NUR 2063 Pathophysiology Final exam 1

NUR 2063 Pathophysiology Final exam 1
1. Gastritis and Etiology and patho
inflammation of stomach's mucolas lining (can involve entire stomach or region) can be acute
or chronic.
may be caused by h. pylori infection (imbeds in mucosal layer activating toxins and enzymes
that cause inflammation. NSAIDS, chronic alcohol consumption, stress, trauma, burns, or
infections, autoimmune conditions
2. manifestations of gastritis
indigestion, heartburn, epigastric pain, abdominal cramping, nausea, vomiting, anorexia, fever,
malaise.
hematemesis and dark, tarry stoolsindicate ulceration and bleeding. chronic gastri- tis increases
risk for peptic ulcers, gastric cancer, anemia, and hemorrhage.
3. gastritis diagnosis/treatment
h&p, GI tract x ray, egd,serum h. pylori antibod- ies, h. pylori breath test, stool analysis (h.
pylori and occult blood
treatment-acute is self limiting ususally resolves
meds-antacids, acid-reducing agents, and mucosal barrier agents other strategies include those
for GERD (diet, small meals, antacids)
4. Peptic ulcer disease (PUD)
refers to erosive lesions affecting the muscularis mucosa of the stomach or duodenum. ulcers
vary in size and severity, ranging from superficial erosions to complete penetration through GI
tract wall
5. peptic ulcer disease etiology and patho
ETIOLOGY
most commonly H.pylori and NSAID use.
PATHO
develops because of an imbalance between destructive forces and protective mechanisms

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