Midterm Exam: NR572 / NR 572
(Latest Update 2025 / 2026) Advanced Acute Care Management | Questions and Answers | Grade A | 100% Correct – Chamberlain
Question:
Antidote for Acetaminophen
Answer:
N-acetylcysteine (NAC) Recommended for patient at risk for hepatic toxicity
Question:
antidote for digoxin
Answer:
Digibind
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Question:
Antidote for opioids
Answer:
Naloxone (Narcan)
Question:
Antidote for benzodiazepines
Answer:
Flumazenil (Romazicon)
Question:
Antidote for beta blocker
Answer:
Glucagon (epi will not work, receptor sites have been blocked)
Question:
Antidote for calcium channel blocker
Answer:
Calcium gluconate
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Question:
Antidote for iron
Answer:
Deferoxamine (Desferal)
Question:
Antidote for cyanide
Answer:
Hydroxocobalamin
Question:
Amyotrophic Lateral Sclerosis (ALS)
Answer:
Progressive neurodegenerative disorder of motor neurons in the spinal cord and brainstem. Caused by death of motor neurons. Upper motor neurons are located in the brain. Lower motor, neurons expand from brain and spinal cord to muscles.
Symptoms include slowness, spasticity, muscle weakness, muscle atrophy (shrinking).
No cure, medications improve muscle coordination, and physical therapy may slow the progression. 3 / 4
Question:
Guillain-Barré syndrome
Answer:
Inflammation of the myelin sheath of peripheral nerves
Characterized by numbness and tingling that begins in lower extremities and ascends upward. Affects motor and sensory nerves and can cause weakness or paralysis, difficulty speaking or swallowing, severe pain, rapid heart rate, impaired bladder, and bowel function/control, and difficulty breathing.
Question:
Multiple Sclerosis (MS)
Answer:
Inflammatory disease that affects central nervous system. Characterized by visible brain lesions called plaques.The location of plaques can vary, but are frequently symmetrical and tend to affect areas of white matter adjacent to the brain ventricles. The plaque locations cause various neurological symptoms, including vision disturbances, muscle weakness, and cognitive deficiencies.MS attacks the myelin sheath, which is the protein-rich coating that insulates neurons. Destruction of the myelin sheath leads to impaired neuron function and eventually cell death. Neuron death causes visible lesions called plaques.
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