{"id":130487,"date":"2023-12-18T05:41:04","date_gmt":"2023-12-18T05:41:04","guid":{"rendered":"https:\/\/learnexams.com\/blog\/?p=130487"},"modified":"2023-12-18T05:41:07","modified_gmt":"2023-12-18T05:41:07","slug":"final-exam-nur631-nur-631-latest-2023-2024-update-advanced-physiology-and-pathophysiology-study-guide-questions-and-verified-answers-100-correct-gcu","status":"publish","type":"post","link":"https:\/\/www.learnexams.com\/blog\/2023\/12\/18\/final-exam-nur631-nur-631-latest-2023-2024-update-advanced-physiology-and-pathophysiology-study-guide-questions-and-verified-answers-100-correct-gcu\/","title":{"rendered":"Final Exam: NUR631\/ NUR 631 (Latest 2023\/2024 Update) Advanced Physiology and Pathophysiology Study Guide| Questions and Verified Answers| 100% Correct \u2013 GCU"},"content":{"rendered":"\n<figure class=\"wp-block-image size-large\"><a href=\"https:\/\/learnexams.com\/search\/study?query=\" target=\"_blank\" rel=\"noopener\"><img decoding=\"async\" src=\"https:\/\/learnexams.com\/blog\/wp-content\/uploads\/2023\/12\/FINAL-EXAM-NUR631-NUR-631-LATEST-20232024-UPDATE-ADVANCED-PHYSIOLOGY-AND-PATHOPHYSIOLOGY-STUDY-GUIDE-QUESTIONS-AND-VERIFIED-ANSWERS-100-CORRECT-GCU-725x1024.png\" alt=\"Final Exam: NUR631\/ NUR 631 (Latest 2023\/2024 Update) Advanced Physiology and Pathophysiology Study Guide| Questions and Verified Answers| 100% Correct \u2013 GCU\" class=\"wp-image-130488\"\/><\/a><\/figure>\n\n\n\n<p>Final Exam: NUR631\/ NUR 631 (Latest 2023\/2024 Update) Advanced Physiology and Pathophysiology Study Guide| Questions and Verified Answers| 100% Correct \u2013 GCU<\/p>\n\n\n\n<p>Final Exam: NUR631\/ NUR 631 (Latest<br>2023\/2024 Update) Advanced Physiology and<br>Pathophysiology Study Guide| Questions and<br>Verified Answers| 100% Correct \u2013 GCU<br>Q: Patient comes in with chronic wounds that aren&#8217;t healing. What are you thinking?<br>Answer:<br>-Diabetes<br>-Something else is going on with those patient\/other comorbidities<br>Q: If a newborn doesn&#8217;t have enough collectin-like protein what kind of infec- tion might they<br>develop?<br>Answer:<br>-Pneumonia<br>-Anything RESPIRATORY related<br>Q: Where do B lymphocytes grow up and develop<br>Answer:<br>Bone marrow<br>Q: I go over to a friend&#8217;s house to get chickenpox. What type of immunity is that?<br>Answer:<br>Active acquired immunity\/Body will make antibodies<br>Q: What is happening at a cellular level with a type two sensitivity reaction?-<\/p>\n\n\n\n<p>Answer:<br>-Antibodies are attaching to the cellular surface of the antigen.<br>-release of histamines and IgE<br>Q: What does Rhogram do?<br>Answer:<br>-Stops hemolytic anemia<br>-Give to Rh &#8211; moms with a Rh + baby<br>Q: If we have a patient that went under some organ transplant, why would we have tissue<br>damage?<br>Answer:<br>Start to see TH1 cells release too many cytokines so we will see the cytotoxic effects. Cytokines<br>will actually attack the endothelial cells.<br>Q: What is an exotoxin?<br>Answer:<br>When bacteria is growing they release exotoxins.<br>Q: What do we have that helps us fight fungal infections?<br>Answer:<br>-Phagocytes<br>-T-Lymphocytes<br>Q: Why, if I&#8217;m really stressed, am I more likely to get sick?<br>Answer:<br>Stress releases cortisol, cortisol increases, helper T cells are suppressed.<\/p>\n\n\n\n<p>Q: Stressed patient (long period of time), what can we see develop with lab results?<br>Answer:<br>-Hypoglycemia<br>-Cortisol increase causes hyperglycemia initially, but with chronic stress cortisol levels become<br>depleted causing hypoglycemia. (Adrenal insufficiency)<br>Q: What happens when we have cellular metabolism that just isn&#8217;t working right? What will<br>develop?<br>Answer:<br>Build up of waste<br>Q: What is the least likely IL to cause endothelial cells to go into that proin- flammatory state?<br>Answer:<br>IL4<br>Q: If you had a patient that developed MODS, what substances stimulate the normal endothelial<br>cells to go into a proinflammatory state?<br>Answer:<br>Interleukins tumor necrosis factor<br>IL-6<br>Q: How dehydrated does a child have to be before we see them have low<br>BP<br>Answer:<br>10%<\/p>\n\n\n\n<p>Q: Why a patient can develop a reperfusion injury.<br>Answer:<br>Once we&#8217;ve had oxygen cut off for a while we can actually see damage happen from that<br>reexposure to oxygen<br>Q: Why do burn patients go into kidney failure?<br>Answer:<br>Damaged muscles produce<br>Myoglobin<br>Myoglobin is huge and hard for kidneys to filter out. Not meant to filter it.<br>Q: What type of cancer do we recommend exercise to decrease prevalence?-<br>Answer:<br>Colon CA<br>Q: In children, exposure to what virus develops a carcinogenic relationship?-<br>Answer:<br>EBV<br>Q: Risk factors of kids being exposed to and might develop CA due to (SE- LECT ALL THAT<br>APPLY)<br>Answer:<br>Ionizing radiation<br>Chemo<br>Cigarette smoke<br>EBV<br>Powered by <a href=\"https:\/\/learnexams.com\/search\/study?query=\" target=\"_blank\" rel=\"noopener\">https:\/\/learnexams.com\/search\/study?query=<\/a><\/p>\n\n\n\n<p>Where do we hold the DNA inside of a cell?<br>The nucleus<\/p>\n\n\n\n<p>What is cell suicide?<br>apoptosis<\/p>\n\n\n\n<p>What are some things we see clinically in a patient presenting with carbon monoxide poisoning?<br>-Normal SpO2<br>-Tissue damage<\/p>\n\n\n\n<p>What do we have: ABG w\/ pH 7.25, bicarb 28, CO2 is 60?<br>Respiratory acidosis<\/p>\n\n\n\n<p>How do we get molecules to between intracellular &amp; extra cellular?<br>Hydrostatic pressure<br>(If it&#8217;s water: osmotic pressure)<\/p>\n\n\n\n<p>If patient has large volume of vomiting, what would we see?<br>Metabolic Alkalosis<\/p>\n\n\n\n<p>Why does metabolic alkalosis develop from vomiting?<br>Losing acid, retaining Bicarb<\/p>\n\n\n\n<p>What do we look for on an amnio to see neural tube defects?<br>Alpha-fetal protein<\/p>\n\n\n\n<p>Clinical characteristics of Trisomy 21<br>Wide eyes<br>Wide neck<br>Short stature<br>Low IQ<br>Low nasal bridge<br>Low set ears<br>Cardiac defects<\/p>\n\n\n\n<p>How could a newborn have type 1 diabetes<br>Autoimmune\/ Cannot be prevented<\/p>\n\n\n\n<p>We have a certain gene that takes care of maintenance of other cells<br>Housekeeping genes<\/p>\n\n\n\n<p>Patient comes in with chronic wounds that aren&#8217;t healing. What are you thinking?<br>-Diabetes<br>-Something else is going on with those patient\/other comorbidities<\/p>\n\n\n\n<p>If a newborn doesn&#8217;t have enough collectin-like protein what kind of infection might they develop?<br>-Pneumonia<br>-Anything RESPIRATORY related<\/p>\n\n\n\n<p>Where do B lymphocytes grow up and develop<br>Bone marrow<\/p>\n\n\n\n<p>I go over to a friend&#8217;s house to get chickenpox. What type of immunity is that?<br>Active acquired immunity\/Body will make antibodies<\/p>\n\n\n\n<p>What is happening at a cellular level with a type two sensitivity reaction?<br>-Antibodies are attaching to the cellular surface of the antigen.<br>-release of histamines and IgE<\/p>\n\n\n\n<p>What does Rhogram do?<br>-Stops hemolytic anemia<br>-Give to Rh &#8211; moms with a Rh + baby<\/p>\n\n\n\n<p>If we have a patient that went under some organ transplant, why would we have tissue damage?<br>Start to see TH1 cells release too many cytokines so we will see the cytotoxic effects. Cytokines will actually attack the endothelial cells.<\/p>\n\n\n\n<p>What is an exotoxin?<br>When bacteria is growing they release exotoxins.<\/p>\n\n\n\n<p>What do we have that helps us fight fungal infections?<br>-Phagocytes<br>-T-Lymphocytes<\/p>\n\n\n\n<p>Why, if I&#8217;m really stressed, am I more likely to get sick?<br>Stress releases cortisol, cortisol increases, helper T cells are suppressed.<\/p>\n\n\n\n<p>Stressed patient (long period of time), what can we see develop with lab results?<br>-Hypoglycemia<br>-Cortisol increase causes hyperglycemia initially, but with chronic stress cortisol levels become depleted causing hypoglycemia. (Adrenal insufficiency)<\/p>\n\n\n\n<p>What happens when we have cellular metabolism that just isn&#8217;t working right? What will develop?<br>Build up of waste<\/p>\n\n\n\n<p>What is the least likely IL to cause endothelial cells to go into that proinflammatory state?<\/p>\n\n\n\n<p>IL4<\/p>\n\n\n\n<p>If you had a patient that developed MODS, what substances stimulate the normal endothelial cells to go into a proinflammatory state?<br>Interleukins<br>tumor necrosis factor<br>IL-6<\/p>\n\n\n\n<p>How dehydrated does a child have to be before we see them have low BP<br>10%<\/p>\n\n\n\n<p>Why a patient can develop a reperfusion injury.<br>Once we&#8217;ve had oxygen cut off for a while we can actually see damage happen from that reexposure to oxygen<\/p>\n\n\n\n<p>Why do burn patients go into kidney failure?<br>Damaged muscles produce Myoglobin<br>Myoglobin is huge and hard for kidneys to filter out. Not meant to filter it.<\/p>\n\n\n\n<p>What type of cancer do we recommend exercise to decrease prevalence?<br>Colon CA<\/p>\n\n\n\n<p>In children, exposure to what virus develops a carcinogenic relationship?<br>EBV<\/p>\n\n\n\n<p>Risk factors of kids being exposed to and might develop CA due to (SELECT ALL THAT APPLY):<br>Ionizing radiation<br>Chemo<br>Cigarette smoke<br>EBV<\/p>\n\n\n\n<p>What are the granulocytes that have those vasoactive amines like histamines, what are those granulocytes called?<br>Basophils<\/p>\n\n\n\n<p>Why would a patient develop pernicious anemia<br>can&#8217;t absorb Vitamin B-12<\/p>\n\n\n\n<p>What is aplastic anemia, what will develop?<br>Pancytopenia (low everything)<br>Low erythrocytes<br>Low leukocytes<br>Low thrombocytes<\/p>\n\n\n\n<p>What kind of inherited disorder is Hemophilia A<br>X linked recessive<\/p>\n\n\n\n<p>Why is Hemophilia A only seen in Men?<br>Because it is X-linked recessive<\/p>\n\n\n\n<p>Why will we see paresthesia develop in our b12 deficient patients?<br>-Myelin sheath degeneration\/damage<br>-Happens right in the spinal cord<\/p>\n\n\n\n<p>What is happening at the cellular level in a sickle cell patient? What happens in their DNA?<br>Valine replaces one of the amino acids (glutamate). That is what will sycle the cells.<\/p>\n\n\n\n<p>Where do we make oxytocin?<br>Posterior Pituitary<\/p>\n\n\n\n<p>What genetic material will translocate in CML(Chronic Myelogenous Leukemia)?<br>Genes 9 and 22 will abnormally fuse and create a protein called BCR-ABL-1<\/p>\n\n\n\n<p>Why do we give vasopressors to shock patients?<br>-Vasoconstriction though ADH<br>-Increase BP<\/p>\n\n\n\n<p>What regulates our calcium? Which hormone?<br>PTH (parathyroid hormone)<\/p>\n\n\n\n<p>If a patient came in with really high levels of ADH, what type of cancer would I suspect?<br>Small cell carcinoma of the stomach<\/p>\n\n\n\n<p>If you have a patient that comes in saying I haven&#8217;t gotten my period in 6 months, abnormal hair growth, osteoporosis, what tests should you run?<br>CT their head to make sure they don&#8217;t have a tumor<br>looking for a Pituitary Adenoma<\/p>\n\n\n\n<p>Why do Diabetics pee so much<br>Tying to get rid off all that glucose<\/p>\n\n\n\n<p>Why do we see so many of those microvascular complications in our diabetic patients, at a cellular level?<br>-Hyperplasia<br>-Basement membranes start to thicken<\/p>\n\n\n\n<p>Why do our diabetics need to get regular eye exams?<br>We will see retinopathy develop as a result of retinal ischemia<\/p>\n\n\n\n<p>S\/S of hypothyroidism (low and slow)<br>Constipation<br>Lethargy<br>Low heart rate<\/p>\n\n\n\n<p>Why are obese people at a higher risk for developing high blood pressure.<br>Make more angiotensin<\/p>\n\n\n\n<p>If you did a test where you poked me with the end of a safety pin and I couldn&#8217;t tell if it was sharp or dull, what part of my brain is affected?<br>Pons<\/p>\n\n\n\n<p>What part of my brain controls involuntary muscle control like my posture and balance?<br>Cerebellum<\/p>\n\n\n\n<p>If I smile and I only have half a smile which nerve is affected?<br>Facial nerve #7<\/p>\n\n\n\n<p>If I had an injury to my spinal cord and damaged upper motor nerves, what would you see?<br>Initially full paralysis, then gradual partial recovery<\/p>\n\n\n\n<p>Differentiation between upper and lower motor neuron injury<br>LOWER: Permanent paralysis<br>UPPER: Reversible paralysis<\/p>\n\n\n\n<p>What is it called that separates the cerebellum from its cerebrum?<br>Tentorium cerebelli<\/p>\n\n\n\n<p>If you had a defect in the arachnoid villi, what would you expect to be wrong with your patient?<br>Too little absorption of CSF<\/p>\n\n\n\n<p>What part of my brain allows me to focus my attention?<br>Pre-Frontal<\/p>\n\n\n\n<p>What kind of seizure: altered LOC, dreamlike trans (impaired).<br>Complex focal seizure<\/p>\n\n\n\n<p>If there is capillary permeability increase in the brain, what type of cerebral edema will we see?<br>Vasogenic cerebral permeability<\/p>\n\n\n\n<p>If a patient had a CVA and they weren&#8217;t able to feel the objects and unable to identify objects, what is that called?<br>Tactile agnosia<\/p>\n\n\n\n<p>What is the theory that regulates pain transmission from cns in and out of the spinal cord?<br>Gate control theory<\/p>\n\n\n\n<p>Who will need the most amount of pain meds: Rotator cuff injury, gall bladder, or CRUSH injury?<br>CRUSH because there is multiple systems involved.<\/p>\n\n\n\n<p>What kind of nerve fibers transmit pain?<br>Delta-A fibers<\/p>\n\n\n\n<p>Where do those pain neurons live in the spinal cord?<br>Dorsal root ganglia<\/p>\n\n\n\n<p>What kind of endogenous opioids do I have in myself<br>Endorphins<\/p>\n\n\n\n<p>System is affected from major depressive disorders<br>Hypothalmic Pituitary adrenal system (HPA)<\/p>\n\n\n\n<p>What receptor is impaired in depressed patients<br>Serotonin<\/p>\n\n\n\n<p>Patient has lithium toxicity, what electrolyte is affected?<br>Sodium (results in Hyponatremia)<\/p>\n\n\n\n<p>What type of inherited disorder is DMD (duchenne muscular disorder)<br>X-linked recessive<\/p>\n\n\n\n<p>What is the link between depression and cortisol secretion?<br>Cortisol increases&#8212;Causes inflammation&#8211;causes depression<\/p>\n\n\n\n<p>Normal PR interval<br>&lt;0.2 seconds<\/p>\n\n\n\n<p>What does the Pericardium do?<br>encloses the heart for protection<\/p>\n\n\n\n<p>Papillary muscles within the heart function as:<br>-Help close valves<br>-Prevent backward expulsion of the AV valves<\/p>\n\n\n\n<p>Cardiac cycle, issue in phase 0, what electrolyte imbalance do I expect to see?<br>Sodium &#8211; LOW &#8211; hyponatremia<\/p>\n\n\n\n<p>Raynaud&#8217;s disease<br>-Vasospasms, seen mostly in fingers<br>-lack of blood flow<\/p>\n\n\n\n<p>My patient comes into the ER \u2105 chest pain\u2026what labs am I ordering?<br>Troponin<br>BNP<\/p>\n\n\n\n<p>Patient c\/o chest pain and the troponin is normal\u2026what&#8217;s the next step?<br>Serial troponins due to possible delayed elevation<\/p>\n\n\n\n<p>If I see a pulseless paradoxus, what are some differentials?<br>Pericardial effusion, because there is low blood volume in all 4 chambers<\/p>\n\n\n\n<p>A patient has COPD and we see increased pulmonary vascular resistance due to what type of heart failure?<br>Right sided heart failure<\/p>\n\n\n\n<p>Most common cardiac complication of Down syndrome<br>Ventricle septal defect<\/p>\n\n\n\n<p>What does a crescendo\/decrescendo systolic ejection murmur sound like?<br>It&#8217;s going to be specific to the 2nd heart sound.<br>along the left sternal border\/2nd-3rd intercostal space<\/p>\n\n\n\n<p>What defect commonly has crescendo\/decrescendo systolic ejection murmur?<br>Atrial septal defects<\/p>\n\n\n\n<p>How does conjugation of bilirubin occur?<br>-fat soluble becomes water soluble<br>-unconjugated bilirubin becomes conjugated bilirubin<\/p>\n\n\n\n<p>What do you want to look for if a patient comes in, zero GI complaints but has projectile vomiting all over the place?<br>-Increased intercranial pressure<br>-Something in the brain<br>CT needed<\/p>\n\n\n\n<p>If I have a lactase deficiency, what kind of diarrhea will I develop?<br>Watery\/osmotic<\/p>\n\n\n\n<p>How do I document bright red bleeding from the rectum?<br>Hematochezia<\/p>\n\n\n\n<p>S\/Sx of a small bowel obstruction<br>-Abdominal distention\/fullness (early sign)<br>-Nausea\/vomiting (late signs)<\/p>\n\n\n\n<p>What bacteria runs rampant in daycares?<br>Impetigo<\/p>\n\n\n\n<p>What disorder do we generally see with hepatic fat accumulation in the liver?<br>Alcoholic cirrhosis<\/p>\n\n\n\n<p>What are the early s\/sx of hepatitis (prodromal)<br>-Fatigue<br>-Vomiting<br>-hyper Algeria (overall nerve pain)<\/p>\n\n\n\n<p>What is it called when we don&#8217;t have complete fusion of the Nadine dial and the inner maxillary process?<br>Cleft lip\/palate<\/p>\n\n\n\n<p>Why will we see physiological jaundice in a newborn?<br>Immature liver<br>Cannot get rid of the bilirubin<br>Resulting in hyperbilirubinemia<\/p>\n\n\n\n<p>What does Surfactant do?<br>Reduces surface tension<br>Keeps Alveoli open<\/p>\n\n\n\n<p>Why would you see clubbing with respiratory conditions?<br>Chronic hypoxic state<\/p>\n\n\n\n<p>If you had a patient that had long term COPD (chronically hypoxia), what are some physical signs of COPD?<br>-Barrel chested<br>-clubbed nails<br>-SOB on exertion<br>-Cyanosis<br>-Frothy cough<\/p>\n\n\n\n<p>If a patient has a pleural effusion and we find that they have transudative exudate but not very many symptoms, what do I want to look for?<br>Protein level (lab draw)<\/p>\n\n\n\n<p>Bronchiectasis that causes constriction and dilation in the bronchi.<br>Varicose bronchiectasis<\/p>\n\n\n\n<p>If I have a patient that is diagnosed with ARDS (acute respiratory distress syndrome), what am I going to be on high alert for them also developing?<br>Pneumonia<\/p>\n\n\n\n<p>What if I diagnose a patient with emphysema but they say they&#8217;ve never smoked?<br>Check to see if there&#8217;s a genetic predisposition to emphysema<\/p>\n\n\n\n<p>What is it called if there is fluid in the pleural space?<br>Pleural Effusion<\/p>\n\n\n\n<p>Working a patient up for Pulmonary HTN, what are some findings?<br>SOB with activity<br>JVD<br>Peripheral edema<\/p>\n\n\n\n<p>Why do I worry about a young infant that has nasal congestion?<br>They&#8217;re nose breathers; can&#8217;t eat if they can&#8217;t breathe<\/p>\n\n\n\n<p>Why do my cystic Fibrosis patients have all of that mucous plugging?<br>-Too much defective chloride secretion<br>-Too much sodium absorption<\/p>\n\n\n\n<p>Why do we care about natriuretic peptides? What does it tell us?<br>It affects the renin and aldosterone<\/p>\n\n\n\n<p>If we have a patient with heart failure, what&#8217;s happening to the renin &amp; aldosterone?<br>The natriuretic peptides are going to inhibit the renin &amp; aldosterone<\/p>\n\n\n\n<p>What do we test if I want to know a patient&#8217;s renal function?<br>GFR<\/p>\n\n\n\n<p>How does potassium (K+) and magnesium (Mg+) affect formation of kidney stones?<br>They affect crystal growth<br>will stop stones from forming<\/p>\n\n\n\n<p>What am I looking for on an urinalysis to differentiate between pyelonephritis and cystitis?<br>White blood cell casts (will be present with pyelo)<\/p>\n\n\n\n<p>What change in the labs for kidneys that is normal for an older person?<br>UA- Specific gravity is going to be higher in an elderly person. This is normal process.<\/p>\n\n\n\n<p>If I have a patient w\/ blood in their urine, + RBC casts, excessive proteinuria\u2026What do I want to do next?<br>Renal biopsy<br>Suspecting Glomerular nephritis<\/p>\n\n\n\n<p>Why will patients who have glomerulonephritis get soda-colored urine?<br>All the RBCs have not broken down<\/p>\n\n\n\n<p>What part of the kidney does the IgG affect when we&#8217;ve got certain nephropathies like glomerulonephritis? Your IgG is going to deposit itself where?<br>Basement membrane<br>IgG is heavy and sinks to bottom<\/p>\n\n\n\n<p>Why will we see urine reflux in children? Why is it more common?<br>It&#8217;s an anatomy thing; the ureters are shorter in children<\/p>\n\n\n\n<p>If I have a urinary obstruction and it&#8217;s due to a spinal cord injury, what do we call that?<br>Neurogenic bladder<\/p>\n\n\n\n<p>Where is an obstructed bladder coming from?<br>Any kind of obstruction:<br>Prerenal<br>intrarenal<br>postrenal<\/p>\n\n\n\n<p>If I hurt my shoulder, what&#8217;s the part of my shoulder that I want to avoid hurting because it&#8217;s going to regenerate the most slowly and heal the least?<br>Articular cartilage<\/p>\n\n\n\n<p>What glycoprotein do we have that&#8217;s going to move around our hormones and other metabolites so that our bone cells can form properly?<br>Bone Albumin<\/p>\n\n\n\n<p>What cells are going to maintain our bone matrix for us?<br>Osteocytes<\/p>\n\n\n\n<p>What is a pathological fracture?<br>Fracture not due to mechanical stress\/insult\/injury<\/p>\n\n\n\n<p>What do you want to check on a patient that was found by neighbors, saying that she&#8217;d been laying on the floor for several hours?<br>CK level (this checks for muscle breakdown)<br>May develop Rhabdomyolysis<\/p>\n\n\n\n<p>Why do women develop osteoporosis more than men?<br>Women have a higher oxidative stress and higher intracellular reactive oxygen.<\/p>\n\n\n\n<p>What is not working properly in a person that has Paget&#8217;s disease?<br>Problems with bone resorption and bone formation\/too much\/too fast<\/p>\n\n\n\n<p>If I saw on an Xray that the radiologist read something sequestrum, what does that mean?<br>Devascularized, devitalized bone on Xray<\/p>\n\n\n\n<p>What is ankylosing spondylitis?<br>Chronic inflammation that causes the spine and SI joints to just start fusing and stiffening<\/p>\n\n\n\n<p>What is it called on a newborn when I go to rotate their hips and the hips are going to stay in contact w\/ the acetabulum but it is not going to be seated very well?<br>Subluxation<\/p>\n\n\n\n<p>If I have a patient that their skin when they hurt themselves it becomes very elevated and rounded, what is it called?<br>Keloid skin<\/p>\n\n\n\n<p>Tell me 2 things an allergic contact dermatitis and stasis dermatitis have in common:<br>-Inflammation of the skin (redness)<br>-Itching<\/p>\n\n\n\n<p>What do I call the skin lesions that are thick, silvery, and scaly?<br>(Plaque) psoriasis<\/p>\n\n\n\n<p>What do I call it if I have a big collection of infected hair follicles?<br>Carbuncle<\/p>\n\n\n\n<p>What do I call it if I have one infected hair?<br>Gruncle<\/p>\n\n\n\n<p>If you had chicken pox early on in life, what do you need to educate your patient that they can develop later on?<br>Herpes Zoster (shingles)<\/p>\n\n\n\n<p>Describe what tinea corporis (ring worm) looks like?<br>Red, circular lesion; scaly<\/p>\n\n\n\n<p>What is Kaposi sarcoma<br>Vascular malignancy<\/p>\n\n\n\n<p>What disorder is kaposi sarcoma associated with?<br>HIV\/AIDS<\/p>\n\n\n\n<p>Where will we see the kaposi sarcoma be located at usually?<br>In the legs<\/p>\n\n\n\n<p>What is kaposi sarcoma associated with?<br>Almost always going to be related to some kind of drug-induced immunosuppression<\/p>\n\n\n\n<p>Where would you see an onychomycosis?<br>A nail bed<br>nail fungus<\/p>\n\n\n\n<p>If I have a history of PID and now I come in with right-sided pain, dark red vaginal discharge, I&#8217;ve missed 2 periods, what is going on?<br>Ectopic pregnancy<\/p>\n\n\n\n<p>What term do we use if we&#8217;re talking about benign uterine tumors (fibroids)?<br>leiomyomas<\/p>\n\n\n\n<p>You have a patient who is in her third trimester of pregnancy and there is all of a sudden bright red vaginal bleeding, no pain. What are the differential diagnosis?<br>Placenta previa<\/p>\n\n\n\n<p>If we diagnose a woman with a cervical carcinoma in situ, what does that mean?<br>Full epithelial thickness of the cervix<\/p>\n\n\n\n<p>Tell me how we figure out the GTPAL system<br>G: Gestation- How many pregnancies you&#8217;ve had, including a current pregnancy<br>T- How many term deliveries<br>P- number of preterm births<br>A- Abortions (including miscarriages)<br>L- Living children<\/p>\n\n\n\n<p>Ovarian cancer is related to a mutation of which gene?<br>BRCA-1<\/p>\n\n\n\n<p>Be able to figure out due date using Nagel&#8217;s rule<br>Add 7 days past the first day of your last period, then subtract 3 months<br>The test will give us a date, we must be able to figure out the due date<\/p>\n\n\n\n<p>If you were doing a breast exam and the woman had squishy feeling, easily palpated and movable areas, what would you diagnose her with?<br>Breast cysts<\/p>\n\n\n\n<p>If my patient has gonorrhea, they probably also have what?<br>Chlamydia<\/p>\n\n\n\n<p>what is Balanitis?<br>Swollen glands on penis and foreskin<\/p>\n\n\n\n<p>If you diagnose a man with balantis, what is the next thing that you would think of?<br>Diabetes<\/p>\n\n\n\n<p>How do we treat trichomoniasis?<br>Flagyl<\/p>\n\n\n\n<p>If I were doing a testicular exam and I felt a firm, non-tender testicular mass, give me a differential diagnosis.<br>Testicular cancer<\/p>\n\n\n\n<p>I have a patient that I am doing an exam on and I see those strawberry spots on her vagina and cervix\u2026what am I diagnosing her with?<br>Trichomoniasis<\/p>\n\n\n\n<p>If a guy comes in and he&#8217;s complaining of fatigue, has diffuse low back pain, fever, urinary retention, what are you going to order?<br>Urinalysis\/UTI<\/p>\n\n\n\n<p>What organism did we say is the culprit for syphilis?<br>Treponema pallidum<\/p>\n\n\n\n<p>Give me some risks for prostate cancer (select all that apply)<br>Diet<br>Over the age of 65 y\/o<br>Family history<br>African american<\/p>\n\n\n\n<p>If I have syphilis and I already have bone and tissue lesions, what stage am I in?<br>Tertiary<\/p>\n\n\n\n<p>How do we document if a man can&#8217;t retract his foreskin over the glands?<br>Phimosis<\/p>\n\n\n\n<p>What does gonorrhea look like in a newborn?<br>Conjunctivitis \u2192 Corneal ulcerations<\/p>\n\n\n\n<p>If you had a patient that came in and said that they had this new lesion, it has a depressed center, it&#8217;s got rolled borders, and its living right on their face, what is your biggest suspicion\/differential diagnosis?<br>Basal cell carcinoma<\/p>\n\n\n\n<p>What does Preeclampsia look like?S\/S<br>Edema(hands\/face)<br>Clonus<br>Headache<br>High B\/P<br>RUQ Pain from protienuria<\/p>\n\n\n\n<p>What can happen if we do not treat PID (pelvic inflammatory disease)?<br>Scarring of the fallopian tubes due to inflammation<\/p>\n\n\n\n<p>An elevated, firm, rough lesion with flat top surface that is more than 1 cm in diameter is called:<br>plaque found in psoriasis and seborrheic and actinic keratosis.<\/p>\n\n\n\n<p>Elevated circumscribed, superficial lesion filled with serous fluid, less than 1 cm in diameter<br>vesicle- varicella and herpes zostrer<\/p>\n\n\n\n<p>Pathogenetic mechanism of POS<br>anovulation, elevation of levels of androgens, hyperandrogenism, and polycytic ovaries<\/p>\n\n\n\n<p>Hirschprung&#8217;s Disease (Megacolon)<br>congenital ganglionic megacolon- neonatal obstuction<\/p>\n\n\n\n<p>Marasmus<br>A wasting away of body tissues in the infant&#8217;s first year, caused by severe protein-calorie deficiency.<\/p>\n\n\n\n<p>Koplik spots are a diagnostic indicator of<br>lusters sm. Size red papules w\/ white centers in the buccal mucosa by lower molars -rubeola measles<\/p>\n\n\n\n<p>variococele<br>swelling and distention of veins of the spermatic cord, somewhat resembling varicose veins of the legs #1 cause of sterility<\/p>\n\n\n\n<p>What medical term is used to identify an inflammatory disorder of the skin that is often considered synonymous with dermatitis and characterized by pruritus with lesions that have an indistinct border?<br>Eczema and Atopic Dermatitits<\/p>\n\n\n\n<p>spermatocele<br>a cyst that develops in the epididymis and is filled with a milky fluid containing sperm<\/p>\n\n\n\n<p>Type of Necrosis associated with gangrene?<br>Caseous Liqueactive Necrosis<\/p>\n\n\n\n<p>3 substances that influence calcium regulation in the body<br>Vit D, calcitonin, and PTH<\/p>\n\n\n\n<p>Risk for autosomal dominant to occur<br>50%<\/p>\n\n\n\n<p>What cancer does HHV-8 cause<br>Kaposi&#8217;s sarcoma<\/p>\n\n\n\n<p>BRCA1 and BRCA2<br>breast cancer 1 and 2 &#8211; genetic mutations associated with increased risk for breast and ovarian cancer<\/p>\n\n\n\n<p>hypomethylation<br>Reduced methylation of DNA. This results in the activation of oncogenes genes and the resulting formation of tumors.<\/p>\n\n\n\n<p>Which component of the plasma protein system tags pathogenic microorganisms for destruction by neutrophils and macrophages?<br>C3b<\/p>\n\n\n\n<p>We have an expert-written solution to this problem!<br>cells that fight against parasitic infections<br>Eosinophills<\/p>\n\n\n\n<p>Which leukocytes are in. and IgE hypersenisitivity<br>Eosinophills<\/p>\n\n\n\n<p>SLE, MS, IBD, are all what<br>auto immune diseases<\/p>\n\n\n\n<p>Treponema pallidum<br>causes syphilis<\/p>\n\n\n\n<p>What does stress-induced sympathetic stimulation of the adrenal medulla cause secretion of?<br>release cathecholamines<\/p>\n\n\n\n<p>We have an expert-written solution to this problem!<br>fluid in the tunica vaginalis<br>hydrocele<\/p>\n\n\n\n<p>Most commonly reported symptom with any tye of cancer<br>fatigue<\/p>\n\n\n\n<p>arterial circle of willis<br>These vessels supply blood to the brain (mostly the cerebrum) without clots in a circle by multiple routes in the middle of the brain<\/p>\n\n\n\n<p>Philadelphia chromosome causes<br>CML<\/p>\n\n\n\n<p>Where does ADH act?<br>renal collecting duct<\/p>\n\n\n\n<p>An elevated, firm, circumscribed area; less than 1 cm in diameter<br>papule<\/p>\n\n\n\n<p>hematopoietic growth factors<br>correct chronic renal failure<br>increase peripheral stem cells for transplantation<br>Accelerate cell proliferation after bone marrow engraftment<\/p>\n\n\n\n<p>Which type of cancer is cause by helicobacter<br>stomach cancer<\/p>\n\n\n\n<p>patholgical fracture<br>a break at the site of a preexisting abnormality, usually by force that would not fracture a normal bone<\/p>\n\n\n\n<p>which hormone exerts antiapoptotic effects on osteoblasts but proapoptotic effects on osteoclasts<br>Estrogen deficiency<\/p>\n\n\n\n<p>ankylosing spondylitis<br>chronic, progressive arthritis with stiffening of joints, primarily of the spine<\/p>\n\n\n\n<p>People with gout are at high risk for:<br>renal calculi<\/p>\n\n\n\n<p>Which protein is absent in DMD<br>dystrophin<\/p>\n\n\n\n<p>The most common childhood bone cancer<br>Osteosarcoma<\/p>\n\n\n\n<p>3 conditions that jaundice can be contributed to<br>acute inflammation of the liver, hemolytic anemia, cholestasis.<\/p>\n\n\n\n<p>Bilateral uterteral obstruction is not associated with which type of renal injury<br>prerenal<\/p>\n\n\n\n<p>x-ray showing punched out lesions<br>multiple myeloma<\/p>\n\n\n\n<p>silicosis<br>disease due to silica or glass dust in the lungs; occurs in mining occupations TLC decreased, FEV1 decreased, and v\/q decreased<\/p>\n\n\n\n<p>A patient presents with right ventricular hypertrophy seen as a right axis deviation on the EKG and a loud systolic murmur heard at the left sternal border. What valve is the cause?<br>Mitral Senosis<\/p>\n\n\n\n<p>sources;<\/p>\n\n\n\n<p><a href=\"https:\/\/yaveni.com\/\" target=\"_blank\" rel=\"noopener\">https:\/\/yaveni.com\/<\/a><\/p>\n\n\n\n<p><a href=\"https:\/\/www.gcu.edu\/\" target=\"_blank\" rel=\"noopener\">https:\/\/www.gcu.edu\/<\/a><\/p>\n","protected":false},"excerpt":{"rendered":"<p>Final Exam: NUR631\/ NUR 631 (Latest 2023\/2024 Update) Advanced Physiology and Pathophysiology Study Guide| Questions and Verified Answers| 100% Correct \u2013 GCU Final Exam: NUR631\/ NUR 631 (Latest2023\/2024 Update) Advanced Physiology andPathophysiology Study Guide| Questions andVerified Answers| 100% Correct \u2013 GCUQ: Patient comes in with chronic wounds that aren&#8217;t healing. What are you thinking?Answer:-Diabetes-Something else [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"site-sidebar-layout":"default","site-content-layout":"","ast-site-content-layout":"default","site-content-style":"default","site-sidebar-style":"default","ast-global-header-display":"","ast-banner-title-visibility":"","ast-main-header-display":"","ast-hfb-above-header-display":"","ast-hfb-below-header-display":"","ast-hfb-mobile-header-display":"","site-post-title":"","ast-breadcrumbs-content":"","ast-featured-img":"","footer-sml-layout":"","ast-disable-related-posts":"","theme-transparent-header-meta":"","adv-header-id-meta":"","stick-header-meta":"","header-above-stick-meta":"","header-main-stick-meta":"","header-below-stick-meta":"","astra-migrate-meta-layouts":"default","ast-page-background-enabled":"default","ast-page-background-meta":{"desktop":{"background-color":"","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"tablet":{"background-color":"","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"mobile":{"background-color":"","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""}},"ast-content-background-meta":{"desktop":{"background-color":"var(--ast-global-color-5)","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"tablet":{"background-color":"var(--ast-global-color-5)","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""},"mobile":{"background-color":"var(--ast-global-color-5)","background-image":"","background-repeat":"repeat","background-position":"center center","background-size":"auto","background-attachment":"scroll","background-type":"","background-media":"","overlay-type":"","overlay-color":"","overlay-opacity":"","overlay-gradient":""}},"footnotes":""},"categories":[25],"tags":[],"class_list":["post-130487","post","type-post","status-publish","format-standard","hentry","category-exams-certification"],"_links":{"self":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/posts\/130487","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/comments?post=130487"}],"version-history":[{"count":0,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/posts\/130487\/revisions"}],"wp:attachment":[{"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/media?parent=130487"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/categories?post=130487"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/www.learnexams.com\/blog\/wp-json\/wp\/v2\/tags?post=130487"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}