{"id":130547,"date":"2023-12-18T07:40:35","date_gmt":"2023-12-18T07:40:35","guid":{"rendered":"https:\/\/learnexams.com\/blog\/?p=130547"},"modified":"2023-12-18T07:40:37","modified_gmt":"2023-12-18T07:40:37","slug":"final-exam-nur2755-nur-2755-new-2023-2024-multidimensional-care-iv-complete-guide-with-questions-and-verified-answers-100-correct-rasmussen","status":"publish","type":"post","link":"https:\/\/www.learnexams.com\/blog\/2023\/12\/18\/final-exam-nur2755-nur-2755-new-2023-2024-multidimensional-care-iv-complete-guide-with-questions-and-verified-answers-100-correct-rasmussen\/","title":{"rendered":"Final Exam: NUR2755\/ NUR 2755 (New 2023 \/ 2024) Multidimensional Care IV |Complete Guide with Questions and Verified Answers| 100% Correct – Rasmussen"},"content":{"rendered":"\n

Final Exam: NUR2755\/ NUR 2755 (New 2023 \/ 2024) Multidimensional Care IV |Complete Guide with Questions and Verified Answers| 100% Correct – Rasmussen<\/p>\n\n\n\n

Final Exam: NUR2755\/ NUR 2755 (New 2023
\/ 2024) Multidimensional Care IV |Complete
Guide with Questions and Verified Answers|
100% Correct – Rasmussen
QUESTION
Earliest sign of increased intracranial pressure
Answer:
Decreased level of con- sciousness
QUESTION
Increased intracranial pressure early signs
Answer:
-EARLIEST SIGN: Decreased level of consciousness
-Restlessness
-Changes in speech
-Confusion
-Headache
-Nausea and vomiting \u2019 projectile
QUESTION
Increased intracranial pressure late signs
Answer:
-Pupillary changes \u2019 can mean herniation
-Cranial nerve dysfunction
-Ataxia
-Cushing’s triad (very late sign)
QUESTION
Increased intracranial pressure interventions<\/p>\n\n\n\n

Answer:
-Low stimulation
-Semi-fowlers \u2019 30 degrees
-Head in neutral position
-Do not cluster activities
-Suction only as needed
-Teach patient not to cough or blow their nose
-Dim lighting
-Stool softeners
-Do not bend or bare down
QUESTION
Increased intracranial pressure treatment
Answer:
IV mannitol given through a filter because it crystallizes at room temperature
QUESTION
Cushing’s triad
Answer:
Severe hypertension, widened pulse pressure (difference be- tween the systolic and diastolic
blood pressure), bradycardia, irregular respirations
QUESTION
Pulse pressure
Answer:
Difference between systolic and diastolic pressure
QUESTION
Ischemic stroke
Answer:<\/p>\n\n\n\n

Caused by the occlusion of a cerebral artery by either a thrombus or an embolus.
QUESTION
Thrombotic stroke
Answer:
Occur secondary to the development of a blood clot on an atherosclerotic plaque in a cerebral
artery that gradually shuts off the artery and causes ischemia distal to the occlusion
QUESTION
Embolic stroke
Answer:
Caused by an embolus traveling from another part of the body to a cerebral artery. Blood to the
brain distal to the occlusion is immediately shut off causing neurologic deficits or a loss of
consciousness to instantly occur.
QUESTION
Hemorrhagic stroke
Answer:
Occur secondary to a ruptured artery or aneurysm. The prognosis for a client who has
experienced a hemorrhagic stroke is poor due to the amount of ischemia and increased ICP
caused by the expanding collection of blood. Patients will complain of the “worst headache of
my life”.
QUESTION
Alexia
Answer:
Inability to understand written words<\/p>\n\n\n\n

QUESTION
Stroke diagnostics
Answer:
A non-contrast computed tomography (CT) scan (WITH- OUT CONTRAST) is the initial
diagnostic test and should be performed within 25 minutes from the time of client arrival to the
emergency department
QUESTION
Homonymous hemianopsia interventions
Answer:
-Instruct them to use a scanning technique (turning head from the direction of the unaffected
side to the affected side) when eating and ambulating
-Rotate their plate so that they can see it
-Talk to them on their good side
QUESTION
Ischemic stroke treatment
Answer:
-Give thrombolytics within 4.5 hours of initial man- ifestations
-Low-dose aspirin is given within 24-48 hours following an ischemic stroke to prevent further
clot formation
QUESTION
Stroke prevention
Answer:
-Smoking cessation
-Heart-healthy diet rich in fruits and vegetables and low in saturated fats
-Regular activity, including planned exercise; example: walking at least 30 minutes most days of
the week
-Reduction in alcohol consumption
-Reduction of salt in diet
Powered by https:\/\/learnexams.com\/search\/study?query=<\/a><\/p>\n\n\n\n

\"\"<\/a><\/figure>\n\n\n\n

asystole flat line but patient has pulses present
check EKG attachments<\/p>\n\n\n\n

management of angina
MONA<\/p>\n\n\n\n

all patients get<\/p>\n\n\n\n

morphine<\/p>\n\n\n\n

oxygen<\/p>\n\n\n\n

nitrates<\/p>\n\n\n\n

aspirin<\/p>\n\n\n\n

CABG complications
hypotension<\/p>\n\n\n\n

hypothermia<\/p>\n\n\n\n

hypertension<\/p>\n\n\n\n

bleeding<\/p>\n\n\n\n

cardiac tamponade<\/p>\n\n\n\n

change in level of consciousness<\/p>\n\n\n\n

CK MB in MI
30-70 units<\/p>\n\n\n\n

detectable 4-6 hours duration 3 days<\/p>\n\n\n\n

Troponin T (MI)
0.2<\/p>\n\n\n\n

detectable 3 hours<\/p>\n\n\n\n

duration 14-21 days<\/p>\n\n\n\n

Troponin I (MI)
0.03<\/p>\n\n\n\n

decidable 3 days<\/p>\n\n\n\n

duration 7-10<\/p>\n\n\n\n

cholesterol
less than 200<\/p>\n\n\n\n

HDL 35-80
LDL less than 130<\/p>\n\n\n\n

triglycerides 35-160<\/p>\n\n\n\n

P wave
atrial depolarization and contract<\/p>\n\n\n\n

PR interval
travel time from atria to ventricle<\/p>\n\n\n\n

QRS
ventricle depolarization and contraction<\/p>\n\n\n\n

T wave
ventricle repolarization<\/p>\n\n\n\n

EKG little box
0.04 seconds<\/p>\n\n\n\n

big box
0.20 seconds<\/p>\n\n\n\n

atrial fibrillation
irregular R-R intervals<\/p>\n\n\n\n

PR interval : NONE<\/p>\n\n\n\n

atrial rate: over 350<\/p>\n\n\n\n

risk for blood clots<\/p>\n\n\n\n

atrial flutter
atrial rate: 250-350<\/p>\n\n\n\n

saw tooth pattern<\/p>\n\n\n\n

2,3,4 flutter waves<\/p>\n\n\n\n

supra ventricular tachycardia
p waves buried may not be seen<\/p>\n\n\n\n

ventricular tachycardia
monomorphic: same shape<\/p>\n\n\n\n

polymorphic: different shape<\/p>\n\n\n\n

rate 100-250<\/p>\n\n\n\n

dead rhythm
asystole<\/p>\n\n\n\n

VFIB<\/p>\n\n\n\n

ventricular tachycardia<\/p>\n\n\n\n

PEA<\/p>\n\n\n\n

asystole
no electrical activity<\/p>\n\n\n\n

check leads<\/p>\n\n\n\n

VFIB
not organized<\/p>\n\n\n\n

treatment: CPR, SHOCK, code meds<\/p>\n\n\n\n

ventricular tachycardia
pulseless<\/p>\n\n\n\n

treatment: CPR, defibrillator<\/p>\n\n\n\n

Pulseless Electrical Activity
no pulse<\/p>\n\n\n\n

first degree av block
pr interval is prolonged<\/p>\n\n\n\n

second degree av block type 1
PR progressively longer<\/p>\n\n\n\n

second degree av block type ii
usually wide QRS<\/p>\n\n\n\n

third degree av block
complete block<\/p>\n\n\n\n

VTACH
rapid response call<\/p>\n\n\n\n

VTACH and VFIB are shockable<\/p>\n\n\n\n

asystole IS NOT<\/p>\n\n\n\n

cardio version on VTACH<\/p>\n\n\n\n

QRS complex with no p wave
lack of sinus beats<\/p>\n\n\n\n

fibrillation or flutter waves<\/p>\n\n\n\n

atrial fibrillation manifestations
Sensations of a fast, fluttering or pounding heartbeat (palpitations)<\/p>\n\n\n\n

Chest pain. Dizziness. SOB<\/p>\n\n\n\n

hemorrhagic stroke
occur secondary to a ruptured artery or aneurysm<\/p>\n\n\n\n

thrombotic stroke
occur secondary to the development of a blood clot on an atherosclerotic plaque in a cerebral artery<\/p>\n\n\n\n

gradually shuts off artery and causes ischemia<\/p>\n\n\n\n

emboli stroke
embolus traveling from another part of the body to cerebral artery<\/p>\n\n\n\n

ischemic strokes
can be reversed with fibrinolytic therapy using alteplase<\/p>\n\n\n\n

3-4.5 hr of initial manifestation<\/p>\n\n\n\n

contraindicated by active bleeding<\/p>\n\n\n\n

GBS
A rare acute inflammatory disorder that affects the axons and or myelin of the PNS<\/p>\n\n\n\n

Results in ascending muscle weakness or paralysis<\/p>\n\n\n\n

demyelination of peripheral nerves<\/p>\n\n\n\n

GBS paralysis occurs
outwards- inwards<\/p>\n\n\n\n

respiratory arrest can occur in later stages<\/p>\n\n\n\n

ensure airway latency<\/p>\n\n\n\n

symptoms of GBS
tingling of lower extremity<\/p>\n\n\n\n

Initial muscle weakness and pain<\/p>\n\n\n\n

Ascending paralysis<\/p>\n\n\n\n

Autonomic dysfunction<\/p>\n\n\n\n

GBS causes
autoimmune disease
viral infection
vaccination<\/p>\n\n\n\n

Glasgow Coma Scale
lowest score 3- practically dead<\/p>\n\n\n\n

highest is 15<\/p>\n\n\n\n

coma- 8<\/p>\n\n\n\n

GCS
eye opening – 1 to 4<\/p>\n\n\n\n

verbal – 1 to 5<\/p>\n\n\n\n

motor – 1 to 6<\/p>\n\n\n\n

myasthenia gravis
a rare progressive autoimmune disease characterized by muscle weakness as a result of impaired acetylcholine receptors<\/p>\n\n\n\n

Affects the voluntary muscles of the body, especially those that control the eyes, mouth, throat and limbs<\/p>\n\n\n\n

common symptoms of maysthenia graves
involvement of eye muscles<\/p>\n\n\n\n

ocular palsies<\/p>\n\n\n\n

Ptosis<\/p>\n\n\n\n

Diplopia<\/p>\n\n\n\n

weak or incomplete eye closure<\/p>\n\n\n\n

tensilon testing
Myasthenic crisis is due to undermedication<\/p>\n\n\n\n

Within 30 to 60 seconds after injection of Tensilon, most myasthenic patients show marked improvement in muscle tone that lasts 4 to 5 minutes.<\/p>\n\n\n\n

myasthenia crisis
undermedication<\/p>\n\n\n\n

cholinergic crisis
overmedication<\/p>\n\n\n\n

MS
Chronic autoimmune disease affecting the myelin sheath and conduction pathway of the CNS<\/p>\n\n\n\n

Characterized by periods of remission and exacerbation<\/p>\n\n\n\n

Hallmark Characteristics
Demyelination<\/p>\n\n\n\n

Diffuse patchy areas of plaque in white matter of CNS<\/p>\n\n\n\n

ALS
an adult onset upper and lower motor neuron disease characterized by progressive weakness, muscle wasting, and spasticity eventually leading to paralysis<\/p>\n\n\n\n

progressive neurodegenerative disease<\/p>\n\n\n\n

ALS symptoms
Early symptoms\u2014fatigue while talking, tongue atrophy, dysphagia, weakness of the hands and arms, fasciculations, nasal quality of speech, dysarthria.<\/p>\n\n\n\n

cushing triad
sign of increased ICP<\/p>\n\n\n\n

-Widening\/decreased of pulse pressure
-Slowing HR<\/p>\n\n\n\n