&#8217;s disease<br>-Anterior pituitary hyposecretion<br>-Hypothyroidism<\/p>\n\n\n\n<p>increased cortisol levels<br>-Hyperthyroidism<br>-cushing&#8217;s syndrome<br>-Stress d\/t trauma\/surgery<br>-carcinoma<br>-Overproduction of ACTH d\/t tumors in ant. Pituitary<br>-Adrenal adenoma<br>-Obesity<\/p>\n\n\n\n<p>ACC\/AHA Stage 1 HTN<br>130-139 or 80-89<\/p>\n\n\n\n<p>ACC\/AHA Stage 2 HTN<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>140 OR &gt;90<\/p>\n<\/blockquote>\n\n\n\n<p>First line HTN medication<br>thiazide diuretics<\/p>\n\n\n\n<p>Management of HTN urgency<br>oral meds<br>clonidine (Catapres)<\/p>\n\n\n\n<p>Treatment of HTN emergency<br>IV Nicardipine, Nitroprusside or labetalol<br>decreased 25% first hour<br>then to 160\/100 next 2-6 hr<\/p>\n\n\n\n<p>What is Prinzmetal&#8217;s angina?<br>coronary artery vasospasm<\/p>\n\n\n\n<p>Lipid panel normals<br>Normal LDL &lt;100 or &lt;70 if DM HDL&gt;40 (M), &gt;50 (F)<br>and triglycerides &lt;150<\/p>\n\n\n\n<p>Meds for angina<br>nitrates, beta blockers, calcium channel blockers<\/p>\n\n\n\n<p>What is the Pooled Cohort Equation used for<br>Est 10 year ASCVD risk<\/p>\n\n\n\n<p>High intensity statins<br>Atorvastatin 40-80 mg<br>Rosuvastatin 20-40 mg<\/p>\n\n\n\n<p>Therapeutic INR<br>2-3<\/p>\n\n\n\n<p>Therapeutic PTT<br>1.5-2 x normal or control values<\/p>\n\n\n\n<p>Contraindications for TPA<br>Neuro surgery\/head trauma within 3 months<br>Prior ICH<br>BP: &gt;185 or Diastolic &gt;110<br>Lumbar puncture recently<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>3 hours<br>If it is a hemorrhagic stroke<br>Seizures<br>Abnormal glucose levels &lt;50 &gt;400<br>Any known bleeding disorders<br>Active internal bleeding<br>Suspected dissection<br>Malingnant intercranial neoplasmq<\/p>\n<\/blockquote>\n\n\n\n<p>Medication management of PVD<br>Cilostazol (pletal)<\/p>\n\n\n\n<p>Normal ABI<br>0.9-1.3<\/p>\n\n\n\n<p>ABI indicative of PVD<br>&lt;1<\/p>\n\n\n\n<p>ECG changes in pericarditis<br>ST segment elevation and T wave inversion<br>PR depression<\/p>\n\n\n\n<p>Treatment of pericarditis<br>NSAIDs<br>Indomethacin<br>Corticosteroids if NSAID failure after several wks<br>ABX &#8211; only if bacterial<\/p>\n\n\n\n<p>Major complication of pericarditis<br>cardiac tamponade<\/p>\n\n\n\n<p>Causes of night sweats in adults<br>endocarditis<br>TB<br>menopause<br>HIV\/AIDS<br>leukemia<\/p>\n\n\n\n<p>Fever<br>malaise<br>night sweats<br>WT loss<br>endocarditis<\/p>\n\n\n\n<p>Top fiver killers of adults in US<br>Heart disease (CAD)<br>Cancer<br>Accidents<br>Lower Respiratory disease (Asthma&amp;COPD)<br>CVA<\/p>\n\n\n\n<p>Cranial nerves<br>I: Olfactory, smell, Sensory<br>II: optic, vision, sensory<br>III: oculomotor, EOM, motor<br>IV: trochlear, downward and inward eye movement, motor<br>V: Trigeminal, mastication, sensation of face, scalp, cornea, and mucous membranes, both<br>VI: abducens, lateral eye movement, motor<br>VII: Facial, facial sensation and movement, both<br>VIII: acoustic, hearing and equilibrium, sensory<br>IX: Glossopharyngeal, phonation, gag, carotid reflex, swallowing, 1\/3 taste, both<br>X: Vagus, talking, swallow, carotid reflex, both<br>XI: Spinal Accessory, shoulder and head movement, motor<br>XII: Hypoglossal, tounge movement, motor<\/p>\n\n\n\n<p>Signs of vertebrobasilar stroke<br>vertigo, ataxia, dizziness, visual field deficits, wkns, confusion<\/p>\n\n\n\n<p>Potassium considerations in burn pts<br>monitor for hyperkalemia firs 24-48 hr<br>monitor for hypokalemia 3 days post burns<\/p>\n\n\n\n<p>How to remove tar in tar burns<br>petroleum products &#8211; ie bacitracin, petroleum jelly<\/p>\n\n\n\n<p>signs of temporal arteritis<br>Visual LOSS!<br>Swollen pale disc with blurred margin on eye exam<br>Jaw claudication<br>Age over 50<br>scalp tenderness<br>fever<br>Temporal artery nodular or enlarged and tender<\/p>\n\n\n\n<p>Eye cup : disk<br>1:2<\/p>\n\n\n\n<p>Cause of increased cup size in eye<br>galucoma<\/p>\n\n\n\n<p>A\/V ratio in the eye<br>2:3 or 4:5, opposite in body, arteries are brighter red and narrower<\/p>\n\n\n\n<p>Signs of diabetic retinopathy<br>cotton wool spots<br>ruptured microaneurysm<br>hard exudates<\/p>\n\n\n\n<p>AV nicking is most commonly seen on retinal exam of which chronic condition?<br>HTN<\/p>\n\n\n\n<p>Differentiation of Conjunctivitis and treatments<br>Chemical &#8211; none, flush with NS<br>Bacterial &#8211; Purulent, ABX &#8211; levofloxacin, ciprofloxacin, moxifloxacin, tobramycin, gentamycin)<br>Viral &#8211; watery &#8211; saline drops, decongestants\/antihistamines, mast cell stabilizers, NSAIDS<br>Herpetic &#8211; bright red and irritated &#8211; refer to opthalmologist<br>Gonococcal or chlamydial &#8211; Copious purulent &#8211; Ceftriaxone 250 mg IM + Azithromycin<\/p>\n\n\n\n<p>Gonococcal and chlamydia treatment<br>Ceftriaxone 250 mg IM + azithromycin<\/p>\n\n\n\n<p>Normal intraocular pressure (IOP)<br>10-20 mmHg<\/p>\n\n\n\n<p>Treatment of open angle glaucoma<br>-prostaglandins (lantanoprost, bimatoprost): inc outflow<br>-beta blockers (timolol, bextalol): dec aqeuous humor production; or<br>-pilocarpine to inc aqeuous humor outflow<br>-carbonic anhydrase inhib can work too<br>-if meds fail, lsaer trabeculoplasty or trabeculectomy<\/p>\n\n\n\n<p>treatment of closed angle glaucoma<br>-medical emergency! goal: decrease IOP<\/p>\n\n\n\n<h1 class=\"wp-block-heading\">1: mannitol<\/h1>\n\n\n\n<p>-eyedrops: timolol, pilocarpine, apraclonidine<br>-systemic meds: (oral\/IV acetazolamide, IV mannitol)<br>-curative: Laser peripheral iridotomy (create hole in peripheral iris)<\/p>\n\n\n\n<p>Major differences in acute\/closed angle glaucoma and cataract exam<br>similar presentation except cataracts are painless<\/p>\n\n\n\n<p>Adult Portion<\/p>\n\n\n\n<p>Contraceptive Options<\/p>\n\n\n\n<p>What would cause break through bleeding on Day 1-9 of cycle?<br>deficiency of estrogen<\/p>\n\n\n\n<p>What would cause break through bleeding on Day 20-21 of cycle<br>deficiency of progesterone<\/p>\n\n\n\n<p>What are the absolute contraindications of COC?<br>hx thromboembolic disorders<br>CVA<br>CAD<br>breast CA<br>estrogen-dep neoplasia<br>pregnancy<br>liver tumor; impaired LF<br>previous cholelithiasis during pregnancy<br>undiagnosed, abnormal uterine bleeding<\/p>\n\n\n\n<p>What type of pill for migraine HAs, breastfeeding, contraindication to COC?<br>progestin-only mini pill<\/p>\n\n\n\n<p>When should a diaphragm\/cervical cap be refitted?<br>gain or loss of 20lbs<\/p>\n\n\n\n<p>How long should a diaphragm be left in vagina following intercourse?<br>6 hours<\/p>\n\n\n\n<p>EENT<\/p>\n\n\n\n<p>If cup is more than 1\/2 the size of the disc diameter, consider <em>__<\/em><br>glaucoma<\/p>\n\n\n\n<p>Raised area at where an artery and vein meet<br>nicking = hypertension<\/p>\n\n\n\n<p>infection of the eye usually by S. aureus<br>abrupt onset<br>painful<br>erythema<br>localized tender mass in eyelid<br>hordeolum (stye)<\/p>\n\n\n\n<p>management of hordeolum (stye)<br>warm compresses<br>topical bacitracin or erythromycin<\/p>\n\n\n\n<p>beady nodule on eye lid, infection or retention cyst of meibomian glad, usually on upper lid<br>painless<br>swelling, tenderness, sensitive to light, increased tearing<br>chalazion<\/p>\n\n\n\n<p>management of chalazion<br>warm compresses<br>refer for surgical removal<\/p>\n\n\n\n<p>Staph infection or seborrheic dermatitis of lid edge<br>red, scaly, greasy flakes<br>thickened, crusted lid margins<br>burning, itching, tearing<br>blepharitis<\/p>\n\n\n\n<p>management of blepharitis<br>hot compress<br>topical bacitracin or erythromycin<br>scrub lashes and lids<\/p>\n\n\n\n<p>treatment for bacterial conjunctivitis<br>antibiotic drops- levo, oflo, cipro floxacin, or tobramycin or gentamycin soln<\/p>\n\n\n\n<p>gonorrhea in eye<br>Ceftriaxone (rocephin) 250 mg IM<\/p>\n\n\n\n<p>chlamydia in eye<br>1 gm azithromycin<\/p>\n\n\n\n<p>treatment for allergic conjunctivitis<br>po antihistamines<\/p>\n\n\n\n<p>increased intraocular pressure<br>glaucoma<\/p>\n\n\n\n<p>type of glaucoma:<br>-asymptomatic<br>-incr IOP<br>-cupping of disc<br>-decr peripheral vision<br>open-angle<\/p>\n\n\n\n<p>type of glaucoma:<br>-extreme pain<br>-blurred vision<br>-halos around lights<br>-pupil dilated or fixed<br>closed-angle<\/p>\n\n\n\n<p>Tonometry screening nationally recommended by age<br>40<\/p>\n\n\n\n<p>clouding and opacification of the normally clear lens of the eye<br>-painless<br>-clouded, blurred vision<br>-halos around lights<br>-night vision difficulty<br>-sensitivity to light and glare<br>-fading\/yellowing of colors<br>-diplopia (double vision) in single eye<br>-no red reflex<br>cataract<\/p>\n\n\n\n<p>separation of the light-sensitive membrane in the back of the eye (retina) from its supporting layers<br>-flashes of light (photopsia), esp. peripheral<br>-floaters in eye<br>-blurred vision<br>-shadow or blindness in a part of visual field<br>retinal detachment<\/p>\n\n\n\n<h1 class=\"wp-block-heading\">1, #2, #3 causes of otitis, sinusitis, &amp; CAP<\/h1>\n\n\n\n<ol class=\"wp-block-list\">\n<li>Strep pneumoniae<\/li>\n\n\n\n<li>Haemophilus influenzae<\/li>\n\n\n\n<li>Moraxella catarrhalis<\/li>\n<\/ol>\n\n\n\n<p>type of chronic otitis media consisting of peeling layers of scaly or keratinized epithelium; if untreated, may erode the middle ear, leading to nerve damage and deafness<\/p>\n\n\n\n<p>squamous epithelium lined sac, filled with desquamated keratin<br>painless otorrhea, hearing loss, canal filled with mucous and granulation tissue<br>tympanic membrane perforation 90% of cases<br>cholesteatoma<\/p>\n\n\n\n<p>What are some medications for the management of vertigo?<br>diazepam (valium)<br>meclizine HCl (antivert)<br>diphenhydramine (benadryl)<br>scopolamine patch<br>antiemetics<\/p>\n\n\n\n<p>sensorineural hearing loss, vertigo, &amp; tinnitus<br>Meniere&#8217;s disease<\/p>\n\n\n\n<p>Weber test normal<br>sound heard bilaterally, does not lateralize<\/p>\n\n\n\n<p>Rinne test normal<br>AC &gt; BC<\/p>\n\n\n\n<p>Conductive loss Weber &amp; Rinne<br>Weber- sound materializes to affected ear<br>Rinne- Abnormal in affected ear (AC &lt; BC)<\/p>\n\n\n\n<p>Sensorineural loss Weber &amp; Rinne<br>Weber- sound materializes to the unaffected ear<br>Rinne- normal in affected ear<\/p>\n\n\n\n<p>watery rhinorrhea<br>erythematous nasal mucosa<br>sneezing, nasal\/sinus blockage, HA, sore throat, cough, malaise<br>common cold; viral rhinitis<\/p>\n\n\n\n<p>anterior cervical adenopathy<br>macropapular rash<br>fever<br>strep throat<\/p>\n\n\n\n<p>Centor criteria for strep (FLEA)<\/p>\n\n\n\n<p>very specific test- the negatives test negative<br>if 2 or more yes- do rapid strep<br>Fever &gt;100.4<br>Lack of cough<br>Phargynotonsillar Exudate<br>Anterior cervical adenopathy<\/p>\n\n\n\n<p>management of strep throat<br>PCV V<br>erythromycin if PCN allergic<\/p>\n\n\n\n<p>ABRUPT onset of fever, HA, myalgia, coryza, anorexia, malaise, cough<br>influenza<\/p>\n\n\n\n<p>management of flu<br>neuraminidase inhibitors<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>zanamivir (relenza) inhaler<\/li>\n\n\n\n<li>oseltamivir (tamiflu) oral<br>shorten duration of s\/s by 2 days<\/li>\n<\/ol>\n\n\n\n<p>white tonsillar exudates<br>posterior cervical adenopathy<br>fever, chills, SEVERE malaise\/fatigue, anorexia, pharyngitis, splenomegaly<br>Mono (Epstein-Barr virus)<\/p>\n\n\n\n<p>management of mono<br>supportive care<br>prednisone\/steroid taper for severely enlarged tonsils<br>avoid contact sports for 3 weeks- months (to prevent splenic rupture)<\/p>\n\n\n\n<p>inflammation of mucous membranes lining one or more of the paranasal sinuses<\/p>\n\n\n\n<p>recent URI, with relapse<br>red nasal mucosa<br>pain\/pressure over face, cheeks, teeth<br>postnasal drip<br>fever<br>HA- supine or bending<\/p>\n\n\n\n<p>decreased transillumination<br>sinusitis<\/p>\n\n\n\n<p>management of bacterial sinusitis<br>amox w\/ clav (Augmentin)<br>clarithromycin (Biaxin)<\/p>\n\n\n\n<p>Skin<\/p>\n\n\n\n<p>circumscribed flat area of skin<br>different in color &amp; texture from surrounding tissue<br>&lt;1cm in size<\/p>\n\n\n\n<p>ex. freckle, petechiae, flat nevi<br>macule<\/p>\n\n\n\n<p>large macule, &gt;1cm in size<\/p>\n\n\n\n<p>ex. mongolian spot, cafe au lait<br>patch<\/p>\n\n\n\n<p>small, solid, elevated lesion<br>&lt;1cm in diameter<\/p>\n\n\n\n<p>ex. ant bite, elevated nevus, verruca (wart)<br>papule<\/p>\n\n\n\n<p>elevation of skin<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>1cm in diameter<\/p>\n<\/blockquote>\n\n\n\n<p>ex. psoriasis<br>plaque<\/p>\n\n\n\n<p>a visible accumulation of purulent fluid under skin<br>&lt;1cm<\/p>\n\n\n\n<p>ex. acne, impetigo<br>pustule<\/p>\n\n\n\n<p>circumscribed elevation of skin containing serous fluid<br>&lt;1cm<\/p>\n\n\n\n<p>herpes, varicella<br>vesicle<\/p>\n\n\n\n<p>solid mass of skin<br>observed as an elevation or can be palpated<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>1cm in diameter<br>often extends into the dermis<\/p>\n<\/blockquote>\n\n\n\n<p>ex. xanthoma, fibroma<br>nodule<\/p>\n\n\n\n<p>&#8220;blister&#8221;<br>circumscribed elevation containing fluid<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>1cm in diameter<br>extends only into the epidermis<\/p>\n<\/blockquote>\n\n\n\n<p>ex. burns, superficial blister, contact dermatiti<br>bulla<\/p>\n\n\n\n<p>elevated white or pink compressible papule or plaque<br>a red, axon-mediated flare often surrounds it<br>commonly associated with allergic reactions<\/p>\n\n\n\n<p>ex. PPD test, mosquito bites<br>wheal<\/p>\n\n\n\n<p>any closed cavity or sac<br>contains fluid or semisolid material<br>normal or abnormal epithelium<\/p>\n\n\n\n<p>ex. sebaceous cyst<br>cyst<\/p>\n\n\n\n<p>a localized collection of purulent fluid in a cavity formed by disintegration or necrosis of tissues<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>1cm in size<br>abscess<\/p>\n<\/blockquote>\n\n\n\n<p>&#8220;mass&#8221;<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>a few cm in diameter<br>firm or soft<br>benign or malignant<br>tumor<\/p>\n<\/blockquote>\n\n\n\n<p>LIVE vaccines<br>varicella<br>MMR<br>zoster<br>flu mist<\/p>\n\n\n\n<p>most frequently used topical antibiotic for acne<br>clindamycin<\/p>\n\n\n\n<p>PO antibiotics for acne<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>tetracycline- don&#8217;t use in pregnancy and &lt;9yo<\/li>\n\n\n\n<li>erythromycin, minocycline, doxycycline, clindamycin<\/li>\n<\/ol>\n\n\n\n<p>2 major skin pathogens<br>Staph<br>Strep<\/p>\n\n\n\n<p>boil that looks like a spider bite<br>MRSA<\/p>\n\n\n\n<p>inflamed area of skin<br>cellulitis<\/p>\n\n\n\n<p>most common pathogens of cellulitis<br>Group a strep<br>s. aureus<\/p>\n\n\n\n<p>management of CA-MRSA<br>TMP-SMZ (bactrim)<br>doxy\/mino<br>clindamycin<\/p>\n\n\n\n<p>if area of very low CA-MRSA infection<br>dicloxacillin<br>or cephalexin (Keflex)<\/p>\n\n\n\n<p>management of Group A strep<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>TMP-SMZ plus beta lactam (PCN, amox, or 1st gen cephalosporin like keflex)<\/li>\n\n\n\n<li>doxy\/mino plus beta lactam<\/li>\n\n\n\n<li>clinda<\/li>\n<\/ol>\n\n\n\n<p>Which antibiotic will cover both CA-MRSA &amp; Strep<br>clinda<\/p>\n\n\n\n<p>looks like a big sunburn<br>bright, red, warm<br>rapid progression of erythema<br>usually cause by Strep<br>erysipelas<\/p>\n\n\n\n<p>Staph infection<br>groin or axilla<br>hidradenitis suppurativa<\/p>\n\n\n\n<p>What treatment for minor skin infections?<br>consider topical- bacitracin, bactroban (mupirocin)<\/p>\n\n\n\n<p>1st gen cephalosporin- cephalexin (Keflex)<br>PCNase- resistant PCN (dicloxacillin)<br>alternatives: clinda or amox-clav<\/p>\n\n\n\n<p>What pathogen is impetigo usually caused by?<br>staph aureus<\/p>\n\n\n\n<p>What is paronychia?<br>staph around nail fold<\/p>\n\n\n\n<p>bruise under finger nail<br>and its treatment<br>subungual hematoma<br>trephination- drill a hole<\/p>\n\n\n\n<p>inflammation of the superficial tissues of the penile head caused by Candida albicans<\/p>\n\n\n\n<p>and its treatment<br>candida balanitis<\/p>\n\n\n\n<p>miconazole, clotrimazole, steroids, fluconazole<\/p>\n\n\n\n<p>irritation of the fold of the skin, commonly occurring in warm, moist body areas<br>&amp; its treatment<br>candida intertrigo<br>drying with talc or cornstarch<br>topical antifungals<br>po antifungals<\/p>\n\n\n\n<p>treatment for capitus<br>selenium shampoo<\/p>\n\n\n\n<p>What 2 meds for post-herpectic neuralgia<br>gabapentin (neurontin)<br>pregabalin (lyrica)<\/p>\n\n\n\n<p>small patches in sun-exposed parts of body<br>premalignant<br>rough, flesh colored, pink or hyperpigmented<br>actinic keratosis<\/p>\n\n\n\n<p>What can AKs progress to?<br>squamous cell carcinoma<\/p>\n\n\n\n<p>management of AKs<br>liquid nitrogen<\/p>\n\n\n\n<p>firm, irregular papule or nodule<br>develop over a few months<br>3-7% metastasize<br>prolonged, sun-exposed areas in fair skinned<br>keratitic, scaly bleeding<br>squamous cell carcinoma<\/p>\n\n\n\n<p>common on geros<br>benign, not painful lesions<br>beige, brown or black plaques<br>stuck on appearance<br>seborrheic keratoses<\/p>\n\n\n\n<p>treatment of seborrheic keratoses<br>none<br>liquid nitro<\/p>\n\n\n\n<p>most common skin CA<br>slow growing<br>waxy, pearly appearance<br>central depression or rolled edge<br>telangiectatic vessels<br>basal cell carcinoma<\/p>\n\n\n\n<p>mortality rate highest of all skin CAs<br>dx at 40yo<br>metastasize anywhere<br>malignant melanoma<\/p>\n\n\n\n<p>What is the initial treatment for psoriasis?<br>tar\/salicylic acid shampoo<br>topical steroids (BMTZ)<br>UVB light exposure<\/p>\n\n\n\n<p>mild, acute inflammatory disorder<br>more common in females during spring and fall<br>pruritic rash on trunk and proximal extremities<br>initial lesion 2-10cm: &#8220;herald patch&#8221;<br>generalized rash within 1-2 weeks<br>lesions in Christmas tree pattern<br>eruption lasts 4-8 weeks<br>pityriasis rosea<\/p>\n\n\n\n<p>What tests do you order if you suspect lyme?<br>ELISA initial<br>Western blot confirmatory<\/p>\n\n\n\n<p>management of lyme<br>doxy<br>amox<\/p>\n\n\n\n<p>What kind of rash do you have with lyme vs. rocky mountain spotted fever?<br>lyme- erythema migrans<br>RMSF- maculopapular rash, petechial rash<\/p>\n\n\n\n<p>What tests for RMSF?<br>PCR<br>immunohistochemical IHC staining<br>Indirect immunofluorescence assay (IFA) for R. rickettsii antigen<\/p>\n\n\n\n<p>sudden onset flu-like symptoms<br>1st lesions on oral mucosa\/palate, face, or forearms<br>centrifugal distribution with greatest concentration of lesions on face and distal extremities<br>all lesions in same stage of development<br>scabs leading to deep, pitted scars<br>excruciating pain<br>small pox<br>no cure, only supportive<\/p>\n\n\n\n<p>pruritic papule leading to ulcer surrounded by vesicles<br>arms\/hands, followed by face and neck<br>black necrotic eschar with edema<br>cutaneous anthrax- report to heath department<\/p>\n\n\n\n<p>antibiotic for anthrax<br>PCN<br>cipro<br>doxy<\/p>\n\n\n\n<p>Endocrine<\/p>\n\n\n\n<p>insulin dependent<br>juvenile onset<br>human leukocyte antigens (HLA-DR3 or HLA-DR4)<br>islet cell antibodies<br>ketones<br>poly&#8217;s- uria, dipsia, phagia<br>DM1<\/p>\n\n\n\n<p>lab findings DM1<br>random glucose &gt;200 with polyuria, polydipsia, weight loss<br>ketonemia, ketouria<br>BUN\/creat elevated<\/p>\n\n\n\n<p>What lab specific to DM 1?<br>human leukocyte antigens (HLA-DR3 or HLA-DR4)<\/p>\n\n\n\n<p>normal BUN<br>normal creat<br>10-20<br>.5-1.5<\/p>\n\n\n\n<p>which one fluctuates?<br>BUN- elevated with dehydration<\/p>\n\n\n\n<p>most sensitive indicator of renal fxn?<br>serum creatinine<\/p>\n\n\n\n<p>good a1c<br>6<\/p>\n\n\n\n<p>What percent of diet should be carbs?<br>55-60%<\/p>\n\n\n\n<p>when should insulin be started?<br>ketones<\/p>\n\n\n\n<p>how should insulin be split up during the day? include dosage<br>0.5 u\/kg\/day with 2\/3 of dose in am, 1\/3 evening<\/p>\n\n\n\n<p>review pg. 228-230<\/p>\n\n\n\n<p>nocturnal hypoglycemia develops a stimulating surge of counter regulatory hormones.<br>hypoglycemic at 0300, rebounds with elevated blood glucose at 0700<br>Somogyi effect<\/p>\n\n\n\n<p>treatment of Somogyi<br>reduce\/omit bedtime dose of insulin<\/p>\n\n\n\n<p>tissue becomes desensitized to insulin nocturnally; progressive elevation of glucose at night<br>elevated glucose at 0700<br>Dawn phenomenon<\/p>\n\n\n\n<p>treatment of Dawn phenomenon<br>add\/increase the bedtime dose of insulin<\/p>\n\n\n\n<p>most common presentation of hyperthyroidism?<br>Grave&#8217;s<\/p>\n\n\n\n<p>most common presentation of hypothyroidism?<br>Hashimoto&#8217;s<\/p>\n\n\n\n<p>most important lab for hyperthyroid? Most important for hypothyroid?<br>Hyper- T3, Hypo- T4<\/p>\n\n\n\n<p>hyperthyroidism<br>TSH ?<br>T3 ?<br>TSH down<br>T3 up<\/p>\n\n\n\n<p>hypothyroid<br>TSH ?<br>T4 ?<br>TSH up<br>T4 down<\/p>\n\n\n\n<p>symptomatic treatment of hyperthyroid<br>propranolol (inderal) 10-80mg qid<\/p>\n\n\n\n<p>meds of hyperthyroid<br>thiourea drugs- methimazole (tapazole) 30-60mg qd in 3 doses<br>propylthiouracil 300-600mg qd in 4 doses<br>radioactive iodine 131-I to destroy goiters<br>lugol&#8217;s solution 2-3gtts po qd x10d to reduce vascularity of gland<\/p>\n\n\n\n<p>ACTH hyper secretion by pituitary<br>adrenal tumors<br>chronic administration of glucocorticoids<br>Cushing&#8217;s syndrome<\/p>\n\n\n\n<p>s\/s of Cushings<br>central obesity<br>moon face, buffalo hump<br>acne<br>poor wound healing<br>purple striae<br>hirsutism<br>HTN from vasoconstriction<br>weakness<br>amenorrhea<br>impotence<br>HA<br>polyuria, thirst<br>labile mood<br>frequent infections<\/p>\n\n\n\n<p>deficient cortisol, androgens, aldosterone<br>autoimmune destruction of adrenal gland<br>mets CA<br>bilateral adrenal hemorrhage from anticoagulant therapy<br>pituitary failure resulting in decreased ACTH<br>Addison&#8217;s disease (primary adrenocortical insufficiency)<\/p>\n\n\n\n<p>s\/s of Addison&#8217;s<br>hyperpigmentation in buccal mucosa and skin creases (knuckles, nail beds, nipples, palmar creases, and posterior neck)<br>diffuse tanning and freckles<br>orthostasis and hypotension<br>scant axillary and pubic hair<br>rapid worsening of chronic s\/s (acute)<br>fever (acute)<br>LOC changes (acute)<\/p>\n\n\n\n<p>outpatient management of Addison&#8217;s<br>glucocorticoid and mineralocorticoid replacement<\/p>\n\n\n\n<p>hydrocortisone (glucocorticoid)<br>fludrocortisone acetate (Florinef) mineralocorticoid<\/p>\n\n\n\n<p>labs of Cushings<br>hyperglycemia<br>hypernatremia<br>hypokalemia<\/p>\n\n\n\n<p>labs of Addisons<br>hypoglycemia<br>hyponatremia<br>hyperkalemia<\/p>\n\n\n\n<p>Musculoskeletal<\/p>\n\n\n\n<p>What lab would be important before high-dose NSAIDs?<br>renal fxn<br>BUN &amp; creat- esp creat<\/p>\n\n\n\n<p>knee locking &amp; immediate swelling<br>meniscal tear<\/p>\n\n\n\n<p>palpable\/audible click when knee is raised slowly with one foot externally rotated<br>test for medial meniscal injury<br>McMurray&#8217;s test<\/p>\n\n\n\n<p>knee in 20-30 degree flexion, grasp leg wit one hand with anterior force to proximal tibia while opposite hand stabilizes the thigh (graded 1+ to 3+)<br>assess for anterior\/posterior cruciate ligament tear<br>Lachman (drawer) test<\/p>\n\n\n\n<p>patient prone<br>flex knee 90 degrees, pressure on heel with one hand while rotation the lower leg internally and externally<br>pain or click is positive for medial or lateral collateral ligament damage and\/or meniscus injury<br>Apley&#8217;s grind test<\/p>\n\n\n\n<p>up to how many steroid injections per year?<br>3<\/p>\n\n\n\n<p>What are the dangers of too many steroid injections?<br>breaks down joint tissue<\/p>\n\n\n\n<p>endocrine dysfunction- Addison&#8217;s crisis- no steroid made by body- hypotension<\/p>\n\n\n\n<p>Heberden&#8217;s nodes and Bouchard&#8217;s nodes seen with which type of arthritis?<br>osteo<\/p>\n\n\n\n<p>Heberden&#8217;s are where<br>distal interphalangeal nodes (DIPs)<\/p>\n\n\n\n<p>Bouchard&#8217;s are where<br>proximal interphalangeal nodes (PIPs)<\/p>\n\n\n\n<p>test for carpal tunnel that involves tapping over median nerve<br>Tinel&#8217;s sign<\/p>\n\n\n\n<p>put them in the pharaoh position for 1 min (wrist flexion)<br>Phalen&#8217;s test<\/p>\n\n\n\n<p>quad muscle weak<br>pain radiates into medial malleolus, numbness along the same path especially medial aspect of knee<br>*decr\/absent patellar reflex<br>screening exam: have patient squat and rise<br>L3-L4 disk pathology<\/p>\n\n\n\n<p>weakness with dorsiflexion of great toe and foot<br>pain radiated into lateral calf; numbness of dorsum of foot and lateral calf<br>screening exam: have patient walk on heels of foot<br>L4-L5 disk pathology<\/p>\n\n\n\n<p>weakness of plantar flexion of great toe and foot<br>pain along buttocks, lateral leg and lateral malleolus; numbness to lateral aspect of foot and in posterior calf<br>diminished\/absent Achilles reflex<br>screening exam: have patient walk on toes<br>L5-S1 disk pathology<\/p>\n\n\n\n<p>benign neuroma causing a compression neuropathy of an inter metatarsal plantar nerve, most commonly of the 3rd or 4th inter metatarsal spaces<br>high heels with closed toes<br>&#8220;standing on a pebble&#8221;<br>shooting pain affecting the contiguous halves of 2 toes<br>tingling\/numbness in the toes<br>morton&#8217;s neuroma<\/p>\n\n\n\n<p>inflammation of plantar fascia, the thick tissue on the bottom of the foot that connects the heel bone to toes and creates the arch of the foot<br>foot arch pain\/problems<br>common in runners<br>pain and stiffness in the bottom of the heel<br>pain worse in the am<br>plantar fasciitis<\/p>\n\n\n\n<p>rupture of growth plate at the tibial tuberosity as a result of stress on the patellar tendon<br>painful limp with pain below knee cap<br>one or both legs<br>pain worse with activities<br>swelling<br>Osgood-Schlatter disease<\/p>\n\n\n\n<p>management of Osgood-Schlatter disease<br>RICE<br>NSAIDs\/tylenol<br>rare cases- surgery referral<\/p>\n\n\n\n<p>an inflammatory disorder involving pain and stiffness in shoulder and usually the hip<br>over 50yo<br>may be associated with temporal arteritis<br>stiffness in neck, shoulders, and hips<br>loss of ROM in area<br>fatigue, anemia, mild fever<br>polymyalgia rheumatica<\/p>\n\n\n\n<p>What lab will be increased with polymyalgia rheumatica?<br>ESR<\/p>\n\n\n\n<p>management of polymyalgia rheumatica<br>corticosteroids<br>symptomatic treatment<\/p>\n\n\n\n<p>STDs<\/p>\n\n\n\n<p>What is the initial test for HIV?<br>ELISA<\/p>\n\n\n\n<p>What is the confirmatory test for HIV?<br>Western Blot<\/p>\n\n\n\n<p>What is the normal CD4 lymphocyte count?<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>800<\/p>\n<\/blockquote>\n\n\n\n<p>What is the CD4 count that is considered AIDS?<br>&lt;200<\/p>\n\n\n\n<p>What is the ideal viral load?<br>&lt;5000<\/p>\n\n\n\n<p>You want the <strong><em>_ to be high and the _<\/em><\/strong> to be low?<br>CD4<br>viral count<\/p>\n\n\n\n<p>What is the leading killer of AIDs patients?<br>Pneumocystis jirovecii (PCP pneumonia)<br>they take bactrim qd for this<\/p>\n\n\n\n<p>When does the CDC recommend AART be started?<br>CD4 350<\/p>\n\n\n\n<p>gram-negative bacillus<br>co-factor for HIV<br>painful, erythamatous base with halo<br>ulcers may be necrotic or severely erosive<br>chancroid<\/p>\n\n\n\n<p>the degree to which those who have a disease screen\/test positive<br>sensitivity<\/p>\n\n\n\n<p>the degree to which those who do not have a disease screen\/test negative<br>specificity<\/p>\n\n\n\n<p>if there is a question about sensitivity, you would mark hose who DO\/DO NOT have it<br>DO, remember +<\/p>\n\n\n\n<p>if there is a question about specificity, you would mark those who DO\/DO NOT have it<br>DO NOT, remember &#8211;<\/p>\n\n\n\n<p>Treatment of chancroid<br>1 gm azithromycin<br>or<br>250 mg IM ceftriaxone (rocephin)<br>or<br>Cipro 500 mg PO bid x3 days<\/p>\n\n\n\n<p>The 2 STDs where it hurts when they pee<br>Gonorrhea and chlamydia<\/p>\n\n\n\n<p>What 4 things cause dyspareunia in women?<br>chlamydia<br>PID<br>menopause<br>trich<\/p>\n\n\n\n<p>treatment for chlamydia<br>1gm azithromycin x1 dose<br>or<br>doxycycline 100mg bid x7 days<br>or<br>alternatives erythromycin, ofloxacin, levofloxacin<\/p>\n\n\n\n<p>treatment for gonorrhea<br>Ceftriaxone (rocephin) 250mg IM x1<br>plus give 1gm azithro PO x1 to cover chlamydia<\/p>\n\n\n\n<p>fever, malaise, dysuria, painful\/pruritic ulcers on genitalia<br>lasting for usually 12 days<br>initial herpes outbreak<\/p>\n\n\n\n<p>recurrence with less painful ulcers lasting for <em>__<\/em> days<br>5<\/p>\n\n\n\n<p>which vir drug especially useful for asymptomatic viral shedding of HSV 2<br>valacyclovir<\/p>\n\n\n\n<p>immunotypes L1, L2, or L3 of Chlamydia trachomatis<br>2-3 mm painless vesicle, bubo, or non-indurated ulcer<br>regional adenopathy<br>lymphogranuloma venereum (LGV)<\/p>\n\n\n\n<p>treatment of lymphogranuloma venereum (LGV)<br>Doxy 100 mg bid x21 days<br>aspirate buboes to prevent ulcerations<\/p>\n\n\n\n<p>lesions are 1-5mm smooth, rounded, firm, shiny flesh-colored to pearly-white papules<br>common on trunk and anogenial region<br>molluscum contagiosum<\/p>\n\n\n\n<p>treatment of molluscum<br>cryo with liquid nitro<\/p>\n\n\n\n<p>treatment of syphilis<br>benzathine PCN G 2.4 million units IM<br>PCN allegic- doxy 100 mg bid or erythro 500 mg qid<\/p>\n\n\n\n<p>GYN<\/p>\n\n\n\n<p>malodorous, frothy yellow-green discharge, pruritus, vaginal erythema, &#8220;strawberry patches&#8221; on cervix and vagina, dyspareunia, dysuria<br>trichomonas<\/p>\n\n\n\n<p>how to do wet-prep for trich?<br>Normal saline shows motile trichomonads<\/p>\n\n\n\n<p>watery, gray, fishy smelling discharge, vaginal spotting<br>BV<\/p>\n\n\n\n<p>thick, white, curd=like discharge, vulvovaginal erythema with pruritus<br>candidiasis<\/p>\n\n\n\n<p>Wet prep for BV?<br>normal saline shows clue cells<\/p>\n\n\n\n<p>Wet prep for candidiasis?<br>KOH mixture shows pseudo hyphae<\/p>\n\n\n\n<p>treatment of trich<br>metronidazole (Flagyl) 2gm PO x1 or 500mg bid x7days<\/p>\n\n\n\n<p>treatment of BV<br>metronidazole (Flagyl) 500mg bid x7 days or 2gm PO x1<br>gel 0.75% 5gm intravag bid x5 days<br>or<br>clindamycin (Cleocin) vag cream 2% 5gm qhs x 7days or 300mg bid x7days<\/p>\n\n\n\n<p>treatment of candidiasis<br>miconazole or clotrimazole 1 % 5gm vag qhs x7days<br>terconazole 80mg suppository qhs x3days<\/p>\n\n\n\n<p>the flu + gyn<br>+CMT<br>adnexal tenderness<br>abd tenderness<br>fever<br>dyspareunia<br>PID<\/p>\n\n\n\n<p>treatment of PID<br>page 260<\/p>\n\n\n\n<p>What are the 3 major concerns when considering hormonal therapy for menopausal symptoms?<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>breast cancer<\/li>\n\n\n\n<li>uterine cancer<\/li>\n\n\n\n<li>MI\/CAD risk- consider lipid panel, metabolic syndrome<\/li>\n<\/ol>\n\n\n\n<p>if not clear, refer<\/p>\n\n\n\n<p>DEXA<br>what is the normal T score?<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>-1.0 SD<\/p>\n<\/blockquote>\n\n\n\n<p>osteopenia T score<br>-1.0 to -2.5<\/p>\n\n\n\n<p>osteoporosis T score<br>below -2.5<\/p>\n\n\n\n<p>dietary sources of calcium<br>dairy, sardines, salmon w\/ bones, green leafy vegetables, tofu, calcium fortified foods, take vit D<\/p>\n\n\n\n<p>management of lower UTI<br>x3 days<br>TMP-SMZ (Bactrim)<br>Cipro<br>Amox w\/ clav (Augmentin)<\/p>\n\n\n\n<p>other considerations<br>amoxicillin, levofloxacin, nitrofurantoin (Macrobid, Macrodantin),<\/p>\n\n\n\n<p>UTI during pregnancy<br>x7-10 days<\/p>\n\n\n\n<p>amoxicillin<br>nitrofurantoin (Macrobid) until 36 weeks<br>cephalexin (Keflex)- never really used<\/p>\n\n\n\n<p>What is the black box warning about cipro?<br>achilles tendon rupture<\/p>\n\n\n\n<p>usually younger man<br>UTI symptoms<br>marked scrotal edema<br>usually chlamydia in &lt;35yo<br>epididymitis<\/p>\n\n\n\n<p>Positive Prehn&#8217;s sign<br>lift scrotum to symphysis pubis- pain relieved<\/p>\n\n\n\n<p>treatment for epididymitis &lt;35<br>Ceftriaxone (rocephin) 250 IM x1 and doxy (vibramycin) 100mg bid<br>or<br>azithro 1gm PO x1<\/p>\n\n\n\n<p>treatment for epididymitis &gt;35<br>TMP-SMZ (Bactrim) DS bid x10d<br>or<br>Cipro 250mg bid x10d<\/p>\n\n\n\n<p>UTI-like in man<br>usually E.Coli gram &#8211;<br>fever, chills, low back pain, dysuria, urgency\/frequency, nocturne<br>edematous prostate, may be warm, tender\/boggy to palpation, pain<br>acute bacterial prostatitis<\/p>\n\n\n\n<p>antibiotic choices for acute bacterial prostatitis<br>TMP-SMZ (bactrim)<br>or fluoroquinolones- levoflox, norflox, oflox<\/p>\n\n\n\n<p>s\/s<br>urgency\/frequency, nocturia, dribbling, retention<br>bladder distention<br>non-tender prostate symmetrical\/asymetrical enlargement<br>smooth, rubbery consistency<br>BPH<\/p>\n\n\n\n<p>2 med classes and 1 herb for BPH<br>alpha-blockers (relax muscles of bladder and prostate)- terazosin (hytrin), prazosin (minipress), tamsulosin (flomax)<br>5-alpha-reductatse inhibitors (shrink large prostates)- finasteride (proscar) and dutasteride (avodart)<br>Saw palmetto- effective for some<\/p>\n\n\n\n<p>Psychosocial<\/p>\n\n\n\n<p>difficulty with speech<br>aphasia<\/p>\n\n\n\n<p>inability to perform a previously learned task<br>apraxia<\/p>\n\n\n\n<p>inability to recognize an object<br>agnosia<\/p>\n\n\n\n<p>What are the meds for Alzheimer&#8217;s disease?<br>acetylcholinesterase inhibitors<br>donezepil (aricept)<br>galantamine (razadyne)<br>rivastigmine (exelon)<\/p>\n\n\n\n<p>vise-like or tight quality<br>usually generalized<br>intense about neck or back of head<br>no neuro symptoms<br>several hours<br>tension headache<\/p>\n\n\n\n<p>which cranial nerve associated with migraine?<br>trigeminal- V<\/p>\n\n\n\n<p>What would you use sumatriptan (imitrex) for?<br>migraine and cluster<\/p>\n\n\n\n<p>a degenerative disorder as a result of insufficient amounts of dopamine?<br>parkinson&#8217;s disease<\/p>\n\n\n\n<p>trio of finds for parkinson&#8217;s<br>tremor, rigidity, bradykinesia<\/p>\n\n\n\n<p>GI<\/p>\n\n\n\n<p>Anti-HAV, IgM<br>active Hep A<\/p>\n\n\n\n<p>Anti-HAV, IgG<br>recovered Hep A<\/p>\n\n\n\n<p>ABsAG, HBeAg, Anti-HBc, IgM<br>active Hep B<\/p>\n\n\n\n<p>ABsAG, Anti-HBc, IgM, IgG<br>chronic Hep B<\/p>\n\n\n\n<p>Anti-HBc, Anti-HBsAg<br>recovered Hep B<\/p>\n\n\n\n<p>Anti-HCV, HCV RNA<br>same in chronic and acute Hep C<\/p>\n\n\n\n<p>Cardiac<\/p>\n\n\n\n<p>What happens during S1?<br>mitral\/tricuspid (A\/V) valves close<\/p>\n\n\n\n<p>What happens during S2?<br>aortic\/pulmonic (semilunar) valves close<\/p>\n\n\n\n<p>What is systole?<br>period between S1 and S2<\/p>\n\n\n\n<p>What is diastole?<br>period between S2 and S1<\/p>\n\n\n\n<p>What does S3 mean?<br>Ken-tuck-y, increased fluid states (CHF, pregnancy)<\/p>\n\n\n\n<p>What does S4 mean?<br>Ten-ne-ssee, stiff ventricular wall (MI, left ventricular hypertrophy, chronic hypertension)<\/p>\n\n\n\n<p>Hematologic &amp; oncologic disorders<\/p>\n\n\n\n<p>Hct normal male\/female<br>40-54% M, 37-47% F<\/p>\n\n\n\n<p>TIBC- capacity to store iron<br>250-450<\/p>\n\n\n\n<p>Serum iron<br>50-150<\/p>\n\n\n\n<p>MCV- microcytic\/macrocytic SIZE normal<br>80-100<\/p>\n\n\n\n<p>MCH- expression of av amount &amp; weight of Hgb in a single RBC normal<br>26-34<\/p>\n\n\n\n<p>MCHC- hypochromic\/normochromic normal<br>32-36<\/p>\n\n\n\n<p>2 anemias with low MCV- small in size<br>IDA, thalassemia<\/p>\n\n\n\n<p>2 anemias with high MCV- big- megaloblastic<br>B12 or folate deficiency; also, alcohol, liver fx, drugs<\/p>\n\n\n\n<p>Normocytic anemia<br>anemia chronic disease, sickle cell, renal fx, blood loss, hemolysis<\/p>\n\n\n\n<p>Iron deficiency labs<br>Microcytic, hypochromic<br>Low Hgb, Hct, MCV, MCHC, RBCs, iron, ferritin<br>High TIBC<br>High RDW<\/p>\n\n\n\n<p>Management of IDA<br>ferrous sulfate 300-325mg qd-bid<\/p>\n\n\n\n<p>Foods high in iron<br>raisins, green leafy veggies, red meats, citrus, iron fortified breads\/cereals<\/p>\n\n\n\n<p>Thalassemia labs<br>Microcytic, hypochromic<br>Low Hbg, MCV, MCHC,<br>Normal TIBC, normal ferritin<\/p>\n\n\n\n<p>Thalassemia management<br>Usually nothing<br>RBC transfusion\/splenectomy if severe<br>iron contraindicated<\/p>\n\n\n\n<p>Folic acid def labs<br>Macrocytic, normochromic<br>Hct &amp; RBCs decreased<br>MCV increased<br>MCHC normal<br>Serum folate low<br>RBC folate &lt;100<\/p>\n\n\n\n<p>Folic acid def signs<br>fatigue etc.<br>Glossitis<br>NO neuro symptoms vs B12 does<\/p>\n\n\n\n<p>Treatment folic acid def<br>1mg folate qd<br>foods: bananas, peanut butter, fish, green leafy, iron fortified breads\/cereals<\/p>\n\n\n\n<p>Pernicious anemia labs<br>Hgb, Hct, RBCs decreased<br>MCV increased<br>Serum B12 &lt;0.1<br>Anti IF (intrinsic factor) and anti parietal cell antibody test<br>Schilling test for cause<\/p>\n\n\n\n<p>Pernicious anemia signs<br>glossitis, NEURO signs- paresthesia, loss vibratory sense, fine motor control, + ROMBERG, + BABINSKI<\/p>\n\n\n\n<p>Pernicious anemia treatment<br>B12 100 IM qd x1week<br>then q month for life<\/p>\n\n\n\n<p>anemia of chronic disease labs<br>normocytic, normochromic<br>Hgb, Hct low<br>MCV normal<br>MCHC normal<br>Serum iron &amp; TIBC low (capacity for more iron is low)<br>Ferritin high (stores of iron high)<\/p>\n\n\n\n<p>Anemia chronic disease treatment<br>Treat underlying disease<br>Nutritional support<\/p>\n\n\n\n<p>Sickle cell labs<br>Hgb low<br>peripheral smear shows sickled cells<br>cellulose acetate and citrate agar gel electrophoresis to confirm Hgb genotype<\/p>\n\n\n\n<p>Sickle cell crisis treatment<br>fluids, pain management<\/p>\n\n\n\n<p>leukemia definition<br>neoplasms arising from hematopoietic cells in bone marrow<\/p>\n\n\n\n<p>80% of acute leukemia in adults<br>remission 50-85%<br>long-term survival ~40%<br>Acute nonlymphocytic leukemia (ANL)\/ Acute myelogenous leukemia (AML)<\/p>\n\n\n\n<p>more difficult to cure in adults than children (90% remission in children)<br>pancytopenia with circulating blasts is hallmark of disease (all lab values decreased)<br>Acute lymphocytic leukemia (ALL)<\/p>\n\n\n\n<p>most common leukemia in adults<br>occurs in middle age and old age<br>median survival 10 years<br>lymphocytosis (large increase 42K) is hallmark of disease<br>Chronic lymphocytic leukemia (CLL)<\/p>\n\n\n\n<p>occurs in individuals 40 and older<br>median survival 3-4 years<br>Philadelphia chromosome seen in leukemic cells is hallmark of disease<br>Chronic myelogenous leukemia (CML)<\/p>\n\n\n\n<p>S\/S leukemia<br>asymptomatic<br>fatigue, weakness, anorexia<br>generalized lymphadenopathy<br>weight loss<\/p>\n\n\n\n<p>Labs leukemia<br>Subnormal RBCs and neutrophils<br>Elevated ESR<br>Bone marrow req for confirmation<\/p>\n\n\n\n<p>Management of leukemia<br>chemo<br>bone marrow transplant<\/p>\n\n\n\n<p>Non-Hodgkin&#8217;s lymphoma<br>unknown etiology, may be viral<br>lymphadenopathy<br>common 20-40<br>diffuse small b cells found<\/p>\n\n\n\n<p>Hodgkin&#8217;s desease<br>males, 32 years old<br>cause unknown<br>cervical adenopathy, spreads predictably along lymph groups<br>Reed-Sternberg cells<\/p>\n\n\n\n<p>Lymphoma management<br>radiation<br>chemo<br>sometimes bone marrow transplant<\/p>\n\n\n\n<p>Psychosocial disorders<\/p>\n\n\n\n<p>Depression- In Sad Cages<br>Interest- loss of pleasure<br>Sleep disturbances<br>Appetite changes<br>Depressed mood<br>Concentration difficulty<br>Activity- agitation\/retardation<br>Guilt feelings or low self-esteem<br>Energy loss<br>Suicidal ideation<\/p>\n\n\n\n<p>Alcoholism- Cages questionnaire<br>C- have you ever felt the need to cut down on your drinking?<br>A- have people annoyed you by criticizing your drinking?<br>G- have you ever felt guilty about your drinking?<br>E- have you ever had a drink first thing in the morning to steady your nerves or get rid of a hangover? (eye-opener)<\/p>\n\n\n\n<p>cause of Alzheimer&#8217;s<br>acetylcholine deficiency<\/p>\n\n\n\n<p>meds of Alzheimer&#8217;s<br>acetylcholinesterase inhibitors<br>donezepil (Aricept)<br>galantamine (Razadyne)<br>rivastigmine (Exelon)<br>often prescribed in conjunction with NMDA receptor antagonists such as memantine (Namenda) to improve thinking and activities of daily living<\/p>\n\n\n\n<p>Cognitive defect definitions<\/p>\n\n\n\n<p>test for object recognition<br>recognize a paperclip in the palm<br>steriognosis<\/p>\n\n\n\n<p>draw 6 in palm<br>gravisthesia<\/p>\n\n\n\n<p>aphasia<br>difficulty with speech<\/p>\n\n\n\n<p>apraxia<br>inability to perform a previously learned task<\/p>\n\n\n\n<p>agnosia<br>inability to recognize an object<\/p>\n\n\n\n<p>Cranial nerves mnemonic<br>On Some<br>Old Say<br>Olympus Marry<br>Towering Money<br>Tops But<br>A My<br>Fin Brother<br>And Says<br>German Big<br>Viewed Bras<br>Some Matter<br>Hops Most<\/p>\n\n\n\n<p>CN 7- facial. Which disease?<br>Bell&#8217;s palsy<\/p>\n\n\n\n<p>Entirely sensory<br>1, 2, &amp; 8<\/p>\n\n\n\n<p>Move the eyes<br>3, 4, &amp; 6<\/p>\n\n\n\n<p>CN associated with migraine<br>5-trigeminal<\/p>\n\n\n\n<p>Meds for daily prophylactic therapy of migraines occurring more than 2-3x per month<br>amitriptyline (Elavil)<br>dialproex (Depakote)<br>propranolol (inderal)<br>imipramine (tofranil)<br>clonidine (catapres)<br>verapamil (Calan)<br>topiramate (Topamax)<br>gabapentin (neurontin)<br>methysergide (sansert)<br>magnesium<\/p>\n\n\n\n<p>Meds for acute migraine<br>ASA<br>Sumatriptan (Imitrex) 5mg SQ at onset, repeat in 1h, total of 3x per day<\/p>\n\n\n\n<p>What 2 types of HA do you use sumatriptan (Imitrex) for?<br>migraine<br>cluster<\/p>\n\n\n\n<p>sign of stroke w\/ altered vision- ipsilateral monocular blindness<br>amaurosis fugax<\/p>\n\n\n\n<h1 class=\"wp-block-heading\">1 cause of heart failure<\/h1>\n\n\n\n<p>HTN<\/p>\n\n\n\n<p>Simple partial seizure<br>common w\/ cerebral lesions<br>NO loss of consciousness<br>rarely &gt;1min<br>motor symptoms in one muscle group, spread to entire side of body<br>paresthesias, flashing lights, vocalizations, hallucinations<\/p>\n\n\n\n<p>Complex partial seizure<br>simple partial followed by impaired level of consciousness<br>may have aura, staring, or automatisms such as lip smacking and picking at clothing<\/p>\n\n\n\n<p>absence (petite mal)<br>arrest of motor activity w\/ blank stare<\/p>\n\n\n\n<p>tonic-clonic (grand mal)<br>may have aura<br>Contractions (tonic), loss of consciousnes, then clonic (maintained involuntary contraction of muscle)<br>last 2-5 min<br>incontinence may happen<br>postictal period<\/p>\n\n\n\n<p>status epilepticus<br>medical emergency<br>series of grand mal &gt;10min<br>don&#8217;t gain consciousness between attacks<br>most uncommon, but life-threatening<\/p>\n\n\n\n<p>Labs\/diagnostics<br>EEG- seizure classification<br>CT- for new onset seizures<\/p>\n\n\n\n<p>Management of seizures<br>supportive<br>protect from injuries, nothing in airway<br>Benzos- Diazepam (valium) or lorazepam (ativan)<br>Phenytoin (dilantin)<br>Fosphenytoin (cerebyx) prodrug of dilantin<br>Phenobarb (luminal) if phenytoin unresponsive<\/p>\n\n\n\n<p>Seizure prevention meds<br>Carbamazepine (tegretol)<br>phenytoin (dilantin)<br>phenobarb (luminal)<br>valproic acid (depakene)<br>primidone (mysoline)<br>clonazepam (klonopin)<\/p>\n\n\n\n<p>Parkinson&#8217;s definition<br>degenerative disorder as a result of insufficient amounts of dopamine in the body<\/p>\n\n\n\n<p>trio of findings in Parkinson&#8217;s<br>tremor<br>rigidity<br>bradykinesia<\/p>\n\n\n\n<p>2 med classes in Parkinson&#8217;s<\/p>\n\n\n\n<ol class=\"wp-block-list\">\n<li>increase dopamine- Carbidopa-levodopa, amantadine, tolcapone, pramipexole<\/li>\n\n\n\n<li>anticholinergics tremors\/rigid- benztropine, trihexyphenidyl<\/li>\n<\/ol>\n\n\n\n<p>Myasthenia gravis<br>autoimmune, reduction of number of acetylcholine receptor sites at the neuromuscular junction<br>weakness worse after exercise, better after rest<\/p>\n\n\n\n<p>Myasthenia gravis s\/s<br>***ptosis<br>diplopia, dysarthria, dysphagia, extremity weakness<br>sensory modalities and DTRs normal<\/p>\n\n\n\n<p>Myasthenia gravis labs<br>antibodies to acetylcholine receptors<br>edrophonium (Tensilon) test to differentiate a myasthenic vs. cholinergic crisis<\/p>\n\n\n\n<p>Myasthenia management<br>anti cholinesterase drugs- pyridostigmine<br>immunosuppressives<br>plasmaphoresis<br>ventilator support<\/p>\n\n\n\n<p>MS def<br>autoimmune, attacks myelin<\/p>\n\n\n\n<p>MS s\/s<br>weakness, numbness, tingling, unsteadiness of limb<br>spastic paraparesis<br>diplopia<\/p>\n\n\n\n<p>MS findings<br>Mild lymphocytosis common<br>elevated protein in CSF, CSF IgG<br>MRI<\/p>\n\n\n\n<p>MS management<br>steroids for acute<br>antispasmodics<br>***interferon therapy<br>immunosuppressive therapy<br>plasmapheresis<\/p>\n\n\n\n<p>Bell&#8217;s CN<br>CN 7, usually right side of face<\/p>\n\n\n\n<p>Bell&#8217;s treatment<br>Prednisone taper 7-10 days<br>Acyclovir<br>eye drops and patch if can&#8217;t close eyes<\/p>\n\n\n\n<p>Trigeminal neuralgia<br>stabbing, electric shock in part of face<\/p>\n\n\n\n<p>Trigeminal neuraligia management<br>anti-seizure drugs<br>muscle relaxants<br>TCAs<\/p>\n\n\n\n<p>GI<\/p>\n\n\n\n<p>murphy&#8217;s sign<br>cholecystis<\/p>\n\n\n\n<p>diverticulitis<br>female, older, LLQ px<\/p>\n\n\n\n<p>gnawing epigastric pain<br>epigastric tenderness<\/p>\n\n\n\n<p>H. pylori<br>NSAIDs, ASA, glucocorticoids<br>smokers<br>stress<br>PUD<\/p>\n\n\n\n<p>What kind of ulcers in older 55-65y?<br>gastric<\/p>\n\n\n\n<p>What kind of ulcers in younger 30-55y?<br>duodenal<\/p>\n\n\n\n<p>relief of pain with eating<br>duodenal ulcer<\/p>\n\n\n\n<p>more pain eating<br>gastric ulcer<\/p>\n\n\n\n<p>1st line PUD<br>H2 receptor antagonist<br>$4 drug list<br>cimetidine (tagamet) 800mg qhs<br>ranitidine (zantac) 300mg qhs<br>famotidine (pepcid) 40mg qhs<br>nizatidine (axid) 300mg qhs<\/p>\n\n\n\n<p>2nd consideration for PUD- diagnosed officially<br>PPIs<br>30 min before meals<br>lansoprazole (prevacid)<br>rabeprazole (aciphex)<br>pantoprazole (Protonix)<br>omeprazole (prilosec)<br>dexlansoprazole (dexilant)<br>esomeprazole (nexium)<\/p>\n\n\n\n<p>basic combo therapy for H. pylori eradication<br>2 antibiotics + PPI or bismuth<\/p>\n\n\n\n<p>pain with right thigh extension<br>Psoa&#8217;s sign<\/p>\n\n\n\n<p>pain with internal rotation of flexed right thigh<br>obturator sign<\/p>\n\n\n\n<p>RLQ pain when pressure applied to LLQ<br>positive Rovsing&#8217;s sign<\/p>\n\n\n\n<p>Lower respiratory<\/p>\n\n\n\n<p>normal percussion sound of chest<br>resonance<\/p>\n\n\n\n<p>asthma percussion<br>hyperresonance<\/p>\n\n\n\n<p>mainstay of COPD treatment (chronic bronchitis and emphysema)<br>inhaled anticholinergic- ipratropium bromide (Atrovent)<\/p>\n\n\n\n<p>most common bug in CAP<br>Strep pneumoniae<\/p>\n\n\n\n<p>treatment of CAP<br>healthy, young- macrolide (azithromycin), clarithromycin (Biaxin), erythromycin, or doxycycline<\/p>\n\n\n\n<p>unhealthy, old- fluoroquinolone- levofloxacin (Levaquin), gemifloxacin (Factive), or moxifloxacin (Avelox)<\/p>\n\n\n\n<p>Cardiovascular<\/p>\n\n\n\n<p>ST segment changes with angina vs MI<br>Angina- down sloping\/depression of ST<br>MI- ST elevation<\/p>\n\n\n\n<p>Total cholesterol normal<br>&lt;200<\/p>\n\n\n\n<p>Trigylcerides<br>&lt;150<\/p>\n\n\n\n<p>HDL<\/p>\n\n\n\n<blockquote class=\"wp-block-quote is-layout-flow wp-block-quote-is-layout-flow\">\n<p>40<\/p>\n<\/blockquote>\n\n\n\n<p>LDL<br>Gen pop &lt;130<br>2+ risks &lt;100<br>diabetic, previous MI &lt;70<\/p>\n\n\n\n<p>3 common med categories for the outpatient treatment of angina<br>Nitrates<br>Beta blockers<br>CCBs<\/p>\n\n\n\n<p>Meds for hyperlipidemia<br>Statin<br>Niacin if not controlled<br>If still trouble, time for fibrate (gimfibrazol)<\/p>\n\n\n\n<p>Extra heart sound common with infarct<br>S4<\/p>\n\n\n\n<p>ECG changes with MI<br>Peaked t waves, ST elevation, Q waves<\/p>\n\n\n\n<p>Leads I, aVL<br>lateral MI<\/p>\n\n\n\n<p>II, III, aVF<br>inferior MI- bottom of heart dying<\/p>\n\n\n\n<p>V leads or V3 and V4<br>anterior MI<\/p>\n\n\n\n<p>Cardiac enzymes<br>CK-MB, Troponin I are 100% cardioselective<br>then Troponin T<\/p>\n\n\n\n<p>Hx CAD, 68yoM, hyperlipidemia, smokes 2ppd, diabetic, calf hurts, numbness to toes, pain<br>Exam: shiny, hairless skin, w\/ dependent rubor, and elevational pallor, ulcerations<br>Doppler, ABI, arteriography<br>-stop smoking, exercise<br>-Pentoxifylline, cilostazol<br>PVD-atherosclerotic narrowing<\/p>\n\n\n\n<p>Woman, maybe genetic, aching of LE&#8217;s, relieved by elevation, edema when up on feet, night cramps, trophic changes, brown blotches, dermatitis<br>use stockings, bed rest w\/ elevated legs<br>CVI- impaired venous return<\/p>\n\n\n\n<p>low pitched rumble during 1st heart sound<br>mitral stenosis<\/p>\n\n\n\n<p>EENT<\/p>\n\n\n\n<p>What does it mean if the cup is more that 1\/2 the size of the disc diameter?<br>glaucoma<\/p>\n\n\n\n<p>retinal vessels<br>arteries vs veins<br>arteries brighter and narrower<br>A:V ratio 2:3 or 4:5<\/p>\n\n\n\n<p>raised area where A &amp; V meet<br>nicking = htn<\/p>\n\n\n\n<p>hyperopia<br>farsighted = old people<\/p>\n\n\n\n<p>myopia<br>nearsighted = young people<\/p>\n\n\n\n<p>presbyopia<br>common after 40, difficult maintain clear focus at a near distance due to decr. flexibility of lens and weakening of the ciliary muscles<\/p>\n\n\n\n<p>arcus senilis<br>cloudy appearance of the cornea with a gray\/white arc or circle around the limbus due to deposition of lipid material = hyperlipidemia<\/p>\n\n\n\n<p>hordeolum (stye)<br>infection, S. aureus<br>pain and erythema<br>warm compress<br>topical bacitracin or erythromycin<\/p>\n\n\n\n<p>chalazion<br>beady nodule on eye lid<br>infection or retention cyst of a meibomian gland<br>painless aside from swelling<br>warm compress &amp; surgery referral<\/p>\n\n\n\n<p>blepharitis<br>Staph infection or seborrheic dermatitis of the the lid edge<\/p>\n\n\n\n<p>eyes itching, burning, redness, tearing, blurred vision, swelling of eyelids, foreign body sensation<br>NO PAIN<br>conjunctivitis<\/p>\n\n\n\n<p>Increased intraocular pressure<br>glaucoma<\/p>\n\n\n\n<p>asymptomatic<br>elevated IOP<br>cupping of the disc<br>constriction of visual fields- decr visual fields<br>open-angle<\/p>\n\n\n\n<p>acute<br>extreme pain, blurred vision, halos around lights, pupil dilated\/fixed<br>closed-angle<\/p>\n\n\n\n<p>clouding and opacification of the normally clear lens of the eye<br>cataracts<\/p>\n","protected":false},"excerpt":{"rendered":"<p>You have been treating Jason, 35 y\/o M, for HIV infection, and the course of his treatment requires that he be hospitalized for a few days. His medical bill is paid by his Aunt, Sylvia, who wants to know why her nephew was in the hospital, but Jason is not willing to share that information. [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"default","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","ast-disable-related-posts":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"tablet":{"background-color":"","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"mobile":{"background-color":"","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""}},"ast-content-background-meta":{"desktop":{"background-color":"var(--ast-global-color-5)","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"tablet":{"background-color":"var(--ast-global-color-5)","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"mobile":{"background-color":"var(--ast-global-color-5)","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""}},"footnotes":""},"categories":[],"tags":[],"class_list":["post-111191","post","type-post","status-publish","format-standard","hentry"],"_links":{"self":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/posts\/111191","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/comments?post=111191"}],"version-history":[{"count":0,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/posts\/111191\/revisions"}],"wp:attachment":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/media?parent=111191"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/categories?post=111191"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/tags?post=111191"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}