WEB WOC WOUND CARE EXAM (ACTUAL / ) STUDY
GUIDE QUESTIONS AND VERIFIED 100% CORRECT ANSWERS
arterial ulcer characteristics - --Answers---tips of the toes, pressure points usually small, may have tunneling, punched out appearance dry, pale tissue, may have necrosis well defined, smooth edges minimal exudate
etiology and diagnosis of LEAD - --Answers---Reduced arterial flow to the LE due to atherosclerosis
ABI/TBI to determine evidence of LEAD and severity, further testing needed if disease significant or no wound progress
neuropathic ulcer characteristics - --Answers---plantar aspects of feet, over pressure points round or oblong, punched out appearance necrotic, pink, pale well defined, smooth edges small-mod exudate
wagner's classifications- DFU - --Answers---grade 0: pre-
injury, skin intact, callus grade 1: superficial injury, partial or full thickness 1 / 4
grade 2: probing to ligament, tendon, joint capsule, soft tissue
infection
grade 3 : deep injury with abscess, osteomyelitis, or joint
sepsis
grade 4: gangrene to toes or fore foot
grade 5: gangrene to entire foot *needs amputation*
etiology and diagnosis of LEND - --Answers---damage to nerves causes neuropathy that leads to undetected repetitive pressure/trauma, inflammation with resulting ulceration
ABI/TBI to determine evidence of LEAD and severity, TBI better test due to vessel calcification. X-ray common with initial evaluation, MRI if probe to bone
vasculitic ulcers - --Answers---autoimmune connective tissue disease that targets blood vessels
acute pain, palpable purpura
sickle cell ulcers - --Answers---Inherited hematologic disorder of 100,000 persons in the U.S., mostly African American, diagnosis based upon history and presentation
painful and slow to heal
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Pyoderma gangrenosum - --Answers---unknown etiology, stems from chronic inflammatory skin disease
painful nodule or pustule that breaks into large ulcer with violaceous border and red halo
calcyphylaxis - --Answers---Occurs most commonly in patients with chronic kidney disease on hemodialysis
star shaped, deep, purple ulcers that usually become gangrenous
superficial burn - --Answers---Epidermis only Typically develops from sunburn or scalding
Pain, warmth, redness, swelling, peeling epidermis
superficial, partial thickness burn - --Answers---Epidermis and dermis involved
Blistering, weeping moist skin. Dermal structures are intact.Painful
deep, partial thickness burn - --Answers---Deeper portions of the dermis damaged
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Red with waxy white tissue present, blister formation. Dry red wound bed. Disruption of hair follicles, nails, and glands
full thickness burn - --Answers---Entire dermis involved and extending into subcutaneous tissue; nerves destroyed
Waxy white to gray in color; may have charred (black leathery) appearance. No sensation of pain because tissue is avascular.
toxic epidermal necrolysis - --Answers---Maculopapular rash followed by erythema, painful skin and sloughing of large sheets of epidermis
Rare condition usually associated with a drug reaction.(sulfa, anticonvulsants etc) Involves over 30% of body; +Nikolsky sign
graft versus host disease - --Answers---Maculopapular rash that develops into bullae, generalized erythema and desquamation; painful and pruritic. Starts on palms, soles then spreads to face, chest, arms, shoulders.
A complication of transplantation or blood transfusion.
necrotizing fasciitis - --Answers---Bullae form then rapid progression to necrosis
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